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1 minase, alkaline phosphatase, bilirubin, and gamma-glutamyltransferase).
2 atory measure of alcohol consumption (plasma gamma-glutamyltransferase).
3 rediction were 0.80 for CLivD lab (including gamma-glutamyltransferase), 0.72 for CLivD non-lab (excl
4 ven by a truncated, proximal tubule-specific gamma-glutamyltransferase 1 (ggt or gammaGT) promoter, t
5  common grade 3-4 adverse events were raised gamma-glutamyltransferase (13 [16%] of 80 patients; coho
6 occurring in >3% of patients) were increased gamma-glutamyltransferase (14 [10%] patients in the idal
7 d with -7.94 (-11.1, -4.8) IU/L; P < 0.001], gamma-glutamyltransferase [-15.08 (-15.5, -14.7) compare
8 hydrase 2, erythropoietin, protein-glutamine gamma-glutamyltransferase 2, thymosin beta-10, U-type mi
9 , thrombocytopenia (26 [18%]), and increased gamma-glutamyltransferase (24 [17%]).
10 ing epithelial membrane protein 3 (EMP3) and gamma-glutamyltransferase 5 (GGT5).
11          We prioritized mucin-1, mesothelin, gamma-glutamyltransferase 5, and cathepsin-E as the most
12                We also found that the enzyme gamma-glutamyltransferase-5 (GGT5), which was highly exp
13 resolved, as evidenced by lower median (IQR) gamma-glutamyltransferase [54 U/L (41, 103 U/L) compared
14 ator geminin and the plasma membrane protein gamma-glutamyltransferase 6, two of the core RIGs, are r
15 trast, elevated alanine aminotransferase and gamma glutamyltransferase activities were associated wit
16  including protein endocytosis and increased gamma-glutamyltransferase activity, and cyclic AMP respo
17 oups had significant reductions in levels of gamma-glutamyltransferase and alanine aminotransferase a
18  also associated with lower plasma levels of gamma-glutamyltransferase and alkaline phosphatase than
19 er previously purified T. denticola enzymes, gamma-glutamyltransferase and cystalysin, CGase mediates
20               Increases in biliary and serum gamma-glutamyltransferase and serum bile acids also occu
21 tuin-A (P-trend = 0.06) and CRP in women and gamma-glutamyltransferase and triglycerides in men.
22  body mass index, waist circumference, serum gamma-glutamyltransferase and triglycerides.
23 athione and cysteine through upregulation of gamma-glutamyltransferases and transcriptional repressio
24 ds, high-sensitivity C-reactive protein, and gamma-glutamyltransferase) and novel (adiponectin, E-sel
25 5), alanine aminotransferase (p < or =.001), gamma-glutamyltransferase, and bilirubin (p < or =.05) (
26 minotransferase (ALT), alkaline phosphatase, gamma-glutamyltransferase, and bilirubin.
27 nsitivity C-reactive protein, triglycerides, gamma-glutamyltransferase, and fatty liver, whereas fast
28 poprotein-B, alanine aminotransferase (ALT), gamma-glutamyltransferase, and lymphocytes were associat
29 ine phosphatase, aspartate aminotransferase, gamma-glutamyltransferase, and triglycerides and lower c
30 minotransferase; aspartate aminotransferase; gamma-glutamyltransferase; and, in women, C-reactive pro
31 ociation between MaxAlc and the liver enzyme gamma-glutamyltransferase (beta = 0.012; P = 2.66 x 10-1
32  aminotransferase, alanine aminotransferase, gamma-glutamyltransferase, bile acids, and spleen size,
33 nity and catalytic efficiency of bovine milk gamma-glutamyltransferase (BoGGT) towards different dono
34                                        Serum gamma-glutamyltransferase, C4, and primary bile acids de
35 n for AST (coefficient of variation, 13.9%), gamma-glutamyltransferase (coefficient of variation, 13.
36 of 113 in the chemotherapy group), increased gamma glutamyltransferase concentration (24 [21%] vs one
37 sting energy expenditure increased and serum gamma-glutamyltransferase concentrations decreased in bo
38 tion, tubular reabsorption of phosphate, and gamma-glutamyltransferase-creatinine ratio.
39 roposed to occur via a three enzyme pathway, gamma-glutamyltransferase, cysteinylglycinase (CGase), a
40  proteoliposomes have been prepared in which gamma-glutamyltransferase (EC 2.3.2.2) was functionally
41 ntribution of glutamyl transpeptidase (GGT) (gamma-glutamyltransferase [EC 2.2.2]) to Helicobacter py
42 ions in one patient and a transitory grade 3 gamma-glutamyltransferase elevation in another patient.
43    In the current study, we characterize the gamma-glutamyltransferase enzyme (GGT) as a novel virule
44                     Glycosylasparaginase and gamma-glutamyltransferase enzymes are found to be simila
45 ut mandated when alanine aminotransferase or gamma glutamyltransferase exceeded 100 U/L.
46 markers, such as liver markers (reflected by gamma-glutamyltransferase, fetuin-A, and sex hormone-bin
47 es (seven [4%] and five [3%]), and increased gamma-glutamyltransferase (five [3%] and two [1%]).
48 d greater risk for dementia, but the role of gamma-glutamyltransferase (gamma-GT) in dementia has not
49 erase (AST), alanine aminotransferase (ALT), gamma glutamyltransferase (gammaGT), lactate dehydrogena
50 ferase (ALT), alkaline phosphatase (ALP) and gamma glutamyltransferase (GGT), on diabetes and cardiov
51 Using MR, genetic predictors of ALT, ALP and gamma glutamyltransferase (GGT), were applied to genome-
52 ted serum alanine aminotransferase (ALT) and gamma-glutamyltransferase (GGT) activities are markers o
53              Alanine aminotransferase (ALT), gamma-glutamyltransferase (GGT) and direct bilirubin, ma
54 exons generates unique noncoding 5'-ends for gamma-glutamyltransferase (GGT) cDNAs in several species
55 lved in the strong dose-response relation of gamma-glutamyltransferase (GGT) concentration with incid
56 (ALT), aspartate aminotransferase (AST), and gamma-glutamyltransferase (GGT) elevation was defined as
57                             In this pathway, gamma-glutamyltransferase (GGT) has been proposed to cat
58               The association between AF and gamma-Glutamyltransferase (GGT) was not fully establishe
59 r enzymes alanine aminotransferase (ALT) and gamma-glutamyltransferase (GGT) was observed in the NRPT
60 ations of alanine aminotransferase (ALT) and gamma-glutamyltransferase (GGT) with diabetes.
61 lecting liver fat accumulation [reflected by gamma-glutamyltransferase (GGT), alanine transaminase (A
62                           Mild elevations in gamma-glutamyltransferase (GGT), alkaline phosphatase (A
63 ase (ALT), aspartate aminotransferase (AST), gamma-glutamyltransferase (GGT), and CK-18 were measured
64 formed values of alanine transaminase (ALT), gamma-glutamyltransferase (GGT), and direct bilirubin on
65 ow-density lipoprotein cholesterol, glucose, gamma-glutamyltransferase (GGT), and hsCRP, as compared
66 ansferase, aspartate aminotransferase (AST), gamma-glutamyltransferase (GGT), and total bilirubin, wh
67  (NAFLD) alanine aminotransferase (ALT), and gamma-glutamyltransferase (GGT), with 3 separate measure
68 (ALT), aspartate aminotransferase (AST), and gamma-glutamyltransferase (GGT).
69   The enzyme responsible for this catalysis (gamma-glutamyltransferase [GGT]; EC 2.3.2.2) was purifie
70 racellular iron reduction strategy involving gamma-glutamyltransferase (Ggt1) activity.
71  colorimetric method to measure GS-catalyzed gamma-glutamyltransferase (GT) activity in rat L6 skelet
72 ne-mapping at the GGT1 locus associated with gamma glutamyltransferase in the African ancestry partic
73 transferase, aspartate aminotransferase, and gamma-glutamyltransferase in child serum.
74 grade 3 or worse adverse event was increased gamma-glutamyltransferase in five (12%) patients.
75 >10%) included an aspartate transaminase and gamma-glutamyltransferase increase (16%), hyperglycemia
76 ith long-acting pasireotide monotherapy were gamma-glutamyltransferase increased (four [10%] of 41 pa
77 ferase level (-10.9 vs -36.2 u/L; P = .009), gamma-glutamyltransferase level (-9.4 vs -41.2 u/L; P =
78 % confidence interval [CI], 1.004-1.031) and gamma-glutamyltransferase level (OR, 1.016 per U/L; 95%
79 phatase level less than or equal to 240 U/L, gamma-glutamyltransferase level less than or equal to 13
80 ange, 5-45 U/L [0.08-0.75 mukat/L]), a serum gamma-glutamyltransferase level of 210 U/L (3.50 mukat/L
81 IU/L [0-0.67 mukat/L]) and a mildly elevated gamma-glutamyltransferase level of 52 IU/L (0.88 mukat/L
82 IU/L [0-0.67 ukat/L]), and a mildly elevated gamma-glutamyltransferase level of 52 IU/L (0.88 ukat/L)
83       There was one grade 3 elevation in the gamma-glutamyltransferase level, which was considered by
84 ramate than naltrexone for BMI, craving, and gamma-glutamyltransferase level.
85  aminotransferase, alkaline phosphatase, and gamma-glutamyltransferase levels and the decrease in cal
86  In alcohol-dependent participants, 24-month gamma-glutamyltransferase levels were lower in TEL than
87 ruritus, intrahepatic cholestasis, low serum gamma-glutamyltransferase levels, and characteristic "By
88 T, UDCA, and untreated groups, respectively: Gamma-glutamyltransferase normalized in 53%, 49%, and 52
89 nia (two [8%] in each cohort), and increased gamma-glutamyltransferase (one [4%] in cohort 1, three [
90 ing toxicities included hematuria, increased gamma-glutamyltransferase or ALT, insomnia, and nausea/v
91 abnormal concentrations of the liver enzymes gamma-glutamyltransferase (OR per 1-SD increase in BPA c
92 er biopsy specimens from 3 children with low gamma-glutamyltransferase PFIC before and after PEBD.
93 orrelated with liver enzymes, in particular, gamma-glutamyltransferase (R = 0.70), independent of bod
94 ression) in arachidonate 15-lipoxygenase and gamma-glutamyltransferase transcripts were demonstrated.
95 s, while accounting for measurement error in gamma-glutamyltransferase, using data collected in the C
96 .7 IU/L to 25.1 +/- 20 IU/L); mean levels of gamma-glutamyltransferases were reduced from 51 IU/L bef
97 ic alanine or aspartate aminotransferase, or gamma-glutamyltransferase, without increased bilirubin,