ライフサイエンスコーパス: galactose

1 lly important hexoses (glucose, mannose, and galactose).

2 cetylglucosamine, N-acetylgalactosamine, and galactose).

3 tabolism of its monomers glucose, fucose and galactose.

4 ctose, dextran, and a mixture of mannose and galactose.

5 d galactonate being more discriminative than galactose.

6 rom a commercially available derivative of d-galactose.

7 charomyces cerevisiae, from the simple sugar galactose.

8 cid, S. oralis bound exposed beta-1,4-linked galactose.

9 from 93% for lactose to 98% for glucose and galactose.

10 e 1-phosphate, and GDP-glucose when grown on galactose.

11 rminus can be substituted by an alpha-linked galactose.

12 patterns for fructose, mannose, glucose, and galactose.

13 t this did not generalize to Na-saccharin or galactose.

14 95.77 +/- 0.67% of lactose into glucose and galactose.

15 p11 as CcrG, Campylobacter ChemoReceptor for Galactose.

16 responsible for lowering its affinity toward galactose.

17 ct of galectins comparable with that of free galactose.

18 exhibited reduced capsule production only in galactose.

19 ning alpha2-6-linked sialic acid to terminal galactose.

20 se only and a blended mixture of mannose and galactose.

21 that P1 does not mediate binding to sulfated galactose.

22 D-galactose (2 muM), galactitol (11 muM) and galactose 1-phosphate (0.1 mM), (corresponding to plasma

23 y-dependent growth phenotype and accumulated galactose 1-phosphate, glucose 1-phosphate, and GDP-gluc

24 alactose and its metabolites, galactitol and galactose 1-phosphate, on oocyte quality as well as embr

25 5 A) and complexed with glucose (1.25 A) and galactose (1.8 A).

26 ynthesis, indicating that WbaP(Mx) transfers galactose-1-P to undecaprenyl-phosphate.

27 d as such replenishes the depleted levels of galactose-1-P, as well as the levels of UDP-glucose and

28 drial respiration was sufficient to diminish galactose-1-phosphate levels and, consequently, affects

29 tose exposure because the deletion decreases galactose-1-phosphate levels.

30 UDP-galactose from uridine triphosphate and galactose-1-phosphate.

31 okinase (GALK), phosphorylating galactose to galactose-1-phosphate.

32 of galactose-related metabolites, including galactose-1-phosphate.

33 s cells (CCs), were exposed for 4 hours to D-galactose (2 muM), galactitol (11 muM) and galactose 1-p

34 alacturonic acid (66.0%), arabinose (26.2%), galactose (3.7%), rhamnose (2.7%) and xylose (1.0%).

35 a sweetening syrup made of glucose (30.48%), galactose (33.51%), fructose (16.92%), D-tagatose (10.54

36 The polysaccharide rich in arabinose and galactose (39-54%) and mannoproteins (38-55%) were the m

37 ess this knowledge gap, here we examined UDP-galactose 4'-epimerase (GALE), which interconverts two p

38 GALE encodes UDP-galactose-4-epimerase, an enzyme of galactose metabolism

39 6.97%), glucose (88.90%, 89.31%, 87.68%) and galactose (5.34%, 5.17%, 5.35%).

40 Galactose (58.9-91.2%, w/w) was the main monosaccharide

41 diagnoses of IA include galactose alpha-1,3 galactose (a carbohydrate contained in red meat) allergy

42 mal studies have shown that a high intake of galactose, a breakdown product of lactose, increases ova

43 ium also possesses two transport systems for galactose, a galactose-PTS system and an ABC galactose t

44 utshines its relatives in the ability to use galactose, a milk sugar byproduct.

45 alpha(2,3)-stereoselective sialylation to a galactose acceptor.

46 ing was also observed, consistent with alpha-galactose addition.

47 ze a protein vaccine candidate with an alpha-galactose adjuvant motif in a one-pot cell-free system a

48 ding breath H(2), blood glucose, and urinary galactose after a lactose tolerance test.

49 portant differential diagnoses of IA include galactose alpha-1,3 galactose (a carbohydrate contained

50 Peanut and galactose alpha-1,3-galactose (alpha-gal) are characteri

51 n-human glycan epitopes, galactose-alpha-1,3-galactose (alpha-gal) and Neu5Gc-alpha-2-6-galactose (Ne

52 Peanut and galactose alpha-1,3-galactose (alpha-gal) are characterized by high- or very

53 IgG to galactose-alpha-1,3-galactose (alpha-gal) are highly abundant natural antibo

54 ibodies (Ab) specific to galactose-alpha-1,3-galactose (alpha-gal) are responsible for a delayed form

55 inding glycan allergen galactose-alpha-(1,3)-galactose (alpha-Gal) is associated with IgE-mediated de

56 ific to the carbohydrate galactose-alpha-1,3-galactose (alpha-gal) is known to induce delayed anaphyl

57 Galactose-alpha-1,3-galactose (alpha-gal) syndrome is characterized by the p

58 ting to IgE specific for galactose-alpha-1,3-galactose (alpha-Gal) was first reported 10 years ago in

59 ates to IgE specific for galactose-alpha-1,3-galactose (alpha-Gal), an oligosaccharide that is presen

60 The oligosaccharide galactose-alpha-1,3-galactose (alpha-Gal), present in mammalian proteins and

61 tain the disaccharide galactosyl-alpha-(1,3)-galactose (alpha-Gal).

62 size the oligosaccharide galactose-alpha-1,3-galactose (alpha-Gal).

63 We also observed that galactose-alpha terminated glycans are almost exclusivel

64 other hand, the IgE-binding glycan allergen galactose-alpha-(1,3)-galactose (alpha-Gal) is associate

65 Two non-human glycan epitopes, galactose-alpha-1,3-galactose (alpha-gal) and Neu5Gc-alp

66 IgG to galactose-alpha-1,3-galactose (alpha-gal) are highly abu

67 IgE antibodies (Ab) specific to galactose-alpha-1,3-galactose (alpha-gal) are responsibl

68 f IgE molecules specific to the carbohydrate galactose-alpha-1,3-galactose (alpha-gal) is known to in

69 Galactose-alpha-1,3-galactose (alpha-gal) syndrome is ch

70 an meat allergy relating to IgE specific for galactose-alpha-1,3-galactose (alpha-Gal) was first repo

71 ha-Gal syndrome" relates to IgE specific for galactose-alpha-1,3-galactose (alpha-Gal), an oligosacch

72 The oligosaccharide galactose-alpha-1,3-galactose (alpha-Gal), present in ma

73 inability to synthesize the oligosaccharide galactose-alpha-1,3-galactose (alpha-Gal).

74 ed by specific IgE to the alpha-gal epitope (galactose-alpha-1,3-galactose) ubiquitously expressed on

75 The carbohydrate epitope galactose-alpha-1,3-galactose, located on the Fab region

76 lutinin (PHA-E) are used to identify fucose, galactose, alpha2-6-linked sialic acid, and bisected N-a

77 ~ 50% conversion in 4 h starting with 300 mM galactose (an average productivity of 37 mM h(-1)), and

78 n enrichment of gut bacteria that catabolize galactose, an end product of lactose metabolism, and of

79 rolines (Hyps) are substituted with an alpha-galactose and 1-5 beta- or alpha-linked arabinofuranoses

80 A to the FBS resulted in a release of 2.8 mM galactose and 4.3 mM N-acetylneuraminic acid; these suga

81 which is in turn substituted with a terminal galactose and a second xylose residue.

82 cessive addition of terminal beta-1,4-linked galactose and alpha-2,6-linked sialic acid to N-glycans.

83 accharide composition, mainly constituted of galactose and arabinose, suggesting the presence of arab

84 vity to metabolic stress in media containing galactose and azide, but not glucose, and decreased mito

85 alacturonic acid, L-arabinose, L-rhamnose, D-galactose and D-glucose.

86 hasizes the importance of the terminal alpha-galactose and establishes the phylogenetic breadth of Sk

87 skeletal muscle cells were shown to oxidise galactose and fatty acids normally, indicating that the

88 r affinity for N-glycans containing terminal galactose and for agalactosylated bisected biantennary N

89 so possess transporters that allow growth on galactose and fructose.

90 ynthesizes unusual N-glycans with a range of galactose and fucose modifications on the Man2-3GlcNAc2

91 linear alpha-1,4-linked exopolysaccharide of galactose and GalNAc that is partially deacetylated afte

92 the quantitative response of these genes to galactose and in the position of these genes in the over

93 Our results suggested that D-galactose and its metabolites disturbed the spindle stru

94 Here, we evaluate the effect of D-galactose and its metabolites, galactitol and galactose

95 both extracts with predominance of glucose, galactose and mannose with no uronic acids detection; Fl

96 onsumption compared to their mother, whereas galactose and palmitic acid utilization were similar.

97 GAG), a cationic polymer of alpha-1,4-linked galactose and partially deacetylated N-acetylgalactosami

98 ctrometry indicated reduced incorporation of galactose and sialic acid, as seen in other Golgi homeos

99 an SRRP is required to bind beta-1,4-linked galactose and the first time that one of these adhesins

100 is of the 1,2-cis-glycosidic linkage between galactose and the linker (spacer) molecule and final pur

101 plates/m for glucose to 326,000 plates/m for galactose and the relative standard deviation (RSD) for

102 th beta1-3/beta1-4 linkages between glucose, galactose and their respective hexosamines.

103 X-ray crystal structures in complex with galactose and unhydrolyzed Galalpha1-3Gal revealed the p

104 ion 10R was composed of rhamnose, arabinose, galactose and uronic acid in 2.8:65.8:28.5:3M ratio, res

105 ion 50R was composed of rhamnose, arabinose, galactose and uronic acid in 4.3:56.2:37.4:2M ratio, res

106 ed N-acetylgalactosamine with beta1,3-linked galactose and variable sialylation.

107 d of arabinose, rhamnose, glucose, fructose, galactose and xylose.

108 ere observed between fermentable (glucose or galactose) and nonfermentable (glycerol) carbon sources

109 g/100g for lactose, 0.14 and 0.27mg/100g for galactose, and 0.16 and 0.26mg/100g for glucose.

110 r oligosaccharides (N-acetylneuraminic acid, galactose, and 6'-sialyllactose), linkage-specific siali

111 rying levels of alpha2-6-linked sialic acid, galactose, and bisected N-acetylglucosamine.

112 richment on alternative sugars, glycerol and galactose, and chemical overproduction of betaxanthins,

113 opylidene-protected d-fructose, l-sorbose, d-galactose, and d-allose.

114 substantial decreases in total sialic acid, galactose, and GalNAc levels in glycans.

115 h and low metabolite responses, observed for galactose, and its metabolites galactitol and galactonat

116 , d-glucose, l-allose, d-allose, d-gulose, d-galactose, and l-mannose are delineated, and for all eig

117 kage information for three hexoses (glucose, galactose, and mannose), three pentoses (xylose, arabino

118 The glycan sequence N-acetylgalactosamine, galactose, and sialic acid was consistently expressed on

119 shifting hPSC-CMs from glucose-containing to galactose- and fatty acid-containing medium promotes the

120 of nine determined monosaccharides (fucose, galactose, arabinose, glucose, rhamnose, xylose, mannose

121 Fucose, galactose, arabinose, glucose, sucrose, rhamnose, xylose

122 By-products such as glucose and galactose are generated.

123 ohydrates (such as D-glucose, D-xylose and D-galactose) are extracted on commercial scales, and serve

124 es when glucose or raffinose was replaced by galactose as the carbon source.

125 for the chemotactic response of C. jejuni to galactose, as shown using wild type, allelic inactivatio

126 GlcNAc, in the form of UDP-GlcNAc, and galactose, as UDP-Gal, are delivered into the Golgi appa

127 osed of a highly fucosylated and xylosylated galactose backbone with arabinan and mannoglucuronan bra

128 led to the discovery of novel monosaccharide galactose-based antagonists, with the strongest-binding

129 group of glycans with a common terminal beta-galactose (beta-Gal).

130 by which Fap1 contributes to beta-1,4-linked galactose binding remains to be defined; however, bindin

131 gonize the cancer-promoting activity of beta-galactose-binding galectins.

132 ouse model system to investigate the role of galactose-binding lectin 1 (Gal1) in immunotherapy resis

133 ems, the sensor and the transporter, utilize galactose-binding proteins that also bind glucose with t

134 idine methyl ester linker, and an acetylated galactose bonded to one of the aromatic nitrogen atoms o

135 Catabolism of galactose by Streptococcus pneumoniae alters the microbe

136 We radiolabeled a dendritic galactose carbohydrate with (18)F ((18)F-labeled galacto

137 ng growth arrest, likely due to overly rapid galactose catabolism and metabolic overload.

138 lus utilizes mono- and disaccharides such as galactose, cellobiose, lactose, and galactosyl-glycerol.

139 activation, and cellular adaption to lithium-galactose challenge.

140 Effective hepatic blood flow (EHBF) by galactose clearance, wet-dry weights, cytokines, histopa

141 two strong hydrogen bonds between ppGBP and galactose compared with glucose may be responsible for l

142 at Gan1D allows the cell to utilize residual galactose-containing disaccharides, taking advantage of

143 X-ray crystallographic structure of the beta-galactose-containing gelator, along with other results f

144 alectins because the polysaccharides contain galactose-containing side chains that might bind this cl

145 al) deficiency, resulting in accumulation of galactose-containing substrates and onset of the progres

146 gal deficiency, resulting in accumulation of galactose-containing substrates and onset of the progres

147 Galactose content of extracted polysaccharides can be mo

148 expressed on CD11b/CD18 such as biantennary galactose could represent novel targets for selective ma

149 hallenge whereas values obtained for urinary galactose/creatinine were lower than the existing litera

150 Plasma glucose and urinary galactose/creatinine were unreliable (AUC < 0.70) after

151 ers (breath H(2), plasma glucose and urinary galactose/creatinine) discriminated between lactase pers

152 which N-acetyl-d-glucosamine, l-fucose, or d-galactose (D-Gal) are replaced with d-glucose and/or l-r

153 Exogenous application of D-galactose (D-Gal) causes an increase in beta-1,4-galacta

154 relevant underivatized hexoses, d-glucose, d-galactose, d-mannose, and d-fructose, using only mass sp

155 minor monosaccharides found were d-xylose, d-galactose, d-mannose, d-glucose, d-arabinose, d-rhamnose

156 IgA1 glycoforms with some galactose-deficient (Gd) HR O-glycans play a key role in

157 circulating immune complexes of IgG bound to galactose-deficient IgA1 (Gd-IgA1).

158 B cells, it failed to reduce serum levels of galactose-deficient IgA1 and antigalactose-deficient IgA

159 lactose-deficient IgA1 or antibodies against galactose-deficient IgA1 did not change.

160 Serum levels of galactose-deficient IgA1 or antibodies against galactose

161 -based targeting moieties including mannose, galactose, dextran, and a mixture of mannose and galacto

162 ited Gal1 produced during previous growth in galactose directly interferes with Gal80 repression to p

163 nt changes in plasticity between glucose and galactose distributed throughout the promoter, suggestin

164 and D' contain an intact gene encoding a UDP-galactose epimerase (galE1) and a truncated remnant (gal

165 ferases encoded by lic2A and lgtC each add a galactose epitope bound by IgM that results in antibody-

166 demonstrated for H-type I and II; alpha(1,3)-galactose epitopes were prepared, and the pentasaccharid

167 bit delayed UPR activation after lithium and galactose exposure because the deletion decreases galact

168 ccharomyces cerevisiae adapting to growth in galactose for up to 8 generations.

169 sponse in PTDH3 activity between glucose and galactose from becoming larger.

170 he recombinant GH43 variants did not release galactose from cell wall-extracted glycoproteins or AGP-

171 eta-Galactosylceramidase (GALC) removes beta-galactose from galactosylceramide and other sphingolipid

172 for MGDG Synthase (MGD1) which adds to it a galactose from UDP-Gal.

173 ophosphorylase (UGP) alternatively makes UDP-galactose from uridine triphosphate and galactose-1-phos

174 beta-galactosidases, is able to remove both galactoses from XLLG oligosaccharides.

175 tions of each of seven saccharides (glucose, galactose, fructose, sucrose, trehalose, raffinose, and

176 from red macroalgae and are composed of a d-galactose (G unit) and l-galactose (L unit) alternativel

177 titotal ATG, but also antigalactose-alpha1-3-galactose (Gal) and anti-Neu5Gc antibodies, 2 xenocarboh

178 s specific for pig xenoantigens, alpha-(1,3)-galactose (GAL) and N-glycolylneuraminic acid (Neu5Gc),

179 luorinated glucose (Glc), mannose (Man), and galactose (Gal) derived by systematically exchanging eve

180 s with low VWF, with a secondary increase in galactose (Gal) exposure.

181 Promoters of seven galactose (GAL) metabolic genes from S. cerevisiae, when

182 studies have shown that the induction of the galactose (GAL) metabolic network does not solely depend

183 oses derived from glucuronic acid (GlcA) and galactose (Gal).

184 Following previous growth in galactose, GAL gene transcriptional memory confers a str

185 Glucose- (glc-) and galactose- (gal-) PAS 10-mer structures are synthesized

186 m kinetics and urinary excretion of lactose, galactose, galactitol, and galactonate in 14 healthy men

187 Free sucrose, lactose, galactose, glucose and fructose were determined in yoghu

188 The neutral polysaccharides consist of galactose, glucose and mannose whereas the acidic polysa

189 Finally, providing specific nutrients (galactose/glucose) to MuSCs directly controlled their fa

190 unique positioning of the 3-O-sulfated beta-galactose headgroup.

191 ption and that mutant WTA lacked appreciable galactose in all except one mutant - which retained a le

192 heir monosaccharide head groups, glucose and galactose in mammalian cells, gives rise to an analytica

193 umaric acid derivatives L-5-oxoproline and d-galactose in Sweetheart cherries were found.

194 tive site cleft and a decreased affinity for galactose in the -2 subsite, respectively, compared to Y

195 hondrial respiration (replacing glucose with galactose in the medium) and glucose deprivation sensiti

196 yme that hydrolyzes lactose into glucose and galactose in the small intestine, where they are absorbe

197 To this end, we further show that the galactose in UDP-galactose is incorporated into mature,

198 henodeoxycholic acid- and GW4046-induced FXR-galactose-induced gene 4 luciferase reporter activity (P

199 cells, the size distributions of cells with galactose-induced Whi5 expression and wild-type cells ar

200 In Saccharomyces cerevisiae, galactose-inducible rare-cutting endonucleases are commo

201 een the HAS1-TDA1 alleles specifically under galactose induction and saturated growth.

202 Galactose induction requires cell cultivation in subopti

203 this allows the cell to control the flux of galactose into the cell in the presence of glucose.

204 alpha-Linked galactose is a common carbohydrate motif in nature that

205 cose is absent from the nasopharynx, whereas galactose is abundant.

206 d, we further show that the galactose in UDP-galactose is incorporated into mature, de novo glycans.

207 DPG), kaempferol and UDPG, quercetin and UDP-galactose, isoliquiritigenin and UDPG, and luteolin and

208 First, GAL1 coding for galactose kinase is deleted to eliminate galactose utili

209 We used a modified galactose kinase markerless gene deletion approach and f

210 are composed of a d-galactose (G unit) and l-galactose (L unit) alternatively linked by alpha-1,3 and

211 3,6-anhydro-alpha-L-galactose (L-AHG) is one of the main monosaccharide cons

212 with three ligands of different affinities: galactose, lactose and N-acetyl-lactosamine using trypto

213 re inhibitory for biofilm formation, whereas galactose, lactose, and low concentrations of sialic aci

214 Macrophage galactose lectin (MGL) together with AMR mediated cleara

215 receptors mannose receptor (MR), macrophage galactose lectin (MGL), and the Toll-like receptors (TLR

216 otein synthesis, and ricin B can bind to the galactose ligand on the cell membrane of host cells.

217 omogeneous distribution of bioactive RGD and galactose ligands required for spheroid tethering and fo

218 In the presence of galactose, lithium ions activate the unfolded protein re

219 The carbohydrate epitope galactose-alpha-1,3-galactose, located on the Fab region of cetuximab, was i

220 In glucose or galactose, loss of ldh increased capsule production, whe

221 s the hydrolysis of lactose into glucose and galactose making it useful for lactose intolerant patien

222 ccharides (N-acetylneuraminic acid (Neu5Ac), galactose, mannose, and fucose) and significantly (p < 0

223 patient-derived fibroblasts in glucose-free galactose medium revealed a respiratory chain defect in

224 apitulated the delay phenotype in 1% glucose-galactose medium, and most had partial effects when test

225 delay when tested individually in 1% glucose-galactose medium.

226 odes UDP-galactose-4-epimerase, an enzyme of galactose metabolism and glycosylation responsible for t

227 ol and galactonate are 2 products of hepatic galactose metabolism that are candidate markers for the

228 in ubx4Delta strain, thereby reestablishing galactose metabolism, UPR activation, and cellular adapt

229 reticulum functions are intertwined through galactose metabolism.

230 Galactose-modified thymidine, LNA-T, and 2'-amino-LNA-T

231 Dendritic galactose moieties have a high affinity for galectin-exp

232 ng sites enables simultaneous binding of two galactose moieties, which strongly enhances binding.

233 Fecal anti-galactose/N-acetylgalactosamine lectin immunoglobulin A

234 3-galactose (alpha-gal) and Neu5Gc-alpha-2-6-galactose (Neu5Gc) have been shown to be antigenic when

235 ected N-glycans that also contain a terminal galactose on the alpha1-6-linked mannose branch, this le

236 re noted, whereas the presence or absence of galactose on the Fc glycan of IgG1 did not alter Fcgamma

237 id residues from host glycoproteins, exposed galactose on the surface of septal epithelial cells, the

238 ility to macrophages than NPs decorated with galactose only and a blended mixture of mannose and gala

239 )C NMR which revealed alpha and beta form of galactose or glucose in MOS, respectively.

240 solubilized glycosyltransferases that attach galactose or sialic acid.

241 f this promoter in media containing glucose, galactose, or glycerol as a carbon source.

242 Galactose oxidase (GAO) contains a Cu(II)-ligand radical

243 s alcohol oxidation mediated by Cu/TEMPO and galactose oxidase.

244 lization precursors are obtained from beta-d-galactose pentaacetate in a nine-step sequence featuring

245 e, N-acetyl-D-galactosamine, D-glucose and D-galactose, present on the cell surface.

246 esses two transport systems for galactose, a galactose-PTS system and an ABC galactose transporter.

247 In galactose, pyruvate metabolism was shunted toward acetyl

248 and homocysteine; and soluble carbohydrates: galactose, raffinose and cellobiose.

249 of tagatose from lactose with a tagatose and galactose ratio of 9:1 in the reaction broth.

250 potentially interfere with the high affinity galactose-recognition element that plays a critical role

251 ase activity and causing the accumulation of galactose-related metabolites, including galactose-1-pho

252 galactosidase BoGal36A increased the rate of galactose release by about 10-fold compared with the rat

253 The number of terminal galactose residues could be interpreted as a stepwise de

254 cross-link surface glycoproteins by binding galactose residues that are normally hidden below termin

255 orrelated directly to the number of terminal galactose residues that remained.

256 inopyranose (d-Arap) caps the LPG side-chain galactose residues, blocking interaction with the midgut

257 expressing terminal N-acetylgalactosamine or galactose residues.

258 position of arabinose or the O-6 position of galactose residues.

259 hor is modified by the addition of 4-6 alpha-galactose residues.

260 1, whose reducing end sugars are glucose and galactose, respectively.

261 ding to plasma concentrations in patients on galactose-restricted diet) and compared to controls.

262 icant parameter affecting linearly yield and galactose/rhamnose contents.

263 ed to 59 samples of Grana Padano PDO cheese: galactose showed the highest concentration and variabili

264 I backbone and two of them from the branched galactose-sialic acid disaccharide contained in this seq

265 longer substrates and is less restricted by galactose side-groups than the mannanase BoMan26A of the

266 a novel class of pathognomonic marker due to galactose stress in affected neonates.

267 ions of two pairs of ancient paralogs of the GALactose sugar utilization network in two yeast species

268 nucleatum host-cell binding and entry using galactose sugars, l-arginine, neutralizing membrane prot

269 al gene expression of genes encoding several galactose-sulfurylases, carbohydrate-sulfotransferases,

270 Here, we confirm the clinical benefit of galactose supplementation in PGM1-CDG-affected individua

271 as well as the levels of UDP-glucose and UDP-galactose, the nucleotide sugars that are required for E

272 in both species-the GAL genes are induced by galactose-there are major differences in both the quanti

273 ed "-Omics" analyses showed that addition of galactose to culture medium improves total oxidative cap

274 us sakei L-arabinose isomerase (LsLAI) for D-galactose to D-tagatose isomerization-that is limited by

275 oss of galactokinase (GALK), phosphorylating galactose to galactose-1-phosphate.

276 spond to the external concentration ratio of galactose to glucose, a phenomenon of unknown mechanism

277 code the enzymes needed for cells to convert galactose to glucose.

278 , and a fitness detriment during the glucose-galactose transition but a benefit when glucose is in ex

279 The ABC galactose transport system is regulated by a three-compo

280 In cardiomyocytes, galactose (transported through SGLT1) did not activate N

281 an acetyl-CoA transporter (pfact) and a UDP-galactose transporter (pfugt).

282 galactose, a galactose-PTS system and an ABC galactose transporter.

283 ing tracer-based metabolomics, we found that galactose treatment of PGM1-CDG fibroblasts metabolicall

284 The human macrophage galactose-type lectin (MGL) is a C-type lectin character

285 The macrophage galactose-type lectin (MGL) is a C-type lectin that bind

286 inked glycans and their receptor, macrophage galactose-type lectin (MGL), on CD163(+) TAMs in gliobla

287 of diverse origin, i.e. the human macrophage galactose-type lectin, a plant lectin, Pisum sativum agg

288 o the alpha-gal epitope (galactose-alpha-1,3-galactose) ubiquitously expressed on glycolipids/glycopr

289 alactosyltransferase and uridine diphosphate galactose (UDP-Gal) for global and site-specific analysi

290 be either mono-functional, synthesising UDP-galactose (UDP-Gal), or bi-functional, synthesising UDP-

291 ns corresponding to the externally available galactose units (20%).

292 e hydrolase), can cleave alpha-(1->3)-linked galactose units from a linear blood group antigen.

293 for galactose kinase is deleted to eliminate galactose utilization.

294 duced hydrolysis of lactose into glucose and galactose varied depending on the concentration, hydroly

295 decreased galactosylation in glycan chains, galactose was administered to individuals with PGM1-CDG

296 GC-FID results proved that galactose was the dominant sugar in the extracted polysa

297 le, maltose, fructose, sucrose, lactose, and galactose) was observed.

298 se, xylose, and glucose, whereas mannose and galactose were present in small amounts.

299 reversible reactions: interconversion of UDP-galactose with UDP-glucose and interconversion of UDP-N-

300 l alpha2-3-linked sialic acid and underlying galactose yielding a terminal N-acetyl glucosamine.