ライフサイエンスコーパス: gastric biopsy

1 receptor gamma rearrangement present in the gastric biopsies.

2 ith symptoms of gastritis, routinely undergo gastric biopsies.

3 to accurately diagnose H. pylori presence on gastric biopsies.

4 red formalin-fixed, paraffin-embedded (FFPE) gastric biopsies.

5 t esophagogastroduodenoscopy procedures with gastric biopsies.

6 plications, potentially reducing unnecessary gastric biopsies.

7 be applied on regular whole slide images of gastric biopsies.

8 pathologic analysis of random post-treatment gastric biopsies.

9 cytoplasm of all bacteria examined in human gastric biopsies.

10 se and HspB within bacteria present in human gastric biopsies.

11 Examination of an FD patient's gastric biopsy also revealed reduced and disoriented axo

12 ence of mucosal dysplasia is noted on random gastric biopsy and may serve as a histologic marker in t

13 Stool, gastric biopsy, and serum samples were collected from 22

14 d transmission electron microscopy of serial gastric biopsies at 0, 3, 5, 8, and 12 months.

15 ssue samples were obtained during sequential gastric biopsies beginning at 3 weeks postinoculation an

16 Pylori infection, proven by gastric biopsy, between AF patients and control group an

17 EPIYA motifs were detected in total DNA from gastric biopsies by PCR.

18 A node and gastric biopsy confirmed diffuse large-B-cell lymphoma,

19 From a cohort of 4-7 month-old monkeys, gastric biopsy cultures identified 44% of animals infect

20 ls of NOD1, CXCL8, IRF1, and CXCL10 in human gastric biopsies displaying severe gastritis, when compa

21 es, high-magnification endoscopy with random gastric biopsies, endoscopic ultrasonography, CT, and PE

22 The study included 428 gastric biopsies form dyspeptic patients, who did not re

23 ired formalin-fixed paraffin-embedded (FFPE) gastric biopsies from 100 H. pylori-positive patients wi

24 e United States who underwent esophageal and gastric biopsies from 2008 through 2010.

25 We obtained gastric biopsies from 30 patients in Australia.

26 D74 expression was increased dramatically on gastric biopsies from H. pylori-positive patients and ga

27 rDNA PCR fragment was amplified from 90% of gastric biopsies from histological H. pylori positive pa

28 Here we obtained gastric biopsies from multiple stomach regions of 16 H.

29 released by reductive beta-elimination from gastric biopsies from rhesus monkeys.

30 upper gastrointestinal endoscopy with random gastric biopsies, high-magnification endoscopy with rand

31 e was confirmed in 992 H. pylori isolates in gastric biopsy material from infected patients.

32 scopy due to various non-emergent causes had gastric biopsies obtained at three adjacent sites.

33 Gastric biopsies of 814 H. pylori infected patients naiv

34 Gastric biopsies of 814 patients infected with H. pylori

35 otype using a molecular approach directly on gastric biopsies of dyspeptic patients attending consecu

36 tions using a molecular approach directly on gastric biopsies of dyspeptic patients consecutively att

37 with clinical isolates and in vivo by using gastric biopsies of infected and noninfected individuals

38 We performed endoscopy with gastric biopsy on 15 healthy pet cats that were rigorous

39 y, VZV DNA and proteins were not detected in gastric biopsies or saliva.

40 isk MEN-1 patients, were correlated with the gastric biopsy results.

41 Gastric biopsies revealed chronic active gastritis with

42 Gastric biopsy samples (n = 6 EoG, n = 7 control) were s

43 shed by detection of CMV inclusion bodies in gastric biopsy samples and by hybridization with a CMV p

44 ed mucin oligosaccharides were released from gastric biopsy samples by beta-elimination and profiled

45 Gastric biopsy samples contained CD3(+) T cells that wer

46 We studied gastric biopsy samples from 78,985 unique patients.

47 Similar studies were performed on gastric biopsy samples from H. pylori-infected and uninf

48 Gastric biopsy samples from patients infected with VacA(

49 Gastric biopsy samples taken at baseline and 3, 6, 12, 1

50 Gastric biopsy samples were collected from patients with

51 ctive gastritis and intestinal metaplasia in gastric biopsy samples were inversely associated with Ba

52 38 803 total gastric biopsy samples were obtained, of which 1163 (3%)

53 as assessed by both histology and culture of gastric biopsy samples.

54 rophy with or without intestinal metaplasia, gastric biopsies should be obtained according to a syste

55 Multiple gastric biopsies showed a small-sized lymphoid infiltrat

56 A gastric biopsy specimen from each subject was tested for

57 Gastric biopsy specimens and blood samples from 185 subj

58 lserine receptor BAI1 was expressed in human gastric biopsy specimens and gastric phagocytes.

59 Gastric biopsy specimens and peripheral blood samples we

60 These results were then confirmed with gastric biopsy specimens and saliva from patients with c

61 heir ability to reliably detect H. pylori in gastric biopsy specimens and salivary samples.

62 Genomic DNA preparations from these gastric biopsy specimens and the corresponding H. pylori

63 tative culture and histologic examination of gastric biopsy specimens from 29 H. pylori-infected dysp

64 Mapped gastric biopsy specimens from 378 H. pylori-positive sub

65 ogen Helicobacter pylori, was quantitated in gastric biopsy specimens from 41 H. pylori-infected pati

66 dy, formalin-fixed, paraffin-embedded (FFPE) gastric biopsy specimens from a cohort of individuals fr

67 is study, 28 H. pylori strains isolated from gastric biopsy specimens from a high-gastric-cancer-risk

68 of disease, from gastritis to carcinoma, and gastric biopsy specimens from Colombian and Honduran coh

69 e in epithelial cell p27(kip1) expression in gastric biopsy specimens from H. pylori-infected patient

70 To test this, we analyzed gastric biopsy specimens from H. pylori-positive and -ne

71 hogenesis, eosinophils have been detected in gastric biopsy specimens from patients with AIG.

72 iption polymerase chain reaction analysis of gastric biopsy specimens from patients with and without

73 Gastric biopsy specimens from the stomach antrum and fun

74 The patterns from 13 of the 19 gastric biopsy specimens matched those of the H. pylori

75 Nineteen gastric biopsy specimens obtained from patients undergoi

76 Gastric biopsy specimens of 68 peptic ulcer disease (PUD

77 cross- sectional study was performed on 800 gastric biopsy specimens of cows, sheep, goats and human

78 immunohistochemistry on mononuclear cells in gastric biopsy specimens of infected but not uninfected

79 IL-18 levels in the gastric biopsy specimens showed similar patterns to thos

80 oarray screening of H. pylori-infected human gastric biopsy specimens to identify candidate genes inv

81 DNA from gastric biopsy specimens was analyzed similarly for comp

82 cter pylori using DNA isolated from infected gastric biopsy specimens was approximately equal to geno

83 that IL-17C expression in H. pylori-infected gastric biopsy specimens was predominantly localized to

84 RNA from 41 H. pylori-positive gastric biopsy specimens was reverse transcribed to cDNA

85 Four hundred two gastric biopsy specimens were analyzed using histopathol

86 Clinical data and gastric biopsy specimens were collected from 9 consecuti

87 Gastric biopsy specimens were collected from patients wh

88 One hundred twenty-six urease-negative gastric biopsy specimens were evaluated for the presence

89 Gastric biopsy specimens were examined by immunohistoche

90 2016 to January 2017, 133 H. pylori-infected gastric biopsy specimens were identified histologically

91 -1), KATO III cells, and cells isolated from gastric biopsy specimens were infected with H pylori.

92 med before, during, and after treatment, and gastric biopsy specimens were obtained for quantitative

93 One hundred twenty-eight gastric biopsy specimens were positive in genotyping PCR

94 distinguish H. pylori strains directly from gastric biopsy specimens without culture of the organism

95 icobacter species present in urease-negative gastric biopsy specimens, 16S rDNA amplicons were cloned

96 anscript profiles and histologic features of gastric biopsy specimens, as well as blood eosinophil co

97 Using cultured cell lines, gastric biopsy specimens, primary cells, and human enter

98 In human gastric biopsy specimens, the vacA i1 allele was strongl

99 rformed in H. pylori-infected and uninfected gastric biopsy specimens.

100 approximately 4.5-fold in H. pylori-infected gastric biopsy specimens.

101 isolates with known cagA status and in human gastric biopsy specimens.

102 ICC numbers were determined from gastric biopsy specimens.

103 sensitivities and specificities of >90% with gastric biopsy specimens.

104 concordance by culture and histology) coded gastric biopsy specimens.

105 ifferent patterns, respectively, from the 19 gastric biopsy specimens.

106 d for directly typing H. pylori strains from gastric biopsy specimens.

107 ary BE, EAC, normal esophageal squamous, and gastric biopsy tissues (n = 89).

108 in our practice, a judicious performance of gastric biopsies to detect gastric PCCs should be adopte

109 YP2C19 were determined on DNA extracted from gastric biopsies, using PCR.

110 sceptibility test results from pre-treatment gastric biopsies was a pre-condition for matching 52 aiH

111 usceptibility test results from pretreatment gastric biopsies was a precondition for matching 52 aiHp

112 H. pylori isolates obtained from endoscopic gastric biopsy was determined by using agar dilution.

113 Gastric biopsies were collected before treatment for det

114 Gastric biopsies were collected for quantitative culture

115 Gastric biopsies were obtained from patients and culture

116 Full thickness gastric biopsies were obtained laparoscopically from two

117 a 5-year period and included those for whom gastric biopsies were performed.

118 Gastric biopsies were taken by a gastroenterologist from

119 Six standard gastric biopsies were taken.

120 In the MALT lymphoma group, time-matched gastric biopsies were used as reference standard and sho

121 Patients with and without H pylori on gastric biopsy were compared, and odds of esophageal eos

122 Single-cell suspensions obtained by gastric biopsy were stimulated with phorbol 12,13-dibuty

123 s, thereby potentially reducing the need for gastric biopsies when test results are negative.

124 s and freshly isolated epithelial cells from gastric biopsies with specific antibodies.

125 d of their H pylori infection as assessed by gastric biopsy, with elimination of gastritis; median an