ライフサイエンスコーパス: gallate

1 related with epicatechin and epicatechin-3-O-gallate.

2 hin and epicatechin, but not epicatechin-3-O-gallate.

3 industry, BHT, alpha-tocopherol, and dodecyl gallate.

4 constant, followed by (-)-epigallocatechin-3-gallate.

5 redox-active cofactors such as ascorbate and gallate.

6 ls, epigallocatechin gallate and epicatechin gallate.

7 the antioxidants ascorbic acid and n-propyl gallate.

8 some EGCG epimerized to (+)-gallocatechin-3-gallate.

9 y the anti-amyloid compound epigallocatechin-gallate.

10 he abundance of EGC, theanine and afzelechin gallate.

11 eviously to be better surfactants than alkyl gallates.

12 In addition, about 130 genes in the large gallate 1-beta-glucosyltransferase (GGT) superfamily wer

13 Phenolic compounds, including propyl gallate, 1-o-galloylglycerol, ferulic, gallic, caffeic,

14 tion) than amylose (IC(50): epigallocatechin gallate = 20.41 +/- 0.25 uM, quercetagetin = 30.15 +/- 2

15 he green tea extract, (-)-epigallocatechin-3-gallate [(2R,3R)-5,7-dihydroxy-2-(3,4,5-trihydroxyphenyl

16 tea-derived flavanol, (-)-epigallocatechin 3-gallate [(2R,3R)-5,7-dihydroxy-2-(3,4,5-trihydroxyphenyl

17 pigallocatechin (2) and epigallocatechin-3-O-gallate (3).

18 nalysis using luteolin (12) and (-)-catechin gallate (37) as model compounds revealed that FabG was i

19 abundant being gallic acid, (-)-epicatechin-gallate, 4-p-coumaroylquinic acid, quercetin-3-O-galacto

20 uced higher decrease in (-)-epigallocatechin gallate (71% and 79%) and quercetin-3-glucoside (29% and

21 (1475-2,070 mug/g extracts) and epicatechin gallate (885-1,603 mug/g extracts); while all-trans-lute

22 6)-membrane interactions by epigallocatechin gallate, a known modulator of amyloid peptide aggregatio

23 We recently discovered that ethyl gallate, a nonflavonoid phenolic antioxidant found in fo

24 n tea extracts containing epigallocatechin 3-gallate, a potent DYRK1A inhibitor, to adult mBACtgDyrk1

25 Epigallocatechin-3-gallate, a potent Mnb/Dyrk1A inhibitor in vitro, apparen

26 recently showed that (-)-epigallocatechin-3-gallate, a tea polyphenol, not only inhibits L-DOPA meth

27 y analysis also showed that epigallocatechin gallate ameliorated vesicle disruption by PrP(106-126).

28 Catechins [(-)-epigallocatechin gallate and (-)-epicatechin gallate)] were most efficien

29 -)-epicatechin gallate, (-)-epigallocatechin gallate and (-)-epigallocatechin protected at lowest con

30 high binding energies for (-)-epicatechin-3-gallate and (-)-epigallocatechin-3-gallate for the bindi

31 ddition of the antioxidants epigallocatechin gallate and alpha-lipoic acid reduces polyglutamine aggr

32 lavan-3-ols, most of which are gallocatechin gallate and catechin.

33 the green tea polyphenols, epigallocatechin gallate and epicatechin gallate.

34 verages were incubated with epigallocatechin gallate and green tea extract at 62 or 37 degrees C for

35 Interestingly, whereas epigallocatechin gallate and heparin prevent membrane damage as judged by

36 a oligomers is inhibited by epigallocatechin gallate and increased by the A2V mutation, previously re

37 2,3-trihydroxybenzene moiety, such as methyl gallate and myricetin.

38 We found that in both protocols, ethyl gallate and norepinephrine improved mean arterial pressu

39 ain inhibition mechanism of epigallocatechin gallate and punicalagin was through complexation with st

40 pplementation of combined epigallocatechin-3-gallate and resveratrol (EGCG+RES) increased energy expe

41 by two amyloid inhibitors (epigallocatechin gallate and scyllo-inositol) that are currently in clini

42 he green tea polyphenol (-)-epigallocatechin gallate and the red wine extract resveratrol both remode

43 , (+)-catechin, gallocatechin, gallocatechin gallate and theasinensin B, and lower levels of (-)-epic

44 , (+)-catechin, gallocatechin, gallocatechin gallate and theasinensin B, and lower levels of (-)-epic

45 ely (p<0.05) correlated with (-)-epicatechin gallate and total phenolic contents.

46 n acyl-hydrolase hydrolyzing HTs, CT monomer gallates and depsides - has been reported in microbes on

47 nti-peroxide activity of gallic acid, methyl gallate, and alpha-tocopherol in a bulk Kilka fish oil a

48 ( approximately 100%), for Trolox, n-propyl gallate, and ascorbic acid the extent of geminate recomb

49 han, isonicotinylhydrazide, epigallocatechin gallate, and curcumin as reducing and stabilizing agents

50 ecially epigallocatechin galate, epicatechin gallate, and epicatechin) than tea infused at 80 degrees

51 n hazelnut shells were catechin, epicatechin gallate, and gallic acid, as quantified by high performa

52 atrol, Curcumin, Quercetin, Epigallocatechin Gallate, and Genistein) for further examination of their

53 including (+)-catechin, (-)-epigallocatechin gallate, and green tea extract were added to washed MFGs

54 (TFdiG), theaflavin-3'-gallate, theaflavin-3-gallate, and theaflavin inhibited PL with IC50 of 1.9, 4

55 ration containing 65% (-)-epigallocatechin-3-gallate] and caffeine on 4-(methylnitrosamino)-1-(3-pyri

56 nd of (-)-epicatechin and (-)-epicatechin-3O-gallate as extension units (positive correlation).

57 tannins [procyanidin B2, procyanidin B2 3'-O-gallate (B2g) and procyanidin trimer (catechin-4-8-catec

58 ent- and bitter-tasting (-)-epigallocatechin gallate, bitter-tasting caffeine, and the umami-tasting

59 ts reveal that polyphenols (epigallocatechin gallate, bromophenol blue, and resveratrol) and glycosam

60 ctivity of gallic acid and a series of alkyl gallates (C4-C18) and glycosylated alkyl gallates (C4-C1

61 kyl gallates (C4-C18) and glycosylated alkyl gallates (C4-C18) on fish oil-in-water emulsions was stu

62 Medium-size alkyl gallates (C6-C12) were the best antioxidants.

63 levels of catechin, epicatechin, epicatechin gallate, caffeine, theobromine and theophylline.

64 compounds, such as gallic acid, epicatechin gallate, catechin, epicatechin and isoquercitrin, were i

65 > ECG > EGCG >= GCG when compared to the non-gallated catechins (C, EC, GC, and EGC).

66 tor of glucose uptake into Caco-2 cells, and gallated catechins the most potent: CG > ECG > EGCG >= G

67 n (C), methyl gallate (MG), and catechin-3-O-gallate (CG).

68 more active (IC50 = 31 microM) than catechin gallate (CG, IC50 = 53 microM) or epicatechin gallate (E

69 of hydrolysable polyphenols (gallic acid 3-O-gallate, cinnamic acid, hesperidin, myricetin 3-O-rhamno

70 uclear perfluoroarylborate, -aluminate, and -gallate cocatalysts/activators B(C6F5)3 (3), B(o-C6F5C6F

71 n and tau aggregation, that epigallocatechin gallate decreases aSyn aggregation, and that dynasore re

72 The gallate degradation pathway ofPseudomonas putidaKT2440 r

73 othiocyanates; G) and (-)-epigallocatechin-3-gallate (E) were investigated in colon (LoVo and CaCo-2)

74 (EC), epigallocatechin (EGC), epicatechin 3-gallate (ECG) and epigallocatechin 3-gallate (EGCG) and

75 etics (in the order of minutes), epicatechin gallate (ECG) and epigallocatechin gallate (ECGC) exhibi

76 (-)-Epicatechin-3-gallate (ECG) functioned similar to EGCG by completely b

77 ocatechin-3-gallate (GCG), and epicatechin-3-gallate (ECG)) and not the catechol-type catechins (cate

78 gallocatechin gallate (EGCG) and epicatechin gallate (ECG)) inhibit GDH in vitro and that EGCG blocks

79 zodioxol-5-amine (MSNBA) and (-)-epicatechin-gallate (ECG).

80 allate (CG, IC50 = 53 microM) or epicatechin gallate (ECG, IC50 = 76 microM) against the colorectal a

81 n C, (-)-epicatechin EC, (-)-epicatechin-3-O-gallate, ECG] and oligomers [B1, B2, B3, B4 dimers and t

82 icatechin gallate (ECG) and epigallocatechin gallate (ECGC) exhibited very rapid binding (in the orde

83 epicatechin or its analogue epigallocatechin gallate (EGCG) (0.25% w/v in drinking water) was adminis

84 mg/daclatisvir (DCV) 60 mg/epigallocatechin gallate (EGCG) 400 mg without ribavirin (RBV); and Dacta

85 Epigallocatechin-3-gallate (EGCG) accounts for almost 50% of the total cate

86 in, (-)-epicatechin, or (-)-epigallocatechin gallate (EGCG) added as chain breakers.

87 on conferred by sugars to epigallocatechin-3-gallate (EGCG) against deterioration.

88 , ACS1, and the antioxidant epigallocatechin gallate (EGCG) all stimulated GSH levels and significant

89 llocatechin (EGC) and (-)-epigallocatechin-3-gallate (EGCG) and butylated hydroxytoluene (BHT) correl

90 more, stems contained lower epigallocatechin gallate (EGCG) and caffeine (ca. 75 and 56%, respectivel

91 echin 3-gallate (ECG) and epigallocatechin 3-gallate (EGCG) and caffeine in 29 commercial green tea s

92 ituents of green tea, (-)-epigallocatechin-3-gallate (EGCG) and caffeine, on intestinal tumorigenesis

93 its aggregation, such as epigallocatechin-3-gallate (EGCG) and dopamine (DA).

94 polyphenols from green tea (epigallocatechin gallate (EGCG) and epicatechin gallate (ECG)) inhibit GD

95 the four compounds tested, epigallocatechin gallate (EGCG) and epicatechin gallate were found to inh

96 tituents, in particular (-)-epigallocatechin gallate (EGCG) and other polyphenols with 1,2-benzenedio

97 e: propyl gallate (PG), (-)-epigallocatechin gallate (EGCG) and quercetin (Q).

98 the polyphenolic compounds epigallocatechin gallate (EGCG) and silibinin bind to specific conformers

99 tea catechins such as (-)epigallocatechin-3-gallate (EGCG) are known to improve energy metabolism at

100 -unexplored property of (-)-epigallocatechin gallate (EGCG) as a chiral solvating agent for enantiodi

101 Here, we identify (-)-epigallocatechin-3-gallate (EGCG) as a new inhibitor of hepatitis C virus (

102 The STAT1 inhibitor epigallocatechin gallate (EGCG) attenuated STAT1 phosphorylation in cispl

103 est green tea-derived (-)-epigallocatechin-3-gallate (EGCG) can attenuate neuronal damage mediated by

104 an ingredient of green tea, epigallocatechin gallate (EGCG) could attenuate oxidative stress-induced

105 Green tea polyphenol, epigallocatechin-3-gallate (EGCG) differentially regulates the cellular gro

106 The G3BP1 inhibitor epigallocatechin gallate (EGCG) disrupted existing G3BP1-cGAS complexes a

107 (-)-Epigallocatechin gallate (EGCG) effectively reduces the cytotoxicity of t

108 green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG) exerts a beneficial role on reducing brai

109 It was reported that epigallocatechin-3-gallate (EGCG) exhibits anti-inflammatory properties, bu

110 hat prostate tumor specific epigallocatechin-gallate (EGCg) functionalized radioactive gold nanoparti

111 ea-derived polyphenol (-)-epigallocatechin-3-gallate (EGCG) has been extensively studied for its anti

112 The flavonoid epigallocatechin gallate (EGCG) has previously been shown to redirect the

113 etinamide (4-HPR) and (-)-epigallocatechin-3-gallate (EGCG) in altering expression of oncogenic micro

114 The polyphenol epigallocatechin-3-gallate (EGCG) in combination with doxorubicin (Dox) exh

115 evaluate the efficacy of epigallocatechin-3-gallate (EGCG) in down-regulating Mcl-1 expression and i

116 tive botanicals including epigallocatechin-3-gallate (EGCG) in green tea polyphenols (GTPs) and sulfo

117 ated green tea polyphenol epigallocatechin-3-gallate (EGCG) in polylactic acid-polyethylene glycol na

118 n of green tea catechin and epigallocatechin gallate (EGCG) in soy lecithin liposomes was examined at

119 techins epicatechin (EC) or epigallocatechin gallate (EGCG) inhibited formation of highly reactive in

120 the green tea polyphenol epigallocatechin-3 gallate (EGCG) inhibits growth and transformed phenotype

121 tive green tea polyphenol epigallocatechin-3-gallate (EGCG) inhibits growth in soft agar of breast ca

122 the green tea polyphenol epigallocatechin 3-gallate (EGCG) inhibits growth of NF639 Her-2/neu-driven

123 Epigallocatechin-3-gallate (EGCG) is a candidate for treatment of Alzheimer

124 Epigallocatechin-3-gallate (EGCG) is a candidate therapeutic for Down syndr

125 Epigallocatechin gallate (EGCG) is a major polyphenol in green tea that h

126 that the commonly used (-)-epigallocatechin gallate (EGCG) is a much less efficient amyloid inhibito

127 Epigallocatechin gallate (EGCG) is a powerful antioxidant and commonly us

128 (-)-Epigallocatechin-3-gallate (EGCG) is a well-known chemoprevention factor.

129 ated that the GT polyphenol epigallocatechin gallate (EGCG) is capable of antagonizing AhR-mediated g

130 gents like the polyphenol epigallocatechin-3-gallate (EGCG) is emerging as an experimental secondary

131 The green tea catechin epigallocatechin-3-gallate (EGCG) is generally considered to be the biologi

132 Epigallocatechin gallate (EGCG) is often described as the most potently c

133 (-)-Epigallocatechin gallate (EGCG) is one of the major green tea catechins t

134 Epigallocatechin gallate (EGCG) is the major active polyphenol in green t

135 Epigallocatechin-3-gallate (EGCG) is the major polyphenol in green tea.

136 Epigallocatechin-3-gallate (EGCG) is the most biologically potent polypheno

137 The green tea component epigallocatechin-3-gallate (EGCG) may be beneficial in autoimmune diseases;

138 TE) supplementation high in epigallocatechin gallate (EGCG) on blood lipids in healthy postmenopausal

139 effect of the polyphenol epigallocatechin 3-gallate (EGCG) on hCT fibrillation was also investigated

140 the green tea polyphenol epigallocatechin-3 gallate (EGCG) on mammary tumor cells were assessed.

141 esveratrol, curcumin, and epigallocatechin-3-gallate (EGCG) on the aggregation of Abeta(17-36) peptid

142 yphenolic amyloid inhibitor epigallocatechin gallate (EGCG) on the aggregation pathway.

143 400muM) and influence (-)-epigallocatechin-3-gallate (EGCG) oxidation (400muM) in Tween- or sodium do

144 green tea shoot (GL) while epigallocatechin gallate (EGCG) recorded higher levels in GL.

145 In contrast, epigallocatechin-3-gallate (EGCG) signals ECs via the 67 kDa laminin-recept

146 th a standardized dose of epigallocatechin-3-gallate (EGCG) was administered using the standard phase

147 e its antioxidant activity, Epigallocatechin Gallate (EGCG) was esterified with stearic acid.

148 f the catechins, especially epigallocatechin gallate (EGCG) were found in green teas.

149 Tannic acid (TA) and epigallocatechin gallate (EGCG) were recently reported as promising TMEM1

150 mponent in green tea, (-)-epigallocatechin-3-gallate (EGCG)'s potential benefits to human health have

151 vestigated the effects of epigallocatechin-3-gallate (EGCG), a bioactive green tea polyphenol, on EGF

152 Epigallocatechin gallate (EGCG), a bioactive polyphenol in green tea, mim

153 (-)-Epigallocatechin-3-gallate (EGCG), a catechin found in green tea, has been

154 ine whether the antioxidant epigallocatechin gallate (EGCG), a catechin-base flavonoid derived from g

155 was to provide support for epigallocatechin gallate (EGCG), a component of green tea, to be consider

156 nd in vivo treatment with Epigallocatechin-3-gallate (EGCG), a Dyrk1a inhibitor, modulated trisomic N

157 uded three phenolics: (-)-epigallocatechin-3-gallate (EGCG), a flavanoid polyphenol found in green te

158 orted previously that (-)-epigallocatechin-3-gallate (EGCG), a green tea polyphenol antioxidant, stim

159 entified in this study that epigallocatechin gallate (EGCG), a green tea-derived catechin, acts as a

160 Epigallocatechin-3-gallate (EGCG), a main catechin of green tea, has been s

161 (-)-Epigallocatechin gallate (EGCG), a major biologically active constituent

162 (-)-Epigallocatechin-3-gallate (EGCG), a major component of green tea, protects

163 t studies have shown that epigallocatechin-3-gallate (EGCG), a major constituent of green tea extract

164 Epigallocatechin gallate (EGCG), a major form of tea catechins, possesses

165 (-)-Epigallocatechin-3-O-gallate (EGCG), a major ingredient of green tea, has bee

166 (-)-Epigallocatechin-3-gallate (EGCG), a major polyphenol component of green te

167 Epigallocatechin-3-gallate (EGCG), a polyphenol extracted from green tea, i

168 Epigallocatechin-3-gallate (EGCG), a polyphenol, influences cutaneous wound

169 are mainly attributed to epigallocatechin-3-gallate (EGCG), a polyphenolic compound from the group o

170 Treatment with epigallocatechin-3-gallate (EGCG), a polyphenolic compound of green tea, re

171 oneally injected with (-)-epigallocatechin-3-gallate (EGCG), a redox-active polyphenol from green tea

172 ermine to what extent (-)-epigallocatechin-3-gallate (EGCG), a widely used dietary supplement, modula

173 We found that epigallocatechin-3-gallate (EGCG), an anti-inflammatory compound found in g

174 s, and the polyphenol (-)-epigallocatechin-3-gallate (EGCG), and has been implicated in a number of d

175 f a model polyphenol, (-)-epigallocatechin-3-gallate (EGCG), and matrix pH (2-7) on the net anti-/pro

176 cysteine (NAC), curcumin, epigallocatechin-3 gallate (EGCG), and p38 inhibitor SB203580 were added to

177 ajor green tea catechin, epigallo-catechin 3-gallate (EGCG), and the major dietary protein and allerg

178 r green tea catechin, (-)-epigallocatechin-3-gallate (EGCG), but effects were observed only at microm

179 igate the interactions of epigallocatechin-3-gallate (EGCG), found in green tea, with Abeta polypepti

180 hibitor, green tea flavonol epigallocatechin-gallate (EGCG), from gestation to adulthood suppressed 3

181 id, caffeine, curcumin, (-)-epigallocatechin gallate (EGCG), gallic acid, propyl gallate, resveratrol

182 yrogallol-type catechins (epigallocatechin-3-gallate (EGCG), gallocatechin-3-gallate (GCG), and epica

183 nin, baicalein, curcumin, epigallocatechin 3-gallate (EGCG), genistein, quercetin, and resveratrol bo

184 tea extracts, catechin (-)-Epigallocatechin gallate (EGCg), has been reported to be biologically act

185 tuent of green tea, (-)-epigallocatechin-3-O-gallate (EGCG), has been shown to have cancer-preventive

186 molecules, FK506 and (-)-epigallocatechin-3-gallate (EGCG), known to inhibit alphaSYN fibril formati

187 d its major catechin, (-)-epigallocatechin-3-gallate (EGCG), on collagen homeostasis in keloid fibrob

188 MT) inhibitors RG108, (-) epigallocatechin-3-gallate (EGCG), or curcumin.

189 yphenolic constituent (-)-epigallocatechin-3-gallate (EGCG), possesses remarkable cancer chemoprevent

190 their active ingredient, epigallocatechin-3-gallate (EGCG), protects cells from subsequent OGD/R-ind

191 gallocatechin (EGC) and (-)-epigallocatechin gallate (EGCG), respectively.

192 gallocatechin (EGC) and (-)-epigallocatechin gallate (EGCG), respectively.

193 Epigallocatechin gallate (EGCg), the main antimicrobial tea catechin, has

194 It was hypothesized that epigallocatechin-gallate (EGCG), the main catechin present in green tea,

195 jection (20 mg/kg) of (-)-epigallocatechin-3-gallate (EGCG), the main polyphenolic constituent of gre

196 Here, we report that (-)-epigallocatechin-3-gallate (EGCG), the main polyphenolic constituent of gre

197 orted previously that (-)-epigallocatechin-3-gallate (EGCG), the major and most active component in g

198 Epigallocatechin 3-gallate (EGCG), the major polyphenol found in green tea,

199 Epigallocatechin gallate (EGCG), the major polyphenol in green tea and th

200 we provide evidence that epigallocatechin-3-gallate (EGCG), the major polyphenol in green tea, is a

201 opical application of (-)-epigallocatechin-3-gallate (EGCG), the major polyphenol of green tea, preve

202 studies indicate that (-)-epigallocatechin-3-gallate (EGCG), the major polyphenol present in green te

203 t for the first time that epigallocatechin-3-gallate (EGCG), the major polyphenolic constituent of gr

204 Epigallocatechin-3-gallate (EGCG), the major tea polyphenolic compound, is

205 (-)-Epigallocatechin-3-gallate (EGCG), the principal polyphenol in green tea, h

206 Epigallocatechin-3-gallate (EGCG), the principal polyphenol isolated from g

207 Epigallocatechin-3-gallate (EGCG), the principal polyphenol present in gree

208 ons with dopamine (DA) or epigallocatechin-3-gallate (EGCG), two inhibitors of AS aggregation, indica

209 tea flavan-3-ol substrates, epigallocatechin gallate (EGCG), was employed.

210 en tea extract containing epigallocatequin-3-gallate (EGCG), which improves executive function in you

211 polyphenols, curcumin and epigallocatechin-3-gallate (EGCG), with that by the endogenous substrate he

212 We successfully synthesized epigallocatechin gallate (EGCG)-loaded nanoparticles (Enano), which were

213 described polyphenol, (-)-epigallocatechin-3-gallate (EGCG).

214 isothiocyanate (BITC), and epigallocatechin gallate (EGCG).

215 ng site of SULT1A1 bound to epigallocatechin gallate (EGCG).

216 n, epicatechin, and (-)-epigallocatechin-3-O-gallate (EGCG)] and bioflavonoids (quercetin, fisetin, a

217 c acid-GA, catechin-CAT and epigallocatechin gallate-EGCG) from processed and digested porridges with

218 5-MTHF) in combination with epigallocatechin-gallate-enriched extract (EGCGe) and epigallocatechin-en

219 mpferol, quercetin, epicatechin, epicatechin gallate, epigallocatechin gallate, genistein, daidzein,

220 erythrocyte hemolysis while (-)-epicatechin gallate, (-)-epigallocatechin gallate and (-)-epigalloca

221 degrees C: catechins [(-)-epigallocatechin-3-gallate, (-)-epigallocatechin, (-)-epicatechin-3-gallate

222 Gallate ester derivatives of the novel C4- and C8-substi

223 Catechins and their gallate esters are a class of polyphenolic compounds.

224 ate, (-)-epigallocatechin, (-)-epicatechin-3-gallate], flavones (kaempferol, kaempferol-3-glucoside,

225 atechin-3-gallate and (-)-epigallocatechin-3-gallate for the binding site on BSA near Trp213.

226 locatechin-3-gallate (EGCG), gallocatechin-3-gallate (GCG), and epicatechin-3-gallate (ECG)) and not

227 echin, epicatechin gallate, epigallocatechin gallate, genistein, daidzein, caffeic acid, gallic acid)

228 ated with 20 micromol/L (-)-epigallocatechin gallate (green tea) was restored back to wild-type level

229 and polyphenols (vanillin, epigallocatechin gallate, green tea extract, and protocatechualdehyde) at

230 in the order punicalagin > epigallocatechin gallate > quercetagetin, with minimal interference using

231 oil system decreased in the order of methyl gallate>gallic acid>alpha-tocopherol.

232 The addition of Trolox, BHA, and propyl gallate had no significant effect on furan formation fro

233 Similarly, (-)-epicatechin-3-gallate had the highest effect on the Circular Dichroic

234 , where an alternative structure for Fe(III) gallate has been commonly cited.

235 Recently, (-)-epigallocatechin gallate has been found to be a potent inhibitor of the p

236 Epigallocatechin gallate has been shown to be an inhibitor of the proteal

237 enols, such as catechin and epigallocatechin gallate, have been used to augment the action of traditi

238 al agent (IC50=29.5 muM), followed by methyl gallate (IC50=38.0 muM, log P=-0.23) and alpha-tocophero

239 sing the DYRK1A inhibitor epigallocatechin-3-gallate improved Ts65Dn skeletal phenotypes.

240 age capacity in vivo Proanthocyanidin trimer gallate in particular modified lipid desaturation in C.

241 as smaller than that of (-)-epigallocatechin gallate in Phase III clinical trials and about one order

242 The antioxidant efficiency of GA and gallates in the emulsified systems used, correlated posi

243 in C2, epigallocatechin and epigallocatechin gallate) in a nutritional context to prevent Celiac Dise

244 dant capacity than their corresponding alkyl gallates, in emulsions prepared with lecithin or Tween-2

245 green tea polyphenol, (-)-epigellocathecin-3-gallate, is also inhibited by apigenin, suggesting that

246 -galloyl catechin (GC), catechin (C), methyl gallate (MG), and catechin-3-O-gallate (CG).

247 enomena standpoint, gallic acid (GA), methyl gallate (MG), and their combination alone and together w

248 in the presence of gallic acid (GA), methyl gallate (MG), MG/GA combinations (75:25, 50:50, and 25:7

249 Proanthocyanidin trimer gallate modulates lipid deposition and fatty acid desatu

250 eptide, a tetraethylene glycol spacer, and a gallate moiety was designed, synthesized, and characteri

251 ds: rifampicin, curcumin, epigallocatechin-3-gallate, myricetin, and scyllo-inositol, in cells expres

252 aptoethanol (beta-ME), Trolox (TX), n-propyl gallate (n-PG), and ascorbic acid (AA).

253 para-nitrobenzoic acid (pNBA), and n-propyl gallate (nPG)--on bilayer properties using a gramicidin

254 ative, oligomerized (-)-epigallocatechin-3-O-gallate (OEGCG) as a carrier for oral lycopene delivery.

255 ors NADH and NADPH and is sensitive to octyl gallate (Ogal), a plastidial terminal oxidase inhibitor.

256 e green tea catechin, (-)-epigallocatechin-3-gallate or (-)-EGCG, has been shown to act as a proteaso

257 te treatment protocol, we administered ethyl gallate or norepinephrine after a sustained approximatel

258 the infusion and randomly administered ethyl gallate or norepinephrine in respective groups.

259 Contrary, epigallocatechin gallate, pelargonidin and catechin, with less impressive

260 the enzymatic transesterification of propyl gallate (PG) and tripalmitin under solvent-free conditio

261 Literature mentions propyl gallate (PG) as a non-toxic synthetic antioxidant that c

262 ity of gallic acid (GA) and its ester propyl gallate (PG) in the presence of UV-A light against Esche

263 ized by the enzymatic glycerolysis of propyl gallate (PG) using a food-grade lipase (Lipozyme(R) 435)

264 The most active antioxidants were: propyl gallate (PG), (-)-epigallocatechin gallate (EGCG) and qu

265 pure OPC dimer, trimer, tetramer, and their gallates (pOPCs).

266 the phenolic compounds catechin, epicatechin gallate, procyanidin B1, rutin, gallic acid, caffeic aci

267 Epigallocatechin gallate, quercetagetin and punicalagin were weaker inhib

268 1 and B2, catechin, epicatechin, epicatechin gallate, quercetin 3-beta-d-glucoside, delfinidin 3-gluc

269 Coumaroylquinic acid, epicatechin gallate, quercetin, and six other phenolics were identif

270 ularly in the late treatment protocol, ethyl gallate resulted in a lower heart rate, a lower troponin

271 Exposure to trimer gallate resulted in the transcriptional down-regulation

272 2 by the green tea compound epigallocatechin gallate results in an increase in [Ca(2+)](ER) due to in

273 catechin gallate (EGCG), gallic acid, propyl gallate, resveratrol, and alpha-tocopherol) were investi

274 Among these polyphenols, (-)-epicatechin-3-gallate showed the highest Stern-Volmer modified quenchi

275 llagic acid, limonin, oleanolic acid, propyl gallate, sinapic acid and ursolic acid demonstrated sign

276 In the emulsion system, methyl gallate still behaved better than gallic acid, but the h

277 3 pOPCs (dimer gallate, trimer and/or trimer gallate) suggested the absence of synergistic potential.

278 polyphenols, tannic acid (TA), theaflavin-3'-gallate (TF2B) and theaflavin-3,3'-digallate (TF3) exhib

279 st that phenolic antioxidants, such as ethyl gallate, that inhibit hydrogen peroxide signaling, may r

280 utrient screen, EGCG ((-)-epigallocatechin 3-gallate), the major phytochemical in green tea, emerged

281 The fate of (-)-epicatechin-3-O-gallate, the main flavan-3-ol in green tea, is unclear b

282 flavin-3,3'-digallate (TFdiG), theaflavin-3'-gallate, theaflavin-3-gallate, and theaflavin inhibited

283 he antiaggregation compound epigallocatechin gallate, thereby maintaining the PrPSc as primarily nono

284 toxicity; moreover, despite epigallocatechin gallate treatment, pooled oM1000 remained oligomeric and

285 combination exposure of 2 or 3 pOPCs (dimer gallate, trimer and/or trimer gallate) suggested the abs

286 Four alkyl gallate triphenylphosphonium lipophilic cations were syn

287 nt-based compounds (hippuric acid and propyl gallate) using cell- and membrane-based transport inhibi

288 hemodynamic and biochemical effects of ethyl gallate vs. those of the commonly used vasopressor, nore

289 stem, the natural product epigallocatechin 3-gallate was found to block Wnt signaling, independent of

290 ssays (MTT) we found that C8-propyl-catechin gallate was more active (IC50 = 31 microM) than catechin

291 Pinoresinol, ethyl caffeate and ethyl gallate were detected for the first time in these oils.

292 gallocatechin gallate (EGCG) and epicatechin gallate were found to inhibit GDH with nanomolar ED(50)

293 he thermosensitive compound epigallocatechin gallate were recovered without significant losses.

294 Glucosyl alkyl gallates were shown previously to be better surfactants

295 efficacy of CPC separation, three glucoside gallates were subsequently isolated by HPLC chromatograp

296 epigallocatechin gallate and (-)-epicatechin gallate)] were most efficient in the inhibition of AAPH-

297 acid, vanillic acid, and epigallocatechin-3-gallate, were effective inhibitors of the activity of ca

298 ehavior of the antioxidant activity of alkyl gallates when increasing alkyl chain length was observed

299 nt of glioma cells with (-)-epigallocatechin gallate, which targets the ATP-binding domain of GRP78 a

300 ll ink (IGI) is an amorphous form of Fe(III) gallate.xH2O (x = approximately 1.5-3.2).