ライフサイエンスコーパス: gastric distension

1 rog/kg) administration, or CCK combined with gastric distension.

2 hindbrain through a mechanism that involves gastric distension.

3 n to a meal represent satiety or feelings of gastric distension.

4 ude hormones, lipid mediators, nutrients and gastric distension.

5 ake and attenuates the hypophagic effects of gastric distension.

6 t cause further pressure increases evoked by gastric distension.

7 through the esophageal orifice with minimal gastric distension.

8 ngth and pressure measured before and during gastric distension.

9 Gastric distension (30 ml) evoked LOS relaxation (70 +/-

10 mbly in decerebrate, unanaesthetized ferrets.Gastric distension (30 ml) evoked LOS relaxation (70 +/-

11 urons expressing GLUR2/3 was similar for the gastric distension (59.8-65.6%) and duodenal linoleic ac

12 Gastric distension (6 ml) evoked a much larger intragast

13 Gastric distension (6 ml) evoked an increase of 9.0 +/-

14 ercentage of neurons in the NTS activated by gastric distension (63.9+/-2.9%), linoleic acid (62.8+/-

15 The combination of gastric distension and cholecystokinin (CCK) enhances bo

16 Chitin consumption causes gastric distension and cytokine production by stomach tu

17 Gastric distension and duodenal nutrient stimuli produce

18 f differing gastric stability would increase gastric distension and reduce appetite compared with a c

19 Similar to in vivo studies, gastric distension caused a smaller increase in intragas

20 We hypothesize that gastric distension causes progressive effacement of the

21 Gastric distension causes progressive shortening of the

22 When combined, CCK and gastric distension enhanced Fos-LI in the DVC greater th

23 ous behavioral data, these results show that gastric distension enhances CCK-induced neuronal activat

24 esult in gastro-oesophageal reflux, that is, gastric distension-evoked lower oesophageal sphincter (L

25 esult in gastro-oesophageal reflux, that is, gastric distension-evoked lower oesophageal sphincter (L

26 aded decrease in the EGJ distensibility with gastric distension following fundoplication.

27 Graded gastric distensions (GD, 20, 40, 60 mm Hg, 20 s) were in

28 ease in intragastric pressure in response to gastric distension in the denervated, vascularly isolate

29 Likewise, ondansetron attenuated Fos-LI by gastric distension in the DVC, specifically within the n

30 inking (fully recapitulating the symptoms of gastric distension) in part via signalling to the parave

31 overall DVC enhanced Fos-LI induced by CCK + gastric distension, in particular at the NTS and AP nucl

32 NPY (0.03--3 nmol), significantly inhibited gastric distension-induced pyloric relaxation in a dose-

33 ch, acting through the Y1 receptor, inhibits gastric distension-induced pyloric relaxation in rats ex

34 Taken together, these findings suggest that gastric distension-induced pyloric relaxation is mediate

35 modulating the effects of hyperglycaemia on gastric distension-induced pyloric relaxation was invest

36 Gastric distension-induced pyloric relaxation was signif

37 se (3 micromol) also significantly inhibited gastric distension-induced pyloric relaxation without af

38 the inhibitory effects of hyperglycaemia on gastric distension-induced pyloric relaxation.

39 from 5.4 to 12.8 mM, significantly inhibited gastric distension-induced pyloric relaxation.

40 The effect of gastric distension on the EGJ was measured, before and a

41 s to assess in normal subjects the effect of gastric distension on the LES length and pressure and it

42 X) caused a significant pressure increase by gastric distension, reaching 17.0 +/- 1.7 cmH2O, suggest

43 The pressure increase evoked by gastric distension remained high 2 weeks after TV in viv

44 LD activation temporally coincident with the gastric distension stimulus.

45 , semirecumbent positioning, minimization of gastric distension, subglottic suctioning, avoidance of

46 rkedly increased vagal afferent responses to gastric distension that could be rescued with GABA(B) re

47 gs show an increase in the EGJ pressure with gastric distension that was 2-3 times higher than the in

48 The pressure increase evoked by gastric distension was significantly enhanced by L-NAME,

49 The pressure increase evoked by gastric distension was significantly enhanced in vivo by

50 After antral denervation, gastric distension with a non-nutrient solution is an ad

51 Progressive gastric distension with air resulted in progressive shor