ライフサイエンスコーパス: gamma-radiation

1 nergy protons from solar flares and not from gamma radiation.

2 te flour with intense pulsed light (IPL) and gamma radiation.

3 nate and acquired immune responses following gamma radiation.

4 oietic progenitor cells from lethal doses of gamma radiation.

5 ure the apoptotic response of individuals to gamma radiation.

6 cient mice were more sensitive to total body gamma radiation.

7 media and great resistance to both beta and gamma radiation.

8 ein (GFP)-Rad51 foci following DNA damage by gamma radiation.

9 ir ability to increase p53 after exposure to gamma radiation.

10 was also carried out using 0, 1, 5 and 10 Gy gamma radiation.

11 effects of UV and the cytostatic effects of gamma radiation.

12 rans cultures recover from acute exposure to gamma radiation.

13 us stimuli and reduce sensitivity of mice to gamma radiation.

14 tion lesions is almost twice as great as for gamma radiation.

15 uscles lacking satellite cell activity after gamma radiation.

16 e (Thr-68) and cannot be activated following gamma radiation.

17 sponse to DNA-damaging agents such as UV and gamma radiation.

18 transcriptomic response of C. neoformans to gamma radiation.

19 p53 is activated and its levels increased by gamma radiation.

20 of DNA strand breaks caused by adriamycin or gamma radiation.

21 th trk A or when these cells were exposed to gamma radiation.

22 chemical dosimetry for low dose detection of gamma radiation.

23 so higher after (56)Fe radiation relative to gamma radiation.

24 Cockayne Syndrome (CS) fibroblasts following gamma-radiation.

25 elanin does not protect E. dermatitidis from gamma-radiation.

26 IEC-6 intestinal epithelial cells following gamma-radiation.

27 s demonstrated in IEC-6 cells following 4 Gy gamma-radiation.

28 followed by gradual capsular stripping with gamma-radiation.

29 ts with TNFalpha, vinblastine, etoposide and gamma-radiation.

30 to apoptosis induced by TNFalpha, TRAIL and gamma-radiation.

31 y 100-fold more efficient in killing HC than gamma-radiation.

32 and UV-B light but only mildly sensitive to gamma-radiation.

33 e was compared with that induced by external gamma-radiation.

34 rosarcoma HT1080 cells following exposure to gamma-radiation.

35 15 months for patients with ISR treated with gamma-radiation.

36 stimuli, including cisplatin, etoposide, and gamma-radiation.

37 ive MRK attenuates the G(2) arrest caused by gamma-radiation.

38 lls from normal individuals upon exposure to gamma-radiation.

39 ls than in wild type cells after exposure to gamma-radiation.

40 e response effect and optimal time points of gamma-radiation.

41 greater sensitivity than wild type cells to gamma-radiation.

42 5C in the nucleus after exposure of cells to gamma-radiation.

43 ses point sources of 137Cs that emit 662-keV gamma-radiation.

44 cific reduction in clonogenic survival after gamma-radiation.

45 with doses of 0 to 18 Gy of either beta- or gamma-radiation.

46 sues of mice exposed to cobalt-60 total-body gamma-radiation.

47 iderable amounts of hydrogen when exposed to gamma-radiation.

48 o lethal and supralethal doses of total body gamma-radiation.

49 m, mainly driven by effects at high doses of gamma-radiation.

50 he hPer2/hp53 complex even when treated with gamma-radiation.

51 perties of this food were examined following gamma-radiation.

52 y correlated with the exposure dose of X- or gamma-radiations.

53 The effects of different doses of gamma radiation (0-20kGy) on the color and lipid oxidati

54 hat irradiation with relatively low doses of gamma-radiation (0.2Gy and 1Gy) does not lead to loss of

55 ation and compared the response with low LET gamma-radiation ((137)Cs; 0.5 Gy/min; 2 Gy).

56 ch more robust transcriptional response than gamma-radiation (2000 cGy) when evaluated 2 h after the

57 emia was induced using omeprazole, following gamma-radiation, 5-fluorouracil, and dextran sulphate so

58 How this bacterium can grow under chronic gamma radiation [50 grays (Gy) per hour] or recover from

59 e-strand breaks (DSBs) caused by exposure to gamma radiation across archaea, bacteria, and eukaryotes

60 NQO1(-/-) mice exposed to gamma-radiation also showed lymphoma tissues (32%) and l

61 ex2-altered cells were not hypersensitive to gamma-radiation, an agent that causes DSBs throughout th

62 ose) or 1.0 Gy (moderate dose) of whole-body gamma radiation and allowed to develop for 16 weeks.

63 rotein is induced 15- to 30-fold in cells by gamma radiation and chemical mutagens but not by UV trea

64 osed to a lethal dose (9.75 Gy) of Cobalt-60 gamma radiation and euthanized at four time points, name

65 ed in the phosphorylation of BRCA1 following gamma radiation and hydroxyurea treatment.

66 No strong link has been established between gamma radiation and its effect on mast cell survival and

67 the Gamma Forest were radiated with 1.8 kGy gamma radiation and survival microbial community analyze

68 er replication stresses such as UV exposure, gamma radiation and treatment with hydroxyurea.

69 (ARF)) showed normal downstream responses to gamma radiation and underwent cell cycle arrest.

70 Higher tail moments of gamma-radiation and benzo(a)pyrene diol epoxide-induced

71 In general, effects of combinations of gamma-radiation and Cd seem to be antagonistic at lower

72 ells displayed a deregulated p53 response to gamma-radiation and decreased regulation of downstream t

73 derived from BS donors are resistant to both gamma-radiation and doxorubicin-induced cell killing, an

74 mage, resulting in sensitization of cells to gamma-radiation and doxorubicin.

75 However, gamma-radiation and Fe(2+)-EDTA produced different propo

76 se in DNA, but the large differences between gamma-radiation and Fe(2+)-EDTA suggest that factors oth

77 two hydroxyl radical-mediated DNA oxidants, gamma-radiation and Fe(2+)-EDTA, produced nucleoside 5'-

78 Furthermore, both gamma-radiation and Fe2+-EDTA/H2O2 showed relatively mod

79 al-induced guanine oxidation associated with gamma-radiation and Fe2+-EDTA/H2O2.

80 thod was applied to plasmid DNA treated with gamma-radiation and peroxynitrite.

81 o the p53 mutation and are hypersensitive to gamma-radiation and reactive oxygen species due to the K

82 Cells were exposed to 5Gy gamma-radiation and repair followed for up to 60 minutes

83 MKP-1 was induced by gamma-radiation and repressed radiation-induced pro-apop

84 Both gamma-radiation and RIT caused cell death via an apoptot

85 ransfectants was preferentially inhibited by gamma-radiation and specific classes of apoptosis induce

86 mice were eliminated with sublethal doses of gamma-radiation and then reconstituted with syngeneic BM

87 and Histoplasma capsulatum (HC) to external gamma-radiation and to the organism-specific mAbs 18B7 a

88 hyperactivate ATM, ATR, and caspase-2 after gamma-radiation and trigger a caspase-2-dependent apopto

89 utilized: pharmacological, radiosurgery with gamma radiation, and external beam radiation.

90 tantial protective effect of melanin against gamma radiation, and the general gene expression pattern

91 pore inactivation was achieved with 8 kGy of gamma radiation, and up to 1.69 log(10)CFU/g reductions

92 for bleomycin, benzo[a]pyrene diol epoxide, gamma-radiation, and 4-nitroquinoline-1-oxide sensitivit

93 ere cultured for 90 hours, exposed to 1.0 Gy gamma-radiation, and harvested at 3 hours after gamma-ra

94 -deoxyribose oxidation products generated by gamma-radiation are similar for purified DNA and cells.

95 that plant seeds treated with high doses of gamma radiation arrest development as seedlings, the cau

96 odurans strain became almost as sensitive to gamma radiation as the ddrA knockout strain.

97 f commercial cranberry syrup irradiated with gamma radiation at a rate of 5 kGy and stored for 6 mont

98 s failed to undergo apoptosis in response to gamma radiation at both 28 and 37 degrees C.

99 c DNA than wild type cells after exposure to gamma-radiation at doses of 2 or 5 Gy.

100 After exposure to gamma radiation, bacteria isolated from the site with in

101 w that mouse embryonic stem cells exposed to gamma-radiation bear the effects of the insult for weeks

102 pressors of the uvh1 mutant's sensitivity to gamma radiation but do not affect the susceptibility of

103 53BP1 foci formation is not restricted to gamma-radiation but is also detected in response to UV r

104 e were susceptible to induction of tumors by gamma-radiation, but most tumors retained and expressed

105 Phosphorylation of Nbs mediated by gamma-radiation, but not that induced by hydroxyurea or

106 e of eight TCCs with mutant p53 responded to gamma radiation by elevation of p53, p21(WAF1), or mdm2

107 Bcl-2 can protect endothelial cells against gamma-radiation by a cytochrome c-independent signaling

108 ated in intestinal epithelia following 14 Gy gamma-radiation by Western blotting and immunohistochemi

109 matitidis, we demonstrate that resistance to gamma-radiation can be greatly increased through repeate

110 Same doses of gamma-radiation caused dramatic hair loss in wild-type m

111 reduced 53BP1 foci formation in response to gamma-radiation compared with cells expressing wild-type

112 n tumor latency, spectrum or frequency after gamma-radiation, compared to their control counterparts.

113 g human primary cells to continuous ionising gamma-radiation delivered at 6-20 mGy/h.

114 Gamma radiation did not affect the thermal properties of

115 tor tolerance induction and the selection of gamma radiation dose is critical for potential clinical

116 The total gamma-radiation dose accumulated by the fruits during th

117 00 MeV/nucleon) and results were compared to gamma radiation doses of 2 or 5 Gy, which are equitoxic

118 rial community was exposed to four different gamma radiation doses ranging from 0.46 to 3.96 kGy to t

119 ns, more than 90% of the OTA was degraded by gamma-radiation doses >/=2.5kGy, and a 2-fold reduction

120 HCl3, CH2Cl2) to enable instant detection of gamma radiation down to the 0.01 Gy level.

121 deficient fibroblasts were hypersensitive to gamma radiation, doxorubicin, and hydrogen peroxide and

122 en at the same position; and (3) both UV and gamma-radiation efficiently induce LOH at doses of radia

123 Gamma radiation, electron beam and X-rays have emerged a

124 an be labeled with 99mTc, a widely available gamma-radiation-emitting radionuclide, for intravenous i

125 es of same food component absorbed different gamma radiation energy though exposed to same radiation

126 Exposure of mice and HL-60 cells to gamma radiation enhanced the levels of NQO2, which led t

127 We observed that cells exposed to gamma radiation exhibited increased levels of L1 retrotr

128 r endothelial cells (HDMEC), when exposed to gamma-radiation, exhibited a time-dependent activation o

129 tivity during the D. radiodurans response to gamma radiation exposure are unknown.

130 vels of cyclin D1, we infer that relative to gamma radiation exposure to (56)Fe radiation induced mar

131 nt of resistance of E. dermatitidis to acute gamma-radiation exposure and the major mechanisms it use

132 at persisted in the mouse lens samples after gamma-radiation exposure increased with decreasing dose-

133 at the ability of E. dermatitidis to survive gamma-radiation exposure is determined by the prior and

134 Intriguingly, exposure of NQO1(-/-) mice to gamma-radiation failed to induce C/EBPalpha and Pu.1, as

135 serovar Typhimurium, were exposed to 25 kGy gamma radiation for complete sterilization.

136 millisievert/year (mSv/y)], larger levels of gamma radiation for the island of Rongelap (mean = 19.8

137 We find low levels of gamma radiation for the settled island of Enewetak [mean

138 First, what is the lowest dose of x- or gamma-radiation for which good evidence exists of increa

139 scatter of a combination of fast-neutron and gamma radiation from steel samples of a variety of thick

140 revealed a consistent pattern; Fe2+-EDTA and gamma-radiation generated MDA but not base propenals or

141 hnia magna, is affected by acute exposure of gamma radiation (GR) in combination with the polycyclic

142 untreated allogeneic splenic leukocytes; (3) gamma radiation group mice received gamma irradiated (2,

143 se rates were lower than those in the active gamma-radiation group and similar to those in the placeb

144 Ionising gamma radiation has been shown to reduce recurrence of r

145 Ionizing gamma radiation has several therapeutic indications incl

146 defective repair for both nonspecific DSBs (gamma-radiation hypersensitivity and genomic instability

147 hermore, these mutants are unable to reverse gamma-radiation hypersensitivity of BRCA1-null human bre

148 Twenty-five patients were treated with gamma radiation in a dose of 15 Gy, and 26 were treated

149 inococcus radiodurans following treatment by gamma radiation in an environment lacking nutrients.

150 Ps with or without PEGylation into mice; the gamma radiation in blood specimens and dissected organs

151 ): 1) to evaluate the actual distribution of gamma radiation in human in-stent restenosis (ISR) lesio

152 We examined the effects of intravascular gamma radiation in patients with in-stent restenosis of

153 ave shown the effectiveness of both beta and gamma radiation in preventing recurrent restenosis in pa

154 The core circadian genes are induced by gamma radiation in wild-type mice but not in mPer2 mutan

155 sence of Puma, HSCs were highly resistant to gamma-radiation in a cell autonomous manner.

156 of investigating the effects of exposure to gamma-radiation in a multistressor context.

157 umor latency, progression and lifespan after gamma-radiation in Atm heterozygous mice compared with t

158 obility and growth, responded to exposure to gamma-radiation in combination with the heavy metal cadm

159 ypt survival was also demonstrated following gamma-radiation in FVB/N mice rendered hypergastrinemic

160 arious inter-strand cross-linking agents and gamma-radiation in vitro.

161 mycin C, methyl methanesulfonate, and UV and gamma-radiation, indicating that mammalian pol zeta help

162 Gamma-radiation induced a BLM-regulated pathway that sel

163 Gamma-radiation induced browning inhibition in minimally

164 gamma-radiation induced the death of P. falciparum in a

165 ytes from patients with RA were resistant to gamma radiation-induced apoptosis, a process known to be

166 Gamma radiation-induced clustered DNA damage containing

167 uman and murine mast cells were resistant to gamma radiation-induced cytotoxicity and, importantly, t

168 In the current study, we showed that gamma radiation-induced expression of p21(Waf1/Cip1) in

169 m quiescence and regenerative potential upon gamma radiation-induced injury.

170 This contributed to the development of gamma radiation-induced myeloproliferative disease in NQ

171 Disruption of the NQO2 gene in mice leads to gamma radiation-induced myeloproliferative diseases.

172 helial cells expressing Bcl-2 also inhibited gamma-radiation-induced activation of p38 MAPK and p53 a

173 The median gamma-radiation-induced and benzo(a)pyrene diol epoxide-

174 LR9 engagement on murine CD4 T cells reduces gamma-radiation-induced apoptosis as judged by decreased

175 int kinase 1 (Chk1) is sufficient to restore gamma-radiation-induced apoptosis in p53 mutant zebrafis

176 n that LNCaP cells are entirely resistant to gamma-radiation-induced apoptosis, but can be sensitized

177 ryonic fibroblasts and osteosarcoma cells to gamma-radiation-induced apoptosis, with an increase in s

178 ion of pRb2/p130 increased the percentage of gamma-radiation-induced apoptotic cells from 27 to 47%.

179 ersion of virulence, and molecular basis for gamma-radiation-induced cell death in malaria parasites.

180 suggest a role for pRb2/p130 in glioblastoma gamma-radiation-induced cell death, indicating that the

181 inoblastoma family member pRb2/p130 enhances gamma-radiation-induced cell death.

182 pression levels in these cell lines; and (c) gamma-radiation-induced chromatid breaks were counted as

183 cid (LPA) and its analog, Radioprotein-1, on gamma-radiation-induced colonic epithelial barrier dysfu

184 embryonic fibroblasts from MMP-9-/- mice on gamma-radiation-induced damage of DNA.

185 reaks (DSBs) are formed during processing of gamma-radiation-induced DNA clustered damage sites.

186 reventing centrosome amplification following gamma-radiation-induced DNA damage and does so by transc

187 In response to gamma-radiation-induced DNA damage, organisms either act

188 important determinant of cell fate following gamma-radiation-induced DNA damage.

189 Next, gamma-radiation-induced G2 delay and apoptosis were test

190 We found a dose-response relationship for gamma-radiation-induced G2 delay and apoptosis.

191 Therefore, we concluded that gamma-radiation-induced G2 delay, apoptosis, p53 increas

192 ontrols, we investigated the relationship of gamma-radiation-induced G2-M arrest and lung cancer risk

193 ion and hospital controls), a lower level of gamma-radiation-induced G2-M arrest was associated with

194 gamma-Radiation-induced G2-M arrest was measured as the

195 The mean percentage of gamma-radiation-induced G2-M arrest was significantly lo

196 cond, wortmannin treatment strongly inhibits gamma-radiation-induced hyperphosphorylation and foci fo

197 The median gamma-radiation-induced increases of cells in the S and

198 Also observed were joint effects between gamma-radiation-induced increases of S and G(2) phase fr

199 In addition, exposure of cells to gamma-radiation induces MRK activity.

200 lly expressed, wild-type BRCA1 decreased the gamma radiation (IR) sensitivity and increased the effic

201 Using a gamma radiation (IR)-induced replication stress T-cell l

202 Gamma radiation is a commonly used adjuvant treatment fo

203 The use of gamma radiation is a technique for preserving food that

204 This study demonstrates that chronic LDR gamma radiation is genotoxic in an exposure scenario rea

205 mune cells, susceptibility of lymphocytes to gamma radiation is well known.

206 we find that greatly increased resistance to gamma-radiation is achieved in E. dermatitidis through d

207 Although pathogen inactivation by gamma-radiation is an attractive approach for whole-orga

208 Gamma-radiation is linked to large-scale structural vari

209 tients with in-stent restenosis treated with gamma-radiation is well tolerated and associated with a

210 posure of mice and HL-60 cells to 3 Grays of gamma-radiation led to increased NQO1 that stabilized C/

211 suggested that exposure of NQO1(-/-) mice to gamma-radiation led to myeloproliferative disease.

212 Distributions of gamma radiation levels are provided, and hot spots are d

213 External gamma radiation levels on Bikini Island significantly ex

214 We report measurements of background gamma radiation levels on six islands in the northern Ma

215 Our results show low external gamma radiation levels on some islands in the Enewetak A

216 eed this standard (P = <<0.01), and external gamma radiation levels on the other islands are below th

217 ans, supporting the hypothesis that even LDR gamma radiation may induce cancer.

218 We perform ordinary kriging on external gamma radiation measurements to provide interpolated map

219 Bcl-2 level and decreased etoposide- and UV/gamma radiation-mediated DNA fragmentation.

220 (N-methyl-N'-nitro-N-nitrosoguanidine), and gamma radiation, none of which resulted in a decrease in

221 tion (TLR) and angiographic restenosis after gamma radiation of restenotic lesions.

222 a high-fat diet, exposed to low-dose (60)Co gamma-radiation of 25 mGy at 2 mo of age, and evaluated

223 Moreover, LC-MS/MS results showed that gamma-radiation of d(GT) under anaerobic condition yield

224 We report on measurements of external gamma radiation on 9 islands in 4 atolls in the northern

225 detailed sequence analysis of the effects of gamma radiation on an entire human chromosome, which giv

226 A damage in vivo, we compared the effects of gamma radiation on DNA synthesis on whole-body sections

227 here is little information on the effects of gamma radiation on mast cells, which are important in bo

228 8 mrem/y = 0.198 mSv/y), and relatively high gamma radiation on the island of Bikini (mean = 184 mrem

229 ygous mice were exposed to 7.5 Gy of (137)Cs-gamma radiation on their right sides, and Aprt-deficient

230 ascular ultrasound to evaluate the effect of gamma-radiation on recurrent in-stent restenosis.

231 synthesis was inhibited 24 h after UV light, gamma radiation or DNA cross-linking by cisplatin in hum

232 Activation of IKKbeta by gamma radiation or tumor necrosis factor-alpha led to in

233 gh-level ULBP1 expression was not induced by gamma radiation or UV radiation.

234 either a 700 cGy dose of Cobalt-60 ((60)Co) gamma-radiation or 600 cGy, 250 kVp x-irradiation.

235 e significantly more resistant to killing by gamma-radiation or 9- amino camptothecin than were cells

236 response to treatment with dexamethasone or gamma-radiation or in response to anti-CD3/anti-CD28 sti

237 PI and nontoxic doses of the ROS generators, gamma-radiation or t-butyl-hydroperoxide, attenuated the

238 M) at DNA damage sites in cells treated with gamma-radiation or the radiomimetic drug neocarzinostati

239 eated cells and in cells treated with either gamma-radiation or ultraviolet (UV) radiation.

240 ltured cells to survive after treatment with gamma-radiation or with the topoisomerase-I inhibitor to

241 lls exposed to low doses of alpha radiation, gamma radiation, or chemical mutagens in the presence an

242 Four days following 14 Gy gamma-radiation, or 2 injections of 400 mg/kg 5-fluorour

243 o densely ionizing 350 MeV/amu Si-particles, gamma-radiation, or sham-irradiated and transplanted 3 d

244 d soil microcosms to weekly bursts of (60)Co gamma radiation over six weeks, at three levels of expos

245 60Co gamma-radiation produces a 1.8-fold increase in the yiel

246 link between Ub protein ligase activity and gamma-radiation protection function of BRCA1, and provid

247 Intracoronary gamma-radiation reduces recurrent in-stent restenosis (I

248 Intracoronary gamma-radiation reduces recurrent in-stent restenosis.

249 itizes radioresistant LNCaP and PC3 cells to gamma radiation, regardless of the status of p53.

250 cal sensor systems for low dose detection of gamma radiation remains highly desired for medical radia

251 Precise detection of low-dose X- and gamma-radiations remains a challenge and is particularly

252 th cytokine-independent survival and partial gamma-radiation resistance.

253 and apoptotic index were 10 h and 48 h after gamma-radiation, respectively.

254 ort also shows continuously recorded (137)Cs gamma radiation response of a unidirectionally-biased pi

255 Exposure of NQO1(-/-) mice to gamma-radiation resulted in reduced apoptosis in granulo

256 10 microM HA14-1 for 1 h followed by 1-6 Gy gamma radiation, resulted in a highly synergistic (combi

257 Gamma-radiation results in cell cycle arrest and apoptos

258 Parallel studies with gamma-radiation revealed levels of MDA similar to those

259 e 1 null (NQO1(-/-)) mice exposed to 3 Gy of gamma-radiation showed an increase in neutrophils, bone

260 These data suggest that MRK may mediate gamma-radiation signaling leading to cell cycle arrest a

261 eutron backscatter and the Compton-scattered gamma radiations, simultaneously.

262 N and HC proved to be extremely resistant to gamma-radiation such that significant killing was observ

263 Cells that are the most resistant to gamma radiation, such as Nrf2 gain-of-function mutant ce

264 r, many fungi manifest extreme resistance to gamma-radiation, such that the doses of several thousand

265 hypersensitive to the cytostatic effects of gamma radiation, suggesting that NHEJ is indeed a critic

266 bserve a dramatic transcriptomic response to gamma-radiation that mobilizes pathways involved in morp

267 e to survive for a long term after high-dose gamma-radiation that normally would pose 100% lethality

268 When exposed to gamma radiation, the halogenated solvents decompose into

269 esistant to the growth inhibitory effects of gamma radiation, the sog1 mutation affects the proper de

270 th the cytotoxic drug 5-fluorouracil or with gamma-radiation, the bcl-w-null animals exhibited substa

271 Under gamma-radiation, the hydrogen bonds are cleaved, resulti

272 gamma-Radiation therapy can effectively prevent recurren

273 The results of our study support the use of gamma-radiation therapy for the treatment of in-stent re

274 Intracoronary gamma-radiation therapy reduces recurrent in-stent reste

275 We studied the effects of intracoronary gamma-radiation therapy versus placebo on the clinical a

276 For gamma-radiation, there was a 0.99 correlation between th

277 After gamma radiation, these mice show a marked increase in tu

278 and thrombosis rates were compared with the gamma-radiation-treated (n=125) and the placebo patients

279 res minus the percentage of mitotic cells in gamma-radiation-treated cultures from the same subject.

280 The suitability of post-packaging gamma radiation treatment for preserving total folates o

281 st after transforming growth factor-beta and gamma-radiation treatment.

282 ma-radiation, and harvested at 3 hours after gamma-radiation treatment.

283 h ISR treated with ICRT who were enrolled in gamma radiation trials.

284 Here we show that DNA damage induced by gamma-radiation triggers the phosphorylation of nuclear

285 s specifically phosphorylated in response to gamma-radiation, ultraviolet light and exposure to hydro

286 Intracoronary gamma-radiation used as adjunct therapy for patients wit

287 ent stent implantation and were treated with gamma-radiation using 192Ir.

288 hese data demonstrates that genes induced by gamma radiation, UV radiation, and the zinc-induced p53

289 The cellular p53 response to gamma radiation was also assessed by immunoblotting.

290 samples were collected from mice exposed to gamma radiation was analyzed.

291 s stabilization of TAp63gamma in response to gamma radiation was significantly decreased in the absen

292 In food matrices, the elimination of OTA by gamma-radiation was found more difficult, as radiation d

293 ypt survival in INS-GAS mice following 14 Gy gamma-radiation was inhibited by administration of a CCK

294 y alterations, their downstream responses to gamma radiation were studied in vitro.

295 tly more efficient in killing CN and HC than gamma-radiation when based on the mean absorbed dose to

296 lease histamine in response to high doses of gamma radiation, whereas other reports suggest that mast

297 ion of human breast and lung cancer cells to gamma radiation, which was further enhanced by Rad001.

298 early clinical benefits after intracoronary gamma radiation with 192Ir seem durable at 5-year clinic

299 cer because it simultaneously emits beta and gamma radiations with proper energy and half-life; there

300 y randomized to receive either intracoronary gamma-radiation with (192)Ir (15 Gy) or placebo.