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1 and fusion during the secretory cycle of the gastric parietal cell.
2 and membrane remodeling processes typical of gastric parietal cells.
3 e membrane trafficking and acid secretion in gastric parietal cells.
4 uter medullary collecting duct (OMCD) and in gastric parietal cells.
5 ing tubulovesicles to the apical membrane of gastric parietal cells.
6 bition of the hydrogen/potassium pump in the gastric parietal cells.
7 dney outer medullary collecting duct and the gastric parietal cells.
8 tly expressed on the basolateral membrane of gastric parietal cells.
9 tially distributed to the apical membrane of gastric parietal cells.
10 egulators, IQGAP1 and IQGAP2, are present in gastric parietal cells.
11  that contain the H,K-ATPase in unstimulated gastric parietal cells.
12 ows for activation of polarized secretion in gastric parietal cells.
13 SCAR) located on the basolateral membrane of gastric parietal cells.
14 ly identified one such protein in studies of gastric parietal cells.
15 entified a 78 kDa AKAP which was enriched in gastric parietal cells.
16 bly blocking ATP4A, a P-type H+/K+ ATPase in gastric parietal cells.
17 roton pump responsible for acid secretion by gastric parietal cells.
18 (2% carbonyl iron), dietary iron deficiency (gastric parietal cell ablation), and HH (HFE -/-).
19  polarized distribution of actin isoforms in gastric parietal cells and association of gastric ezrin
20 at VacA permeabilizes the apical membrane of gastric parietal cells and induces hypochlorhydria.
21 ts demonstrate that ezrin is a major AKAP in gastric parietal cells and may function to tether type I
22 apical membrane-cytoskeletal interactions in gastric parietal cells and thereby causes hypochlorhydri
23 /K(+) ATPase characteristic of the mammalian gastric parietal cell, and the molecular mechanisms of a
24             The prevalence of positive serum gastric parietal cell antibody (PCA) was 61.8%.
25 se bearing a transgenic TCR specific for the gastric parietal cell antigen, H(+)K(+)-ATPase, to induc
26 AQP1 in intrahepatic cholangiocytes, AQP4 in gastric parietal cells, AQP3 and AQP4 in colonic surface
27                                              Gastric parietal cells are polarized epithelial cells in
28 dney collecting duct, airway epithelium, and gastric parietal cells, as well as in astrocytes and ske
29 iously, AKAP120 was identified from a rabbit gastric parietal cell cDNA library; however, a monoclona
30                                           In gastric parietal cells, cholinergically induced increase
31                           Acid secreted from gastric parietal cells determines mucosal injuries which
32 of acid-secreting cells similar to mammalian gastric parietal cells has been identified by a unique u
33 ody, using lysates of purified (>95%) canine gastric parietal cells in primary culture.
34 H-K-ATPase exclusively on the apical pole of gastric parietal cells in Slc26a9(-/-) mice, in contrast
35                        Acid secretion by the gastric parietal cell is regulated by paracrine, endocri
36                        The H,K-ATPase of the gastric parietal cell is the most critical component of
37                                      Loss of gastric parietal cells is a critical precursor to gastri
38 The H,K-adenosine triphosphatase (ATPase) of gastric parietal cells is targeted to a regulated membra
39 disorder characterized by the destruction of gastric parietal cells, leading to the loss of intrinsic
40 tivation of E-cadherin (Cdh1) and p53 in the gastric parietal cell lineage.
41 tiated cells such as functional hepatocytes, gastric parietal cells, or gut epithelial cells.
42                       Here, we show that the gastric parietal cell (PC) is a key cellular component o
43 ed the MHC class II molecule presentation of gastric parietal cell (PC)-specific H(+)/K(+)-ATPase, wh
44 2A), zinc transporter 8, thyroid peroxidase, gastric parietal cells (PCAs), tissue transglutaminase,
45  tamoxifen leads to apoptosis of >90% of all gastric parietal cells (PCs) and metaplasia of zymogenic
46 te that Hip1r is abundantly expressed in the gastric parietal cell, predominantly localizing with F-a
47     These mice develop autoantibodies to the gastric parietal cell proton pump, H/K ATPase, and aberr
48                               Stimulation of gastric parietal cells results in exocytic recruitment o
49     H+/K(+)-ATPase is the proton pump in the gastric parietal cell that is responsible for gastric ac
50                                          The gastric parietal cell was the first system where a regul
51  of Trpml1 and its role in acid secretion by gastric parietal cells were analyzed using biochemical,