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1 cient synaptic inhibition upon activation of gamma-aminobutyric acid receptors.
2 also includes the metabotropic glutamate and gamma-aminobutyric acid receptors.
3 dopamine and muscarinic cholinergic, but not gamma-aminobutyric acid receptors.
4 by picrotoxin, an antagonist of the GABA(A) (gamma-aminobutyric acid) receptor.
5 entiate neuronal inhibition through GABA(A) (gamma-aminobutyric acid) receptors.
6 onal cell-surface antibodies, mainly against gamma-aminobutyric acid receptors (53% vs 11%; P < .001)
7 ethyl-d-aspartate receptor in 4 patients and gamma-aminobutyric acid receptor A in 1 patient of 111 p
10 piramate-a glutamate receptor antagonist and gamma-aminobutyric acid receptor agonist-would result in
11 he known subunit stoichiometry of the A-type gamma-aminobutyric acid receptor allowed us to create re
12 acts as a non-competitive antagonist of the gamma-aminobutyric acid receptor and has shown prolonged
13 enhance inhibitory transmission mediated by gamma-aminobutyric acid receptors and have anticonvulsan
15 hreshold dose of picrotoxin, a use-dependent gamma-aminobutyric acid receptor antagonist, reduces cor
17 ter-operated ion channels, such as the GABA (gamma-aminobutyric acid) receptor, are important in fast
18 le-associated protein 1 light chain 3 (LC3), gamma-aminobutyric acid receptor-associated protein (GAB
20 of reduced lysosomal degradation of RhoB in Gamma-aminobutyric acid receptor-associated protein (GAB
23 by disinhibition because local antagonism of gamma-aminobutyric acid receptors blocked responses to C
24 etylcholine receptor for neonicotinoids, the gamma-aminobutyric acid receptor/chloride channel for po
25 and pharmacological evidence that ionotropic gamma-aminobutyric acid receptors contribute to fluid tr
26 +/- 9% enhancement) than it was at enhancing gamma-aminobutyric acid receptor current (3 +/- 3% enhan
27 n N-methyl-D-aspartate receptor currents and gamma-aminobutyric acid receptor currents within the sam
28 E cells juxtaposed next to cohorts of normal gamma-aminobutyric acid receptor expressing secretory ce
30 nscriptional control of the mammalian type A gamma-aminobutyric acid receptor (GABA(A)R) subunit gene
32 amic activation process for the metabotropic gamma-aminobutyric acid receptor (GABA(B)R) based on ext
34 the Ca(2+)-sensing receptor (CaR) and type B gamma-aminobutyric acid receptor (GABA-B-R) from human e
35 xpression of delta-subunit containing type A gamma-aminobutyric acid receptors (GABA(A)(delta)R) incr
41 which drive neuronal excitation, and type A gamma-aminobutyric acid receptors (GABA(A)Rs), which are
44 cently become appreciated that activation of gamma-aminobutyric acid receptors (GABA-Rs) on ss-cells
46 oxication causes changes in the rodent brain gamma-aminobutyric acid receptor (GABAAR) subunit compos
48 stsynaptic potentials mediated by ionotropic gamma-aminobutyric acid receptors (GABAARs) and glycine
52 t rapid antidepressants cause a shift in the gamma-aminobutyric acid receptor (GABABR) signaling path
54 ange the type of excitability: a depolarized gamma-Aminobutyric acid receptor (GABAR) reversal potent
58 peptide (AgRP) neurons because inhibition of gamma-aminobutyric acid receptor in the ARC did not prev
59 igated the role of regionally discrete GABA (gamma-aminobutyric acid) receptors in the sedative respo
60 n of unknown function isolated previously as gamma-aminobutyric acid receptor-interacting factor 1 (G
62 macologically distinct ionotropic receptors: gamma-aminobutyric acid receptors, levamisole-sensitive
63 The effect is caused by a decrease in type A gamma-aminobutyric acid receptor-mediated inhibition of
65 sed by cocaine-induced reduction of GABA(A) (gamma-aminobutyric acid) receptor-mediated inhibition of
67 ated the expression of functional ionotropic gamma-aminobutyric acid receptors on the apical plasma m
69 ministration model, we studied glutamate and gamma-aminobutyric acid receptor regulation in the synap
71 lar endothelial growth factor, integrin, and gamma-aminobutyric acid receptor signaling cascades, plu
72 s, and behavioral results suggest that local gamma-aminobutyric acid receptor signaling mediates the
74 ficant association with polymorphisms in the gamma-aminobutyric acid receptor subunit B3 gene (GABRB3
75 on chromosome 15 to an area surrounding the gamma-aminobutyric acid-receptor subunit genes, in AutD,
76 e identified with differential expression of gamma-aminobutyric acid receptor subunits and transporte
77 ved from studies of anesthetic resistance in gamma aminobutyric acid receptors, tandem pore potassium
78 inactivation of BLA by microinfusion of the gamma-aminobutyric acid receptor type A agonist muscimol
79 physiological studies have demonstrated that gamma-aminobutyric acid receptors type A (GABA(A)) media
80 steroids on neural transmission mediated by gamma-aminobutyric acid receptors within forebrain neuro