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1 and controlling bleeding from esophageal and gastric varices.
2 ch, established on hemodynamic physiology of gastric varices.
3 enal malignancy (48 and 32 respectively) and gastric varices aetiologies (2.8) when compared with oth
4 s (aHR, 0.4; 95% CI, 0.2-0.9; P = 0.048), or gastric varices (aHR, 0.5; 95% CI, 0.3-0.7; P = 0.022) p
5           Five of the 12 patients had proved gastric varices and five were presumed to have varices o
6 TIPS) on portal hemodynamics, esophageal and gastric varices, and hepatic function have not been full
7 g, ascites, thrombocytopenia, esophageal and gastric varices, anemia, and increased levels of liver e
8                                              Gastric varices are encountered less frequently than eso
9 intestinal bleedings from ulcers or esophago-gastric varices are life threatening medical conditions
10                               Esophageal and gastric varices are the commonest endoscopic findings, w
11       Most patients bleed from esophageal or gastric varices, but bleeding from ectopic varices or po
12 he conventional and hemodynamic diagnosis of gastric varices concerning new classifications; explore
13 h actively bleeding esophageal or contiguous gastric varices despite sclerotherapy were assessed for
14 of radiology files revealed 86 patients with gastric varices diagnosed during double-contrast upper g
15                                       Fundic gastric varices failed to resolve in 6 of 12 cases.
16 ocedure for pediatric patients with bleeding gastric varices (GV) associated with advanced liver cirr
17                                              Gastric varices (GV) occur in 20% of patients with porta
18 oup, patients with ascites and/or esophageal/gastric varices had lower serum RvD1 levels compared to
19                              Conventionally, gastric varices have been described based on the locatio
20                                     Isolated gastric varices (IGV) may be located in the fundus (IGV1
21                                Bleeding from gastric varices is treated by injection with cyanoacryla
22                                     Tumorous gastric varices manifest as remarkably similar findings
23                                       Fundic gastric varices often fail to disappear after TIPS.
24 e of the characteristic features of tumorous gastric varices on double-contrast studies so that they
25 cksonville location with esophageal varices, gastric varices, or both seen on CT abdomen and pelvis,
26         In addition, a significant number of gastric varices, peri-esophageal varices, and extralumin
27                             In patients with gastric varices, primary prophylaxis with cyanoacrylate
28                             In patients with gastric varices, primary prophylaxis with cyanoacrylate
29 ents with EV regardless of the size and with gastric varices since these patients display clinically
30 dhesive agents for the treatment of bleeding gastric varices, the successful treatment of early gastr
31 rade 2 varices with red wale markings and/or gastric varices, treated consecutively from February 200
32           The sensitivity of CT in detecting gastric varices was 87%.
33 ers were invalid (n = 239), if esophageal or gastric varices were not reported (n = 25), varices othe
34                               Esophageal and gastric varices were prevalent in 311 (60.6%) and 109 (2
35                               Esophageal and gastric varices were significantly associated with reble
36 vity of CT scans in detecting esophageal and gastric varices when compared to the gold standard of EG
37 e esophageal varices (LEVs) in 60 (40%), and gastric varices without LEVs in 11 (7%).