ライフサイエンスコーパス: galactoside

1 affinity for melibiose or nitrophenyl-alpha-galactoside.

2 of the cytoplasmic cavity in the presence of galactoside.

3 rple was cyanidin 3-xylosyl(feruloylglucosyl)galactoside.

4 the artificial substrate o-nitrophenyl-beta-galactoside.

5 the chromogenic substrate o-nitrophenyl-beta-galactoside.

6 side was rapidly metabolized to peonidin-3-O-galactoside.

7 anomerization to obtain good yields of alpha-galactosides.

8 te -1 to enable the binding of various alpha-galactosides.

9 FV was also affected in the presence of beta-galactosides.

10 modification blocks galectin binding to beta-galactosides.

11 ndary carbon sources such as alpha- and beta-galactosides.

12 recognition domain that interacts with beta-galactosides.

13 naling requires binding to cell surface beta-galactosides.

14 ratios despite intense selection on the pure galactosides.

15 posed to substantial amounts of dietary beta-galactosides.

16 ility of PllA as a probe for detecting alpha-galactosides.

17 3-glucuronide (29.66 mg/100 g), cyanidin 3-O-galactoside (130.93 mg/100 g) and loganic acid (303.3 mg

18 yptophan, Wessely-Moser isomers apigenin-6-C-galactoside-8-C-arabinoside & apigenin-6-C-arabinoside-8

19 -galactoside and cyanidin-3-xylosyl-glucosyl-galactoside accounted for the highest amount of the tota

20 its three major lipids, acylated cholesteryl galactoside (ACGal), monogalactosyl diacyglycerol (MGalD

21 Furthermore when galactoside affinity is measured as a function of pH, an

22 Accumulation of galactoside against a concentration gradient does not in

23 LacY catalyzes accumulation of galactosides against a concentration gradient by couplin

24 anslocation of H(+) to drive accumulation of galactosides against a concentration gradient.

25 ns between a tryptophan residue and the beta-galactoside alpha face are observed.

26 beta-1,3-glucosyltransferase (B3GLCT), beta-galactoside alpha-2,3-sialyltransferase 5 (ST3GAL5), and

27 study we identified high expression of beta-galactoside alpha-2,3-sialyltransferase, ST3GAL6, in MM

28 Our results suggest that the beta-galactoside alpha-2,6-sialyltransferase 1 (ST6Gal-I) sia

29 lactosyltransferase 1 (B4GALT1) and ST6 beta-galactoside alpha-2,6-sialyltransferase 1 (ST6GAL1) cata

30 ether inactivation of the UDP-galactose:beta-galactoside-alpha1-3-galactosyltransferase (alpha1,3GT)

31 Knockdown of beta-galactoside alpha2,6-sialyltransferase (ST6Gal-1) by RNA

32 s to inhibit galectin signaling, namely beta-galactoside alpha2,6-sialyltransferase (ST6Gal-I).

33 ation of the Fc fragment is mediated by beta-galactoside alpha2,6-sialyltransferase 1 (ST6Gal-1), act

34 from overexpression of the Golgi enzyme beta-galactoside:alpha2-6-sialyltransferase (ST6Gal-I).

35 prisingly, however, binding of high-affinity galactoside analogues is severely compromised in the mut

36 Here we show that DDAOG, a conjugate of beta-galactoside and 7-hydroxy-9H-(1,3-dichloro-9,9-dimethyla

37 ns, catalyzes the coupled translocation of a galactoside and a H(+) across the cytoplasmic membrane o

38 The effects of the di-galactoside and alternative glucose extension were also

39 ns, catalyzes the coupled translocation of a galactoside and an H(+) across the Escherichia coli memb

40 lix protein LacY that catalyzes symport of a galactoside and an H(+), was studied.

41 atalyzes coupled stoichiometric symport of a galactoside and an H(+).

42 Cyanidin-3-xylosyl-galactoside and cyanidin-3-xylosyl-glucosyl-galactoside

43 lactoside, cyanidin arabinoside, delphinidin galactoside and delphinidin arabinoside.

44 DH-negative bacteria were impaired for alpha-galactoside and galactose metabolism.

45 Quercetin 3-O-rhamnosyl galactoside and glucoside were higher in green (h degree

46 against a concentration gradient by coupling galactoside and H(+) transport (i.e., symport).

47 n, respectively) together with quercetin-3-O-galactoside and quercetin-3-O-glucoside (15 and 140mugg(

48 and caffeic acids and between quercetin-3-O-galactoside and quercetin-3-O-glucoside.

49 in family defined by their affinity for beta-galactosides and by their conserved carbohydrate recogni

50 chia coli catalyzes coupled translocation of galactosides and H(+) across the cell membrane.

51 However, the minerals, total alpha-galactosides and inositol phosphates contents were reduc

52 The content of alpha-galactosides and IPs was determined by high performance

53 ession of Mrt is exclusively up-regulated by galactosides and sucrose.

54 C-arabinoside & apigenin-6-C-arabinoside-8-C-galactoside, and 9,12,13-trihydroxy-trans-10-octadecenoi

55 luteolin 7-O-malonylglucoside, myricetin 3-O-galactoside, and naringenin tri glycoside.

56 Loganic acid, cornuside, cyanidin3-galactoside, and pelargonidin 3-galactoside were determi

57 noninducing species, orthonitrophenyl-beta-D-galactoside; and (3) transform an inducible switch to on

58 ive preferences for growth on different beta-galactosides are observed within Bifidobacterium members

59 way, and could be involved in the sorting of galactoside-bearing glycoconjugates, since it was found

60 pK for binding is approximately 10.5); (iii) galactoside binding and dissociation, not microH+, are t

61 However, galactoside binding by mutants defective in lactose-indu

62 driving forces for alternating access; (iv) galactoside binding involves induced fit, causing transi

63 cal range, but the apparent pK (pK(app)) for galactoside binding is 10.5.

64 Because the pK(a) value for galactoside binding is approximately 10.5, protonation o

65 Galectin-9 is a beta-galactoside binding lectin capable of modulating immune

66 Galectin-3 is a beta-galactoside binding lectin that is highly expressed in f

67 s performed with a model ligand on both beta-galactoside binding lectins showed additional interactio

68 Increasingly, the B-galactoside binding lectins, termed galectins, are being

69 G) analogues as recognition elements of beta-galactoside binding lectins.

70 ase (PI3K) and Ras is suppressed by the beta-galactoside binding protein (betaGBP) molecule, a cytoki

71 We report here that beta-galactoside binding protein (betaGBP), an antiproliferat

72 Galectin-3 (Gal-3), a member of the beta-galactoside binding protein family containing the NWGR a

73 ced cellular expression of the secreted beta-galactoside binding protein Galectin 1-like 2 (Drgal1-L2

74 Alterations in the production of the beta-galactoside binding protein galectin-3 and of MUC2 intes

75 The beta-galactoside binding protein galectin-3 modulates the exp

76 Galectin-3 is a beta-galactoside binding protein that has also been associate

77 gulates intracellular expression of the beta-galactoside binding protein, Galectin-3 (Gal-3).

78 Recently, the overexpression of a beta-galactoside binding protein, galectin-3 (LGALS3), has be

79 plasmic loop IV/V and in the vicinity of the galactoside binding site at the interface of helices IV,

80 Galactoside binding strongly modifies kinetic, energetic

81 conformational changes in LacY initiated by galactoside binding were monitored in real time by Trp q

82 teoliposomes and were compared with rates of galactoside binding.

83 ing 6 of 8 conserved amino acids involved in galactoside-binding activity.

84 Galectin-3 is a member of the beta-galactoside-binding animal lectin family expressed in va

85 Galectin-3 is a member of a beta-galactoside-binding animal lectin family.

86 The beta-galactoside-binding animal lectin galectin-3 is predomin

87 Galectin-7 is a beta-galactoside-binding animal lectin specifically expressed

88 Galectin-3 is a member of a family of beta-galactoside-binding animal lectins expressed abundantly

89 Galectin-3 belongs to a family of beta-galactoside-binding animal lectins expressed in several

90 3 is a member of the galectin family of beta-galactoside-binding animal lectins.

91 Here, we show that galectin-3 (Gal3), a beta-galactoside-binding cytosolic lectin, unifies and coordi

92 ative trait locus for the transcript soluble galactoside-binding lectin 9 (LGALS9) that is in linkage

93 Here, we show that a beta-galactoside-binding lectin [galectin-3 (gal3)] that reco

94 Galectin (Gal)-3 is a beta-galactoside-binding lectin and currently intensely studi

95 Galectin-3 (gal-3) is a beta-galactoside-binding lectin expressed in diverse fibrotic

96 Galectin-1 is a galactoside-binding lectin expressed in multiple tissues

97 Galectin-3 (Gal-3) is a member of the beta-galactoside-binding lectin family and plays an important

98 we show that galectin-12, a member of a beta-galactoside-binding lectin family preferentially express

99 Galectin-1 is a member of the conserved beta-galactoside-binding lectin family that binds galactoside

100 mune surveillance by overexpressing the beta-galactoside-binding lectin galectin-1.

101 acetylglucosamine, a ligand for a ubiquitous galactoside-binding lectin galectin-1.

102 The role played by the beta-galactoside-binding lectin galectin-3 (Gal-3) in airway

103 Several lines of evidence implicate the beta-galactoside-binding lectin galectin-3 in development and

104 e hypothesized that human galectin-3, a beta-galactoside-binding lectin involved in immune regulation

105 Galectin-3 is a soluble ss-galactoside-binding lectin released by activated cardiac

106 Galectin-3 is a beta-galactoside-binding lectin that is highly expressed with

107 Galectin-3 is a beta-galactoside-binding lectin that plays an important role

108 Galectin-3 is a beta-galactoside-binding lectin widely expressed on epithelia

109 show that ablation of galectin-3 (Gal-3), a galactoside-binding lectin, accelerates high-fat diet-in

110 Galectin-3 (GAL3), a beta-galactoside-binding lectin, confers chemoresistance to a

111 Galectin-1, a beta-galactoside-binding lectin, is involved in many physiolo

112 e show that expression of galectin-3, a beta-galactoside-binding lectin, is up-regulated in a mouse m

113 tudy, we demonstrate that galectin-8, a beta-galactoside-binding lectin, is upregulated in the epider

114 Galectin-1 (Gal-1), a beta-galactoside-binding lectin, plays a profound role in mod

115 -1 (Gal-1), an evolutionarily conserved beta-galactoside-binding lectin, plays essential roles in the

116 Galectin-9, a beta-galactoside-binding lectin, promotes immune suppression

117 Galectin-1 is a member of a family of beta-galactoside-binding lectins that are soluble adhesion mo

118 Galectins are beta-galactoside-binding lectins that regulate diverse cell b

119 he prototype galectin family, which are beta-galactoside-binding lectins, exhibit subunit-specific al

120 tin (Gal)-3, a M(r) 31000 member of the beta-galactoside-binding protein family, is a multifunctional

121 Galectin-3 (Gal-3), a member of a beta-galactoside-binding protein family, is involved in RNA p

122 Galectin-7, a member of the beta-galactoside-binding protein family, is primarily express

123 Mammalian beta-galactoside-binding protein Galectin-3 (Gal-3) modulates

124 The galactoside-binding protein galectin-3 is increasingly r

125 The beta-galactoside-binding protein galectin-3 is widely express

126 The beta-galactoside-binding protein galectin-9 is critical in re

127 Galectin-1, a beta-galactoside-binding protein highly expressed in the thym

128 Galectin-3 is a multifunctional beta-galactoside-binding protein implicated in apoptosis, mal

129 Galectin-3 is a beta-galactoside-binding protein implicated in tumor progress

130 LGALS4, encoding a galactoside-binding protein involved in cell-cell and ce

131 Galectin-3 (Gal-3), a beta-galactoside-binding protein is expressed in a specific c

132 Galectin-3 (Gal-3) is a beta-galactoside-binding protein that is involved in cancer p

133 Galectin-1 (Gal-1), a beta-galactoside-binding protein, can alter fate and effector

134 msen-Friedenreich glycoantigen with the beta-galactoside-binding protein, galectin-3.

135 e Lgals3 gene encodes a multifunctional beta-galactoside-binding protein, galectin-3.

136 Galectin-3, a beta-galactoside-binding protein, has been implicated in a va

137 Galectin-3 (Gal-3), a beta-galactoside-binding protein, has been implicated in a va

138 Galectin-3, a beta-galactoside-binding protein, has been implicated in Wnt

139 an galectin-1 (dGal-1), a small dimeric beta-galactoside-binding protein, induces phosphatidylserine

140 Galectin-1 (Gal-1) is a beta-galactoside-binding protein, the expression of which is

141 Galectin-3 (Gal-3), a pleiotropic beta-galactoside-binding protein, was shown to be involved in

142 ted C4S, which binds less galectin-3, a beta-galactoside-binding protein.

143 ), which directly killed NK cells using beta-galactoside-binding protein.

144 Galectin-3 (gal-3), a member of the beta-galactoside-binding proteins family, was identified as a

145 Galectins are a family of beta-galactoside-binding proteins that are frequently altered

146 Galectins are a family of beta-galactoside-binding proteins that are widely found among

147 Galectins are a family of mammalian beta-galactoside-binding proteins that positively and negativ

148 er of the galectin family consisting of beta-galactoside-binding proteins with conserved carbohydrate

149 Consistent with their role as galactoside-binding proteins, the interaction with their

150 roteoliposomes reveals dramatic increases in galactoside-binding rates induced by interaction with th

151 A linear dependence of galactoside-binding rates on sugar concentration is obse

152 Tumor-derived galectin-1 (Gal-1), a beta-galactoside-binding S-type lectin, has been shown to enc

153 d were very poor inhibitors of the canonical galactoside-binding site for the tested galectins, with

154 own inhibitors of galectin-3 target its beta-galactoside-binding site in the carbohydrate recognition

155 r inhibition of the evolutionarily conserved galactoside-binding site of galectins has not been demon

156 HUK-921 bind galectin-3 outside of its beta-galactoside-binding site.

157 we transferred the Gal-1-KO mutation (lectin galactoside-binding soluble 1(-/-) ) from the B6 strain

158 Lectin galactoside-binding soluble 3 binding protein (LGALS3BP,

159 cking Gal-1-mediated angiogenesis or lectin, galactoside-binding, soluble, 1 deficiency results in a

160 The data further show that beta-galactosides block the interaction between FV and Gal8.

161 del was built for the three considered alpha-galactosides both in the seed and in the soaking water,

162 greatly reduced or no growth on sucrose and galactosides but did not affect growth on monosaccharide

163 ntially incapable of oxidizing galactose and galactosides, but instead efficiently catalyse the oxida

164 in compound, followed by cyanidin-3-xyloside-galactoside (C3XG, 49.56-70.12mg/L).

165 on, potentially through vaccination or novel galactoside compounds, could be an effective strategy fo

166 Final alpha-galactoside concentrations were low in all sourdoughs.

167 ntaneous hydrolysis of the 2,4-dinitrophenyl galactosides, confirming earlier studies on the role pla

168 The substrate, a caged D-luciferin-galactoside conjugate, must first be cleaved by beta-gal

169 ns are soluble-type lectins recognizing beta-galactoside containing glycans.

170 alectin family that has an affinity for beta-galactoside containing glycoconjugates.

171 n proteins that show binding to various beta-galactoside-containing glycans.

172 drate-binding protein with affinity for beta-galactoside-containing glycoconjugates, is upregulated u

173 Galectin-8 (Gal8) interacts with beta-galactoside-containing glycoproteins and has recently be

174 basis for PllA's high specificity for alpha-galactoside-containing ligands, and we show that PllA ca

175 gnificant increase, up to 85% in total alpha-galactosides content.

176 uding cyanidin 3-xylosyl-(caffeoyl-glucosyl)-galactoside, cyanidin 3-xylosyl-(p-hydroxybenzoyl-glucos

177 anidin 3-xylosyl-(p-hydroxybenzoyl-glucosyl)-galactoside, cyanidin 3-xylosyl-galactoside+vinylphenol,

178 omatography (CCC) and identified as cyanidin galactoside, cyanidin arabinoside, delphinidin galactosi

179 Peonidin-3-O-galactoside, cyanidin-3-O-arabinoside, and cyanidin-3-O-

180 ide, delphinidin-3-O-glucoside, cyanidin-3-O-galactoside, cyanidin-3-O-glucoside, cyanidin-3-O-arabin

181 that delphinidin-3-glucoside, delphinidin-3-galactoside, delphinidin-3-arabinoside and petunidin-3-a

182 Delphinidin-3-O-galactoside, delphinidin-3-O-glucoside, cyanidin-3-O-gal

183 of cultured malignant T cells induced a beta-galactoside-dependent inhibition of normal T-cell prolif

184 diate that undergoes alternating access; (v) galactoside dissociates, releasing the energy of binding

185 produced coupled reaction-diffusion of alpha-galactosides during the soaking-cooking process with a g

186 ), which must be protonated for LacY to bind galactoside effectively.

187 re compounds (chlorogenic acid, cyanidin-3-O-galactoside, epicatechin, rutin and quercetin) present i

188 The mutant no longer requires galactoside for IIA(Glc) binding as demonstrated by both

189 nize a wide variety of glycans with terminal galactosides for conferring epithelial cell adhesion.

190 Produced, diffused and degraded alpha-galactoside fractions were identified by performing a ma

191 mutant hardly catalyzes transport, but binds galactosides from either side of the membrane with the s

192 The major anthocyanins mono-galactoside, -glucoside and -arabinoside isomers of delp

193 black carrot pomace with cyanidin-3-xyloside-galactoside-glucoside-ferrulic acid (C3XGGF, 60.85-74.22

194 glycan-binding protein (GBP) that binds beta-galactoside glycan structures to orchestrate a variety o

195 Galactoside/H(+) symport across the cytoplasmic membrane

196 c membrane protein, catalyzes stoichiometric galactoside/H(+) symport by an alternating access mechan

197 mutants exhibit poor affinity for sugar, and galactoside/H(+) symport is abolished as well.

198 The lactose permease (LacY) catalyzes galactoside/H(+) symport via an alternating access mecha

199 n H(+) across the Escherichia coli membrane (galactoside/H(+) symport).

200 he cytoplasmic membrane of Escherichia coli (galactoside/H(+) symport).

201 perties of the N-terminal domain to initiate galactoside/H(+) symport.

202 Few side chains in the galactoside/H(+) symporter LacY (lactose permease of Esc

203 Delphinidin-3-galactoside has the highest capacity (13.062 +/- 2.729 m

204 alpha-Galactoside hydrolases mostly show a preference for the

205 -3-O-rhamnogalactoside, rutin, quercetin-3-O-galactoside (hyperoside), quercetin-3-O-glucoside (querc

206 Herein we report that isobutyl-beta-C-galactoside (IBCG) is also a promising inducer of gene e

207 ivers with 5-bromo-4-chloro-3-indolyl beta-d-galactoside in any of the 18 recipient mice analyzed wer

208 y undescribed demonstration of a cholesteryl galactoside in bacteria.

209 the hydrophobic patch displayed by the beta-galactoside in Le(x) is essential in both cases for thei

210 sh group fruits presented mainly quercetin-3-galactoside in their composition, while Rabbiteye group

211 Gal3 bound to N-linked beta-galactosides in Dsg2 extracellular domain and co-sedimen

212 sulfated carbohydrate ligands as 6-sulfated galactosides in the Ca(2+)-dependent binding site.

213 2) is responsible for the breakdown of alpha-galactosides in the lysosome.

214 evidence that it has high affinity for beta-galactosides in vitro.

215 alectin-3, an animal lectin recognizing beta-galactosides, in regulating dendritic cell motility both

216 ceable residue was mutated individually, and galactoside-induced opening or closing of periplasmic or

217 The addition of isopropyl-beta-D-thio-galactoside (IPTG) destabilizes but does not eliminate t

218 competitive inhibitor isopropyl thio-beta-D-galactoside (IPTG) requires the mutant enzyme to adopt i

219 olecules suggest that the O6 hydroxyl on the galactoside is essential for establishing a water-mediat

220 rate-binding proteins with affinity for beta-galactosides, is a key modulator of diverse cell functio

221 a special lectin with high affinity to beta-galactosides, is implicated in protection against ischem

222 e to form an alpha 1-3 link with beta-linked galactosides; it is part of a family of homologous retai

223 ricin glycome is enriched with terminal beta-galactosides, known binding partners for a family of mul

224 mber of the galectin family of secreted beta-galactoside lectins containing a conserved carbohydrate

225 roducing a phenyl aglycon moiety next to the galactoside ligands on both termini did indeed lead to a

226 Thus, beta-galactoside-mediated intravascular heterotypic and homot

227 resents five copies of m-nitrophenyl-alpha-D-galactoside (MNPG) rather than five copies of beta-D-gal

228 ntal conditions, benzochlorin without a beta-galactoside moiety or the related glucose conjugate did

229 of syringetin and laricitrin, and rhamnosyl-galactosides of myricetin, quercetin and isorhamnetin we

230 l-04 binds alpha-(1,6)-linked glucosides and galactosides of varying size, linkage, and monosaccharid

231 the anomeric group, namely, either an alpha-galactoside or an alpha-glucoside.

232 rene-based glycoclusters functionalized with galactosides or fucosides have been synthesized.

233 g concentrations of either lactulose, methyl-galactoside, or a 72:28 mixture of the two.

234 A member of the LacS subfamily of galactoside-pentose hexuronide translocators was identif

235 de, pelargonidin-3-O-glucoside, peonidin-3-O-galactoside, peonidin-3-O-glucoside, cyanidin-3-O-xylosi

236 olymorphism was preserved had switched their galactoside preference.

237 tor of a known ECA via affinity for the beta-galactosides present on this receptor.

238 luminating the structural basis of alpha-1,3-galactoside processing by the family as a whole.

239 ned in the extracts from quince, quercetin-3-galactoside (Q-Ga), quercetin-3-rutinoside (Q-Ru), querc

240 de, quercetin 3-O-rutinoside, quercetin 3'-O-galactoside, quercetin 3'-O-glucoside, quercetin 3'-O-rh

241 constituents gallic acid, rutin, quercetin-3-galactoside, quercetin-3-glucoside, myricetin, quercetin

242 econdary carbon sources, including the alpha-galactosides raffinose and stachyose.

243 ed with nutritional yeast, in terms of alpha-galactosides (raffinose, stachyose, verbascose), inosito

244 phism is protected only in a narrow range of galactoside ratios despite intense selection on the pure

245 24B11 blocked ricin attachment to galactoside receptors on primary mouse splenocytes and o

246 lectin-binding data indicate a possible beta-galactoside-recognized protein specificity of the galact

247 ncubated with lactose (known to bind to beta-galactoside-recognized proteins) prior to the addition o

248 Galectin-8 is a beta-galactoside-recognizing protein having an important role

249 chloro-9,9-dimethylacridin-2-on-7-yl) beta-D-galactoside, reducing the inhibitor concentration to K(i

250 beta-galactosidase and o-nitrophenyl-beta-d-galactoside, reducing their affinity and prejudicing the

251 a 200-800-fold lower catalytic rate on aryl galactosides relative to the WT enzyme.

252 galactoside-binding lectin family that binds galactoside residues on cell surface glycoconjugates.

253 ptations specific to lactulose and to methyl-galactoside, respectively.

254 eferentially recognizes alpha-2,6 sialylated galactosides showed strong binding reactivity with undif

255 ns needed for utilization of alpha- and beta-galactosides, slowed growth on diverse carbon sources, a

256 ch control attachment of a galalpha1-4gal di-galactoside structure (lic2A and lgtC phase-on) or an al

257 lactose transfer from UDP-Gal to beta-linked galactosides, such as lactose, and in the absence of an

258 67) because neither VGVAPG-like peptides nor galactoside sugars altered adhesion.

259 mino acids that are crucial for binding beta-galactoside sugars.

260 d respond to the information encoded by beta-galactoside sugars.

261 metastasis via its interactions with various galactoside-terminated glycans.

262 ealed a strict specificity of PllA for alpha-galactoside-terminating glycoconjugates.

263 mbrane; (ii) LacY must be protonated to bind galactoside (the pK for binding is approximately 10.5);

264 We found that di-galactoside, the alternative glucose extension, ChoP, an

265 ng sialic acid to the nonreducing end of the galactosides through a sialyltransferase-catalyzed enzym

266 ctins are a family of lectins that bind beta-galactosides through their conserved carbohydrate recogn

267 pant with respect to the data for thiomethyl galactoside (TMG).

268 lacF lacR double mutant (lacF encodes a beta-galactoside transport protein) grown in medium containin

269 assays, revealed that MRT was a sucrose and galactoside transporter.

270 ould be transported by the overproduced beta-galactoside transporters and cause the induction of alph

271 model in which lacR mutants overproduce beta-galactoside transporters, thereby overwhelming the induc

272 ference is also reflected in the alpha-(1,6)-galactoside uptake profile of the bacterium.

273 buted to the constitutive expression of beta-galactoside utilization genes in lacR mutants.

274 ransporters and cause the induction of alpha-galactoside utilization genes in the presence of both su

275 the ABC transporter encoded by the same beta-galactoside utilization locus.

276 -galactoside+vinylphenol, cyanidin 3-xylosyl-galactoside+vinylcatechol, cyanidin 3-xylosyl-(feruloyl-

277 chol, cyanidin 3-xylosyl-(feruloyl-glucosyl)-galactoside+vinylcatechol, cyanidin 3-xylosyl-galactosid

278 alactoside+vinylcatechol, cyanidin 3-xylosyl-galactoside+ vinylguaiacol, cyanidin 3-xylosyl-(feruloyl

279 acol, cyanidin 3-xylosyl-(feruloyl-glucosyl)-galactoside+vinylguaiacol were found in the shalgam samp

280 yl-glucosyl)-galactoside, cyanidin 3-xylosyl-galactoside+vinylphenol, cyanidin 3-xylosyl-galactoside+

281 alpha-Mannoside or beta-galactoside was immobilized on a gold disk electrode usi

282 Cyanidin-3-galactoside was more reactive (susceptible to hydration

283 a narrow periplasmic opening and an occluded galactoside was obtained, confirming many observations a

284 Among the anthocyanins, cyanidin-3-O-galactoside was rapidly metabolized to peonidin-3-O-gala

285 Cyanidin-3-galactoside was the major anthocyanin (146.9 mg/100 g).

286 Kaempferol-3-O-galactoside was the predominant compound in almond skin

287 e, cyanidin3-galactoside, and pelargonidin 3-galactoside were determined as the most dominant compoun

288 Specialists on each galactoside were isolated from chemostats that maintaine

289 , cyanidin-3-O-arabinoside, and cyanidin-3-O-galactoside were the predominant anthocyanins characteri

290 side and cyanidin-3-xylosyl(sinapoylglucosyl)galactoside) were incubated with pyruvic or caffeic acid

291 ots were cyanidin 3-xylosyl(sinapoylglucosyl)galactoside, whereas the main anthocyanin present in Dee

292 yzes the coupled stoichiometric symport of a galactoside with a cation (either Na(+), Li(+), or H(+))

293 a coli catalyzes stoichiometric symport of a galactoside with an H(+), using a mechanism in which sug

294 out the coupled stoichiometric symport of a galactoside with an H(+), using the free energy released

295 galactose from UDP-galactose to beta-linked galactosides with retention of its alpha configuration.

296 r depectinisation, two more ACNs (cyanidin-3-galactoside-xyloside and cyanidin-3-galactoside-xyloside

297 cid as the major ACN, followed by cyanidin-3-galactoside-xyloside-glucoside-coumaric acid, and cyanid

298 Unclarified BCJ contained cyanidin-3-galactoside-xyloside-glucoside-ferulic acid as the major

299 anidin-3-galactoside-xyloside and cyanidin-3-galactoside-xyloside-glucoside-sinapic acid) were also i

300 side-glucoside-coumaric acid, and cyanidin-3-galactoside-xyloside-glucoside.