ライフサイエンスコーパス: ganglia

1 dbrain boundary, spinal cord and dorsal root ganglia.

2 o calcium mobilization in murine dorsal root ganglia.

3 ent, giving off varicose endings in multiple ganglia.

4 ifelong latent infection in peripheral nerve ganglia.

5 s in receipt of inputs from the nodose vagal ganglia.

6 onditions associated with the mPFC and basal ganglia.

7 r 90% of latent virus from superior cervical ganglia.

8 nces in neuronal gene expressions in sensory ganglia.

9 systems residing in the brain, in our basal ganglia.

10 tal trajectories of tissue iron in the basal ganglia.

11 y the corresponding systems within the basal ganglia.

12 elated and motivational signals in the basal ganglia.

13 neurons form a key network within the basal ganglia.

14 on of tissue iron concentration in the basal ganglia.

15 s learning and action selection in the basal ganglia.

16 n the striatum, the central hub of the basal ganglia.

17 , yet an understudied component of the basal ganglia.

18 in sensory, sympathetic and parasympathetic ganglia.

19 the distal colon is devoid of enteric neural ganglia.

20 ine nerve cell bodies were labeled in nodose ganglia.

21 ptosis and microglial activation in infected ganglia.

22 entrally positioned network within the basal ganglia.

23 tum is the main input structure of the basal ganglia.

24 d in the dorsal root, autonomic, and enteric ganglia.

25 ry neurons of the dorsal root and trigeminal ganglia.

26 rebral cortex, the cerebellum, and the basal ganglia.

27 ntion-related visual processing in the basal ganglia.

28 es are the target cells for EBOV in affected ganglia.

29 50-100 nl) were made into L3-L5 dorsal root ganglia.

30 esses cFos expression in the gut sympathetic ganglia.

31 profiling, that localized to either TL or LS ganglia.

32 ing white matter-enhancing lesions and basal ganglia abnormalities that could be related to severe ac

33 Basal ganglia abnormalities were seen in four other patients (

34 e permitted or triggered by changes in basal ganglia activity through gating- or rebound-like mechani

35 normal and pathological patterning of basal ganglia activity.

36 Within trigeminal ganglia, afferents innervating craniofacial muscles inte

37 pa1(+)EECs directly stimulates vagal sensory ganglia and activates cholinergic enteric neurons by sec

38 Human myenteric ganglia and adjacent smooth muscle were isolated by lase

39 sal premotor cortex, as well as in the basal ganglia and anterior cerebellum.

40 RT(+) neurons send axons to the prevertebral ganglia and are polysynaptically connected to the liver

41 (learning and memory that rely on the basal ganglia and associated circuitry) can explain numerous b

42 direct pathways running from cortex to basal ganglia and between basal ganglia and cerebellum in the

43 te that a population of neurons in the vagal ganglia and brainstem are activated via the gut-brain ax

44 Hypomyelination with atrophy of the basal ganglia and cerebellum (H-ABC) is a neurodegenerative di

45 luding Hypomyelination with Atrophy of Basal Ganglia and Cerebellum (H-ABC), a rare hypomyelinating l

46 om cortex to basal ganglia and between basal ganglia and cerebellum in the pathophysiology of movemen

47 ionship between neuronal firing in the basal ganglia and cortical gamma activity during movement, we

48 xpression of Tbx3 from the SAN and (cardiac) ganglia and in neonatal lethality.

49 btype 2 showed increased volume in the basal ganglia and internal capsule, and otherwise normal brain

50 and stable anatomy, except for larger basal ganglia and internal capsule, not explained by antipsych

51 expression patterns and found that the basal ganglia and medium spiny neurons were most enriched for

52 transcriptional profiling of human myenteric ganglia and mouse EN provides a rich foundation for deve

53 perdirect and indirect pathways of the basal ganglia and movement control.

54 d patients demonstrate the role of the basal ganglia and other interconnected structures, such as the

55 with blunted reward activation in the basal ganglia and other regions such as the medial prefrontal

56 he frontal and occipital white matter, basal ganglia and pons was used to obtain a global cerebral sm

57 demonstrate that EBOV can infect peripheral ganglia and results in ganglionopathy in rhesus macaques

58 synaptic vagal sensory input from the nodose ganglia and spinal sensory input from the dorsal horn.

59 y and multi-network integration in the basal ganglia and thalamus of individual human subjects.

60 , myelin and iron concentration of the basal ganglia and thalamus were estimated from 182 MRI dataset

61 ize the functional interaction between basal ganglia and thalamus, we demonstrated that patients with

62 both via ascending projections to the basal ganglia and through descending projections to brainstem

63 of 132 colon afferents (from NG, TL, and LS ganglia) and 128 bladder afferents (from TL and LS gangl

64 be, the diencephalon, cerebral cortex, basal ganglia, and cerebellum.

65 tions of nervous system white matter tracts, ganglia, and nerves, and an enhanced series of 10 flatma

66 nerve endings in circular muscle, myenteric ganglia, and submucosa.

67 neurons innervated mucosal crypts, myenteric ganglia, and submucosa.

68 sterior cingulate and parietal cortex, basal ganglia, and temporal cortex.

69 The basal ganglia are a collection of nuclei in vertebrates that e

70 The basal ganglia are a group of subcortical nuclei that contribut

71 SGCs in sensory ganglia are activated by numerous types of nerve injury

72 of beta frequency band activity in the basal ganglia are associated with slowing of voluntary movemen

73 The basal ganglia are implicated in a range of perceptual function

74 The basal ganglia are important for movement and reinforcement lea

75 decision processes and learning in the basal ganglia are modulated by the motivation.

76 n slices we show that within each bird basal ganglia Area X-projecting (HVC(X)) neurons share similar

77 nnected with the prefrontal cortex and basal ganglia, areas which have been implicated in interval ti

78 as used to show distal colon without enteric ganglia, as well as a transition zone and proximal pull-

79 ns whose cell bodies are located in multiple ganglia associated with the brainstem and spinal cord.

80 By profiling sensory ganglia at single-cell resolution, we find that all soma

81 -specific CD8(+) T cells that infiltrate the ganglia at the onset of latency and contract to a stable

82 einforcement learning models treat the basal ganglia (BG) as an actor-critic network.

83 treatments that decrease pathological basal ganglia (BG) beta oscillations (10-17 Hz in primates), s

84 nigra pars reticulata (SNr), where the basal ganglia (BG) direct and indirect pathways converge, cont

85 PD are generally attributed to altered Basal Ganglia (BG) function.

86 behavioral protocol to understand how basal ganglia (BG) is affected by cannabinoids.

87 As a rodent basal ganglia (BG) output nucleus, the substantia nigra pars r

88 In the basal ganglia (BG), anatomically segregated and topographicall

89 d in the neocortex and cortical lobes, basal ganglia (BG), hippocampi, and thalami.

90 ticulata (SNr), the main output of the basal ganglia, blocks signaled active avoidance, while inhibit

91 s. 32%, 24%, and 38% with (68)Ga-PSMA-11 for ganglia, bone, and unspecific lymph nodes, respectively)

92 he hypothesis that pulsatile output from the ganglia can synchronize islet behavior.

93 Most of our images were of myenteric ganglia, captured using a 20x objective lens.

94 cated in the following areas: level of basal ganglia (caudate nucleus, putamen, corpus callosum, post

95 In contrast, some basal ganglia, cerebellar, and limited cortical areas showed e

96 reated HIV, except a MRS alteration in basal ganglia choline.

97 Therefore, the M2-basal ganglia circuit is critical for the assembly of the moto

98 Within the basal ganglia circuit, the external globus pallidus (GPe) is c

99 us (GPe) is a critical node within the basal ganglia circuit.

100 manner as the hyperdirect and indirect basal ganglia circuitry.

101 y in HVC and occurred independently of basal ganglia circuitry.

102 glutamatergic transmission in cortico-basal ganglia circuits and represents a major target of L-DOPA

103 ial attention, then the involvement of basal ganglia circuits should incorporate the subject's expect

104 critically relies on frontal cortex - basal ganglia circuits.

105 g cognitive, motor, and limbic cortico-basal ganglia circuits.

106 ng prefrontal, anterior cingulate, and basal ganglia connections linked to the limbic system.

107 on for these diverse roles is that the basal ganglia control the level of commitment to particular mo

108 ) developed T1 hyperintensities of the basal ganglia, corresponding to accumulated lipid phagocytes o

109 ephalic signaling center important for basal ganglia development known in other vertebrates (i.e., th

110 eases are associated with dysregulated basal ganglia direct-pathway activity.

111 PNs) are thought to contribute to many basal ganglia disorders, including early-onset neurodevelopmen

112 sing in-situ contact angle measurements, oil ganglia distribution analysis, and three-dimensional vis

113 D and Dystonia are likely amplified by basal ganglia downstream structures.

114 m in molecular signatures of the dorsal root ganglia (DRG) and spinal cord response, not observed at

115 lin-dependent kinase 5 (Cdk5) in dorsal root ganglia (DRG) and spinal dorsal horn.

116 tion of VIP in the corresponding dorsal root ganglia (DRG) and the dorsal horn of the spinal cord.

117 h PC12 cells and chick embryonic dorsal root ganglia (DRG) bodies, as well as the migration of Schwan

118 The dorsal root ganglia (DRG) contain the somas of first-order sensory n

119 s assessed by von Frey assay and dorsal root ganglia (DRG) expression of Calca and Tac1 genes.

120 The dorsal root ganglia (DRG) house the primary afferent neurons respons

121 Resident GFAP(+) glia in dorsal root ganglia (DRG) known as satellite glial cells (SGCs) pote

122 protein levels in fibroblasts in dorsal root ganglia (DRG) meninges and in the epi/perineurium of the

123 erived exosomes communicate with dorsal root ganglia (DRG) neurons.

124 ases the regenerative ability of dorsal root ganglia (DRG) sensory neurons compared to EE or a condit

125 drives pathologic changes in the dorsal root ganglia (DRG) through inflammation, altered metabolism,

126 in primary culture and in mouse dorsal root ganglia (DRG), as determined by the characteristic senes

127 :LUC fusion protein, we isolated dorsal root ganglia (DRG), the primary sensory cell body for periphe

128 In dorsal root ganglia (DRG), there is an increase in CGRP(+), TH(+), a

129 Dorsal root ganglia (DRG), which contain the somata of primary senso

130 ent neurons, with cell bodies in dorsal root ganglia (DRG).

131 y receptor alpha1 (GFRalpha1) in dorsal root ganglia (DRG).

132 round injured sensory neurons in dorsal root ganglia (DRG).

133 e predominantly in thoracolumbar dorsal root ganglia (DRG).

134 ifelong latent infections in the dorsal root ganglia (DRG).

135 alpha6beta4, an nAChR subtype in dorsal root ganglia (DRG).

136 undles, and the transcriptome of dorsal root ganglia (DRGs) provide possible explanations for the inc

137 Transcriptome profiling of dorsal root ganglia (DRGs) revealed 138 differentially regulated gen

138 the distal nerve and axotomized dorsal root ganglia (DRGs).

139 TRPC4 is highly expressed in dorsal root ganglia (DRGs).

140 f tissue iron concentration across the basal ganglia during adolescence and provide evidence that dim

141 e increase in beta oscillations in the basal ganglia during sleep paralleled decreased NREM sleep, in

142 d directed medial prefrontal cortex to basal ganglia effective connectivity is abnormally increased o

143 Basal ganglia encephalitis (BGE), representing a subset of AE

144 The basal ganglia, especially the caudate nucleus 'head' (CDh) of

145 There were no changes in nodose ganglia excitability in TNX deficient mice, suggesting t

146 VANs, located in nodose ganglia, express receptors for various gut-derived pepti

147 nd executive function, mediated by the basal ganglia, extended amygdala, and frontal cortex, respecti

148 hat establish lifelong latency in peripheral ganglia following the initial infection at mucosal surfa

149 modes of information processing in the basal ganglia for different motor and nonmotor functions and o

150 erminating in the celiac-superior mesenteric ganglia form varicose-like structures surrounding indivi

151 the basal ganglia in PD and models of basal ganglia function and dysfunction were proposed.

152 GNIFICANCE STATEMENT Current models of basal ganglia function are often based on a distinction of two

153 ys provides striking predictions about basal ganglia function that have been used to develop deep bra

154 n the striatum play a critical role in basal ganglia function, such as reinforcement and motor learni

155 amine, both of which are essential for basal ganglia function.

156 of either sex, we found that the main basal ganglia GABAergic output in the midbrain, the substantia

157 wever, is increased motor cortical and basal ganglia gamma synchrony.

158 my of the medial prefrontal cortex and basal ganglia has been extensively studied and the former has

159 ntral pallidum, located in the ventral basal ganglia, has long been recognized as an obligatory node

160 While autonomic ganglia have been extensively studied in rats instead of

161 The basal ganglia have been implicated in action selection and tim

162 While prefrontal cortex and basal ganglia have been implicated in interval timing in the s

163 protein with age in several cortices, basal ganglia, hippocampus, and midbrain, a decrease with age

164 ndomly distributed among neuronal subsets in ganglia, implying that improved delivery to all neuronal

165 e white matter in children, and in the basal ganglia in adults.

166 es of patients with Hirschsprung disease had ganglia in multiple layers and thick nerve fiber bundles

167 of potential volumetric changes of the basal ganglia in patients with PD who underwent staged STN DBS

168 iological changes that occurred in the basal ganglia in PD and models of basal ganglia function and d

169 Recent work has implicated the primate basal ganglia in visual perception and attention, in addition

170 fibrils (PFFs) into the stellate and celiac ganglia induces spreading of alpha-Syn pathology only th

171 he integration of limbic, sensory, and basal ganglia information to guide effective response strategi

172 firing rate for understanding cortico-basal ganglia information transfer.

173 uggest that the broken balance between basal ganglia inhibition and thalamus synchronization can info

174 xamine this hypothesis, rats received nodose ganglia injections of an adeno-associated virus (AAV) ex

175 Few neurons in nodose ganglia innervate the uterus.

176 Cortico-basal ganglia interactions continuously shape the way we move.

177 ontocerebellar Hypoplasia with typical basal ganglia involvement on neuroimaging.

178 normative developmental trajectory of basal ganglia iron concentration during adolescence and its as

179 results suggest a prolonged period of basal ganglia iron enrichment that extends into the mid-twenti

180 Atypical iron concentration in the basal ganglia is associated with neurodegenerative disorders i

181 of herpes simplex virus 1 in the trigeminal ganglia, leading to dissemination of virus to, and infec

182 ec; right, 1.15 x10(3)mum(2)/sec), and basal ganglia (left, 1.26 x10(3)mum(2)/sec; right, 1.23 x10(3)

183 +/- 0.32; right, 1.45 years +/- 0.33), basal ganglia (left, 1.79 years +/- 0.31; right, 1.70 years +/

184 ed inflammatory processes in the dorsal root ganglia, likely by activating stress-response proteins,

185 nectivity between the habenula and the basal ganglia, limbic, and sensory systems was already present

186 Viscera receive innervation from sensory ganglia located adjacent to multiple levels of the brain

187 r excess activity of inhibitory corticobasal ganglia loops is unclear.

188 demonstrate that activity through the basal ganglia may play an important and distinct role among th

189 iatum is the main input nucleus of the basal ganglia, mediating motor and cognitive functions.

190 sease, leading to abnormal function of basal ganglia motor circuits and the accompanying characterist

191 PSD-95 was reduced in the brainstem, basal ganglia, neocortex, and cerebellum within 13 dpi, indica

192 action or inhibition recruit a fronto-basal-ganglia network just like stopping.

193 nogo signal recruited the same fronto-basal-ganglia network which is usually assigned to stopping.

194 like global stopping recruit a fronto-basal-ganglia network.

195 y of actions can be implemented in the basal ganglia network.

196 ous findings on the essential role for basal ganglia networks in absence seizures, in particular the

197 beta bursting, both locally and across basal ganglia networks, impacts on motor performance in this c

198 beta bursting, both locally and across basal ganglia networks, may impact on motor performance.SIGNIF

199 promise information coding capacity in basal ganglia networks.

200 tion linked to abnormalities in corticobasal ganglia networks.

201 l methods to detail the progression of basal ganglia neuron type-specific pathology and the deficits

202 receptor (nAChR) is enriched in dorsal root ganglia neurons and is an attractive non-opioid therapeu

203 Jugular ganglia neurons wire into a central circuit that is nota

204 ato mice label 80% of nodose and dorsal root ganglia neurons.

205 Early in this pathway, in the lateral line ganglia, neurons respond almost exclusively to the simpl

206 acolumbar (TL), lumbosacral (LS), and nodose ganglia (NG) in male and female mice.

207 Dorsal root ganglia nociceptors protect against STm colonization, in

208 The thalamus also receives inputs from basal ganglia nuclei (BG) involved in value-based decision mak

209 ontrol the flow of information through basal ganglia nuclei that eventually project back to the mPFC

210 und that beta dynamics differed across basal ganglia nuclei.

211 Beta dynamics therefore differ across basal ganglia nuclei.

212 Myenteric ganglia occupied 34% +/- 4% of myenteric plexus.

213 ubstance P and S100beta density in myenteric ganglia of HFD mice were increased at week 8, but not at

214 ntribute to the pain state in the trigeminal ganglia of injured mice.

215 ASYMP HLA transgenic rabbits, the trigeminal ganglia of non-protected SYMP HLA transgenic rabbits had

216 of beta frequency band activity in the basal ganglia of patients with Parkinson's disease (PD) are as

217 tures of a cell bridge linking two autonomic ganglia of the neck, namely, the nodose ganglion (NG) an

218 ns improved with lesions involving the basal ganglia or thalamus.

219 iesterase 10A (PDE10A) activity in the basal ganglia orchestrates the control of coordinated movement

220 Herein, we generate dorsal root ganglia organoids (DRG organoids) by in vitro differenti

221 ed single-unit activity from connected basal ganglia output and thalamic nuclei (globus pallidus-inte

222 ergence, illuminating how synchrony of basal ganglia output during motor learning or in pathological

223 he ventral thalamus, a major target of basal ganglia output, is often assumed to be permitted or trig

224 and resulted in reduced direct pathway basal ganglia output.

225 e impact of the subthalamic nucleus on basal ganglia output; then, at ~160 ms, suppression was detect

226 nal medulla or in sympathetic, paravertebral ganglia outside the medulla.

227 was sympathetic hyperinnervation in stellate ganglia (p = 0.02) but not ventricles (p = 0.2) of PVC-C

228 Further study of basal ganglia pathophysiology is required to better understand

229 ting this complex balancing act is the basal ganglia pathway, but its roles have not yet been examine

230 d upon the direct but not the indirect basal ganglia pathway.

231 The basal ganglia play an important role in decision making and se

232 o the subthalamic nucleus (STN) of the basal ganglia, play a key role in inhibiting impulsive respons

233 Neural ganglia regeneration and structure, bowel motility, epit

234 ignals in the ventral pallidum (VP), a basal ganglia region functionally linked to reward-seeking beh

235 Basal ganglia regions may be relatively preserved in TS owing

236 tion using R2* relaxometry within four basal ganglia regions, including the caudate, putamen, nucleus

237 cross patients, but not in cortical or basal ganglia regions.

238 tween the medial prefrontal cortex and basal ganglia related to depression.

239 linergic stimulation of islets by pancreatic ganglia resets these endocrine units, producing synchron

240 ly, explant of latently infected dorsal root ganglia revealed a decreased and delayed reactivation ph

241 bal transcriptional profiling of dorsal root ganglia revealed differential expression, notably in reg

242 and spinal cord glia as well as dorsal root ganglia satellite glia have been identified important- i

243 ch project to the celiac-superior mesenteric ganglia, significantly increase splenic nerve activity a

244 We also observed that oil ganglia size was reduced under tertiary mode of low sali

245 Ganglia-specific transcriptional profiles were identifie

246 ocomotion by releasing dopamine in the basal ganglia, spinal networks, and the mesencephalic locomoto

247 related to the smaller volume of some basal ganglia structures.

248 plex virus 1 (HSV-1) from neurons in sensory ganglia such as the trigeminal ganglia (TG) is influence

249 The nodose and jugular vagal ganglia supply sensory innervation to the airways and lu

250 ortex with reward prediction errors in basal ganglia support exploration of latent task representatio

251 The temporal evolution of cortical and basal ganglia synchronization is cell type-selective, which co

252 The current model of the basal ganglia system based on the 'direct', 'indirect' and 'hy

253 eta band, restrict the capacity of the basal ganglia system to encode physiologically relevant inform

254 s providing a novel perspective on the basal ganglia system.

255 In isolated ganglia, T cells showed phase-dependent IPSPs during dop

256 rus during acute infection in the trigeminal ganglia (TG) and brain stem compared to the control-vacc

257 cavities, sensory neurons within trigeminal ganglia (TG) are an important site for latency.

258 ns in sensory ganglia such as the trigeminal ganglia (TG) is influenced by virus-specific CD8(+) T ce

259 ) in naturally VZV-infected human trigeminal ganglia (TG).

260 latency is sensory neurons within trigeminal ganglia (TG).

261 oaches and ganglion types [DRG vs trigeminal ganglia (TG)].

262 re demonstrated bilaterally within the basal ganglia, thalami, corpus callosum, occipital, temporal,

263 tion and iron concentration within the basal ganglia-thalamic circuit over 2 years post-SCI.

264 nd reorganizational changes across the basal ganglia-thalamic circuitry occur early after SCI and pro

265 ally increased beta bursts in cortical-basal ganglia-thalamic circuits are associated with rigidity a

266 ated within dopamine-modulated cortico-basal ganglia-thalamic circuits in schizophrenia.

267 o-thalamic connectivity within cortico-basal-ganglia-thalamic circuits.

268 Thus, in the healthy state, basal ganglia-thalamic communication during learned movement i

269 ) is a critical component of a cortico-basal ganglia-thalamo-cortical loop regulating limbic and cogn

270 PD), gamma oscillatory activity in the basal ganglia-thalamo-cortical network is altered and the LTP-

271 gamma oscillations are altered in the basal ganglia-thalamo-cortical network.

272 We will then describe the basal ganglia-thalamocortical circuit, the major locus of PD-r

273 a key component of the limbic cortico-basal ganglia-thalamocortical loop.

274 tive of the interactive pathways among basal ganglia, thalamus and cortex, to explore the imprinting

275 l lesions found on neuroimaging in the basal ganglia, thalamus, and brainstem and by a loss of motor

276 The basal ganglia, thalamus, and cerebral cortex form an interconn

277 ronization in the insular, cerebellum, basal ganglia, thalamus, operculum, frontoparietal cortices, a

278 nt hippocampal inhibition of amygdala, basal-ganglia, thalamus, orbital frontal cortex, inferior fron

279 changes in interactive profiles of the basal ganglia-thalamus network in the current history group ma

280 th the thalamocortical network and the basal ganglia-thalamus network with resting state functional M

281 to the classic indirect pathway of the basal ganglia that also targets the STN.

282 show that a major input station of the basal ganglia, the caudate nucleus, plays a causal role in int

283 In the primate basal ganglia, the caudate tail (CDt) encodes the historical v

284 These include the striatum of the basal ganglia, the dorsolateral prefrontal cortex (DLPFC), the

285 Within the basal ganglia, the globus pallidus pars externa (GPe) has been

286 Atypical basal ganglia tissue iron levels have been linked to impaired

287 PTGER2 (also called EP2) in the dorsal root ganglia to promote visceral hypersensitivity.

288 Despite clear evidence linking the basal ganglia to the control of outcome insensitivity (i.e., h

289 d worsening was identified in post-ATI basal ganglia total choline MRS, suggesting an alteration in n

290 n and pain are found in afferents across all ganglia types, suggesting that conscious sensation and h

291 benign lesions most frequently observed were ganglia, unspecific lymph node, and bone lesions, at a r

292 gray matter as well as hippocampal and basal ganglia volumes in NMDARE children.

293 uronal tracer (DiI) and dye uptake in nodose ganglia was examined.

294 In this study, peripheral ganglia were collected from 12 rhesus macaques that succ

295 a) and 128 bladder afferents (from TL and LS ganglia) were analyzed.

296 role in this process is played by the basal ganglia, where neural activity and plasticity are modula

297 ugh their ascending projections to the basal ganglia, which in turn project to the mesencephalic loco

298 lso expressed strongly in the adult thoracic ganglia while sema1a.1 was only weakly expressed and ple

299 lly or caudally along many rows of myenteric ganglia with little circumferential displacement, giving

300 ngs together models of learning in the basal ganglia with the incentive salience theory in a single s