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1 DRS) subscale PIGD (Postural Instability and Gait Disorder)].
2 erse phenotypes such as tremor, weakness and gait disorder.
3 upper motor neurons and causing progressive gait disorder.
4 parkinsonism together with DOPA-unresponsive gait disorders.
5 cally and genetically heterogeneous group of gait disorders.
6 terminant of cognitive decline, dementia and gait disorders.
7 6.4 years) were significantly younger, while gait disorders (43.2 years) had a significantly later ag
8 iovascular side effects and improved spastic gait disorders after brain injury in a disease model.
9 aucasian patient, with a complex progressive gait disorder and adrenal insufficiency, was referred fo
14 e motor learning in patients with functional gait disorders and probe whether abnormal postural mecha
15 tation potential in patients with functional gait disorders and related disorders (e.g. fear of falli
18 ntine nucleus may help treat the balance and gait disorder in atypical parkinsonian disorders in the
19 the study was to distinguish the hypokinetic gait disorder in idiopathic normal pressure hydrocephalu
21 paired visual sampling may contribute to the gait disorder in PD, especially when navigating through
23 inhibition, age and postural instability and gait disorder score (Movement Disorders Society Unified
24 and gait symptoms (Postural Instability and Gait Disorder score-Movement Disorders Society Unified P
27 ferent ataxias present with a characteristic gait disorder that reflects cerebellar motor dysfunction
28 ar, the pedunculopontine nucleus, to address gait disorders that respond poorly to levodopa and conve
29 ohort of people with Parkinson's disease and gait disorder to derive spatial covariance networks of c
30 differentiate clinical entities with similar gait disorders utilizing instrumented gait analysis proc
31 pid and severe disease progression; onset of gait disorders was 3 times more rapid (p < 0.0001) and t
34 ugh late-onset progressive action tremor and gait disorder with CNS atrophy was recently reported in