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1 xpressing cells around and within the spiral ganglion.
2 ve primary sensory neurons in the trigeminal ganglion.
3 than those from neurons at the border of the ganglion.
5 administration, increases in GP and stellate ganglion activity and blood pressure during apnea were a
8 These include specific subtypes of retinal ganglion and horizontal cells, suggesting that in this c
10 in Magel2, a PWS gene, within the trigeminal ganglion and regions that are anatomically relevant to f
11 riginate in the periphery, where dorsal root ganglion and trigeminal ganglion neurons feed pain infor
12 smaller proportion of nitrergic neurons per ganglion, and reduced markers of neurogenesis compared w
13 ic activity in the crustacean stomatogastric ganglion, and use these results to derive hypotheses for
14 Processes from neurons in the rear of the ganglion are more directed and grow faster than those fr
15 ratus, we demonstrate that chick dorsal root ganglion axons exhibit a tension buffering or strain-sof
16 essing, intrinsically photosensitive retinal ganglion cell (ipRGC) subtypes are two RGC types that ar
18 action of Xenopus tadpoles, a single retinal ganglion cell (RGC) axon misprojects to the ipsilateral
20 growth factor-1 (IGF1) by initiating retinal ganglion cell (RGC) axon regeneration after axotomy.
23 ieved to be the major contributor to retinal ganglion cell (RGC) death, the endpoint of optic neuropa
25 describes a novel paradigm to reduce retinal ganglion cell (RGC) degeneration underlying glaucoma.
26 nto cone bipolar cell (BC) axons and retinal ganglion cell (RGC) dendrites, but makes the majority of
27 of intraocular pressure (IOP) causes retinal ganglion cell (RGC) dysfunction and death and is a major
28 sion protein optic atrophy 1 (Opa1), retinal ganglion cell (RGC) dysfunction and visual loss occur by
29 The visual message conveyed by a retinal ganglion cell (RGC) is often summarized by its spatial r
30 mber of Nissl-stained neurons in the retinal ganglion cell (RGC) layer in the Caribbean and Chilean f
31 has previously been used to stratify Retinal Ganglion Cell (RGC) populations in histological samples
32 rodents, Rbfox2 is expressed in all retinal ganglion cell (RGC) subtypes, horizontal cells, as well
33 erged in which the three most common retinal ganglion cell (RGC) types captured much of the variance
34 l information is encoded in distinct retinal ganglion cell (RGC) types in the eye tuned to specific f
36 tablished role in the development of retinal ganglion cell (RGCs) types, the main transducers of visu
38 rod photoreceptor (~80,000 rods mm(-2) ) and ganglion cell (~1,800 cells mm(-2) ) densities across th
39 etinal nerve fiber layer (pRNFL) and macular ganglion cell + inner plexiform layer (GCIPL) thinning i
40 ally record optogenetically restored retinal ganglion cell activity in the fovea of the living primat
41 map from the Cirrus OCT (Carl Zeiss Meditec) Ganglion Cell Analysis (GCA) was extracted, and structur
43 lusters were defined as locations from where ganglion cell axons enter the optic nerve head within a
46 o automatically and accurately count retinal ganglion cell axons in optic nerve (ON) tissue images fr
47 We show that in the absence of Dcc, some ganglion cell axons stalled at the optic disc, whereas o
49 studies showing age-related loss of retinal ganglion cell axons, we showed a significant decline in
50 advantage of the retinal direction-selective ganglion cell circuit, where directionally tuned inhibit
52 aphy (OCT)-based measurements of the macular ganglion cell complex (GCC) in healthy children facilita
55 er thickness; but similar nerve fiber layer, ganglion cell complex, inner nuclear layer, and outer pl
56 ingle cone by mid-gestation and bipolar cell-ganglion cell connectivity undergoing a more protracted
65 re analyzed relative to previously published ganglion cell distributions in this species, showing a p
67 with a loss of structural markers of retinal ganglion cell health in a multiethnic Asian population.
68 udies indicate that there are 30-50 types of ganglion cell in mouse retina, whereas only a few years
69 T) showed, in both eyes, a thickening of the ganglion cell layer (GCL) with a hyperreflective opacity
70 papillary retinal nerve fiber layer, macular ganglion cell layer (mGCL), and macular inner plexiform
71 ers (retinal nerve fiber layer thickness and ganglion cell layer - inner plexiform layer thickness).
72 ganglion cell layer volume (GCL, p = 0.003), ganglion cell layer - inner plexiform layer volume (GCL-
73 that specifically labels all neurons in the ganglion cell layer but is largely excluded from otherwi
74 red stimulation increased activity in cones, ganglion cell layer neurons, and cortical neurons, and e
75 ecies level was also elevated in the retinal ganglion cell layer of aged M(1) receptor-deficient mice
76 es such as total retinal volume (p = 0.037), ganglion cell layer volume (GCL, p = 0.003), ganglion ce
77 he retinal nerve fiber layer and the retinal ganglion cell layer with spectral-domain optical coheren
78 M1-like cells typically had somas in the ganglion cell layer, with 23% displaced to the inner nuc
79 se retina, with alpha equaling ~0.050 in the ganglion cell layer, ~0.122 in the inner plexiform layer
80 ve fibre layer thickness (mRNFL) and macular ganglion cell layer-inner plexiform layer thickness were
83 ; (b) the outer plexiform, inner nuclear and ganglion cell layers are the strongest biomarkers for di
86 M(1) receptor deficiency results in retinal ganglion cell loss in aged mice via involvement of oxida
89 lls may contribute to the melanopsin retinal ganglion cell loss previously described and to the distu
91 , which is associated with increased retinal ganglion cell loss, retinal nerve fiber layer thinning,
93 in peripapillary retinal nerve fiber layer, ganglion cell plus inner plexiform layer (GCIPL), whole-
94 esterase is demonstrated to increase retinal ganglion cell survival in vivo in mice of both sexes fol
97 twork motif in which the signals of distinct ganglion cell types are partially mixed at the output st
102 fiber layer (RNFL) thickness, rim area, and ganglion cell-inner plexiform layer (GC-IPL) thickness m
103 umpapillary RNFL (cpRNFL) thickness, macular ganglion cell-inner plexiform layer (GCIPL) thickness an
105 re classified as either predominantly macula ganglion cell-inner plexiform layer (mGCIPL), predominan
108 sured how populations of direction-selective ganglion cells (DSGCs) from the retinas of male and fema
109 quency of 4-7 Hz. nob ON direction-selective ganglion cells (DSGCs), which detect global motion and p
113 taining intrinsically photosensitive retinal ganglion cells (ipRGCs) are thought to play a role, how
116 These intrinsically photosensitive retinal ganglion cells (ipRGCs) have well-established roles in a
117 essing, intrinsically photosensitive retinal ganglion cells (ipRGCs) synchronize our biological clock
120 o the brain by dimming-sensitive OFF retinal ganglion cells (OFF-RGCs) that respond to light decremen
121 on selectivity of On-Off direction-selective ganglion cells (On-Off DSGCs) against noisy backgrounds
122 FICANCE STATEMENT ON-OFF direction-selective ganglion cells (ooDSGCs) in the mammalian retina are typ
124 mitochondria-associated function in retinal ganglion cells (RGCs) and the resulting optic nerve rema
129 nostic ability of OCT parameters and retinal ganglion cells (RGCs) count in identify glaucomatous dis
134 aracterized by a progressive loss of retinal ganglion cells (RGCs) in the eye, which ultimately resul
136 unequivocally that a small subset of retinal ganglion cells (RGCs) project to the opposite retina and
139 nar (PIm) is innervated by widefield retinal ganglion cells (RGCs), and this pathway is not a collate
141 f optic nerve axons and apoptosis of retinal ganglion cells (RGCs), however, the precise mechanisms a
142 chronic neurodegenerative disease of retinal ganglion cells (RGCs), is a leading cause of irreversibl
143 odulate input from photoreceptors to retinal ganglion cells (RGCs), rendering each RGC type selective
145 ely even topographic distribution of retinal ganglion cells (RGCs)-the output neurons of the eye.
152 isplayed a massive postnatal loss of retinal ganglion cells and a large fraction of photoreceptors.
153 ings from monosynaptically connected retinal ganglion cells and LGN neurons in male/female cats durin
155 ressing intrinsically photosensitive retinal ganglion cells are characterized by sluggish activation
157 tion that determine directional responses in ganglion cells are shaped by two 'core' mechanisms, both
158 egulatory reprogramming in zebrafish retinal ganglion cells at specific time points along the axon re
159 coupled cell types were sustained ON center ganglion cells but showed distinct light response proper
161 density of OFF-midget bipolar and OFF-midget ganglion cells can support one-to-one connections to 1.0
162 receptive fields of human midget and parasol ganglion cells divide naturalistic movies into adjacent
164 o these two functional realms and melanopsin ganglion cells have begun to challenge the boundary betw
166 ve shown that individual types of melanopsin ganglion cells have the potential to impact image-formin
169 procal correlated firing between heterotypic ganglion cells in multielectrode array recordings during
171 aspects of the six known types of melanopsin ganglion cells in the mouse retina and to highlight thei
173 e of electrical coupling between heterotypic ganglion cells introduces a network motif in which the s
175 ction of miR-223-3p in vivo in mouse retinal ganglion cells protects their axons from degeneration in
176 ckness near birth, implying that the retinal ganglion cells reserve is affected by intrauterine proce
177 , Sox2 and Klf4 genes (OSK) in mouse retinal ganglion cells restores youthful DNA methylation pattern
178 tomical evidence that two different types of ganglion cells share information via electrical coupling
179 ctivity distributed across feature-selective ganglion cells such that signals representing distinct s
181 ar (DB) types DB3a and DB3b (M pathway), and ganglion cells were counted along the temporal horizonta
182 fic subtypes of horizontal cells and retinal ganglion cells were overrepresented, suggesting that Thr
183 onfer intrinsic light sensitivity to retinal ganglion cells when photoreceptors have degenerated and
184 phologically distinct types of mouse retinal ganglion cells with overlapping excitatory synaptic inpu
186 ing to the predominant oxygenation status of ganglion cells within the superficial inner retina, whet
187 rgic synapses from AII amacrine cells to OFF ganglion cells) is sufficient for fast, mesopic rod-driv
188 uch that the densities of early-born retinal ganglion cells, amacrine and horizontal cells, as well a
189 ed in several bipolar cell subtypes, retinal ganglion cells, and some amacrine cell subtypes but not
190 rences in the function of midget and parasol ganglion cells, consistent asymmetries between their ON
191 rently six known types (M1-M6) of melanopsin ganglion cells, each with unique morphology, mosaics, co
192 rburst cholinergic and GABAergic synapses to ganglion cells, form the basis for a parallel mechanism
193 I amacrine and melanopsin-containing retinal ganglion cells, in control and PD eyes from human donors
194 rted to occur only between homotypic retinal ganglion cells, in line with the concept of parallel pro
195 ich exhibit elevated IOP and loss of retinal ganglion cells, Tek(+/-);Ptprb(+/-) mice have elevated T
197 eye, followed by neural sampling by retinal ganglion cells, to demonstrate the perceptual effects of
199 ibed in horizontal, OFF-bipolar, amacrine or ganglion cells, which could not be fully blocked in the
215 s spatial resolution is lost at the level of ganglion cells.SIGNIFICANCE STATEMENT We make accurate m
216 ye disease characterized by death of retinal ganglion cells; lowering IOP is the only proven treatmen
217 CN V) differentiation and altered trigeminal ganglion (CNgV) cellular composition prefigure these dis
218 ent and reactivation using human dorsal root ganglion-derived neuronal HD10.6 cells as an in vitro mo
220 onal preparations, such as whole dorsal root ganglion (DRG) and hindpaw tissues, revealed only a few
221 riptome analyses of rodent whole dorsal root ganglion (DRG) have revealed sex differences, mostly in
222 istribution of mammalian PATs in dorsal root ganglion (DRG) neurons and, strikingly, found that only
223 arkably decreased RNA binding in dorsal root ganglion (DRG) neurons compared with wild-type and non-p
224 ed sodium channels (VGSC) on the dorsal root ganglion (DRG) neurons controlling electrical impulses m
226 mutation, which is known to make dorsal root ganglion (DRG) neurons hyperexcitable, but different pai
227 chemogenetic activation of adult dorsal root ganglion (DRG) neurons improves axon growth on an in vit
229 ormed on small-diameter (<30 um) dorsal root ganglion (DRG) neurons, cultured from fentanyl-primed ra
230 fentanyl (0.5 nm) was applied to dorsal root ganglion (DRG) neurons, cultured from opioid-primed rats
235 protein levels of P2X(3) in the dorsal root ganglion (DRG), and the whole cell patch clamp was used
236 urons with different positions in the spiral ganglion employ different guidance mechanisms, with evid
238 ng behavior and innervated by the trigeminal ganglion including the lateral periodontium, rostral per
239 ual neurons in each sympathetic prevertebral ganglion innervated the proximal or distal colon, with p
242 finitively shows that an identified cerebral ganglion neuron that is a member of a CPG underlying swi
244 d sodium channel Nav1.7 underlie dorsal root ganglion neuronal hyperexcitability and pain in a subset
245 ologically distinct classes of type I spiral ganglion neurons (SGNs) are necessary to encode sound in
246 pment, primary auditory neurons named spiral ganglion neurons (SGNs) are surrounded by otic mesenchym
247 damages the postsynaptic terminals of spiral ganglion neurons (SGNs) on cochlear inner hair cells (IH
248 blished RNA-sequencing dataset of geniculate ganglion neurons and by in situ hybridization, we demons
249 Na(V)1.7 is highly expressed in dorsal root ganglion neurons and is obligatory for nociceptive signa
250 -S Na+ current density in medium dorsal root ganglion neurons and, importantly, mechanical allodynia
251 sensitization of native TRPV1 in dorsal root ganglion neurons as well as of recombinant TRPV1 express
252 ically distinct from other classes of spiral ganglion neurons because they extend a peripheral axon b
254 instance, in the developing cochlea, spiral ganglion neurons extend their peripheral processes throu
255 y, where dorsal root ganglion and trigeminal ganglion neurons feed pain information into the CNS.
256 ved in the functional changes of dorsal root ganglion neurons following vincristine treatment and it
259 p recordings of small and medium dorsal root ganglion neurons from vincristine-treated animals reveal
261 This finding advances our knowledge of how ganglion neurons generate uncharacteristic electrical im
262 d increased Trpm3 mRNA levels in dorsal root ganglion neurons innervating the inflamed paw, and augme
263 n, genetically deleting GluN1 in dorsal root ganglion neurons or alpha2delta-1 genetic KO similarly a
264 man by a subpopulation of TRPV1+ dorsal root ganglion neurons specialized in detecting painful stimul
265 pharmacology to investigate rat dorsal root ganglion neurons using two models of peripheral nerve in
266 labelling of bladder-projecting dorsal root ganglion neurons was used to investigate expression of 5
267 odium current, in small-diameter dorsal root ganglion neurons, an effect that was attenuated by a PI3
269 the adult mouse cochlea including the spiral ganglion neurons, suggesting changes in expression level
270 ated activity in inner hair cells and spiral ganglion neurons, which begins at birth and follows a ba
278 omic ganglia of the neck, namely, the nodose ganglion (NG) and the superior cervical ganglion (SCG) i
279 stem, oral sensory neurons of the geniculate ganglion project via the chorda tympani nerve to innerva
281 This protein expression is recognized by ganglion-resident HSV-1-specific CD8(+) T cells that mai
283 RT2 accumulated in the nuclei of dorsal root ganglion sensory neurons and prevented neuronal cell dea
284 stablished a co-culture system of trigeminal ganglion sensory neurons and vascular endothelial cells
285 localized in the organ of Corti (OC), spiral ganglion (SG), stria vascularis (SV), and afferent nerve
286 Pou4f1 after SGN formation does not disrupt ganglion size or morphology, change the distribution of
287 d nerve activity (NA) from the left stellate ganglion (SNA), left cardiac vagus (VNA), and arterial b
289 (GM) neuron in the crustacean stomatogastric ganglion (STG) operates like a single electrotonic compa
290 ss the safety and efficacy of sphenopalatine ganglion stimulation for treatment of chronic cluster he
292 relationship between attained sphenopalatine ganglion stimulation intensity and the primary outcome i
293 ic transcriptome signature in the trigeminal ganglion (TG) that includes Rictor, the rapamycin-insens
294 e virus enters latency within the trigeminal ganglion (TG), from which it can reactivate throughout t
296 y to drug therapy treated with left stellate ganglion transcutaneous magnetic stimulation (TCMS) to r