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1 n or neuroadaptive consequences of long-term gambling.
2 , for instance, during foraging, trading, or gambling.
3 risk-seeking behaviors, most pertinently in gambling.
4 utcomes confer vulnerability to pathological gambling.
5 g [F(1,9) = 9.2, P = 0.01] in the context of gambling.
6 show promise in the treatment of disordered gambling.
7 ional human behaviors such as overeating and gambling.
8 ondition marked by excessive and problematic gambling.
9 risk-taking in humans, such as pathological gambling.
10 s are less likely to be risk aversive during gambling.
11 l investments, career and health choices, or gambling.
12 erized by risky choices such as pathological gambling.
13 ncertainty on decision making extends beyond gambling.
14 h as drug abuse, overeating, or pathological gambling.
15 factors have been established for adolescent gambling.
16 in the treatment of adults with pathological gambling.
17 ed about the risks associated with excessive gambling.
18 owledge of the growing problem of adolescent gambling.
19 ficant reduction in severity of pathological gambling.
20 addressed in effective prevention of problem gambling.
21 order extend to those with problem (at-risk) gambling.
22 n-making, such as addiction and pathological gambling.
23 ification of the dangers of products used in gambling.
24 -related effects of neuroscience research on gambling.
25 aborations are needed to reduce the harms of gambling.
26 striatal dopamine synthesis in pathological gambling.
27 th a lifetime history of DSM-IV pathological gambling, 36%-39% did not experience any gambling-relate
29 reward anticipation and in individuals with gambling addiction during reward outcome is in line with
31 e ventral striatum, whereas individuals with gambling addiction showed decreased activation in the do
33 anticipation, individuals with substance and gambling addictions showed decreased striatal activation
34 similar to what is observed in substance and gambling addictions, the neural and behavioral mechanism
37 terview-based diagnoses of past-year problem gambling and alcohol, cannabis, and nicotine dependence
42 neural discriminators (including those from gambling and emotion tasks), it suggests the involvement
44 npharmacological treatments for pathological gambling and is based on a review of the literature publ
45 evaluation signals subsequent choices during gambling and is essential for dynamically adjusting deci
46 ve-behavioral therapy (CBT) for pathological gambling and its general principles and provides an exam
49 pted suicide misestimate expected rewards on gambling and probabilistic learning tasks.OBJECTIVES-To
50 impulse control disorders (eg, pathological gambling and shopping, binge eating, and hypersexuality)
53 relationship between diminished control over gambling and the adverse socioeconomic consequences of g
54 d prognostic uses of neuroscience in problem gambling and the provision of novel tools (eg, virtual r
56 atric disorders such as impulsivity, problem gambling, and addiction suggests that a common mechanism
57 disorder, Tourette's syndrome, pathological gambling, and addictions remain poorly understood, limit
58 luding substance use disorders, pathological gambling, and attention deficit hyperactivity disorder.
61 while they watched videos depicting cocaine, gambling, and sad scenarios to investigate the neural co
63 trol disorders (ICD), including pathological gambling, are common in Parkinson's disease (PD) and ten
66 nd disorders including addiction, compulsive gambling, attention-deficit/hyperactivity disorder, and
67 in Gambling Disorder or in at-risk (problem) gambling: attentional inhibition, motor inhibition, disc
69 a from a subset of twin pairs discordant for gambling behavior was used to control for genetic and fa
70 influencing risky choices that may moderate gambling behaviors in humans, particularly in casino and
74 ling twins and their problem or pathological gambling brothers, but adjusted co-twin analyses resulte
75 impulsivity may extend to problem (at-risk) gambling, but further studies are needed to confirm such
76 may be associated with the addictiveness of gambling, but little is known about the neurocognitive m
77 ntrol disorders (ICDs), including compulsive gambling, buying, sexual behavior, and eating, can occur
78 expected value had a stronger influence over gambling choices in adolescents relative to adults, an e
79 for addictive disorders such as pathological gambling, cigarette smoking, and drug and alcohol abuse.
81 sts develop pathological behaviours, such as gambling, compulsive eating, shopping, or disinhibited s
83 y contrast, Bayesian analysis indicated that gambling decisions were better explained by models that
85 neurobiological markers in individuals with gambling disorder (GD) could reflect transdiagnostic vul
90 also not clear whether cognitive deficits in Gambling Disorder extend to those with problem (at-risk)
93 e-third of the individuals with pathological gambling disorder in these two nationally representative
94 y among individuals with DSM-IV pathological gambling disorder in two large and representative U.S. n
99 examining the following cognitive domains in Gambling Disorder or in at-risk (problem) gambling: atte
101 of impulsivity to neurobiological models of Gambling Disorder, a comprehensive meta-analysis of all
103 has a formal indication for the treatment of gambling disorder, although placebo-controlled trials su
105 ted dexamphetamine-induced opioid release in gambling disorder, suggesting that this dysregulation in
112 th other substance use disorders, and moving gambling disorders to the chapter formerly reserved for
113 en the comorbidity between substance use and gambling disorders, surprisingly little is known about t
116 f individuals with a history of pathological gambling eventually recover, most without formal treatme
117 eversing a loss-induced effect (causing more gambling for gains than losses and the reverse) while le
120 ng logs of numerous solely probability-based gambling games and extract the wager and odds distributi
122 acy of naltrexone treatment for pathological gambling has been replicated in a double-blind, placebo-
123 From the perspective of personality, problem gambling has much in common with the addictive disorders
126 ry were estimated among individuals from the Gambling Impact and Behavior Study (N=2,417) and the Nat
139 increasing overall prevalence of adolescent gambling, it is imperative that pediatricians appreciate
141 y environmental effects on HRQoL and problem gambling.Main Outcome Measure Health-related quality of
142 arge epidemiological surveys of pathological gambling may eventually overturn the established wisdom
145 new pharmacological agents for pathological gambling, N-acetyl cysteine and modafinil, produced sign
149 fail, as in mental disorders such as problem gambling or addiction, the results can be disastrous.
150 Fourteen impulse control disorder (problem gambling or compulsive shopping) and 14 matched Parkinso
155 addictions, that certain activities such as gambling or video-game play may be considered addictive
156 ncertainty when updating their beliefs about gambling outcome and translating these beliefs into acti
161 ed from clinician-administered tasks and the gambling paradigm, as compared to questionnaire data.
162 than 18 years; recreational substance use or gambling; participants at risk for addictive behaviors;
166 ause of shared characteristics, pathological gambling (PG) has been variously conceptualized as an ob
167 ll individuals in treatment for pathological gambling (PG), but relatively little is known about the
168 oth cocaine dependence (CD) and pathological gambling (PG), few studies have directly investigated ne
171 has trouble explaining why, in slot machine gambling, players are motivated by extended play to spen
173 e, value-independent) gambling propensity, a gambling preference scaling with the amount/variance, an
174 amounts of money in 15 patients with problem gambling (PRGs) and 16 healthy control subjects (HCs).
177 diction disorders, the cues inherent in many gambling procedures are thought to play an important rol
178 substance use disorder, and many commercial gambling products incorporate salient win-paired cues.
179 mpany reward delivery, similar to commercial gambling products, show greater preference for risky opt
180 tcomes are proximal to the jackpot, increase gambling propensity and may be associated with the addic
181 pended on a baseline (ie, value-independent) gambling propensity, a gambling preference scaling with
182 ncreased just the value-independent baseline gambling propensity, leaving the other components unaffe
185 generally lacking from these procedures are gambling related cues that may moderate risk preferences
188 tudy investigated brain regions underpinning gambling-related cognitive distortions, contrasting pati
190 may be further increased by the presence of gambling-related cues in the environment, but the extent
191 at have highlighted the transitory nature of gambling-related problems have called into question whet
192 cal gambling, 36%-39% did not experience any gambling-related problems in the past year, even though
193 red gambling refers to the full continuum of gambling-related problems that includes PG disorder.
194 ilored larger-but-later rewards while visual gambling-related scenes were presented in the background
195 the environment, but the extent to which the gambling relatedness of task settings affects reward res
196 Considerations for future directions in gambling research, with a view towards translating neuro
198 Furthermore, the negative relation between gambling severity and amygdala activation in gain expect
199 hly significant negative correlation between gambling severity scores and right amygdala activation a
201 include hypersexuality as well as compulsive gambling, shopping, and eating, and these behaviors are
207 rates of treatment-seeking for pathological gambling suggests that natural recovery might be common.
209 rticipants, while they took part in the Iowa Gambling Task (IGT), a monetary decision making task tha
210 ociation with cognition, especially the Iowa Gambling Task (IGT), and with schizophrenic psychopathol
211 sion making can be quantified using the Iowa Gambling Task (IGT), which requires choosing between adv
214 Rats trained to perform a version of the rat gambling task (rGT) in which salient audiovisual cues ac
216 ing win-associated audiovisual cues to a rat gambling task (rGT) would influence decision making.
220 onths, who played 60 turns of the Children's Gambling Task and provided regular estimates on their pe
221 loyed a simple two-alternative forced choice gambling task and quantified the frequency at which part
223 ormance in specific domains of the Cambridge Gambling Task correlated inversely and specifically with
224 aking over 3 million decisions, who played a gambling task designed to measure the latent causes and
226 ealthy control subjects (n = 23) completed a gambling task featuring a decision between a gamble and
227 elucidate its neural basis, we used a novel gambling task in conjunction with functional magnetic re
229 e, we investigate human decision-making in a gambling task in which the informational value of each t
230 ty of single neurons in monkeys performing a gambling task in which the reward outcome of each choice
231 and adult participants performed a monetary gambling task in which they chose to accept or reject ga
232 Using functional imaging during a simple gambling task in which we constantly changed risk, we sh
234 d dopamine release to conditioned cues and a gambling task on [(11)C]raclopride positron emission tom
235 acquired while the participants performed a gambling task predicting whether they would win or lose
236 ementary eye field (SEF) of monkeys during a gambling task that allowed us to distinguish chosen good
237 (20 males and 20 females) while performing a gambling task that involved monetary loss and gain.
238 al circuits encoding bias, we administered a gambling task to 10 participants implanted with intracer
240 atement: We used a rodent analog of the Iowa Gambling Task to determine whether the addition of audio
241 We combined functional imaging with a simple gambling task to vary expected reward and risk simultane
242 fy time-series for emotion task vs. no-task, gambling task vs. no-task and emotion task vs. gambling
243 imaging (fMRI) data during performance of a gambling task where blocks comprise values drawn from on
244 ices made by two rhesus macaques in a visual gambling task while we varied the delay between trials.
245 ing vmPFC neurons while macaques performed a gambling task with asynchronous offer presentation.
246 Following the two nights, they performed a gambling task with no immediate feedback; for each round
248 individuals make poor decisions on the Iowa Gambling Task, a reward-related decision-making task tha
250 e between the good and bad decks of the Iowa Gambling Task, and that healthy participants even have c
256 e risky, disadvantageous options on the Iowa Gambling Task, is associated with greater risk of relaps
259 s) recorded in humans during a probabilistic gambling task, we show that individuals' behavioral tend
260 ala (BLA) in acquisition of the rGT and Iowa Gambling task, we used a contralateral disconnection les
279 striatal dopamine release to incentive cues, gambling tasks and reward prediction, and possible inhib
280 to be robustly risk-seeking in computerized gambling tasks typically used for electrophysiology.
281 of sleep restriction affects neither general gambling tendency, nor two of the main predictions of pr
282 erized by a persistent, recurrent pattern of gambling that is associated with substantial distress or
283 se data indicate that near-misses invigorate gambling through the anomalous recruitment of reward cir
286 differences existed between the non-problem gambling twins and their problem or pathological gamblin
290 vated for cocaine videos in CD participants, gambling videos in PG participants, and sad videos in co
295 ndred seven persons with DSM-IV pathological gambling were randomly assigned to receive nalmefene (25
296 e Compulsive Scale Modified for Pathological Gambling) were analyzed by using a linear mixed-effects
297 ion was phrased in terms of gains and toward gambling when the decision was phrased in terms of losse
298 ng in the stop-signal paradigm reduces risky gambling when the training and gambling task are separat
299 that they are dysfunctional in pathological gambling with poor self-control, we studied gamblers wit
300 therapy, compulsive buying and pathological gambling, with eight patients exhibiting more than one i