コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 on that stimulated the corresponding retinal ganglion cell.
2 a private-line connection with an OFF midget ganglion cell.
3 individual dendrites of direction-selective ganglion cells.
4 tor mediated stimulation in the same retinal ganglion cells.
5 re one of the major types of primate retinal ganglion cells.
6 mode of action or a direct impact on retinal ganglion cells.
7 he involved cell types as sustained ON alpha-ganglion cells.
8 ed in compromised differentiation of retinal ganglion cells.
9 ls for transmission to the brain via retinal ganglion cells.
10 blindness due to the degeneration of retinal ganglion cells.
11 smission, which is propagated to the retinal ganglion cells.
12 te with endosomes along the axons of retinal ganglion cells.
13 cells, as well as a small number of retinal ganglion cells.
14 ntracellular recordings from macaque retinal ganglion cells.
15 ansmission is sufficient to sensitize nearby ganglion cells.
16 atial resolution is lost at the level of the ganglion cells.
17 rd drive from photoreceptors to amacrine and ganglion cells.
18 nnervation of the visual thalamus by retinal ganglion cells.
19 in via the output neurons of the retina, the ganglion cells.
20 arousal modulates the firing of some retinal ganglion cells.
21 is interaction is present in primary retinal ganglion cells.
22 rod photoreceptor (~80,000 rods mm(-2) ) and ganglion cell (~1,800 cells mm(-2) ) densities across th
24 her sex during ultrasonic stimuli that drive ganglion cell activity and observed micron scale displac
25 ally record optogenetically restored retinal ganglion cell activity in the fovea of the living primat
27 uch that the densities of early-born retinal ganglion cells, amacrine and horizontal cells, as well a
28 an overall decrease in the number of retinal ganglion cells, amacrine cells, and an increase in the n
29 map from the Cirrus OCT (Carl Zeiss Meditec) Ganglion Cell Analysis (GCA) was extracted, and structur
30 isplayed a massive postnatal loss of retinal ganglion cells and a large fraction of photoreceptors.
31 ings from monosynaptically connected retinal ganglion cells and LGN neurons in male/female cats durin
33 , comprising the retinal nerve fiber (RNFL), ganglion cell, and inner plexiform layers, can be correl
34 ed in several bipolar cell subtypes, retinal ganglion cells, and some amacrine cell subtypes but not
35 ressing intrinsically photosensitive retinal ganglion cells are characterized by sluggish activation
39 tion that determine directional responses in ganglion cells are shaped by two 'core' mechanisms, both
42 o evaluate the diagnostic ability of macular ganglion cell asymmetry to diagnose preperimetric glauco
43 egulatory reprogramming in zebrafish retinal ganglion cells at specific time points along the axon re
44 lusters were defined as locations from where ganglion cell axons enter the optic nerve head within a
47 o automatically and accurately count retinal ganglion cell axons in optic nerve (ON) tissue images fr
48 Cue stimulation of growing Xenopus retinal ganglion cell axons induces rapid dissociation of riboso
49 We show that in the absence of Dcc, some ganglion cell axons stalled at the optic disc, whereas o
51 studies showing age-related loss of retinal ganglion cell axons, we showed a significant decline in
52 coupled cell types were sustained ON center ganglion cells but showed distinct light response proper
55 density of OFF-midget bipolar and OFF-midget ganglion cells can support one-to-one connections to 1.0
56 advantage of the retinal direction-selective ganglion cell circuit, where directionally tuned inhibit
58 aphy (OCT)-based measurements of the macular ganglion cell complex (GCC) in healthy children facilita
59 lary retinal nerve fiber (cpRNFL) thickness, ganglion cell complex (GCC) thickness, and visual field
61 ganglion cell/inner plexiform layer (GCIPL), ganglion cell complex (GCC), and full macular thickness
65 er thickness; but similar nerve fiber layer, ganglion cell complex, inner nuclear layer, and outer pl
66 ingle cone by mid-gestation and bipolar cell-ganglion cell connectivity undergoing a more protracted
67 rences in the function of midget and parasol ganglion cells, consistent asymmetries between their ON
69 The visual response properties of retinal ganglion cells correlated well with those of their disyn
74 ship between the topographic distribution of ganglion cell density and the nonuniform spatial integra
76 and stereology, we sought to measure how the ganglion cell density varies across the retina of the Nu
80 re analyzed relative to previously published ganglion cell distributions in this species, showing a p
81 receptive fields of human midget and parasol ganglion cells divide naturalistic movies into adjacent
82 sured how populations of direction-selective ganglion cells (DSGCs) from the retinas of male and fema
83 the mammalian retina, ON direction-selective ganglion cells (DSGCs) respond preferentially to slow im
84 Two types of mammalian direction-selective ganglion cells (DSGCs), ON and ONOFF, operate over diffe
85 quency of 4-7 Hz. nob ON direction-selective ganglion cells (DSGCs), which detect global motion and p
87 rently six known types (M1-M6) of melanopsin ganglion cells, each with unique morphology, mosaics, co
88 Here, we identify a novel marker for retinal ganglion cells encoding directional motion that is evolu
89 sicular GABA/glycine transporter) in retinal ganglion cells enhances the activity of inner retinal DA
92 rburst cholinergic and GABAergic synapses to ganglion cells, form the basis for a parallel mechanism
96 o these two functional realms and melanopsin ganglion cells have begun to challenge the boundary betw
98 ve shown that individual types of melanopsin ganglion cells have the potential to impact image-formin
99 with a loss of structural markers of retinal ganglion cell health in a multiethnic Asian population.
100 glia, bipolar cells, amacrine cells, retinal ganglion cells, horizontal cells, astrocytes, and microg
102 udies indicate that there are 30-50 types of ganglion cell in mouse retina, whereas only a few years
103 of Neuron, Rhoades et al. (2019) describe a ganglion cell in primate retina that reports visual inpu
104 naptic connectivity of melanopsin-expressing ganglion cells in four post mortem human donor retinas.
107 procal correlated firing between heterotypic ganglion cells in multielectrode array recordings during
109 aspects of the six known types of melanopsin ganglion cells in the mouse retina and to highlight thei
110 racterize the chromatic tuning of OFF midget ganglion cells in the near peripheral retina that receiv
112 wth in vitro and for the survival of retinal ganglion cells in vivo following optic nerve injury.
113 I amacrine and melanopsin-containing retinal ganglion cells, in control and PD eyes from human donors
114 rted to occur only between homotypic retinal ganglion cells, in line with the concept of parallel pro
115 tial organization of cone signals in retinal ganglion cells, including how signals from cones of diff
116 gion of interest (R = -0.78, P < 0.0001) and ganglion cell inner plexiform layer region of interest (
117 of interest (R = -0.74, P < 0.0001) and the ganglion cell inner plexiform layer region of interest (
118 etinal nerve fiber layer (pRNFL) and macular ganglion cell + inner plexiform layer (GCIPL) thinning i
119 tinal nerve fiber layer and 4mum for macular ganglion cell + inner plexiform layer are robust thresho
121 fiber layer (RNFL) thickness, rim area, and ganglion cell-inner plexiform layer (GC-IPL) thickness m
122 a, central subfield thickness (CST), macular ganglion cell-inner plexiform layer (GC-IPL) thickness,
123 umpapillary RNFL (cpRNFL) thickness, macular ganglion cell-inner plexiform layer (GCIPL) thickness an
125 re classified as either predominantly macula ganglion cell-inner plexiform layer (mGCIPL), predominan
126 FL at baseline (P = .006) or thinner average ganglion cell-inner plexiform layer (P = .028) along wit
128 measurements for ganglion cell layer (GCL), ganglion cell/inner plexiform layer (GCIPL), ganglion ce
129 3 was the gene most strongly associated with ganglion cell/inner plexiform layer atrophy (P = 0.004)
130 y, we conducted a similar set of analyses of ganglion cell/inner plexiform layer thinning in a replic
131 e of electrical coupling between heterotypic ganglion cells introduces a network motif in which the s
132 essing, intrinsically photosensitive retinal ganglion cell (ipRGC) subtypes are two RGC types that ar
133 ated by intrinsically photosensitive retinal ganglion cells (ipRGCs) and is critical for driving seve
135 taining intrinsically photosensitive retinal ganglion cells (ipRGCs) are thought to play a role, how
138 These intrinsically photosensitive retinal ganglion cells (ipRGCs) have well-established roles in a
140 essing, intrinsically photosensitive retinal ganglion cells (ipRGCs) synchronize our biological clock
142 ressing intrinsically photosensitive retinal ganglion cells (ipRGCs), but the relevant downstream bra
145 romatic primate retina, the "midget" retinal ganglion cell is the classical substrate for red-green c
146 erve atrophy resulting from death of retinal ganglion cells is the most prominent ocular manifestatio
147 rgic synapses from AII amacrine cells to OFF ganglion cells) is sufficient for fast, mesopic rod-driv
148 ts was noted, with schitic spaces within the ganglion cell layer (13/17 eyes; 76.5%) observed to be p
149 nd that Cpne5, 6, and 9 are expressed in the ganglion cell layer (GCL) and inner nuclear layer (INL)
151 T) showed, in both eyes, a thickening of the ganglion cell layer (GCL) with a hyperreflective opacity
152 a, the retinal nerve fiber layer (RNFL), the ganglion cell layer (GCL), and the choroidal thickness (
153 , and superpixels thickness measurements for ganglion cell layer (GCL), ganglion cell/inner plexiform
154 papillary retinal nerve fiber layer, macular ganglion cell layer (mGCL), and macular inner plexiform
155 ers (retinal nerve fiber layer thickness and ganglion cell layer - inner plexiform layer thickness).
156 ganglion cell layer volume (GCL, p = 0.003), ganglion cell layer - inner plexiform layer volume (GCL-
157 ing cells have their soma exclusively in the ganglion cell layer and include a small proportion of bi
158 that specifically labels all neurons in the ganglion cell layer but is largely excluded from otherwi
159 red stimulation increased activity in cones, ganglion cell layer neurons, and cortical neurons, and e
160 ecies level was also elevated in the retinal ganglion cell layer of aged M(1) receptor-deficient mice
161 e analyzed: nerve fiber layer plexus (NFLP), ganglion cell layer plexus (GCLP), superficial vascular
162 ly correlated with time from surgery for the ganglion cell layer region of interest (R = -0.74, P < 0
163 een the resected brain tissue volume and the ganglion cell layer region of interest (R = -0.78, P < 0
166 es such as total retinal volume (p = 0.037), ganglion cell layer volume (GCL, p = 0.003), ganglion ce
167 he retinal nerve fiber layer and the retinal ganglion cell layer with spectral-domain optical coheren
169 M1-like cells typically had somas in the ganglion cell layer, with 23% displaced to the inner nuc
170 se retina, with alpha equaling ~0.050 in the ganglion cell layer, ~0.122 in the inner plexiform layer
171 ve fibre layer thickness (mRNFL) and macular ganglion cell layer-inner plexiform layer thickness were
175 ; (b) the outer plexiform, inner nuclear and ganglion cell layers are the strongest biomarkers for di
177 subpopulations in both the inner nuclear and ganglion cell layers, respectively, and to distinguish t
179 r, optic nerve crush injury-mediated retinal ganglion cell loss evokes neither peripheral monocyte re
180 M(1) receptor deficiency results in retinal ganglion cell loss in aged mice via involvement of oxida
183 lls may contribute to the melanopsin retinal ganglion cell loss previously described and to the distu
184 sual field defect and corresponding anatomic ganglion cell loss suggests a focal retinal injury.
185 , which is associated with increased retinal ganglion cell loss, retinal nerve fiber layer thinning,
186 ye disease characterized by death of retinal ganglion cells; lowering IOP is the only proven treatmen
187 ological studies showed that some OFF midget ganglion cells may receive sparse input from short (S)-w
188 or predictive ability of VF sensitivity from ganglion cell measurements may be applied to future mode
189 ds, cones, and melanopsin-containing retinal ganglion cells (mRGCs)-have been shown to provide electr
190 ion loss through the degeneration of retinal ganglion cell neurons and their axons in the optic nerve
192 o the brain by dimming-sensitive OFF retinal ganglion cells (OFF-RGCs) that respond to light decremen
193 r subtypes of ON direction-selective retinal ganglion cells (ON-DS RGCs), those preferring ventral re
194 on selectivity of On-Off direction-selective ganglion cells (On-Off DSGCs) against noisy backgrounds
195 FICANCE STATEMENT ON-OFF direction-selective ganglion cells (ooDSGCs) in the mammalian retina are typ
197 in peripapillary retinal nerve fiber layer, ganglion cell plus inner plexiform layer (GCIPL), whole-
198 ction of miR-223-3p in vivo in mouse retinal ganglion cells protects their axons from degeneration in
201 ckness near birth, implying that the retinal ganglion cells reserve is affected by intrauterine proce
202 , Sox2 and Klf4 genes (OSK) in mouse retinal ganglion cells restores youthful DNA methylation pattern
203 one, preserves vision and attenuates retinal ganglion cell (RGC) and axonal loss during EAE optic neu
206 action of Xenopus tadpoles, a single retinal ganglion cell (RGC) axon misprojects to the ipsilateral
207 In the developing mouse optic tract, retinal ganglion cell (RGC) axon position is organized by topogr
209 growth factor-1 (IGF1) by initiating retinal ganglion cell (RGC) axon regeneration after axotomy.
213 ieved to be the major contributor to retinal ganglion cell (RGC) death, the endpoint of optic neuropa
216 describes a novel paradigm to reduce retinal ganglion cell (RGC) degeneration underlying glaucoma.
217 nto cone bipolar cell (BC) axons and retinal ganglion cell (RGC) dendrites, but makes the majority of
218 of intraocular pressure (IOP) causes retinal ganglion cell (RGC) dysfunction and death and is a major
219 sion protein optic atrophy 1 (Opa1), retinal ganglion cell (RGC) dysfunction and visual loss occur by
220 The visual message conveyed by a retinal ganglion cell (RGC) is often summarized by its spatial r
221 mber of Nissl-stained neurons in the retinal ganglion cell (RGC) layer in the Caribbean and Chilean f
222 J2 (tie2-CYP2J2-Tr) protects against retinal ganglion cell (RGC) loss induced by glaucoma and in what
224 has previously been used to stratify Retinal Ganglion Cell (RGC) populations in histological samples
225 rodents, Rbfox2 is expressed in all retinal ganglion cell (RGC) subtypes, horizontal cells, as well
226 erged in which the three most common retinal ganglion cell (RGC) types captured much of the variance
227 l information is encoded in distinct retinal ganglion cell (RGC) types in the eye tuned to specific f
231 tablished role in the development of retinal ganglion cell (RGCs) types, the main transducers of visu
232 crease the background firing rate of retinal ganglion cells (RGCs) and overlay the stimulated respons
233 mitochondria-associated function in retinal ganglion cells (RGCs) and the resulting optic nerve rema
240 nostic ability of OCT parameters and retinal ganglion cells (RGCs) count in identify glaucomatous dis
245 aracterized by a progressive loss of retinal ganglion cells (RGCs) in the eye, which ultimately resul
247 unequivocally that a small subset of retinal ganglion cells (RGCs) project to the opposite retina and
248 r vertebrates, the circuit formed by retinal ganglion cells (RGCs) projecting ipsilaterally (iRGCs) o
252 nar (PIm) is innervated by widefield retinal ganglion cells (RGCs), and this pathway is not a collate
254 ine cells (SACs), propagating across retinal ganglion cells (RGCs), further through visual centers.
255 f optic nerve axons and apoptosis of retinal ganglion cells (RGCs), however, the precise mechanisms a
256 chronic neurodegenerative disease of retinal ganglion cells (RGCs), is a leading cause of irreversibl
257 odulate input from photoreceptors to retinal ganglion cells (RGCs), rendering each RGC type selective
259 ons, and synaptically transmitted to retinal ganglion cells (RGCs), which send information to the bra
260 ely even topographic distribution of retinal ganglion cells (RGCs)-the output neurons of the eye.
270 tomical evidence that two different types of ganglion cells share information via electrical coupling
271 s spatial resolution is lost at the level of ganglion cells.SIGNIFICANCE STATEMENT We make accurate m
273 ctivity distributed across feature-selective ganglion cells such that signals representing distinct s
274 esterase is demonstrated to increase retinal ganglion cell survival in vivo in mice of both sexes fol
277 ich exhibit elevated IOP and loss of retinal ganglion cells, Tek(+/-);Ptprb(+/-) mice have elevated T
279 from a distinct neuronal population, tectal ganglion cells (TGCs), of the optic tectum/superior coll
282 synaptic pathway from S cones to OFF midget ganglion cells through OFF midget bipolar cells remains
283 orm microcircuits with bipolar, amacrine and ganglion cells to process visual information in the inne
284 eye, followed by neural sampling by retinal ganglion cells, to demonstrate the perceptual effects of
285 w that M6 cells are by far the most abundant ganglion cell type labeled in adult pigmented Cdh3-GFP B
286 behaviors, receiving input from >30 retinal ganglion cell types and projecting to behaviorally impor
287 twork motif in which the signals of distinct ganglion cell types are partially mixed at the output st
292 ar (DB) types DB3a and DB3b (M pathway), and ganglion cells were counted along the temporal horizonta
294 fic subtypes of horizontal cells and retinal ganglion cells were overrepresented, suggesting that Thr
295 onfer intrinsic light sensitivity to retinal ganglion cells when photoreceptors have degenerated and
297 ibed in horizontal, OFF-bipolar, amacrine or ganglion cells, which could not be fully blocked in the
298 phologically distinct types of mouse retinal ganglion cells with overlapping excitatory synaptic inpu
300 ing to the predominant oxygenation status of ganglion cells within the superficial inner retina, whet