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1 atient request or adverse effects, including gastric distention).
2  not acutely sensitize animals to subsequent gastric distention.
3 he human brain regions that are activated by gastric distention.
4 d by duodenal distention and were excited by gastric distention.
5 by duodenal distention and were inhibited by gastric distention.
6 al gastric lesions, thanks to the additional gastric distention.
7 on of the enteroenterostomy leading to acute gastric distention.
8                                  Symptomatic gastric distention activates structures implicated in so
9                          Hypersensitivity to gastric distention, an important feature of functional d
10 dation), interlobular septal thickening, and gastric distention and contents (fluid or sediment).
11                     Persistent, asymptomatic gastric distention can be seen in neonates within 48 hou
12 nine infants (both with initial asymptomatic gastric distention) developed feeding intolerance with p
13       The effect of systemic Delta(9)-THC on gastric distention-evoked LOS relaxation was reversed by
14         The effect of systemic delta9-THC on gastric distention-evoked LOS relaxation was reversed by
15 ), significantly attenuated the nadir of the gastric distention-evoked LOS relaxation, and time to re
16 g),significantly attenuated the nadir of the gastric distention-evoked LOS relaxation, and time to re
17 ophageal sphincter relaxation (tLESR) during gastric distention in patients with gastroesophageal ref
18                                              Gastric distention increased the frequency of reflux by
19  by the gastrointestinal system, focusing on gastric distention, intestinal and pancreatic satiation
20                                        Early gastric distention may be predictive of feeding intolera
21                                In all cases, gastric distention resolved with cessation of prostaglan
22 usefulness of the pneumo-computed tomography gastric distention technique in the detection and morpho
23 ealthy volunteers during baseline and distal gastric distentions to 10 mm Hg, 20 mm Hg, threshold pai
24  prostaglandin therapy, persistent (>3 days) gastric distention was noted in four of nine infants.
25 esophageal sphincter relaxations result from gastric distention, which shortens the sphincter to the