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1 xpressing cells around and within the spiral ganglion.
2 ve primary sensory neurons in the trigeminal ganglion.
3 than those from neurons at the border of the ganglion.
4 tsynaptic ascending neurons in the abdominal ganglion(5).
5 administration, increases in GP and stellate ganglion activity and blood pressure during apnea were a
6 n desaturation, a tonic increase in stellate ganglion activity and blood pressure ensued.
7                 Increasing superior cervical ganglion activity by activating Gq-coupled designer rece
8   These include specific subtypes of retinal ganglion and horizontal cells, suggesting that in this c
9 ptor, horizontal, bipolar, amacrine, retinal ganglion and non-neuronal cells.
10 in Magel2, a PWS gene, within the trigeminal ganglion and regions that are anatomically relevant to f
11 riginate in the periphery, where dorsal root ganglion and trigeminal ganglion neurons feed pain infor
12  smaller proportion of nitrergic neurons per ganglion, and reduced markers of neurogenesis compared w
13 ic activity in the crustacean stomatogastric ganglion, and use these results to derive hypotheses for
14    Processes from neurons in the rear of the ganglion are more directed and grow faster than those fr
15 ratus, we demonstrate that chick dorsal root ganglion axons exhibit a tension buffering or strain-sof
16 essing, intrinsically photosensitive retinal ganglion cell (ipRGC) subtypes are two RGC types that ar
17           Early progression involves retinal ganglion cell (RGC) axon dysfunction that precedes frank
18 action of Xenopus tadpoles, a single retinal ganglion cell (RGC) axon misprojects to the ipsilateral
19          Binocular vision depends on retinal ganglion cell (RGC) axon projection either to the same s
20 growth factor-1 (IGF1) by initiating retinal ganglion cell (RGC) axon regeneration after axotomy.
21                                  The retinal ganglion cell (RGC) competence factor ATOH7 is dynamical
22 deficits, which are characterized by retinal ganglion cell (RGC) death and ON degeneration.
23 ieved to be the major contributor to retinal ganglion cell (RGC) death, the endpoint of optic neuropa
24         Optic atrophy resulting from retinal ganglion cell (RGC) degeneration is a prominent ocular m
25 describes a novel paradigm to reduce retinal ganglion cell (RGC) degeneration underlying glaucoma.
26 nto cone bipolar cell (BC) axons and retinal ganglion cell (RGC) dendrites, but makes the majority of
27 of intraocular pressure (IOP) causes retinal ganglion cell (RGC) dysfunction and death and is a major
28 sion protein optic atrophy 1 (Opa1), retinal ganglion cell (RGC) dysfunction and visual loss occur by
29     The visual message conveyed by a retinal ganglion cell (RGC) is often summarized by its spatial r
30 mber of Nissl-stained neurons in the retinal ganglion cell (RGC) layer in the Caribbean and Chilean f
31 has previously been used to stratify Retinal Ganglion Cell (RGC) populations in histological samples
32  rodents, Rbfox2 is expressed in all retinal ganglion cell (RGC) subtypes, horizontal cells, as well
33 erged in which the three most common retinal ganglion cell (RGC) types captured much of the variance
34 l information is encoded in distinct retinal ganglion cell (RGC) types in the eye tuned to specific f
35                             Multiple retinal ganglion cell (RGC) types in the mouse retina mediate pa
36 tablished role in the development of retinal ganglion cell (RGCs) types, the main transducers of visu
37 cone -> midget bipolar interneuron -> midget ganglion cell (the "private line").
38 rod photoreceptor (~80,000 rods mm(-2) ) and ganglion cell (~1,800 cells mm(-2) ) densities across th
39 etinal nerve fiber layer (pRNFL) and macular ganglion cell + inner plexiform layer (GCIPL) thinning i
40 ally record optogenetically restored retinal ganglion cell activity in the fovea of the living primat
41 map from the Cirrus OCT (Carl Zeiss Meditec) Ganglion Cell Analysis (GCA) was extracted, and structur
42                               The OFF-midget ganglion cell array acuity is well-matched to photopic s
43 lusters were defined as locations from where ganglion cell axons enter the optic nerve head within a
44 through which vasculature enters the eye and ganglion cell axons exit.
45                                      Retinal ganglion cell axons forming the optic nerve (ON) emerge
46 o automatically and accurately count retinal ganglion cell axons in optic nerve (ON) tissue images fr
47     We show that in the absence of Dcc, some ganglion cell axons stalled at the optic disc, whereas o
48 in the developing Xenopus brain, and retinal ganglion cell axons turned to follow this gradient.
49  studies showing age-related loss of retinal ganglion cell axons, we showed a significant decline in
50 advantage of the retinal direction-selective ganglion cell circuit, where directionally tuned inhibit
51                                      Macular ganglion cell complex (GCC) and peripapillary retinal ne
52 aphy (OCT)-based measurements of the macular ganglion cell complex (GCC) in healthy children facilita
53                       In Pearson's test, all Ganglion Cell Complex (GCC) thicknesses showed the weake
54 esponding to the central VF loss and macular ganglion cell complex thinning.
55 er thickness; but similar nerve fiber layer, ganglion cell complex, inner nuclear layer, and outer pl
56 ingle cone by mid-gestation and bipolar cell-ganglion cell connectivity undergoing a more protracted
57 rly located retinal area with photoreceptor: ganglion cell convergence as low as 39:1.
58                         To date, ganglion-to-ganglion cell coupling is thought to occur only between
59 addition to secondary vascular damage due to ganglion cell damage.
60 oligodendrocyte loss, and subsequent retinal ganglion cell death.
61       Intraocular pressure-sensitive retinal ganglion cell degeneration is a hallmark of glaucoma, th
62 significantly associated with IOP or retinal ganglion cell density.
63  in myopia to compensate for reduced retinal ganglion cell density.
64 o or outside the GCA grid when corrected for ganglion cell displacement.
65 re analyzed relative to previously published ganglion cell distributions in this species, showing a p
66 wired in the machinery of vertebrate retinal ganglion cell genesis.
67 with a loss of structural markers of retinal ganglion cell health in a multiethnic Asian population.
68 udies indicate that there are 30-50 types of ganglion cell in mouse retina, whereas only a few years
69 T) showed, in both eyes, a thickening of the ganglion cell layer (GCL) with a hyperreflective opacity
70 papillary retinal nerve fiber layer, macular ganglion cell layer (mGCL), and macular inner plexiform
71 ers (retinal nerve fiber layer thickness and ganglion cell layer - inner plexiform layer thickness).
72 ganglion cell layer volume (GCL, p = 0.003), ganglion cell layer - inner plexiform layer volume (GCL-
73  that specifically labels all neurons in the ganglion cell layer but is largely excluded from otherwi
74 red stimulation increased activity in cones, ganglion cell layer neurons, and cortical neurons, and e
75 ecies level was also elevated in the retinal ganglion cell layer of aged M(1) receptor-deficient mice
76 es such as total retinal volume (p = 0.037), ganglion cell layer volume (GCL, p = 0.003), ganglion ce
77 he retinal nerve fiber layer and the retinal ganglion cell layer with spectral-domain optical coheren
78     M1-like cells typically had somas in the ganglion cell layer, with 23% displaced to the inner nuc
79 se retina, with alpha equaling ~0.050 in the ganglion cell layer, ~0.122 in the inner plexiform layer
80 ve fibre layer thickness (mRNFL) and macular ganglion cell layer-inner plexiform layer thickness were
81 ning of the retinal nerve fiber layer and/or ganglion cell layer.
82 on ganglion cells in the Nubian ibex retinal ganglion cell layer.
83 ; (b) the outer plexiform, inner nuclear and ganglion cell layers are the strongest biomarkers for di
84 rs and marked atrophy of the nerve fiber and ganglion cell layers at the central macula.
85 ht-driven responses at the photoreceptor and ganglion cell levels.
86  M(1) receptor deficiency results in retinal ganglion cell loss in aged mice via involvement of oxida
87 otemporally and correlates anatomically with ganglion cell loss on spectral-domain OCT.
88 icant for corresponding focal superior nasal ganglion cell loss on spectral-domain OCT.
89 lls may contribute to the melanopsin retinal ganglion cell loss previously described and to the distu
90 sual field defect and corresponding anatomic ganglion cell loss suggests a focal retinal injury.
91 , which is associated with increased retinal ganglion cell loss, retinal nerve fiber layer thinning,
92                     Further studies on these ganglion cell photoreceptors will no doubt continue to i
93  in peripapillary retinal nerve fiber layer, ganglion cell plus inner plexiform layer (GCIPL), whole-
94 esterase is demonstrated to increase retinal ganglion cell survival in vivo in mice of both sexes fol
95 pecific, and topographically ordered retinal ganglion cell synaptic inputs.
96            We then image activity of retinal ganglion cell terminals and pretectal neurons.
97 twork motif in which the signals of distinct ganglion cell types are partially mixed at the output st
98                     Although many melanopsin ganglion cell types do project to image-forming brain ta
99         The functions of the diverse retinal ganglion cell types in primates and the parallel visual
100                                      Several ganglion cell types, the retinal output neurons, show se
101         The purpose was to study the macular ganglion cell- inner plexiform layer (GC-IPL) thickness
102  fiber layer (RNFL) thickness, rim area, and ganglion cell-inner plexiform layer (GC-IPL) thickness m
103 umpapillary RNFL (cpRNFL) thickness, macular ganglion cell-inner plexiform layer (GCIPL) thickness an
104  of the retinal nerve fiber layer (RNFL) and ganglion cell-inner plexiform layer (GCIPL).
105 re classified as either predominantly macula ganglion cell-inner plexiform layer (mGCIPL), predominan
106                                      Macular ganglion cell-inner plexiform layer complex (GCIPL) and
107 on that stimulated the corresponding retinal ganglion cell.
108 sured how populations of direction-selective ganglion cells (DSGCs) from the retinas of male and fema
109 quency of 4-7 Hz. nob ON direction-selective ganglion cells (DSGCs), which detect global motion and p
110                     Responses of ON- and OFF-ganglion cells (GCs) were recorded extracellularly from
111 r library in human stem cell-derived retinal ganglion cells (hRGCs).
112         Intrinsically photosensitive retinal ganglion cells (ipRGCs) are a subset of cells that parti
113 taining intrinsically photosensitive retinal ganglion cells (ipRGCs) are thought to play a role, how
114         Intrinsically photosensitive retinal ganglion cells (ipRGCs) control non-visual light respons
115 f these intrinsically photosensitive retinal ganglion cells (ipRGCs) have emerged.
116   These intrinsically photosensitive retinal ganglion cells (ipRGCs) have well-established roles in a
117 essing, intrinsically photosensitive retinal ganglion cells (ipRGCs) synchronize our biological clock
118 through intrinsically photosensitive retinal ganglion cells (ipRGCs)(4).
119         Intrinsically photosensitive retinal ganglion cells (ipRGCs), which express the photopigment
120 o the brain by dimming-sensitive OFF retinal ganglion cells (OFF-RGCs) that respond to light decremen
121 on selectivity of On-Off direction-selective ganglion cells (On-Off DSGCs) against noisy backgrounds
122 FICANCE STATEMENT ON-OFF direction-selective ganglion cells (ooDSGCs) in the mammalian retina are typ
123 sive degeneration of optic nerve and retinal ganglion cells (RGC).
124  mitochondria-associated function in retinal ganglion cells (RGCs) and the resulting optic nerve rema
125                The damage or loss of retinal ganglion cells (RGCs) and their axons accounts for the v
126                                      Retinal ganglion cells (RGCs) are a heterogeneous population of
127         We compared acutely isolated retinal ganglion cells (RGCs) at various developmental stages an
128                                      Retinal ganglion cells (RGCs) convey visual signals to 50 region
129 nostic ability of OCT parameters and retinal ganglion cells (RGCs) count in identify glaucomatous dis
130                                      Retinal ganglion cells (RGCs) drive diverse, light-evoked behavi
131                                      Retinal ganglion cells (RGCs) form an array of feature detectors
132                 Using purified adult retinal ganglion cells (RGCs) in culture, we demonstrated here t
133           Counts of Brn3a-expressing retinal ganglion cells (RGCs) in implanted eyes were indistingui
134 aracterized by a progressive loss of retinal ganglion cells (RGCs) in the eye, which ultimately resul
135 phy (OCT) measures the most critical retinal ganglion cells (RGCs) in the human eye.
136 unequivocally that a small subset of retinal ganglion cells (RGCs) project to the opposite retina and
137                                      Retinal ganglion cells (RGCs) serve as a crucial communication c
138                        The number of retinal ganglion cells (RGCs) underlying Ricco's area or crowdin
139 nar (PIm) is innervated by widefield retinal ganglion cells (RGCs), and this pathway is not a collate
140                             In chick retinal ganglion cells (RGCs), downregulation of Arl8B reduces a
141 f optic nerve axons and apoptosis of retinal ganglion cells (RGCs), however, the precise mechanisms a
142 chronic neurodegenerative disease of retinal ganglion cells (RGCs), is a leading cause of irreversibl
143 odulate input from photoreceptors to retinal ganglion cells (RGCs), rendering each RGC type selective
144                                      Retinal ganglion cells (RGCs), the output neurons of the retina,
145 ely even topographic distribution of retinal ganglion cells (RGCs)-the output neurons of the eye.
146 OFF receptive subfields in F-mini-ON retinal ganglion cells (RGCs).
147 hat their eyes send to the brain via retinal ganglion cells (RGCs).
148 ent of cell-specific morphologies in retinal ganglion cells (RGCs).
149 al field loss caused by the death of retinal ganglion cells (RGCs).
150 ignal and noise among populations of retinal ganglion cells (RGCs).
151                In humans, midget and parasol ganglion cells account for most of the input from the ey
152 isplayed a massive postnatal loss of retinal ganglion cells and a large fraction of photoreceptors.
153 ings from monosynaptically connected retinal ganglion cells and LGN neurons in male/female cats durin
154 ulls (math5(-/-)), mutants that lack retinal ganglion cells and retinofugal projections.
155 ressing intrinsically photosensitive retinal ganglion cells are characterized by sluggish activation
156        A small fraction of mammalian retinal ganglion cells are directly photoreceptive thanks to the
157 tion that determine directional responses in ganglion cells are shaped by two 'core' mechanisms, both
158 egulatory reprogramming in zebrafish retinal ganglion cells at specific time points along the axon re
159  coupled cell types were sustained ON center ganglion cells but showed distinct light response proper
160                                              Ganglion cells can form electrical synapses between dend
161 density of OFF-midget bipolar and OFF-midget ganglion cells can support one-to-one connections to 1.0
162 receptive fields of human midget and parasol ganglion cells divide naturalistic movies into adjacent
163 ectrical coupling between different types of ganglion cells exists in the mammalian retina.
164 o these two functional realms and melanopsin ganglion cells have begun to challenge the boundary betw
165                                   Melanopsin ganglion cells have defied convention since their discov
166 ve shown that individual types of melanopsin ganglion cells have the potential to impact image-formin
167 strate an ever-expanding role for melanopsin ganglion cells in image-forming vision.
168                          For parasol retinal ganglion cells in macaque retina, estimated subunits par
169 procal correlated firing between heterotypic ganglion cells in multielectrode array recordings during
170                     Thus, midget and parasol ganglion cells in the human retina efficiently encode ou
171 aspects of the six known types of melanopsin ganglion cells in the mouse retina and to highlight thei
172           We estimated a total of ~1 million ganglion cells in the Nubian ibex retinal ganglion cell
173 e of electrical coupling between heterotypic ganglion cells introduces a network motif in which the s
174 mphocytes and neurons in the gut wall and in ganglion cells of the myenteric plexus.
175 ction of miR-223-3p in vivo in mouse retinal ganglion cells protects their axons from degeneration in
176 ckness near birth, implying that the retinal ganglion cells reserve is affected by intrauterine proce
177 , Sox2 and Klf4 genes (OSK) in mouse retinal ganglion cells restores youthful DNA methylation pattern
178 tomical evidence that two different types of ganglion cells share information via electrical coupling
179 ctivity distributed across feature-selective ganglion cells such that signals representing distinct s
180  and the response characteristics of retinal ganglion cells under a broad range of conditions.
181 ar (DB) types DB3a and DB3b (M pathway), and ganglion cells were counted along the temporal horizonta
182 fic subtypes of horizontal cells and retinal ganglion cells were overrepresented, suggesting that Thr
183 onfer intrinsic light sensitivity to retinal ganglion cells when photoreceptors have degenerated and
184 phologically distinct types of mouse retinal ganglion cells with overlapping excitatory synaptic inpu
185                   Neural sampling by retinal ganglion cells with receptive field size and spacing tha
186 ing to the predominant oxygenation status of ganglion cells within the superficial inner retina, whet
187 rgic synapses from AII amacrine cells to OFF ganglion cells) is sufficient for fast, mesopic rod-driv
188 uch that the densities of early-born retinal ganglion cells, amacrine and horizontal cells, as well a
189 ed in several bipolar cell subtypes, retinal ganglion cells, and some amacrine cell subtypes but not
190 rences in the function of midget and parasol ganglion cells, consistent asymmetries between their ON
191 rently six known types (M1-M6) of melanopsin ganglion cells, each with unique morphology, mosaics, co
192 rburst cholinergic and GABAergic synapses to ganglion cells, form the basis for a parallel mechanism
193 I amacrine and melanopsin-containing retinal ganglion cells, in control and PD eyes from human donors
194 rted to occur only between homotypic retinal ganglion cells, in line with the concept of parallel pro
195 ich exhibit elevated IOP and loss of retinal ganglion cells, Tek(+/-);Ptprb(+/-) mice have elevated T
196 d an inhibitory surround, that propagates to ganglion cells, the retina's projection neurons.
197  eye, followed by neural sampling by retinal ganglion cells, to demonstrate the perceptual effects of
198           Axotomy elevated lipin1 in retinal ganglion cells, which contributed to regeneration failur
199 ibed in horizontal, OFF-bipolar, amacrine or ganglion cells, which could not be fully blocked in the
200 nnervation of the visual thalamus by retinal ganglion cells.
201 in via the output neurons of the retina, the ganglion cells.
202 arousal modulates the firing of some retinal ganglion cells.
203 re one of the major types of primate retinal ganglion cells.
204 is interaction is present in primary retinal ganglion cells.
205  individual dendrites of direction-selective ganglion cells.
206 tor mediated stimulation in the same retinal ganglion cells.
207 he involved cell types as sustained ON alpha-ganglion cells.
208 mode of action or a direct impact on retinal ganglion cells.
209 ed in compromised differentiation of retinal ganglion cells.
210 ls for transmission to the brain via retinal ganglion cells.
211 blindness due to the degeneration of retinal ganglion cells.
212 smission, which is propagated to the retinal ganglion cells.
213 atial resolution is lost at the level of the ganglion cells.
214 rd drive from photoreceptors to amacrine and ganglion cells.
215 s spatial resolution is lost at the level of ganglion cells.SIGNIFICANCE STATEMENT We make accurate m
216 ye disease characterized by death of retinal ganglion cells; lowering IOP is the only proven treatmen
217 CN V) differentiation and altered trigeminal ganglion (CNgV) cellular composition prefigure these dis
218 ent and reactivation using human dorsal root ganglion-derived neuronal HD10.6 cells as an in vitro mo
219 st calcium signals in neurons throughout the ganglion, did not activate myenteric neurons.
220 onal preparations, such as whole dorsal root ganglion (DRG) and hindpaw tissues, revealed only a few
221 riptome analyses of rodent whole dorsal root ganglion (DRG) have revealed sex differences, mostly in
222 istribution of mammalian PATs in dorsal root ganglion (DRG) neurons and, strikingly, found that only
223 arkably decreased RNA binding in dorsal root ganglion (DRG) neurons compared with wild-type and non-p
224 ed sodium channels (VGSC) on the dorsal root ganglion (DRG) neurons controlling electrical impulses m
225                               In dorsal root ganglion (DRG) neurons cultured from rats primed with fe
226 mutation, which is known to make dorsal root ganglion (DRG) neurons hyperexcitable, but different pai
227 chemogenetic activation of adult dorsal root ganglion (DRG) neurons improves axon growth on an in vit
228 phaq)-coupled receptors in mouse dorsal root ganglion (DRG) neurons isolated from both sexes.
229 ormed on small-diameter (<30 um) dorsal root ganglion (DRG) neurons, cultured from fentanyl-primed ra
230 fentanyl (0.5 nm) was applied to dorsal root ganglion (DRG) neurons, cultured from opioid-primed rats
231 PV1-ANO1 channel coupling in rat dorsal root ganglion (DRG) neurons.
232 roximately to promote latency in dorsal root ganglion (DRG) neurons.
233 rgent currents in large-diameter dorsal root ganglion (DRG) neurons.
234 t directly on nociceptors in the dorsal root ganglion (DRG) to cause pain sensitization.
235  protein levels of P2X(3) in the dorsal root ganglion (DRG), and the whole cell patch clamp was used
236 urons with different positions in the spiral ganglion employ different guidance mechanisms, with evid
237 differ significantly at E10.5 just after the ganglion has coalesced.
238 ng behavior and innervated by the trigeminal ganglion including the lateral periodontium, rostral per
239 ual neurons in each sympathetic prevertebral ganglion innervated the proximal or distal colon, with p
240                 By attenuating the abdominal ganglion inputs to pC1 neurons and oviINs, sex peptide d
241 A), we focused on large-diameter dorsal-root ganglion (L-DRG) neurons with myelinated axons.
242 finitively shows that an identified cerebral ganglion neuron that is a member of a CPG underlying swi
243 t and regenerate isolated primary trigeminal ganglion neuronal cells (TGNC).
244 d sodium channel Nav1.7 underlie dorsal root ganglion neuronal hyperexcitability and pain in a subset
245 ologically distinct classes of type I spiral ganglion neurons (SGNs) are necessary to encode sound in
246 pment, primary auditory neurons named spiral ganglion neurons (SGNs) are surrounded by otic mesenchym
247 damages the postsynaptic terminals of spiral ganglion neurons (SGNs) on cochlear inner hair cells (IH
248 blished RNA-sequencing dataset of geniculate ganglion neurons and by in situ hybridization, we demons
249  Na(V)1.7 is highly expressed in dorsal root ganglion neurons and is obligatory for nociceptive signa
250 -S Na+ current density in medium dorsal root ganglion neurons and, importantly, mechanical allodynia
251 sensitization of native TRPV1 in dorsal root ganglion neurons as well as of recombinant TRPV1 express
252 ically distinct from other classes of spiral ganglion neurons because they extend a peripheral axon b
253                 We show that these abdominal ganglion neurons directly activate the female-specific p
254  instance, in the developing cochlea, spiral ganglion neurons extend their peripheral processes throu
255 y, where dorsal root ganglion and trigeminal ganglion neurons feed pain information into the CNS.
256 ved in the functional changes of dorsal root ganglion neurons following vincristine treatment and it
257 stant (TTX-R) sodium currents in dorsal root ganglion neurons following vincristine treatment.
258                   Cultured adult dorsal root ganglion neurons from nSIRT1OE mice, maintained at high
259 p recordings of small and medium dorsal root ganglion neurons from vincristine-treated animals reveal
260  capacity when compared to adult dorsal root ganglion neurons from wild-type mice.
261   This finding advances our knowledge of how ganglion neurons generate uncharacteristic electrical im
262 d increased Trpm3 mRNA levels in dorsal root ganglion neurons innervating the inflamed paw, and augme
263 n, genetically deleting GluN1 in dorsal root ganglion neurons or alpha2delta-1 genetic KO similarly a
264 man by a subpopulation of TRPV1+ dorsal root ganglion neurons specialized in detecting painful stimul
265  pharmacology to investigate rat dorsal root ganglion neurons using two models of peripheral nerve in
266  labelling of bladder-projecting dorsal root ganglion neurons was used to investigate expression of 5
267 odium current, in small-diameter dorsal root ganglion neurons, an effect that was attenuated by a PI3
268 ocytes, cultured hippocampal and dorsal root ganglion neurons, and brain slices.
269 the adult mouse cochlea including the spiral ganglion neurons, suggesting changes in expression level
270 ated activity in inner hair cells and spiral ganglion neurons, which begins at birth and follows a ba
271  Nav1.6-mediated excitability in dorsal root ganglion neurons.
272  expressed in nodose-petrosal and geniculate ganglion neurons.
273 til after reinnervation by distant gustatory ganglion neurons.
274  innervation of muscle fibers by dorsal root ganglion neurons.
275 polymodal and pure mechanosensory trigeminal ganglion neurons.
276 2, is selective for Na(V) 1.6 in dorsal root ganglion neurons.
277 nat2 compound heterozygote superior cervical ganglion neurons.
278 omic ganglia of the neck, namely, the nodose ganglion (NG) and the superior cervical ganglion (SCG) i
279 stem, oral sensory neurons of the geniculate ganglion project via the chorda tympani nerve to innerva
280                               In dorsal root ganglion protein extracts from nSIRT1OE mice, the NAD+-c
281     This protein expression is recognized by ganglion-resident HSV-1-specific CD8(+) T cells that mai
282 dose ganglion (NG) and the superior cervical ganglion (SCG) in a cohort of C57BL/6J mice.
283 RT2 accumulated in the nuclei of dorsal root ganglion sensory neurons and prevented neuronal cell dea
284 stablished a co-culture system of trigeminal ganglion sensory neurons and vascular endothelial cells
285 localized in the organ of Corti (OC), spiral ganglion (SG), stria vascularis (SV), and afferent nerve
286  Pou4f1 after SGN formation does not disrupt ganglion size or morphology, change the distribution of
287 d nerve activity (NA) from the left stellate ganglion (SNA), left cardiac vagus (VNA), and arterial b
288 naptic activity in the brain, suboesophageal ganglion (SOG) and sensory neurons.
289 (GM) neuron in the crustacean stomatogastric ganglion (STG) operates like a single electrotonic compa
290 ss the safety and efficacy of sphenopalatine ganglion stimulation for treatment of chronic cluster he
291            36 patients in the sphenopalatine ganglion stimulation group and 40 in the control group h
292 relationship between attained sphenopalatine ganglion stimulation intensity and the primary outcome i
293 ic transcriptome signature in the trigeminal ganglion (TG) that includes Rictor, the rapamycin-insens
294 e virus enters latency within the trigeminal ganglion (TG), from which it can reactivate throughout t
295                                     To date, ganglion-to-ganglion cell coupling is thought to occur o
296 y to drug therapy treated with left stellate ganglion transcutaneous magnetic stimulation (TCMS) to r
297 lly influenced by preparation approaches and ganglion types [DRG vs trigeminal ganglia (TG)].
298 e prepared by using different approaches and ganglion types.
299                             Single myenteric ganglion volume averaged 3,527,678 +/- 573,832 mm(3) wit
300             Here we show that the trigeminal ganglion, which provides sensory innervation to the face

 
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