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1 berrant and spectacular structures (clubs or galls).
2 imilation and importation of carbon into the gall.
3 ractions in the development of fusiform rust gall.
4 alled tissues and in tissues surrounding the gall.
5 all, horned gall, circular gall and ensiform gall.
6 increasing drought stress tolerance of crown galls.
7 ia, a physiological condition found in crown galls.
8 SAD6-OE-RNAi or by RNA interference in crown galls.
9 resulting in the formation of characteristic galls.
10 ions with forms that feed from leaf and root galls.
11 nfected with the ubc1 mutant did not produce galls.
12  simply diagnoses Agrobacterium-induced root galls.
13            The resulting structure - a plant gall - accommodates various needs of the foreigner, whic
14                  The plant species that make galls also are diverse.
15  was flower-like gall, horned gall, circular gall and ensiform gall.
16 genes are related pathogens that cause crown gall and hairy root diseases, which result from integrat
17 thogens that cause different diseases, crown gall and hairy root.
18 acid by bacterial pathogens that cause crown gall and related diseases.
19 eginning with those factors that cause plant galls and continuing through carbohydrate metabolism to
20 we analyzed the suberin composition of crown galls and found a reduction in the amounts of long-chain
21 lower susceptibility to the RKNs and smaller galls and GCs.
22                        are the largest plant galls and have great economic and medical values.
23                We examined the structures of galls and their functional adaptation using various macr
24                                         Both GALLS and VirE2 contain nuclear localization sequences a
25 with the galactose-inducible promoters GALS, GALL, and GAL1, allowing for low, moderate, and high lev
26 nes produced by Agrobacterium-infected plant galls, and can be applied to easily distinguish Agrobact
27 es were significantly enriched in developing galls, and that expression of many candidate genes invol
28                          The phylloxera leaf gall appears to be phenotypically and transcriptionally
29                                              Galls are an understudied phenomenon in plant developmen
30                      Since Arabidopsis crown galls are covered by a suberin-containing periderm inste
31               Unfortunately for plants, most galls are made for foes, some of which are deeply studie
32                 Agrobacterium-mediated plant galls are often misdiagnosed as nematode-mediated knots,
33 ms that these arthropods use to induce plant galls are poorly understood.
34 lower and/or fruit development in developing galls as opposed to ungalled leaves.
35 at exogenous nopaline produced by plant root galls binds to NocR, resulting in NocR/nopaline complexe
36  the organism was recovered from an infected gall bladder (Aeromonas veronii biotype veronii).
37 confirmed the presence of perforation of the gall bladder and cholecysto-cutaneous fistula.
38 entery, pancreas, portal hepatis, bile duct, gall bladder and jejunum was recorded from the right tho
39 L. monocytogenes can replicate in the murine gall bladder and provide evidence that its replication t
40       Histologic examination of the resected gall bladder and stenotic ureteric segment showed CMV in
41 d in enhanced flux of FC from macrophages to gall bladder bile and feces in vivo.
42 bile of Cel(-/-) mice, and the mass of CE in gall bladder bile was elevated.
43 ake of the tracer in the kidneys, liver, and gall bladder but rapid clearance via the urine/bladder w
44 ere readily identified in the bile ducts and gall bladder by special stains and by in situ hybridizat
45 reased risk of liver cancer, bladder cancer, gall bladder cancer, malignant lymphoma, and lung cancer
46 ial cells, the epidermis and hair follicles, gall bladder epithelium, choroid plexus, and biliary epi
47    FGF19 regulates bile acid homeostasis and gall bladder filling; FGF19 binds only to FGF receptor 4
48 port of cleavage products results in intense gall bladder fluorescence.
49                             Carcinoma of the gall bladder has a guarded prognosis with predominant si
50 al jaundice is rare in KD without associated gall bladder hydrops and tends to occur in older patient
51       Osseous metastasis in carcinoma of the gall bladder is rare and hence bone scintigraphy does no
52 xpressed in embryonic septum transversum and gall bladder mesenchyme.
53 velopmental defects in lung, intestinal, and gall bladder morphogenesis.
54 r stomach, respiratory tract, bile duct, and gall bladder of B6,129 CYP1A2-null and wild-type mice as
55 d spleen towards the left, and the liver and gall bladder on the right.
56 e animals, revealing strong signals from the gall bladder over a period of several days, in diseased
57             The ultrasound findings included gall bladder wall thickening in 66 patients (41.2%).
58    The present study suggests that increased gall bladder wall thickness, pleural effusion, ascites,
59                             In addition, the gall bladder was absent and the extrahepatic bile duct c
60 istula with its opening in the fundus of the gall bladder was revealed.
61  describe two patients with carcinoma of the gall bladder with osteolytic metastasis (stage 4).
62            Parasites are present in the gut, gall bladder, and biliary tree, and biliary epithelial c
63  In vivo, [(11)C]MT107 accumulated in liver, gall bladder, and intestines but only scarcely in hCD80-
64 d from foregut endoderm such as lung, liver, gall bladder, and pancreas.
65 l for the development of the mouse pancreas, gall bladder, and the interhepatic bile ducts.
66 led metabolites accumulates in the liver and gall bladder, consistent with the known routes of excret
67 ed a more selective pattern of expression in gall bladder, intestine, brain, ovary, spleen, and thymu
68 ts are solubilized by bile released from the gall bladder, resulting in the formation of two product
69 e canaliculi, and subsequently stored in the gall bladder.
70 L. monocytogenes may be carried in the human gall bladder.
71 ng mirror-image reversals of heart, gut, and gall bladder.
72 protein expressed by the human intestine and gall bladder.
73  scan or PET CT in cases of carcinoma of the gall bladder.
74 copy to rule out Veress needle injury to the gall bladder.
75 onse: 11 with bile duct cancer and four with gall-bladder carcinoma.
76 akage, fluid accumulation, pleural effusion, gall-bladder wall thickening and rapid haematocrit rise
77                                     Foxf1+/- gall bladders were significantly smaller and had severe
78  mutant for tumor formation, indicating that GALLS can substitute for VirE2.
79 sulting in the development of spindle-shaped galls (cankers) on branches or stems.
80 ategories (i.e. oak, grape seed, grape skin, gall, chestnut, quebracho, tea and acacia).
81 drolysable tannins from various sources (nut galls, chestnut and oak woods) and sulfur dioxide on met
82 evolution trail was flower-like gall, horned gall, circular gall and ensiform gall.
83                                Unlike VirE2, GALLS contains a nucleoside triphosphate binding motif s
84                                Unlike VirE2, GALLS contains ATP-binding and helicase motifs similar t
85                            The presence of a gall cost a ramet an average of 1743 seeds, but the cost
86                               Coral-dwelling gall crabs (Cryptochiridae) are obligate symbionts of st
87 , triggering morphogenetic changes to induce galls, de novo formed 'pseudo-organs' containing several
88                        Our results show that gall development involved the amplification of existing
89 t whether miRNAs play roles in fusiform rust gall development, we cloned and identified 26 miRNAs fro
90 T plays an important role in correct GCs and gall development, where miRNA172 is modulated by auxins.
91 the lipid transfer protein AtLTPI-4 in crown gall development.
92 iral N gene response against bacterial crown gall disease and highlight the importance of achieving t
93  systemic acquired resistance (SAR) on crown gall disease caused by Agrobacterium tumefaciens.
94 um plants that are highly resistant to crown gall disease development.
95 ed to easily distinguish Agrobacterium crown gall disease from nematode disease.
96 ns is a soilborne pathogen that causes crown gall disease in many dicotyledonous plants by transfer o
97 8, the pathogenic bacteria that causes crown gall disease in plants, harbors one circular and one lin
98 is bacterium is the causative agent of crown gall disease in plants.
99 m tumefaciens, the causative agent for crown gall disease of plants has proven a productive model for
100 imary virulence factor responsible for crown gall disease of plants.
101                                        Crown gall disease, caused by the soil bacterium Agrobacterium
102 itro, how whitefly infestation affects crown gall disease.
103       Agrobacterium tumefaciens causes crown gall disease.
104 nt pathogen and the causative agent of crown gall disease.
105  its own DNA into host plants to cause Crown Gall disease.
106  method to produce plants resistant to crown gall disease.
107 e variation in their susceptibility to crown gall disease.
108 pathogenic bacterium that induces the 'crown gall' disease in plants by transfer and integration of a
109                                          The gall-dwelling colonies of a social aphid species (Pemphi
110 columnar ellipsoid body-protocerebral bridge gall (E-PG) neuron and ellipsoid body (EB) R2/R4m ring n
111 so report that, during further maturation of galls, enlargement of host cells invaded by the pathogen
112 e plant KRP6 transcription to the benefit of gall establishment.
113                   In this issue of Immunity, Gall et al. characterize, in a murine model of autoimmun
114                   In the second study, by Le Gall et al., the modulation of epitope immunodominance a
115                        Using the protocol of Gall et al., we developed a robust methodology for ampli
116 es two proteins from one open reading frame: GALLS-FL and a protein comprised of the C-terminal domai
117                        VirD2 interacted with GALLS-FL and localized inside the nucleus, where its pre
118                                              GALLS-FL and VirE2 contain nuclear localization signals
119                                              GALLS-FL tagged with yellow fluorescent protein localize
120             Unlike VirE2, full-length GALLS (GALLS-FL) contains ATP-binding and helicase motifs simil
121 s from a specialist herbivore, the goldenrod gall fly (Eurosta solidaginis).
122 e E,S-conophthorin produced by the goldenrod gall fly as the specific chemical component that elicits
123 cheal cells of the freeze-tolerant goldenrod gall fly, Eurosta solidaginis, chilling to 0 degrees C e
124                                              Gall formation also perturbed vascular development with
125 ance and differentiation, that a decrease in gall formation did not prevent pathogen development.
126  insect infestation on Agrobacterium induced gall formation has not been investigated.
127 we explored the cellular events that underly gall formation in Arabidopsis thaliana with the help of
128 r salicylic acid (SA) synthesis, compromised gall formation indicating an involvement of SA in whitef
129      This finding demonstrates that although gall formation is a typical symptom of the disease and i
130 s exhibited at least a two-fold reduction in gall formation on both stem and crown root.
131 opment of reproductive structures is part of gall formation, we expected to find significantly elevat
132 ents responsible for arthropod-induced plant gall formation.
133 ion has in the study of insect-induced plant gall formation.
134 t ultimately dominate and be responsible for gall formation.
135 ells in a meristematic state was crucial for gall formation; disruption of the VC activity significan
136                       Cross-sections through galls formed by feeding nematodes on rme1 roots were ide
137                     Mostly all fusiform rust galls formed under field conditions are produced as a re
138  in ways that appear to directly benefit the gall former.
139                                  Across both gall-former species we find consistent differences in bo
140  leaf damage by four insect guilds (chewers, gall formers, leaf miners and rollers) on silver birch (
141                                            A gall-forming aphid has an extended post-reproductive lif
142                           Here we report the gall-forming aphid-like parasite phylloxera, Daktulospha
143  underlie the distributions of 10 species of gall-forming arthropods and their ability to adapt to ne
144                                              Gall-forming arthropods are highly specialized herbivore
145 ver, the lack of parallel genetic studies on gall-forming arthropods limits our ability to define the
146                            Endoparasitism by gall-forming insects dramatically alters the plant pheno
147 ilarity strongly affect the distributions of gall-forming species, individually and as a community.
148 ed with wild-type cells that often developed galls from initially chlorotic tissue, plants infected w
149                    Unlike VirE2, full-length GALLS (GALLS-FL) contains ATP-binding and helicase motif
150 ive gene repression events observed in early gall/GCs development are thought to be mediated by post-
151 ::GUS showed restricted promoter activity in galls/GCs that was regulated by auxins through auxin-res
152                       Here, we show that the GALLS gene encodes two proteins from one open reading fr
153  of these domains abolish the ability of the GALLS gene to substitute for virE2.
154                                          The GALLS gene was essential for pathogenicity of A. rhizoge
155 ey transfer T strands efficiently due to the GALLS gene, which complements an A. tumefaciens virE2 mu
156 tion, and defense investment in favor of the galling habit.
157           The manipulation of post-emergence galls had little effect on the galler-abundance and surv
158 oxidizes l-Gal to l-galactono-1,4-lactone (l-GalL), has been purified from pea seedlings and cloned f
159 r and histological features of these petiole galls have been preserved in exquisite detail, including
160 We infer the evolution trail was flower-like gall, horned gall, circular gall and ensiform gall.
161 nd semicircular xylems traces in an ensiform gall in cross sectional views, which would provide more
162 t occurrence of external foliage-feeding and galling in the terrestrial fossil record.
163 te plants remarkably decreased the number of galls in transformed hairy roots inoculated with RKN.
164                                   We propose gall inception for discovering unifying features of the
165 an and Nurudea shiraii on Rhus chinensis and gall induced by Kaburagia rhusicola rhusicola on Rhus po
166                                          The galls induced by Schlechtendaia chinensis, Schlechtendai
167 d foes, talk about molecules that plants and gall-inducers use to get what they want from each other,
168 and cyst nematodes in particular, as well as gall-inducing and leaf-mining insects, manipulate plant
169 ctant of a closely associated herbivore, the gall-inducing fly Eurosta solidaginis, exhibit enhanced
170                                              Gall-inducing insects and nematodes engage in sophistica
171 f tall goldenrod (Solidago altissima) to the gall-inducing tephritid Eurosta solidaginis.
172                                              Gall induction can be viewed as ecosystem engineering si
173 ic and kill E. solidaginis hatchlings before gall induction.
174 ow that the main colorant of historical iron gall ink (IGI) is an amorphous form of Fe(III) gallate.x
175 tilized TERS to identify indigo dye and iron gall ink in situ on Kinwashi paper.
176  In addition, TERS was used to identify iron gall ink on a historical document with handwritten text
177 mponents of a complex chemical mixture, iron gall ink, can be identified.
178 t the inks are related to the family of iron gall inks, whose introduction is commonly attributed to
179 een growth and terpenoids manifested through galling insects supported the GDBH.
180 optera: Cynipidae) feed within inconspicuous galls inside the flowering stems of the prairie perennia
181 ecosystem engineering effect (post-emergence galls) instead of the species itself, we demonstrate the
182                               Post-emergence galls interfered with aphid inquilinism-likely by the pr
183                                            l-GalL is a proposed substrate for ascorbate biosynthesis
184         Because the only known function of l-GalL is ascorbate synthesis, these antisense plants prov
185  modification) likely by killing parasitised galling larvae.
186 by arbuscular mycorrhizal fungi respond in a gall-like manner, and present a research agenda.
187                Notably, chewing, sucking and gall-making herbivores were more affected by top-down th
188 ; Diptera: Cecidomyiidae), a plant parasitic gall midge and a pest of wheat (Triticum spp.), with the
189 cally important gall midge species, the rice gall midge and the Hessian fly, with their host plants,
190 ibility gene for infestation of wheat by the gall midge M. destructor, commonly known as the Hessian
191                   The interaction of a third gall midge species, the orange wheat blossom midge, with
192 e interactions of two economically important gall midge species, the rice gall midge and the Hessian
193 lly anchored genetic map is the first of any gall midge species.
194 Mayetiola destructor), the most investigated gall midge, was the first insect hypothesized to have a
195 netics underlying important aspects of these gall midge-grass interactions, a unique opportunity exis
196                                              Gall midges constitute an important group of plant-paras
197                                              Gall midges induce formation of host nutritive cells and
198 e discoveries suggest that the HF, and other gall midges, may be considered biotrophic, or hemibiotro
199 eat worldwide, and an emerging model for all gall midges, we investigated its antioxidant responses d
200 ago smuts, root knot and cyst nematodes, and gall midges.
201  group, the Eriophyoidea, which includes the gall mites and comprises at least 3,500 Recent species,
202                             Antiquity of the gall mites in much their extant form was unexpected, par
203                             According to the Gall model, 37.4 breast cancers were expected in the mod
204 find consistent differences in body size and gall morphology associated with host plant use, as well
205 nnins from oak, extract of gallotannins from gall nuts and extract of proanthocyanidins from grape se
206      Agrobacterium tumefaciens-derived crown galls of Arabidopsis (Arabidopsis thaliana) contain elev
207 s, analysis of Meloidogyne incognita-induced galls of KRP6-overexpressing lines revealed a role for t
208 n rme1 roots were identical to sections from galls of susceptible tomato roots.
209  strain C58, highly expressed AtLTPI-4 Crown galls of the atltpI-4 loss-of-function mutant were much
210 Drosophila tracheal system, mutations in oak gall (okg) and conjoined (cnj) confer identical defects,
211 mental processes leading to the formation of galls on its underground parts.
212 hly complex species- and generation-specific galls on oaks and other Fagaceae.
213       We tested the effect of post-emergence galls on the galler, as well as on the galler-parasitoid
214 Ns in vivo via significant reduction of root-galls on tomato (Solanum lycopersicum var. Rutgers).
215 le from chrysopine, a newly discovered crown gall opine.
216 ogens that are the causative agents of crown gall or hairy root disease.
217  and expressed in plant cells, causing crown gall or hairy root disease.
218 ene transfer leads to the formation of crown galls or hairy roots, due to expression of transferred T
219 f wild grapevine (Vitis riparia) leaves to a galling parasite, phylloxera (Daktulosphaira vitifoliae)
220  but fewer nodules and nematode-induced root galls per plant, than control hairy roots.
221                                    We assume gall production costs the plant.
222                                          The GALLS protein can complement an A. tumefaciens virE2 mut
223          Instead, these bacteria express the GALLS protein, which is essential for their virulence.
224                          In plant cells, the GALLS proteins interacted with themselves, VirD2, and ea
225                          On some hosts, both GALLS proteins were required to substitute for VirE2.
226  different botanical sources (oak, chestnut, gall, quebracho, tea, grape skin and grape seed) were co
227                                         Many galls resemble flowers or fruits, suggesting that elemen
228 can be viewed as ecosystem engineering since galls serve as habitat for other species.
229   Many are found in cryptic habitats such as galls, several widespread genera are surface feeders on
230 crescentic pit, circular-oval pit, or a true gall) shows that species within crab genera tend to inha
231  the VC activity significantly decreased the gall size.
232 e (Sternorrhyncha: Psyllidae), the commonest galling species associated with B. dracunculifolia, in 1
233 gnificantly increased, particularly in later gall stages.
234  families was significantly repressed in the galled stem.
235 um stems but strongly differed from 50:50 in galled stems, with "+" and "-" enantiomers strongly domi
236 nes in enantiomeric ratios characteristic of galled stems.
237 ervation, cholecystitis and complications of gall stones such as pancreatitis, and ovarian diseases.
238            Despite their lack of similarity, GALLS substituted for VirE2.
239     The patterns of gene expression found in galls suggest that phylloxera exploits vascular cambium
240                                          The galls support species-rich, closed communities of inquil
241 mediated knots, even by experts, because the gall symptoms in both conditions are very similar.
242                      Tannins, especially nut gall tannin, were effective in limiting both methionine
243 ion for discovering unifying features of the galls that plants make for friends and foes, talk about
244 n years ago) a larva of the Holometabola was galling the internal tissue of Psaronius tree-fern frond
245 f SAD6 with fatty acid desaturation in crown galls, the lipid pattern was analyzed of plants with con
246 ycolysis; and fermentation increased in leaf-gall tissues.
247 rns of these miRNAs and their targets in the galled tissues and in tissues surrounding the gall.
248 neering by removing or adding post-emergence galls to different plots of their host plant in the Braz
249 ny of these domains abolished the ability of GALLS to substitute for VirE2.
250                  We assess the importance of gall traits in structuring oak cynipid communities and s
251 rimary metabolism, we also characterized the gall transcriptome to infer the level of global reconfig
252 or the synthesis of mannopine (MOP) by crown gall tumor cells, MocC is essential for the utilization
253 dies on the temperature sensitivity of crown gall tumor development on plants.
254 ntrol the infection process leading to crown gall tumor disease on susceptible plants.
255 erium Agrobacterium tumefaciens causes crown gall tumor formation in plants.
256                       An early step in crown gall tumor formation involves the attachment of Agrobact
257      Isolate Rr 2-17, from a grapevine crown gall tumor, is a member of the Novosphingobium genus tha
258 segment of DNA to a host plant, generating a gall tumor.
259 a gene of the plasmid pSa can suppress crown gall tumorigenesis incited by Agrobacterium tumefaciens.
260                  During the process of crown gall tumorigenesis, Agrobacterium tumefaciens transfers
261 cient, but the ecotype is deficient in crown gall tumorigenesis, transformation to kanamycin resistan
262  DNA or T-DNA) into plant cells during crown gall tumorigenesis.
263 th pJW323 and pTiA6, the initiation of crown gall tumors (i.e., T-DNA transfer) is greatly suppressed
264 host pathogens, the causative agent of crown gall tumors Agrobacterium tumefaciens and the parasitic
265 olved in the biosynthesis of opines in crown gall tumors are always matched by Ti plasmid genes confe
266                       The formation of crown gall tumors by Agrobacterium tumefaciens requires that t
267      Agrobacterium tumefaciens induces crown gall tumors by transferring a piece of its tumor-inducin
268 ciens is a plant pathogen that incites crown gall tumors by transferring to and expressing a portion
269  by the conjugative opines produced by crown gall tumors induced on plants by the bacteria.
270 al, a subset of the opines produced by crown gall tumors initiated on plants by the pathogen, serves
271      Agrobacterium tumefaciens induces crown gall tumors on plants by transferring a nucleoprotein co
272       Agrobacterium tumefaciens causes crown gall tumors on various plants by delivering transferred
273 small carbon compounds produced by the crown gall tumors that are induced by the bacteria.
274     Arabidopsis (Arabidopsis thaliana) crown gall tumors, which develop upon infection with the virul
275  to octopine, a nutrient released from crown gall tumors.
276 f Agrobacterium tumefaciens that cause crown gall tumors.
277  responsible for opine biosynthesis in crown gall tumors.
278  plant genome, ultimately resulting in crown gall tumour formation.
279 ium tumefaciens is well known to cause crown gall tumours at plant wound sites and to benefit from th
280 n which opines, substrates produced by crown gall tumours, induce a quorum-sensing system.
281 s called opines that are released from crown gall tumours.
282 e levels of unsaturated fatty acids in crown galls under hypoxia and drought stress conditions.
283 thesis developed significantly smaller crown galls under normal, but not under high, relative humidit
284 ance and survivorship were not affected, and gall volume changed only slightly-but modified interacti
285   And the possible evolutionary trend of the gall was bigger chamber, more stable mechanical supporti
286                     The development of crown galls was not affected either in SAD6-OE or SAD6-OE-RNAi
287 s in response to Pst, and one of these, stem galls, was found to be simply inherited.
288                                Larvae of the gall wasp Antistrophus rufus Gillette (Hymenoptera: Cyni
289                                          Oak gall wasps (Hymenoptera: Cynipidae, Cynipini) are charac
290                                              Gall wasps pollinate fig trees.
291 thin stems in a complex prairie habitat, and gall wasps themselves apparently influence the plant to
292 ics of the interactions between host plants, gall wasps, and natural enemies.
293 ciated populations of two species of cynipid gall wasps, Belonocnema treatae and Disholcaspis quercus
294 adequate means do not exist to control crown gall, we created resistant plants by introducing transge
295                                         Leaf galling, which is negatively correlated with moisture to
296                                     In crown galls, which endogenously express AtLtpI-4, it is involv
297 ant development to form unique organs called galls, which provide these insects with unique, enhanced
298                IAA induced their activity in galls while PEO-IAA treatment and mutations in AuxRe mot
299 tors in my life, including my father and Joe Gall, who is my "Doktor Vater." In turn, as an establish
300 th to those of female eggs, yet emerged from galls with shorter pedicels than those of female wasps.

 
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