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1 els of chemokines KC and monokine induced by gamma interferon.
2 tor, monocyte chemoattractant protein 1, and gamma interferon.
3 e I interferons (IFN-I), interleukin 12, and gamma interferon.
4 reas C4 synthesis occurred on treatment with gamma interferon.
5  by 28 dpi except interleukin 12 (IL-12) and gamma interferon.
6 ved coma scores, as well as higher levels of gamma interferon.
7 se and cationic amino acid transporter 2 via gamma interferon.
8 rculating inflammatory cytokines, except for gamma-interferon.
9 MCP-1, MCP-2, MCP-3, and monokine induced by gamma-interferon.
10 ursting during transcriptional activation by gamma-interferon.
11  induction of CD25, CD69, interleukin-2, and gamma-interferon.
12         By contrast, activity of the diverse gamma-interferon-activated inhibitor of translation (GAI
13                                    The GAIT (gamma-interferon-activated inhibitor of translation) com
14 ecruitment to a region containing a putative gamma-interferon activation sequence (GAS) element at -4
15 both burst size and frequency in response to gamma-interferon activation.
16 t CTLs failed to upregulate FasL and produce gamma interferon after engagement with target cells and
17 ens, and EPS abrogates systemic induction of gamma interferon after infection.
18  cell-depleted mice resulted in increases in gamma interferon and interleukin-17 production and decre
19 ad elevated splenic CD4(+) T cells producing gamma interferon and interleukin-17A, indicating that PN
20 boratory HIV database (1,300 peptides) using gamma interferon and interleukin-2 (IFN-gamma/IL-2) Fluo
21 e, suggesting Th1 polarization, and produced gamma interferon and other cytokines after reactivation
22              These cells are able to produce gamma interferon and remain metabolically active but hav
23            Individual baboons expressed more gamma interferon and tumor necrosis factor alpha in resp
24       The frequency of CD8 T cells producing gamma interferon and tumor necrosis factor in response t
25 L)-17A secretion by Th17 cells, 2) inhibited gamma-interferon and tumor necrosis factor alpha secreti
26 ) produced less tumor necrosis factor alpha, gamma interferon, and granzyme B.
27 tein 2, RANTES, tumor necrosis factor alpha, gamma interferon, and interleukin-10 were upregulated in
28 L10 [gamma interferon-inducible protein 10], gamma interferon, and lambda interferon) in association
29 ach of these strains was also used to infect gamma interferon- and lipopolysaccharide-activated murin
30 erium avium complex (DMAC) disease with anti-gamma interferon autoantibodies.
31 actic protein-1/CCL2 and monokine induced by gamma interferon/CXCL9 in the hypotensive and normotensi
32 inal wash antibodies and an antigen-specific gamma interferon-dominated Th1-biased T cell response.
33                                 By measuring gamma interferon enzyme-linked immunosorbent spot (ELISP
34 eptides from rDEN2Delta30 and used them in a gamma interferon enzyme-linked immunosorbent spot assay
35 etermined via a newly established guinea pig gamma interferon enzyme-linked immunosorbent spot assay.
36 ear cells from these donors were screened in gamma interferon enzyme-linked immunosorbent spot assays
37                                              Gamma interferon enzyme-linked immunosorbent spot assays
38                                          IFN-gamma (interferon-gamma) primes EC responsiveness to MAC
39 -alpha (tumor necrosis factor-alpha) and IFN-gamma (interferon-gamma) were upregulated in the DC ACT
40 or cytokines/chemokines (IL-2, IL-12, IL-17, gamma interferon, granulocyte-macrophage colony-stimulat
41     Resistance to Toxoplasma is dependent on gamma interferon (IFN-gamma) activation of both hematopo
42                            Neutralization of gamma interferon (IFN-gamma) ameliorated the enhancement
43 Affymetrix microarray analysis revealed that gamma interferon (IFN-gamma) and 16 antiviral interferon
44 rgets that was influenced by the presence of gamma interferon (IFN-gamma) and availability of newly t
45 hat Brucella-specific CD8(+) T cells express gamma interferon (IFN-gamma) and can transition to long-
46 activation of proinflammatory genes, such as gamma interferon (IFN-gamma) and CCL2.
47                                              Gamma interferon (IFN-gamma) and CCL5 were induced in lu
48                             Plasma levels of gamma interferon (IFN-gamma) and CXCL9 (monokine induced
49 function, defined by increased production of gamma interferon (IFN-gamma) and granzyme B and expressi
50  CNS CD4 T cells was associated with reduced gamma interferon (IFN-gamma) and granzyme B expression b
51 e show that activated CD8(+) T cells express gamma interferon (IFN-gamma) and granzymes but that gran
52  splenic T cells secreted significantly more gamma interferon (IFN-gamma) and had significantly more
53 -alpha) in the draining lymph node (DLN) and gamma interferon (IFN-gamma) and IL-10 in the lung.
54  require eosinophils, and was independent of gamma interferon (IFN-gamma) and IL-17.
55 elper-1 (Th1), Th2, and Th17 cells, elevated gamma interferon (IFN-gamma) and interleukin (IL)-17 pro
56                                     Although gamma interferon (IFN-gamma) and interleukin-10 (IL-10)
57 enged with HHV-6 preparations indicated that gamma interferon (IFN-gamma) and interleukin-10 (IL-10)
58                              We screened the gamma interferon (IFN-gamma) and interleukin-10 (IL-10)
59 e to that provoked by LVS but with increased gamma interferon (IFN-gamma) and interleukin-17A (IL-17A
60 and a negative correlation between levels of gamma interferon (IFN-gamma) and interleukin-17A (IL-17A
61 from the mutant-immunized mice produced more gamma interferon (IFN-gamma) and interleukin-4.
62                                              Gamma interferon (IFN-gamma) and interleukin-5 (IL-5) en
63 mmatory cytokines and chemokines, especially gamma interferon (IFN-gamma) and KC, in the lungs compar
64  associated with NK cells and DCs, including gamma interferon (IFN-gamma) and RANTES, were increased
65                                 Induction of gamma interferon (IFN-gamma) and T-bet was observed with
66 tumor necrosis factor alpha (TNF-alpha), and gamma interferon (IFN-gamma) and the memory markers CD27
67                     SC subjects produce less gamma interferon (IFN-gamma) and tumor necrosis factor a
68 ory CD8(+) T cells that were able to secrete gamma interferon (IFN-gamma) and tumor necrosis factor a
69 ting a cytokine storm by their production of gamma interferon (IFN-gamma) and tumor necrosis factor a
70 ociated with increased production of mucosal gamma interferon (IFN-gamma) and tumor necrosis factor a
71 sion is induced by proinflammatory cytokines gamma interferon (IFN-gamma) and tumor necrosis factor a
72 d to a response that included high levels of gamma interferon (IFN-gamma) and tumor necrosis factor a
73  YopE69-77-specific CD8(+) T cells producing gamma interferon (IFN-gamma) and tumor necrosis factor a
74 Th1, Th2, and Th17 cytokines; however, early gamma interferon (IFN-gamma) and tumor necrosis factor a
75                                 Increases in gamma interferon (IFN-gamma) and tumor necrosis factor a
76 ic CD8(+) T lymphocytes capable of producing gamma interferon (IFN-gamma) and tumor necrosis factor a
77 tion of interleukin-17A (IL-17A), IL-22, and gamma interferon (IFN-gamma) as well as the antimicrobia
78  mice were also completely unable to produce gamma interferon (IFN-gamma) at any time points followin
79             Furthermore, genetic deletion of gamma interferon (IFN-gamma) but not IL-22 or antibody-m
80 ty of NK cells to proliferate and to produce gamma interferon (IFN-gamma) but positively associated w
81 monstrated that the functional production of gamma interferon (IFN-gamma) by antigen-specific CD4(+)
82 h was associated with enhanced production of gamma interferon (IFN-gamma) by NK cells.
83 to amplified T cell activation, higher serum gamma interferon (IFN-gamma) concentrations, enhanced in
84               In the livers of C57BL/6 mice, gamma interferon (IFN-gamma) controls intracellular Leis
85 s in vivo, wild-type mice were injected with gamma interferon (IFN-gamma) DNA or colony-stimulating f
86               SIV-specific T cells producing gamma interferon (IFN-gamma) dominated these responses.
87                                              Gamma interferon (IFN-gamma) drives antiparasite respons
88 on in BALB/c mice, whereas neutralization of gamma interferon (IFN-gamma) enhanced protection in C57B
89 saic immunogens induced significantly higher gamma interferon (IFN-gamma) enzyme-linked immunosorbent
90 m HIV-1 Gag and Env were used as antigens in gamma interferon (IFN-gamma) enzyme-linked immunosorbent
91 y enzyme-linked immunosorbent assay (ELISA), gamma interferon (IFN-gamma) enzyme-linked immunosorbent
92 e transcriptases (RT), were identified using gamma interferon (IFN-gamma) enzyme-linked immunosorbent
93 e-specific T-cell activation was measured by gamma interferon (IFN-gamma) enzyme-linked immunosorbent
94 ial epitope targeting rates, as confirmed by gamma interferon (IFN-gamma) enzyme-linked immunosorbent
95 ith primary HIV-1 infection were screened by gamma interferon (IFN-gamma) enzyme-linked immunospot (E
96 22 did not worsen abscesses but did increase gamma interferon (IFN-gamma) expression at these sites.
97 d with decreased survival and decreased lung gamma interferon (IFN-gamma) expression compared to B6 c
98 osuppressive anthrax lethal toxin impairs NK gamma interferon (IFN-gamma) expression, but neither let
99 rt that (i) miR-H28 induced the synthesis of gamma interferon (IFN-gamma) in both infected cells and
100  of interleukin 1beta (IL-1beta), IL-18, and gamma interferon (IFN-gamma) in sera.
101 tions of interleukin 1ra (IL-1ra), IL-6, and gamma interferon (IFN-gamma) increased markedly.
102                                              Gamma interferon (IFN-gamma) is an important driver of i
103 vo approaches reveals that the expression of gamma interferon (IFN-gamma) is epigenetically silenced
104                            We confirmed that gamma interferon (IFN-gamma) is essential for induction
105 n reported that the proinflammatory cytokine gamma interferon (IFN-gamma) is responsible.
106  able to regain long-lasting colonization in gamma interferon (IFN-gamma) knockout mice following int
107 restingly, IgA(-/-) mice had lower pulmonary gamma interferon (IFN-gamma) levels and decreased number
108                  Despite substantial in vivo gamma interferon (IFN-gamma) levels, T-bet-knockout (KO)
109 centages of CD4(+)Tbet(+) cells and elevated gamma interferon (IFN-gamma) mRNA in PBMCs.
110 of the biofilm, even when prestimulated with gamma interferon (IFN-gamma) or TNF-alpha or cocultured
111 KT) cells from coexposed mice expressed less gamma interferon (IFN-gamma) per cell than did those fro
112 to 18 h postinfection, and was unaffected by gamma interferon (IFN-gamma) pretreatment.
113 -derived macrophages (MDM) requires NOX2 and gamma interferon (IFN-gamma) pretreatment.
114 rease P. falciparum-specific in vitro recall gamma interferon (IFN-gamma) production after CHMI, and
115              Further studies have shown that gamma interferon (IFN-gamma) production and activation o
116 s of cognate antigen-specific T cell clones: gamma interferon (IFN-gamma) production and mobilization
117 reatment with anti-CXCL10 antibodies reduced gamma interferon (IFN-gamma) production and Th17 cell in
118                                       Innate gamma interferon (IFN-gamma) production by CD8(+) T cell
119 iters and increased numbers of and increased gamma interferon (IFN-gamma) production by several diffe
120 servation of CD4(+) T cells and an increased gamma interferon (IFN-gamma) production from stimulated
121 ureus and greatly promoted proliferation and gamma interferon (IFN-gamma) production in CD4 and CD8 T
122 ost potent combinations capable of eliciting gamma interferon (IFN-gamma) production in NK cells.
123  evident, while decreased mitogen-stimulated gamma interferon (IFN-gamma) production suggested immuno
124 ciency prevents proper T cell activation and gamma interferon (IFN-gamma) production, which are criti
125 role in CD4+ Th1 lineage differentiation and gamma interferon (IFN-gamma) protein expression by CD4+
126 IL-10)/tumor necrosis factor alpha and IL-12/gamma interferon (IFN-gamma) ratios, and higher antibody
127 vants correlated better with enhanced T-cell gamma interferon (IFN-gamma) recall responses rather tha
128                                              Gamma interferon (IFN-gamma) release assays (IGRAs) are
129 ssing target cells, including stimulation of gamma interferon (IFN-gamma) release, specific target ce
130 enes might mediate the greater virulence and gamma interferon (IFN-gamma) resistance of C. muridarum
131 mune response to malaria by initiating a CD4 gamma interferon (IFN-gamma) response early in a blood-s
132                                          The gamma interferon (IFN-gamma) response, mediated by the S
133 n in rhesus macaques, SC-Ad generated higher gamma interferon (IFN-gamma) responses and higher transg
134 berculosis (TB) vaccine candidates, elicited gamma interferon (IFN-gamma) responses from both TB and
135 /-) mice display organ burdens and pulmonary gamma interferon (IFN-gamma) responses similar to those
136          This strain also resulted in better gamma interferon (IFN-gamma) responses than the strain w
137 pitope and found individuals who had ex vivo gamma interferon (IFN-gamma) responses to the peptide ep
138                              The majority of gamma interferon (IFN-gamma) responses were associated w
139                                         Lung gamma interferon (IFN-gamma) responses were blunted and
140  of HIV-1-specific CD4 T cell proliferation, gamma interferon (IFN-gamma) secretion, and, to a lesser
141 ion of MDMs by lipopolysaccharide (LPS) plus gamma interferon (IFN-gamma) stimulation caused no effec
142 In vivo, the host immune system will release gamma interferon (IFN-gamma) to combat infection.
143                In this study, we report that gamma interferon (IFN-gamma) treatment, but not IFN-alph
144 ypes, their relative expression of TRAIL and gamma interferon (IFN-gamma) was assessed during both ea
145                                              Gamma interferon (IFN-gamma) was detected in serum betwe
146 esponse to viral antigen, while secretion of gamma interferon (IFN-gamma) was more limited in compari
147 er of CD4 T cells and CD8 T cells expressing gamma interferon (IFN-gamma) were observed in IL-17A(-/-
148 hese activated effector CD8 T cells produced gamma interferon (IFN-gamma) when stimulated with dengue
149 ith decreased detection of granzyme B(+) and gamma interferon (IFN-gamma)(+) MAIT cells relative to t
150      This was further validated by depleting gamma interferon (IFN-gamma), a cytokine known to contro
151 pon MAIT cell selection by MR1, secretion of gamma interferon (IFN-gamma), and an innate interleukin
152 s to elicit anti-HIV functions such as CCL4, gamma interferon (IFN-gamma), and CD107a expression.
153 tion, secretion of the inflammatory cytokine gamma interferon (IFN-gamma), and host defense against t
154 chose TLR7, transcription factor p65 (RelA), gamma interferon (IFN-gamma), and IFN-gamma-inducible pr
155 ression of mRNAs for CD3delta, CD4, CD8beta, gamma interferon (IFN-gamma), and inducible nitric oxide
156                   Levels of BALF leukocytes, gamma interferon (IFN-gamma), and interleukin 10 (IL-10)
157  of tumor necrosis factor alpha (TNF-alpha), gamma interferon (IFN-gamma), and interleukin 2 (IL-2),
158 ent on CD8(+) T cells, involves perforin and gamma interferon (IFN-gamma), and is correlated with the
159  tumor necrosis factor alpha (TNF-alpha) and gamma interferon (IFN-gamma), and previous studies have
160 lammatory cytokines IL-22, IL-17a, IL-1beta, gamma interferon (IFN-gamma), and TNF-alpha, as well as
161 in interleukin 1beta (IL-1beta), IL-6, IL-8, gamma interferon (IFN-gamma), and tumor necrosis factor
162 simultaneously produce interleukin-2 (IL-2), gamma interferon (IFN-gamma), and tumor necrosis factor
163  colon; an increase in the levels of CD107a, gamma interferon (IFN-gamma), and tumor necrosis factor
164 mory phenotype and usually expressed CD107a, gamma interferon (IFN-gamma), and tumor necrosis factor
165 R2DL3, and KIR3DL1 NK cells to produce CCL4, gamma interferon (IFN-gamma), and/or CD107a were assesse
166 reased production of interleukin 17 (IL-17), gamma interferon (IFN-gamma), CCL4, and granulocyte-macr
167 ELISpot assay and intracellular detection of gamma interferon (IFN-gamma), CD107(a/b) degranulation,
168 train of C. neoformans engineered to produce gamma interferon (IFN-gamma), denoted H99gamma, demonstr
169 was associated with diminished production of gamma interferon (IFN-gamma), fewer effector CD4(+) and
170 production of interleukin 15 (IL-15), IL-18, gamma interferon (IFN-gamma), granulocyte colony-stimula
171 6), tumor necrosis factor alpha (TNF-alpha), gamma interferon (IFN-gamma), IL-1beta, and inducible ni
172 PD-L1 blockade leads to balanced increase in gamma interferon (IFN-gamma), IL-2, and IL-13 secretion,
173 myristate acetate and ionomycin, we examined gamma interferon (IFN-gamma), IL-4, IL-5, and IL-17 prod
174 oduced significantly higher amounts of IL-2, gamma interferon (IFN-gamma), IL-4, IL-6, and IL-17A in
175 )17 cells could develop in mice deficient in gamma interferon (IFN-gamma), IL-4, or IL-10.
176 er, we also observed increased production of gamma interferon (IFN-gamma), IL-5, IL-9, IL-17, and IL-
177 ing tumor necrosis factor alpha (TNF-alpha), gamma interferon (IFN-gamma), IL-6, and IL-1beta were hi
178 n of the proinflammatory cytokines IL-1beta, gamma interferon (IFN-gamma), IL-6, or IL-10.
179               The T(h)1 and T(h)17 cytokines gamma interferon (IFN-gamma), interleukin-12 (IL-12) p70
180 wed significantly lower expression levels of gamma interferon (IFN-gamma), interleukin-6 (IL-6), and
181 by DH82 cells (P < 0.01), while secretion of gamma interferon (IFN-gamma), interleukin-6 (IL-6), inte
182  of CD4(+) Th1 cells and NKT cells producing gamma interferon (IFN-gamma), it increased the ratio of
183                               Treatment with gamma interferon (IFN-gamma), l-arginine, and tetrahydro
184 xpression of IFN-stimulated genes (ISGs) and gamma interferon (IFN-gamma), likely secondary to defect
185 ll-dependent clearance is likely mediated by gamma interferon (IFN-gamma), since mice deficient in IF
186 iver, infected TLR4(-/-) mice showed reduced gamma interferon (IFN-gamma), tumor necrosis factor (TNF
187 0057(pYA5199) produced significant levels of gamma interferon (IFN-gamma), tumor necrosis factor alph
188 igher in DeltaTgPL1-infected mice, including gamma interferon (IFN-gamma), tumor necrosis factor alph
189  the DLNs soon after immunization, including gamma interferon (IFN-gamma), tumor necrosis factor alph
190 1 response, and specifically, high levels of gamma interferon (IFN-gamma), tumor necrosis factor alph
191 ion groups had higher levels of secretion of gamma interferon (IFN-gamma), tumor necrosis factor alph
192                           Gene expression of gamma interferon (IFN-gamma), tumor necrosis factor alph
193 R. typhi(GFPuv)-infected BALB/c mice elicits gamma interferon (IFN-gamma), tumor necrosis factor alph
194 mokine (C-X-C motif) ligand 10 (CXCL10), and gamma interferon (IFN-gamma), was also significantly upr
195  by treatment with E2, but the proportion of gamma interferon (IFN-gamma)- and tumor necrosis factor
196 ween cytokine-producing T cell subsets with, gamma interferon (IFN-gamma)- and tumor necrosis factor
197 vium subsp. paratuberculosis survival inside gamma interferon (IFN-gamma)-activated bovine macrophage
198                                          The gamma interferon (IFN-gamma)-activated inhibitor of tran
199 lex that promoted translational silencing in gamma interferon (IFN-gamma)-activated myeloid cells.
200     Experimental cerebral malaria (ECM) is a gamma interferon (IFN-gamma)-dependent syndrome.
201 ranscription factor CEBP-beta, regulates the gamma interferon (IFN-gamma)-induced expression of Dapk1
202 ss I (MHC-I) or CD4(+) T lymphocytes through gamma interferon (IFN-gamma)-induced MHC-II.
203                      The fact that immunized gamma interferon (IFN-gamma)-KO mice and wild-type (WT)
204 ls are the primary effector cells regulating gamma interferon (IFN-gamma)-mediated host resistance.
205 umenting a cell-autonomous role for IRF-1 in gamma interferon (IFN-gamma)-mediated inhibition of MNV
206 otein FopC, which is required for evasion of gamma interferon (IFN-gamma)-mediated signaling, is able
207 4-depleted mice also increased the number of gamma interferon (IFN-gamma)-positive CD8(+) central mem
208 ve demonstrated that antigen-specific CD8(+) gamma interferon (IFN-gamma)-positive T-cell responses a
209 ith virus-specific circulating ASFV-specific gamma interferon (IFN-gamma)-producing cells.
210  and (ii) enhanced the numbers of functional gamma interferon (IFN-gamma)-producing CRTAM(+) CFSE(+)
211 ted killing, and decreases the proportion of gamma interferon (IFN-gamma)-producing NK cells that had
212 in response to C. rodentium was dependent on gamma interferon (IFN-gamma)-producing NK1.1(+) cells an
213 lar herpesvirus infection (i.e., day 2), the gamma interferon (IFN-gamma)-producing PLZF(lo)RORgammat
214                                              Gamma interferon (IFN-gamma)-producing T cells were indu
215 p of LANA and STAT1 binding sites in several gamma interferon (IFN-gamma)-regulated genes.
216 and production of ESAT-6- or CFP-10-specific gamma interferon (IFN-gamma)-secreting and tumor necrosi
217 of activated CD8(+) T cells and Gag-specific gamma interferon (IFN-gamma)-secreting CD8(+) cells, low
218     In addition, the frequency and number of gamma interferon (IFN-gamma)-secreting effector memory (
219 The inflammatory reaction is orchestrated by gamma interferon (IFN-gamma)-secreting Th1 cells, and re
220                                 Furthermore, gamma interferon (IFN-gamma)-stimulated primary peritone
221        Furthermore, P. gingivalis suppressed gamma interferon (IFN-gamma)-stimulated release of IP-10
222 and upstream stimulatory factor 1 (USF-1) in gamma interferon (IFN-gamma)-treated hepatocytes.
223                    In this study, using dual gamma interferon (IFN-gamma)-yellow fluorescent protein
224 more interleukin-4 (IL-4) and IL-10 and less gamma interferon (IFN-gamma).
225 induced miR-155 and proinflammatory cytokine gamma interferon (IFN-gamma).
226 n serum levels of interleukin 12 (IL-12) and gamma interferon (IFN-gamma).
227 ell as the ability of the T cells to secrete gamma interferon (IFN-gamma).
228 scued only in the colon of mice deficient in gamma interferon (IFN-gamma).
229 receptor superfamily member 9 (TNFRSF9); and gamma interferon (IFN-gamma).
230 ulmonary CD4(+) T cells capable of secreting gamma interferon (IFN-gamma).
231 fection requires type I (alpha/beta) and II (gamma) interferon (IFN) production.
232                Infected mice mounted innate (gamma interferon [IFN-gamma] and soluble interleukin 2 r
233 ressing genes involved in cellular immunity (gamma interferon [IFN-gamma] and the IFN-gamma-inducible
234 (interleukin-17 [IL-17] and IL-6) and liver (gamma interferon [IFN-gamma] and tumor necrosis factor a
235 terleukin 2 [IL-2], IL-4, IL-10, IL-17A, and gamma interferon [IFN-gamma]) and rendered T cells refra
236 ed substantially higher levels of antiviral (gamma interferon [IFN-gamma], 10-kDa gamma interferon-in
237 r alpha [TNF-alpha], interleukin-12 [IL-12], gamma interferon [IFN-gamma], and IL-6), chemokines (mon
238  expressed latency-specific genes (e.g., the gamma interferon [IFN-gamma], Cxcl9, and Ccl5 genes) are
239 cell-mediated immune cytokine and chemokine (gamma interferon [IFN-gamma], interleukin 10 [IL-10], IL
240 udies with protection from clinical malaria (gamma interferon [IFN-gamma], interleukin-10 [IL-10], an
241 is viable counts and inflammatory mediators (gamma interferon [IFN-gamma], interleukin-1beta [IL-1bet
242 ulating levels of proinflammatory cytokines (gamma interferon [IFN-gamma], tumor necrosis factor alph
243 iently produced multiple effector cytokines (gamma interferon [IFN-gamma], tumor necrosis factor alph
244 evels of degranulation, cytokine expression (gamma interferon [IFN-gamma], tumor necrosis factor alph
245 f systemic and TB antigen-stimulated type 1 (gamma interferon [IFN-gamma], tumor necrosis factor alph
246 whereas recombinant viruses expressing IL-4, gamma interferon, IL-12p35, IL-12p40, or IL-12p70 do not
247  of natural killer (NK) cells, which produce gamma interferon in an IFN-I-dependent fashion, and is i
248 le nitric oxide synthase, interleukin-6, and gamma interferon in kidney tissue.
249 tigen-independent activation and produce IFN-gamma (interferon) in situ.
250 eased interleukin-6 secretion, and decreased gamma interferon-induced protein 10 secretion.
251  increased the production of IL-6 and CXCL10/gamma interferon-induced protein 10, which are implicate
252 iviral (gamma interferon [IFN-gamma], 10-kDa gamma interferon-induced protein [IP-10]) and proinflamm
253 ors (interferon regulatory factor 7, CXCL10 [gamma interferon-inducible protein 10], gamma interferon
254 g as a nuclear pattern recognition receptor, gamma interferon-inducible protein 16 (IFI16) colocalize
255                                          IFN-gamma, interferon-inducible protein-10 (IP-10), tumor ne
256                                              gamma-Interferon-inducible lysosomal thiol reductase (GI
257                                              Gamma-interferon-inducible lysosomal thiol reductase (GI
258                                              Gamma-interferon-inducible lysosomal thiol reductase (GI
259 H2-M molecular chaperone, the proteasome and gamma-interferon-inducible lysosomal thiol reductase rev
260  pleckstrin homology (PH) domains 2 (ADAP2), gamma-interferon-inducible lysosome/endosome-localized t
261 ng the AIM2 (absent in myeloma 2) and IFI16 (gamma-interferon-inducible protein 16) inflammasomes.
262            T-cell responses were screened by gamma interferon/interleukin-2 (IFN-gamma/IL-2) FluoroSp
263  Mphis incubated with lipopolysaccharide and gamma interferon (LPS/IFN-gamma) and with interleukin-4
264  HCV-specific CD8 T-cell activation (CD107a, gamma interferon, macrophage inflammatory protein 1beta,
265 on gamma (IFN-gamma) and monokine induced by gamma interferon (MIG) (CXCL9) in response to mycobacter
266 4, also known as LIGHT), monokine induced by gamma interferon (MIG), and granzyme B (P <0.00001).
267 rotein (MIP)-3alpha, and monokine induced by gamma interferon (MIG).
268 ors in BBB disruption, the administration of gamma interferon-neutralizing antibody ameliorated the e
269  activity of the type I (alpha) and type II (gamma) interferon pathways and enhances the infiltration
270 ls were found in FI-RSV immune mice, whereas gamma interferon-positive (IFN-gamma(+)) and TNF-alpha(+
271 gher among Th17 cells than among IL-17(-) or gamma interferon-positive (IFN-gamma(+)) cells, even upo
272         Furthermore, a greater proportion of gamma interferon-positive (IFN-gamma(+)) effector CD8 an
273 er the first MVA-CMDR boost, vaccine-induced gamma interferon-positive (IFN-gamma(+)) Gag-specific T-
274 nge, the mice developed broad, proteome-wide gamma interferon-positive (IFN-gamma(+)) T cell response
275 tion correlated with high numbers of CD4(+), gamma interferon-positive (IFN-gamma(+)), tumor necrosis
276 crosis factor alpha-positive [TNF-alpha(+)], gamma interferon-positive [IFN-gamma(+)]) CD4(+) effecto
277                       The levels of anti-SIV gamma interferon-positive, CD4(+), and CD8(+) T cells at
278                       The levels of specific gamma interferon-positive, interleukin-10-positive T cel
279 rs of activated CD4+ T cells in all tissues, gamma interferon-producing (IFN-gamma+) CD8+ T cells in
280 okine production, leukocyte infiltrates, and gamma interferon-producing CD4(+) T cells, as well as an
281                                              Gamma interferon-producing cells peaked at day 24 postim
282 e the function of CD8(+) T cells in terms of gamma interferon production or prevent their apoptosis t
283  cells exhibited high functional avidity for gamma interferon production, whereas VP3(173-181)-specif
284 crophage markers, and increased the level of gamma interferon production, while it decreased the leve
285 NLuc infection of AG129 mice (alpha/beta and gamma interferon receptor deficient) showed rapid spread
286 imilar disease development in alpha/beta and gamma interferon receptor knockout mice, including neuro
287 ly reported that mice lacking alpha/beta and gamma interferon receptors (IFN-alpha/betaR and -gammaR)
288  acid hybridization probe, a M. tuberculosis gamma interferon release assay, and is closely related t
289 s have correlated the results of interferon (gamma interferon) release assays (IGRAs) with known mark
290  RSV infection induced an RSV-specific human gamma interferon response in HIS mouse splenocytes.
291 frequency of functional HSV-specific, CD8(+) gamma interferon-secreting cells was significantly decre
292  and TssM produced high-titer IgG and robust gamma interferon-secreting T cell responses against the
293 of Af5517-aspirated mice displayed decreased gamma interferon secretion and increased interleukin-4 t
294 ptibility within tumor necrosis factor alpha/gamma interferon-stimulated macrophages in a STAT3/6-ind
295 g regimen resulted in significantly elevated gamma interferon T cell responses and high-avidity antig
296 are more responsive to induction of MHC-I by gamma-interferon than other neuronal populations.
297  and CAAT mutant promoters were activated by gamma interferon, the Sp1 and Inr mutants were repressed
298  levels of granzyme B, granzyme K, perforin, gamma interferon, tumor necrosis factor alpha, and inter
299 ointestinal tract, but a slight reduction of gamma interferon was observed in the ceca of mice infect
300 roteins, intracellular cytokine staining for gamma interferon was utilized to identify novel RSV-spec

 
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