ライフサイエンスコーパス: gallbladder

1 overall, HR = 0.98, 95% C.I. = 0.62-1.55 for gallbladder).

2 he fundus of the significantly ptotic, large gallbladder.

3 literature indicating tumor location in the gallbladder.

4 ation and defective contraction of the fetal gallbladder.

5 ues, including livers > mesonephros > guts > gallbladder.

6 mation, colonization, and persistence in the gallbladder.

7 s to persist as asymptomatic biofilms in the gallbladder.

8 is followed by carcinoma of the pancreas and gallbladder.

9 the intra- or extrahepatic bile ducts or the gallbladder.

10 n and (250.6-278.5) x10(-5) mm(2)/sec in the gallbladder.

11 f the extrahepatic biliary tract, except the gallbladder.

12 extrusion, for Salmonella maintenance in the gallbladder.

13 estinal peptide-a myorelaxant factor for the gallbladder.

14 ecapitulate a transport function of, primary gallbladder.

15 in the canalicular network, bile ducts, and gallbladder.

16 neoplastic lesion or LOH was found in normal gallbladders.

17 ectopic cystic duct-like epithelia in their gallbladders.

18 Estimated radiation doses were highest for gallbladder (0.27 mSv/MBq), upper large intestine (0.19

19 atio [HR] 1.62, 99% CI 1.56-1.69; p<0.0001), gallbladder (1.31, 1.12-1.52; p<0.0001), kidney (1.25, 1

20 -02mSv/MBq), kidneys (5.26E-02 mSv/MBq), and gallbladder (4.05E-02 mSv/MBq).

21 tic distributions included lungs (100%), the gallbladder (49%) if present, and locations involving he

22 pertension (18.2%) and empyema or perforated gallbladder (6.1%).

23 of BAs in the plasma (45.9%), liver (60.2%), gallbladder (76.3%), small intestine (88.7%), and colon

24 to 2014 was queried for patients with pT2/T3 gallbladder adenocarcinoma who underwent resection.

25 ency laparotomy, which revealed a gangrenous gallbladder adjacent to the duodenum and surrounded by p

26 uding biliary atresia, choledochal cysts and gallbladder agenesis.

27 The Sox17(+/-) gallbladder also showed a drastic reduction in sonic hed

28 re were 94 abdominal or pelvic surgeries (36 gallbladder): among 2,762 comparators there were 134 abd

29 mission when bile flow is diverted through a gallbladder anastomosis to jejunum, ileum or duodenum (s

30 min, followed by an anterior image over the gallbladder and abdomen.

31 l malrotation, midline liver with left-sided gallbladder and an interrupted inferior vena cava with a

32 There was no difference between gallbladder and bile duct tumors (P = .68).

33 Only tumors of the gallbladder and bile ducts were assessed.

34 oma (HCC), intrahepatic bile duct (IBD), and gallbladder and biliary tract cancers outside of the liv

35 sophageal; stomach; colon and rectum; liver; gallbladder and biliary; pancreatic; larynx; tracheal, b

36 as to isolate stem cells from an adult mouse gallbladder and determine whether they were unique, comp

37 of sav1 (-/-) rescued adults reveals loss of gallbladder and formation of dysplastic cell masses expr

38 licability to other tissues, including human gallbladder and heart.

39 Mean IOU for identification of the liver, gallbladder and hepatocystic triangle were: 0.86 (+/-0.1

40 We compared primary gallbladder and IHBD cells by flow cytometry and found p

41 ound to be abundantly expressed in the human gallbladder and in the common bile duct, with only minor

42 ntestine and during chronic infection of the gallbladder and inform the basis for development of ther

43 bladder (P = 0.009), and a lower AUC for the gallbladder and intestines (P = 0.001).

44 increased AUC for liver, and a lower AUC for gallbladder and intestines (P = 0.042, 0.034, and 0.001,

45 time-activity curves of the liver and of the gallbladder and intestines were obtained and correlated

46 obtained and no activity was detected in the gallbladder and intestines.

47 we observed labeled epithelial cells in the gallbladder and lower intestines, as well as Microfold c

48 nd intestines, and to a lesser extent in the gallbladder and pancreatic duct.

49 expressed in mucosal epithelial cells of the gallbladder and small intestine.

50 , along the lower edge of the liver near the gallbladder and the right kidney, the tip touching the d

51 cally important for E. coli to thrive in the gallbladder and upper intestinal tract, where high bile

52 my is an important tool for use in difficult gallbladders and achieves morbidity rates comparable to

53 d to cholic acid or bile, and in vivo in the gallbladders and livers of infected mice.

54 utaneously administered (54)Mn in the liver, gallbladder, and gastrointestinal tract of the KO mice,

55 Rates of prespecified muscle, gallbladder, and hepatic adverse effects and cancer were

56 angerous (No-Go) zones of dissection, liver, gallbladder, and hepatocystic triangle during LC.

57 m) and digestive accessory organs (pancreas, gallbladder, and liver).

58 are synthesized in the liver, stored in the gallbladder, and secreted into the duodenum at meals.

59 s of whole-body PET data in 12 patients, the gallbladder appeared to be the dose-limiting organ, with

60 The primary neuroendocrine tumors of the gallbladder are very rare, representing 0.2% of all neur

61 The gallbladder as content in the case of abdominal hernias

62 lous communication between cystic artery and gallbladder as the cause, a simultaneous cholecystoduode

63 ive dose of less than 0.30 mSv/MBq, with the gallbladder as the critical organ; the human target dose

64 (n = 7), periportal lymph nodes (n = 1), and gallbladder bed (n = 1).

65 We have performed a detailed analysis of gallbladder bile acid and lipid metabolism in Tgr5(-/-)

66 Analysis of gallbladder bile acid composition showed marked increase

67 Serum biochemistries, gallbladder bile acids, and liver sections were examined

68 thereby inducing cholesterol-supersaturated gallbladder bile and accelerating cholesterol crystalliz

69 Cftr-/- mice had abnormal gallbladder bile and duodenal acidity, and overexpressed

70 Interestingly, human gallbladder bile contains high levels of FGF19 (21.9 +/-

71 el surgical procedure in mice, termed distal gallbladder bile diversion to the ileum (GB-IL(dist)), t

72 A less aggressive surgery in which the gallbladder bile is diverted to the proximal ileum, term

73 Conclusion Bowel abnormalities and gallbladder bile stasis were common findings on abdomina

74 Gallbladder bile sterol levels were analyzed in choleste

75 NA in the wall of small intestine, appendix, gallbladder, bile, liver, and urine.

76 esophagus, colorectal, liver, pancreas, and gallbladder/bile duct cancers) in 69,310 nonsmoking and

77 Bacteria in the gallbladder can then seed the intestines and feces, lead

78 Gallbladder cancer (GBC) is an aggressive gastrointestin

79 ausal association of gallbladder stones with gallbladder cancer (GBC) is not yet well established.

80 Gallbladder cancer (GBC) is rare, but lethal.

81 that KPNA2 was significantly upregulated in gallbladder cancer (GBC), and the increased levels were

82 estigated the frequency of DNA repair GAs in gallbladder cancer (GBC), association with tumor mutatio

83 metastases on outcome after R0 resection of gallbladder cancer (GBCA).

84 ture (HR 2.082, P=0.008), obstruction due to gallbladder cancer (HR 31.029, P=0.012) and only partial

85 R, 21.00), pancreatic cancer (HR, 5.26), and gallbladder cancer (HR, 9.19) (P < .001 for all).

86 Preoperative predictors of incidental gallbladder cancer (iGBC) have been poorly defined despi

87 ciated with substantial variation in risk of gallbladder cancer (sibling relative risk 3.15 [95% CI 1

88 incidence, 373 per 10 000 person-years) and gallbladder cancer (SMR, 3.82 [95% CI, 3.31-4.39]; morta

89 ents with extrahepatic cholangiocarcinoma or gallbladder cancer and a microscopically positive surgic

90 ese imaging studies also serve to screen for gallbladder cancer and HCC.

91 IV stricture, biliary obstruction caused by gallbladder cancer and when only partial liver drainage

92 for re-resection for incidentally discovered gallbladder cancer appears to be between 4 and 8 weeks a

93 The discovery cohort comprised 1042 gallbladder cancer cases and 1709 controls and the repli

94 ols and the replication cohort contained 428 gallbladder cancer cases and 420 controls.

95 n study (GWAS), we did a genome-wide scan of gallbladder cancer cases and hospital visitor controls,

96 125b-5p up-regulation promoted cell death in gallbladder cancer cells in the presence of cisplatin.

97 ockdown of miR-125b-5p reduced cell death in gallbladder cancer cells treated with cisplatin.

98 Although the OR for gallbladder cancer could not be calculated, the OR for t

99 years with microscopically confirmed primary gallbladder cancer diagnosed or treated at Tata Memorial

100 Gallbladder cancer is highly lethal, with notable differ

101 tion as a new biomarker for the prognosis of gallbladder cancer patients.

102 Gallbladder cancer represents the most common malignancy

103 eport of common genetic variation conferring gallbladder cancer risk at genome-wide significance.

104 l samples, miR-125b-5p was down-regulated in gallbladder cancer whereas Bcl2 was up-regulated and the

105 firmed cholangiocarcinoma or muscle-invasive gallbladder cancer who had undergone a macroscopically c

106 of 207 patients with incidentally discovered gallbladder cancer who underwent reoperation and had ava

107 Of 449 patients with gallbladder cancer, 207 cases (46%) were discovered inci

108 lence of seropositivity was higher: 100% for gallbladder cancer, 97% of extrahepatic bile duct cancer

109 Although gallstones are recognized causes of gallbladder cancer, associations with other cancers of t

110 increased risk of cholangiocarcinoma (CCA), gallbladder cancer, hepatocellular carcinoma (HCC), and

111 of Bcl2 is correlated with poor prognosis in gallbladder cancer.

112 chemoradiotherapy for patients with resected gallbladder cancer.

113 ents with extrahepatic cholangiocarcinoma or gallbladder cancer.

114 es to identify miR-125b-5p down-regulated in gallbladder cancer.

115 select patients with incidentally discovered gallbladder cancer.

116 fy common genetic susceptibility alleles for gallbladder cancer.

117 olipid transporter genes in the pathology of gallbladder cancer.

118 hich mediates the function of miR-125b-5p in gallbladder cancer.

119 ant epithelioid hemangioendothelioma: n = 1, gallbladder cancer: n = 1 or metastatic disease [colorec

120 d extended/radical cholecystectomy for T2/T3 gallbladder cancer; however, many tumors are discovered

121 nd low pass whole genomes (n = 146) from 167 gallbladder cancers (GBCs) from patients in Korea, India

122 perihilar and distal cholangiocarcinomas and gallbladder cancers in Japanese patients.

123 considered in the differential diagnosis of gallbladder cancers.

124 Bile was collected by gallbladder cannulation, and liver samples were analyzed

125 Gallbladder carcinoma (GBC) is frequent in the Indian su

126 Individuals with gallbladder carcinoma (GBC), the most aggressive maligna

127 in extrahepatic cholangiocarcinoma (EHCC) or gallbladder carcinoma (GBCA) is unknown.

128 tract cancers (i.e., cholangiocarcinoma and gallbladder carcinoma) are associated with significant m

129 ignancies, including incidental diagnosis of gallbladder carcinoma, are on the rise.

130 Polypoidal thickening was seen in gallbladder carcinoma.

131 Lymphadenopathy was seen in 1 case with gallbladder carcinoma.

132 cholelithiasis may indicate XGC rather than gallbladder carcinoma.

133 extrahepatic cholangiocarcinomas as well as gallbladder carcinomas, are a genetically diverse collec

134 frequently altered gene in a series of nine gallbladder carcinomas.

135 evels of importance in biofilm formation and gallbladder colonization and that the ECM diminishes dis

136 ith cholecystolithiasis and patients without gallbladder concretions.

137 ded duodenum filled with fluid and a dilated gallbladder containing dilute bile with high bicarbonate

138 Ultrasound demonstrated the gallbladder containing multiple stones, with wall thicke

139 t-prandial variations in gallbladder volume; gallbladder contraction in response to cholecystokinin-8

140 ibution of embryonic cholecystitis and fetal gallbladder contraction in the early pathogenesis of con

141 of the unique short duration and reversible gallbladder contraction observed in vivo upon administra

142 The defective gallbladder contraction positively correlated with the s

143 Gallbladder cystic mucosal hyperplasia was commonly foun

144 The concept that motor disorders of the gallbladder, cystic duct and sphincter of Oddi can cause

145 latation as long as they involve presence of gallbladder deposits and tumors.

146 ancer death (5 more per 10 000 woman-years), gallbladder disease (20 more per 10 000 woman-years), de

147 dementia (22 more cases [95% CI, 4 to 53]), gallbladder disease (21 more cases [95% CI, 10 to 34]),

148 antly increased, per 10000 person-years, for gallbladder disease (30 more cases [95% CI, 16 to 48]),

149 thrombosis (7 more per 10 000 woman-years), gallbladder disease (33 more per 10 000 woman-years), an

150 Gallbladder disease (GBD) is a highly prevalent conditio

151 ancers in the cholecystectomy cohort and the gallbladder disease cohort compared with a control cohor

152 duced and hazard ratios for pairs defined by gallbladder disease followed by death were increased, th

153 eta-analyses (human height, QTc interval and gallbladder disease); all previous reported association

154 ased risk for stroke, thromboembolic events, gallbladder disease, and urinary incontinence.

155 olism, dementia (in women aged >/=65 years), gallbladder disease, and urinary incontinence; benefits

156 ding acute cholecystitis, chronic acalculous gallbladder disease, high-grade and partial biliary obst

157 ed to patients undergoing surgery for benign gallbladder disease.

158 Mongolia has an extremely high incidence of gallbladder disease.

159 are, on occasion, initially misdiagnosed as gallbladder disease.

160 cholecystectomy in patients with Functional Gallbladder Disorder (FGBD) and the relevance of sphinct

161 soft tissue with diffuse infiltration of the gallbladder, displacement of the transverse colon, hepat

162 iary atresia, as it loses its bile ducts and gallbladder during metamorphosis.

163 patients, which is mechanistically linked to gallbladder dysmotility in critical illness.

164 Gallbladder dysmotility was associated with the impaired

165 The gallbladder ejection fraction correlated positively with

166 metabolic hormone induced by bile salts upon gallbladder emptying after enteral nutrient stimulation.

167 active intestinal peptide and had defects in gallbladder emptying and in levels of secondary BAs, but

168 Gallbladder emptying and the FGF19 response were respect

169 han healthy subjects and nutrient-stimulated gallbladder emptying is impaired with "gallbladder pares

170 tr-/- and CftrDeltaF508 mice have defects in gallbladder emptying that disrupt enterohepatic circulat

171 In 10 of 24 ICU patients, gallbladder emptying was disturbed.

172 Gallbladder emptying was similar in the 3 groups.

173 30 minutes until 1 hour after infusion, and gallbladder emptying was studied by ultrasound.

174 ion, hormone concentrations, and gastric and gallbladder emptying were measured several times over 4

175 G-1 also impairs gallbladder emptying, leading to sluggish gallbladder mo

176 underwent a laparoscopic cholecystectomy for gallbladder empyema and died from severe respiratory fai

177 n of a laparoscopic cholecystectomy due to a gallbladder empyema.

178 ased risk of cancers of the liver, pancreas, gallbladder, endometrium, stomach, kidney, brain (benign

179 ditional deletion of Sox17 in the primordial gallbladder epithelia but not in fetal liver hepatoblast

180 lls were evident on the surface of the mouse gallbladder epithelia up to 21 days postinfection.

181 a was able to adhere to and invade polarized gallbladder epithelial cells apically in the absence and

182 here to and form extensive bacterial foci on gallbladder epithelial cells as early as 12 h postinocul

183 othesize that in addition to gallstones, the gallbladder epithelium aids in the establishment/mainten

184 d carriage showed invasion and damage of the gallbladder epithelium and lamina propria up to 2 months

185 work, we present evidence of the role of the gallbladder epithelium in chronic carriage by a mechanis

186 tence and/or bacterial aggregation in/on the gallbladder epithelium with luminal cell extrusion, for

187 The gallbladder excretes cytotoxic bile acids into the duode

188 Gallbladder explants secrete 500 times more FGF19 than F

189 The anatomopathologic examination of her gallbladder following cholecystectomy showed signs of ch

190 leum G1 lesions, 1 pancreas G2 lesion, and 1 gallbladder G3 lesion.

191 Gallbladder had the highest radiation exposure (21 muSv/

192 Fistula between the cystic artery and gallbladder has been commonly reported to occur after la

193 e were 134 abdominal or pelvic surgeries (37 gallbladder) (hazard ratio HR] = 0.70, 95% confidence in

194 mall intestine in 16 (36%), and the liver or gallbladder in 13 (30%).

195 e concluded that the finding of incarcerated gallbladder in the content of an inguinal hernia is a ra

196 eter less than 6 mm and those with an intact gallbladder in whom the cystic duct would be overlapped

197 pplication of bradykinin every 10-min on the gallbladder induced consistent pressor reflex responses.

198 reoperative severity grade and the extent of gallbladder inflammation on histopathology.

199 .02) and the presence of a gallstone in the gallbladder infundibulum (78% and 22% for conversion and

200 itis) and the presence of a gallstone in the gallbladder infundibulum are associated with conversion

201 e and bacterial survival in vivo and reduced gallbladder interleukin-10 (IL-10) levels.

202 nsport of ingested fluorescent lipids to the gallbladder is blocked.

203 Our work shows that the gallbladder is crucial for spread of P. aeruginosa from

204 ) epithelial cell subpopulation from primary gallbladder is enriched in colony-forming cells, compare

205 a or intervention, between cystic artery and gallbladder is rare with very few reports in literature.

206 On the basis of whole-body PET data, the gallbladder is the dose-limiting organ, with an average

207 fication of resident stem cells in the mouse gallbladder is, to date, unexplored.

208 ultiple myeloma, colorectal, uterine corpus, gallbladder, kidney, and pancreatic cancer) in young adu

209 causality, 41% of uterine and 10% or more of gallbladder, kidney, liver, and colon cancers could be a

210 the bladder, cervix, endometrium, esophagus, gallbladder, kidney, liver, and lung.

211 mach) and 11 site-specific cancers in women (gallbladder, kidney, liver, lung, lymphatic system, ovar

212 pecific cancers in men (bladder, colorectum, gallbladder, kidney, liver, lung, lymphatic system, panc

213 he bladder, breast, colon/rectum, esophagus, gallbladder, kidney, liver, lung, skin (melanoma-only),

214 ients were scheduled for cholecystectomy for gallbladder lithiasis.

215 d chromatography-mass spectrometry of serum, gallbladder, liver, small intestine, and colon of wild-t

216 on of Diseases, Ninth Revision, diagnosis of gallbladder malignancy who underwent a laparoscopic chol

217 Stomach Meridian of Foot-Yangming (SMFY) or Gallbladder Meridian of Foot-Shaoyang (GMFS) in healthy

218 rs gallbladder emptying, leading to sluggish gallbladder motility and promoting the development of bi

219 astrectomy on hunger, satiation, gastric and gallbladder motility, and gastrointestinal hormone respo

220 opathological examination revealed a primary gallbladder neuroendocrine tumor.

221 le cannot be safely identified in "difficult gallbladders." OBJECTIVE: To conduct a systematic review

222 Emergence in the gallbladder occurred later (P = 0.009, highest dose), an

223 serum whereas bile was decreased within the gallbladder of Cic-L(-/-) mice.

224 iated with the presence of gallstones in the gallbladder of infected carriers upon which Salmonella c

225 The gallbladders of Cftr-/- mice were enlarged and had defec

226 rs, was lower in the ileum but higher in the gallbladders of Cftr-/- mice, compared with wild-type mi

227 ate colonization and persistent infection in gallbladders of humans and mouse models of chronic carri

228 he surfaces of cholesterol gallstones in the gallbladders of mice and human carriers.

229 ellent and no activity was identified in the gallbladder or intestine.

230 t transformation in predisposed mice, murine gallbladder organoids, and fibroblasts, with TP53 mutati

231 emergence time of (99m)Tc-mebrofenin in the gallbladder (P = 0.009), and a lower AUC for the gallbla

232 ions of the upper abdomen, ADC values of the gallbladder, pancreas, spleen, and kidney may be compara

233 bdominal regions (left and right liver lobe, gallbladder, pancreas, spleen, and renal cortex and medu

234 lated gallbladder emptying is impaired with "gallbladder paresis" occurring in approximately 20%.

235 noperated group, which was mostly due to non-gallbladder pathologies but total hospital admission tim

236 ution in applying this technique to inflamed gallbladder pathology.

237 n be used as a decision aid to predict which gallbladder patients may benefit from adjuvant CRT.

238 A diagnosis of gallbladder perforation was performed.

239 valence and natural long-term progression of gallbladder polyps in a random sample of the general pop

240 In long-term follow-up, the prevalence of gallbladder polyps increased, with new lesions developin

241 The prevalence of gallbladder polyps was 6.1% (115/1880) in the 2002 study

242 irteen subjects (48.1%, 13/27) no longer had gallbladder polyps, and 14 subjects (51.9%, 14/27) still

243 rvival in vivo without altering the examined gallbladder pro- or anti-inflammatory cytokines.

244 ximately 17 muSv/MBq (62 mrem/mCi), with the gallbladder receiving the highest dose of 190 muSv/MBq.

245 operated group (12.2% vs. 2.0%, P < 0.001), gallbladder-related deaths were significantly lower than

246 nses to RYGB, and patients who underwent the gallbladder removal also lacked some significant improve

247 s related to participation in the programme (gallbladder removal and ruptured Achilles tendon).

248 en cholecystectomy as the primary method for gallbladder removal countrywide in 2011.

249 usness due to drugs for pre-existing angina, gallbladder removal, hospital admission with suspected h

250 holesterol medication use and higher odds of gallbladder removal.

251 often used to diagnose dysfunction (dynamic gallbladder scintigraphy and sphincter manometry) is con

252 Histopathologic examination of the gallbladder showed a spindle cell proliferation with dif

253 Of the 350 gallbladder specimens from gallstone patients, hyperplas

254 Histological examination of the gallbladder specimens was done for preneoplastic lesions

255 ans receiving highest absorbed dose were the gallbladder, spleen, stomach, liver, kidneys, and bladde

256 In addition, the relationship between adult gallbladder stem cells and intrahepatic bile duct (IHBD)

257 Their abdomen CT showed migration of gallbladder stones in the pelvis while paracentesis docu

258 Causal association of gallbladder stones with gallbladder cancer (GBC) is not

259 (20 of 37) revealed a dilated sludge-filled gallbladder, suggestive of bile stasis.

260 rograde cholangiopancreatography (ERCP), and gallbladder surgery during the index admission for patie

261 e the RR was 0.99 (95% C.I. = 0.56-1.77) for gallbladder surgery.

262 or pelvic surgery nor the specific subset of gallbladder surgery.

263 lly ill patients have larger, thicker-walled gallbladders than healthy subjects and nutrient-stimulat

264 In the stomach, kidneys, and gallbladder, the radiotracer showed a rapid initial upta

265 PEBDs), 11 ileal exclusions (IEs), and seven gallbladder-to-colon (GBC) diversions.

266 destruction, focal biliary cirrhosis, micro-gallbladder, vas deferens loss, airway disease, and meco

267 y ill patients (21%), there was no change in gallbladder volume in response to nutrient, and overall

268 at (LE3), different dynamics of postprandial gallbladder volume were induced (P </= 0.001).

269 potent GLP-1 secretagogue, has low effect on gallbladder volume, and improves glucose homeostasis in

270 ects of lipid emulsions on gastric emptying, gallbladder volume, and triglyceride absorption are depe

271 scintigraphy or post-prandial variations in gallbladder volume; gallbladder contraction in response

272 In the critically ill, fasting gallbladder volumes (critically ill, 61 mL [36-100 mL] v

273 Gallbladder volumes were measured with the use of real-t

274 ferences in GLP-1 and PYY concentrations and gallbladder volumes were observed after the capsaicin in

275 ed activities (in h) were the liver (0.117), gallbladder wall (0.046), and small intestine (0.033), a

276 , averaged over both men and women, were the gallbladder wall (0.59 +/- 0.44), small intestine (0.12

277 highest dose exposure (in muGy/MBq) were the gallbladder wall (111 +/- 60) > liver (21 +/- 7), kidney

278 The Hounsfield unit (HU) value of the gallbladder wall and intraluminal bile was measured.

279 ing bioengineered tissue can reconstruct the gallbladder wall and repair the biliary epithelium follo

280 The absence of gallbladder wall enhancement (58% and 40% for conversion

281 The absence of gallbladder wall enhancement (associated with the presen

282 On preoperative CT images, the absence of gallbladder wall enhancement was associated with the pre

283 A cut-off CT density value of the gallbladder wall of more than 31.5 HU, intraluminal bile

284 The absorbed doses to the liver and gallbladder wall were slightly but significantly higher

285 der wall, 0.258 mGy/MBq (0.955 rad/mCi), and gallbladder wall, 0.193 mGy/MBq (0.716 rad/mCi).

286 the upper large intestine, small intestine, gallbladder wall, and lower large intestines were 0.082,

287 stic stranding, wall thickness, THAD, fat in gallbladder wall, cholelithiasis, infiltration, biliary

288 ons in order to search for thickening of the gallbladder wall, pleural effusion, and ascites.

289 tudy was to evaluate the combined hyperdense gallbladder wall-lumen sign on computed tomography (CT)

290 blingual gland, spleen, pancreas, liver, and gallbladder was observed.

291 Three-dimensional images of the gallbladder were acquired at 30-minute intervals from -3

292 ll enhancement, intrahepatic bile ducts, and gallbladder were also evaluated.

293 During surgery, 23/39 (59%) of the gallbladders were found to contain biliary stones or slu

294 By 2013, 62% of gallbladders were removed laparoscopically countrywide a

295 is a procedure that removes portions of the gallbladder when structures of the Calot triangle cannot

296 inosa can spread from the bloodstream to the gallbladder, where it replicates to extremely high numbe

297 pithelial cell population from primary mouse gallbladder with stem cell characteristics and found it

298 The critical organ was the gallbladder, with an average radiation-absorbed dose of

299 observed in the kidneys, bladder, liver, and gallbladder, with mean standardized uptake values at 1 h

300 sed a big mass of hard texture involving the gallbladder, with multiple concrements, hepatoduodenal l