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1                                              Gastroesophageal acid reflux at each of the pH sensors e
2  specific therapy for patients with advanced gastroesophageal adenocarcinoma (GEA).
3            Patients with advanced gastric or gastroesophageal adenocarcinoma need more efficacious an
4 Chemoradiation After Resection of Gastric or Gastroesophageal Adenocarcinoma) assessed whether a post
5 atients with advanced, untreated gastric, or gastroesophageal adenocarcinoma.
6 rognostic factor in patients with resectable gastroesophageal adenocarcinomas with and without neoadj
7 nset in 6 of 16 patients (38%): prostate and gastroesophageal adenocarcinomas, myeloma, melanoma, col
8 ce of the ischemia after gastrointestinal or gastroesophageal anastomosis.
9                        Children experiencing gastroesophageal and extraesophageal reflux are often pr
10   Amplification of the MET proto-oncogene in gastroesophageal cancer (GEC) may constitute a molecular
11              Many miRNAs are dysregulated in gastroesophageal cancer cells via alterations in transcr
12 ative chemotherapy plus surgery for operable gastroesophageal cancer from July 1, 1994, through April
13 ) expression may help to stratify breast and gastroesophageal cancer patients for HER2-targeting ther
14 SI, and survival in patients with resectable gastroesophageal cancer randomized to surgery alone or p
15                                              Gastroesophageal cancer resections are associated with s
16 to reintervene and rescue patients following gastroesophageal cancer resections in England.
17 cer, head and neck squamous cell carcinomas, gastroesophageal cancer, and anal cancer).
18                             The incidence of gastroesophageal cancers is increasing each year, but de
19                         miRNAs contribute to gastroesophageal carcinogenesis by altering expression o
20 ater understanding of the roles of miRNAs in gastroesophageal carcinogenesis could provide insights i
21 oncoding RNAs that have been associated with gastroesophageal carcinogenesis.
22  correlate with the worst prognosis of human gastroesophageal carcinoma.
23 tiated neoplasms, which are similar to human gastroesophageal carcinoma.
24 s study aimed to determine the prevalence of gastroesophageal disease (GERD) and extraesophageal mani
25 PIs) are used for the long-term treatment of gastroesophageal disorders and the non-prescription medi
26                It is used off-label to treat gastroesophageal, endometrial, cervical, prostate, and h
27                                              Gastroesophageal function was assessed by clinical valid
28                                              Gastroesophageal function was evaluated using a clinical
29 sease-specific survival (DSS) for resectable gastroesophageal (GE) junction and gastric adenocarcinom
30 chemotherapy regimen for advanced gastric or gastroesophageal junction (GEJ) adenocarcinoma and is as
31 iate esophageal versus gastric epithelium in gastroesophageal junction (GEJ) biopsies, the histology
32  be valid treatment options in patients with gastroesophageal junction (GEJ) cancer.
33 Lewis esophagectomy (TTIL) for esophageal or gastroesophageal junction (GEJ) cancer.
34 ction of stage IB through IV (M0) gastric or gastroesophageal junction adenocarcinoma were randomly a
35 gnosis in patients with distal esophageal or gastroesophageal junction adenocarcinoma who have receiv
36 rpose After curative resection of gastric or gastroesophageal junction adenocarcinoma, Intergroup Tri
37 ion After a curative resection of gastric or gastroesophageal junction adenocarcinoma, postoperative
38 nts with resectable or metastatic gastric or gastroesophageal junction adenocarcinoma.
39  radiochemotherapy for high risk gastric and gastroesophageal junction adenocarcinoma: Demonstrated s
40 veness in reducing mortality from esophageal/gastroesophageal junction adenocarcinomas has not been e
41 ed by gastroesophageal reflux disease in the gastroesophageal junction and associated with tumorigene
42 ined as eradication of IM (in esophageal and gastroesophageal junction biopsy specimens), documented
43 reviously untreated metastatic esophageal or gastroesophageal junction cancer were randomly assigned
44 otherapy for locally advanced esophageal and gastroesophageal junction cancers is based on a few posi
45             Implantation of a MSA around the gastroesophageal junction has been shown to be a safe an
46 ith adenocarcinoma of the gastric cardia and gastroesophageal junction have no history of reflux.
47                               We studied the gastroesophageal junction in asymptomatic volunteers wit
48 the lower frequency of tumors arising at the gastroesophageal junction in comparison with distal gast
49  carcinomas arising in the stomach or in the gastroesophageal junction in patients with HER2-positive
50                        Adenocarcinoma of the gastroesophageal junction is an aggressive disease, with
51 tients with resectable adenocarcinoma of the gastroesophageal junction treated with or without neoadj
52 thoracic esophageal carcinoma, including the gastroesophageal junction tumors (Siewert I), were rando
53  cancer localized in the distal esophagus or gastroesophageal junction undergoing McKeown TMIE or Ivo
54 ble adenocarcinoma of the lower esophagus or gastroesophageal junction were reviewed.
55                        Manometric values of "gastroesophageal junction" significantly increased at 12
56 c oesophagogastric (gastric, oesophageal, or gastroesophageal junction) cancer.
57 py of local and locally advanced esophageal, gastroesophageal junction, and gastric adenocarcinomas,
58 lumnar junction and 3) 5-10 mm distal to the gastroesophageal junction, as demarcated by the top of t
59 metastatic adenocarcinoma of the stomach and gastroesophageal junction.
60  correlated with the (18)F-FDG uptake at the gastroesophageal junction.
61 asia seen in some cases, particularly at the gastroesophageal junction.
62 ogy, and recurrence patterns associated with gastroesophageal malignancies suggest the need to split
63  with advanced lung, colorectal, pancreatic, gastroesophageal, or breast cancer between 1998 and 2005
64 doscopic capture from patients with gastric, gastroesophageal, or esophageal cancer who are administe
65  moderate to severe abdominal pain, a severe gastroesophageal reflex, and moderate to severe depressi
66  point), obstructive sleep apnea (+1 point), gastroesophageal reflux (+1 point), and depression (+1 p
67 omorbidities were allergic rhinitis (62.4%), gastroesophageal reflux (42.1%), sinusitis (37.9%), nasa
68                          The contribution of gastroesophageal reflux (GER) and vagal function to esop
69          Dental erosion is a complication of gastroesophageal reflux (GER) in adults; in children, it
70                                              Gastroesophageal reflux (GER) is a disorder that is comm
71              The prevalence of abnormal acid gastroesophageal reflux (GER) is higher in patients with
72                                 Asymptomatic gastroesophageal reflux (GER) is prevalent in children w
73 f a disease engendered more controversy than gastroesophageal reflux (GER), a highly prevalent condit
74 sts with erosive tooth wear have significant gastroesophageal reflux (GERD), despite minor reflux sym
75 ocephaly, developed intellectual disability, gastroesophageal reflux and a seizure disorder.
76            There were 7 million diagnoses of gastroesophageal reflux and almost 4 million diagnoses o
77 tissue remodeling is associated with chronic gastroesophageal reflux and constitutes a premalignant l
78                BE is associated with chronic gastroesophageal reflux and esophageal cancer.
79 because of both mechanical effects promoting gastroesophageal reflux and nonmechanical effects.
80 also significant improvements in symptoms of gastroesophageal reflux and rhinitis, bronchial reversib
81 index greater than 4 on pH monitoring and/or gastroesophageal reflux and/or herniated wrap on upper g
82                    Nissen fundoplication for gastroesophageal reflux can be followed by troublesome s
83 ng bowel habit (15%), constipation (13%) and gastroesophageal reflux disease (12%).
84 lipidemia (24.5%), hypertension (21.9%), and gastroesophageal reflux disease (17.3%).
85                              The presence of gastroesophageal reflux disease (26%), esophageal strict
86 ere rhinitis (84.0%), sinusitis (47.8%), and gastroesophageal reflux disease (46.3%).
87 gus (n = 1059) with those from subjects with gastroesophageal reflux disease (gastroesophageal reflux
88 .1; 95% confidence interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI,
89 lin, leptin, and ghrelin are associated with gastroesophageal reflux disease (GERD) and Barrett's eso
90 and evaluate esophageal disorders, including gastroesophageal reflux disease (GERD) and eosinophilic
91              Skinner to the understanding of gastroesophageal reflux disease (GERD) and its complicat
92  might represent an alternative treatment of gastroesophageal reflux disease (GERD) and may provide d
93 agement of extraesophageal manifestations of gastroesophageal reflux disease (GERD) and to compare th
94                 Current diagnostic tests for gastroesophageal reflux disease (GERD) are suboptimal an
95                                Management of gastroesophageal reflux disease (GERD) commonly starts w
96 .4)] were included; 70% had been treated for gastroesophageal reflux disease (GERD) during infancy.
97  surgery, highly variable rates of recurrent gastroesophageal reflux disease (GERD) have been reporte
98 The histologic changes associated with acute gastroesophageal reflux disease (GERD) have not been stu
99                            The prevalence of gastroesophageal reflux disease (GERD) in Africa is not
100         The aim was to compare recurrence of gastroesophageal reflux disease (GERD) in children rando
101 RAs) are frequently used in the treatment of gastroesophageal reflux disease (GERD) in children; howe
102 n (TF) can decrease or eliminate features of gastroesophageal reflux disease (GERD) in some patients
103      There are few data on the prevalence of gastroesophageal reflux disease (GERD) in the United Sta
104                                              Gastroesophageal reflux disease (GERD) increases EAC ris
105                                              Gastroesophageal reflux disease (GERD) is a common comor
106                                              Gastroesophageal reflux disease (GERD) is a common diagn
107                   It has been suggested that gastroesophageal reflux disease (GERD) is a risk factor
108                                              Gastroesophageal reflux disease (GERD) is associated wit
109                     Endoscopic management of gastroesophageal reflux disease (GERD) is being employed
110                                              Gastroesophageal reflux disease (GERD) is caused by gast
111                          The pathogenesis of gastroesophageal reflux disease (GERD) is complex and in
112                                              Gastroesophageal reflux disease (GERD) is defined by rec
113  illustrated by the Montreal classification, gastroesophageal reflux disease (GERD) is much more than
114                                              Gastroesophageal reflux disease (GERD) is prevalent worl
115 ump inhibitor (PPI) therapy in patients with gastroesophageal reflux disease (GERD) is reported in up
116                                              Gastroesophageal reflux disease (GERD) is the most commo
117                                              Gastroesophageal reflux disease (GERD) is the most preva
118                                              Gastroesophageal reflux disease (GERD) is the strongest
119                        Many individuals with gastroesophageal reflux disease (GERD) never visit their
120 f an underlying acid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it pre
121                                   Refractory gastroesophageal reflux disease (GERD) reduces quality o
122                                              Gastroesophageal reflux disease (GERD) seems to increase
123  has been developed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite
124 tailed critique of objective measurements of gastroesophageal reflux disease (GERD) would improve man
125 BS), 3.0; dyspepsia, 1.8; constipation, 3.9; gastroesophageal reflux disease (GERD), 9.7.
126 cluding Crohn disease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic
127 frequency of irritable bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive b
128 rm use of PPIs for three common indications: gastroesophageal reflux disease (GERD), Barrett's esopha
129 this study was to appraise the prevalence of gastroesophageal reflux disease (GERD), esophagitis, and
130 n several pathophysiological states, such as gastroesophageal reflux disease (GERD), functional dyspe
131                                              Gastroesophageal reflux disease (GERD), functional dyspe
132         Barrett esophagus, a complication of gastroesophageal reflux disease (GERD), predisposes pati
133 ed the accuracy of frequency and duration of gastroesophageal reflux disease (GERD), using data from
134                                              Gastroesophageal reflux disease (GERD), which leads to a
135                                       Unlike gastroesophageal reflux disease (GERD), whose symptoms c
136 can individuals have a similar prevalence of gastroesophageal reflux disease (GERD), yet esophageal a
137  defined as a decrease of 50% or more in the Gastroesophageal Reflux Disease (GERD)-Health Related Qu
138  procedure of choice for obese patients with gastroesophageal reflux disease (GERD).
139 y risk factors for both BE and EA is chronic gastroesophageal reflux disease (GERD).
140 responders and treatment-naive patients with gastroesophageal reflux disease (GERD).
141 n accurate method for diagnosing adults with gastroesophageal reflux disease (GERD).
142 s for a range of medical disorders including gastroesophageal reflux disease (GERD).
143 only used in the diagnosis and management of gastroesophageal reflux disease (GERD).
144 ctional gastrointestinal disorders (FGD) and gastroesophageal reflux disease (GERD).
145 a novel device for the surgical treatment of gastroesophageal reflux disease (GERD).
146 halasia, eosinophilic esophagitis (EoE), and gastroesophageal reflux disease (GERD).
147 one combination vs omeprazole monotherapy in gastroesophageal reflux disease (GERD).
148 en fundoplication (CNF) for the treatment of gastroesophageal reflux disease (GERD).
149 dal anti-inflammatory drugs) and symptoms of gastroesophageal reflux disease (GERD).
150 urgical option for patients with early-stage gastroesophageal reflux disease (GERD).
151            We also included 10 patients with gastroesophageal reflux disease (GERD; age, 32-60 y; 7 w
152                                              Gastroesophageal reflux disease (GORD) is a chronic and
153 nths after fundoplication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF
154 ad EoE (n = 17), indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esoph
155 onitoring on therapy in patients with proven gastroesophageal reflux disease [GERD]), to document phy
156 ether patient-identified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting,
157 er improvement in quality of life related to gastroesophageal reflux disease and a 50% or greater red
158 patterns from biopsies of patients with EoE, gastroesophageal reflux disease and controls.
159 ulosis is associated with conditions such as gastroesophageal reflux disease and diabetes mellitus, a
160 allenges to diagnosis, including the role of gastroesophageal reflux disease and proton pump inhibito
161  75-year-old woman with a medical history of gastroesophageal reflux disease and type II diabetes pre
162   The high reoperation rate and incidence of gastroesophageal reflux disease are concerning.
163 pump inhibitors and has focused attention on gastroesophageal reflux disease as a causative factor in
164  We prospectively assessed 100 patients with gastroesophageal reflux disease before and after sphinct
165 iteria consisted of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophagea
166 , screening 50-year-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost ef
167  emerging disease that is distinguished from gastroesophageal reflux disease by the expression of a u
168  suppression, have been confirmed in EoE and gastroesophageal reflux disease cell cultures.
169  95% confidence interval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95%
170 bjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and
171       Early aggressive surgical treatment of gastroesophageal reflux disease decreases the rate of br
172 es for detailed drugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive
173 ugs in 8 drug classes (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, a
174                           Most patients with gastroesophageal reflux disease experience symptomatic r
175 rm outcome of ARS in pediatric patients with gastroesophageal reflux disease have shown good to excel
176  months improved in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality o
177 imary laparoscopic antireflux surgery due to gastroesophageal reflux disease in adults (>18 years).
178 ed strong genetic correlations of BE/EA with gastroesophageal reflux disease in male individuals and
179  related to the acidic environment caused by gastroesophageal reflux disease in the gastroesophageal
180                                              Gastroesophageal reflux disease is a highly prevalent di
181 is (EoE) was historically distinguished from gastroesophageal reflux disease on the basis of histolog
182 ated with EoE (or indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and
183        Diagnosis of GERD was by means of the gastroesophageal reflux disease questionnaire (GERDQ) wh
184 ted with a relatively high rate of recurrent gastroesophageal reflux disease requiring treatment, dim
185       Symptoms improved in group A, with the Gastroesophageal Reflux Disease Symptom Assessment Scale
186  men in the United Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the p
187                               The history of gastroesophageal reflux disease was highly prevalent in
188                                              Gastroesophageal reflux disease was identified as an imp
189                                      De novo gastroesophageal reflux disease was reported in 43.8%.
190 screening ages for patients with symptomatic gastroesophageal reflux disease were older (58 for men a
191 as chronic sinusitis, allergic rhinitis, and gastroesophageal reflux disease were only associated wit
192                                Patients with gastroesophageal reflux disease who have a partial respo
193 structive sleep apnea may be associated with gastroesophageal reflux disease, a strong risk factor fo
194 allergic rhinitis, chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep
195              Risk factors evaluated included gastroesophageal reflux disease, alcohol consumption, sm
196 ibly due to increased abdominal pressure and gastroesophageal reflux disease, although this pathogeni
197 lization for asthma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other
198 lation), and prior diagnoses (eg, pneumonia, gastroesophageal reflux disease, and other comorbidities
199  the individual to irritable bowel syndrome, gastroesophageal reflux disease, and peptic ulcer diseas
200  factors for EAC have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, ob
201 shown to be a safe and effective therapy for gastroesophageal reflux disease, but its effect on the L
202                         Risk factors include gastroesophageal reflux disease, central obesity, and sm
203  terms: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
204 lowing: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
205 l gastrointestinal complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Ba
206   This issue provides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis,
207 on airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunction
208 , including history of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal ble
209  premorbid prevalence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infect
210  44 patients (52%) had objective findings of gastroesophageal reflux disease, such as esophagitis.
211                                 Triggered by gastroesophageal reflux disease, the origin of this meta
212 ic conditions (hypertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabet
213                                   Except for gastroesophageal reflux disease, which showed a higher r
214 ministered a modified version of a validated gastroesophageal reflux disease-specific QOL tool to pat
215             Quality of life, measured by the gastroesophageal reflux disease-specific QOL tool, and r
216 ery effective and long-lasting treatment for gastroesophageal reflux disease.
217         Obesity is associated with a risk of gastroesophageal reflux disease.
218 llow-up after ARS in pediatric patients with gastroesophageal reflux disease.
219 180-degree LAF for the surgical treatment of gastroesophageal reflux disease.
220 st frequently performed surgical therapy for gastroesophageal reflux disease.
221 ved cells is a novel potential treatment for gastroesophageal reflux disease.
222 drome (IBS) frequently overlap with those of gastroesophageal reflux disease.
223 gents widely prescribed for the treatment of gastroesophageal reflux disease.
224  to evaluate for the presence of concomitant gastroesophageal reflux disease.
225  thereby conclusively distinguishing it from gastroesophageal reflux disease.
226 fundoplication is an effective treatment for gastroesophageal reflux disease.
227 places esophageal squamous mucosa damaged by gastroesophageal reflux disease.
228 is a laryngeal symptom that can be caused by gastroesophageal reflux disease; however, treatment outc
229 cough may have abnormal proximal exposure to gastroesophageal reflux documented by HMII that would ha
230  sphincter (UES) to simulated or spontaneous gastroesophageal reflux have shown conflicting results.
231                 Subjects reporting nocturnal gastroesophageal reflux in both 1999 and 2010 had more N
232 he diagnosis of cough, LPR, or asthma due to gastroesophageal reflux is difficult, as no criterion st
233                                              Gastroesophageal reflux is the main risk factor for esop
234  allograft injury from donor-directed Abs or gastroesophageal reflux led to new ColV and KAT Abs post
235                                           As gastroesophageal reflux persists or recurs in 43% of chi
236             There is significant though mild gastroesophageal reflux postoperatively in 46% of patien
237 rative complications, sleep difficulties and gastroesophageal reflux progressively worsened during fo
238 ireflux surgery, 17.7% experienced recurrent gastroesophageal reflux requiring long-term medication u
239  poorly described, and the risk of worsening gastroesophageal reflux requiring revision may be higher
240                                              Gastroesophageal reflux scores were higher in overweight
241 t questionnaire was considered diagnostic of gastroesophageal reflux symptoms (GERD).
242 based on the modified Rome III criteria) and gastroesophageal reflux symptoms (GERS) in a population-
243                           Apoptosis pathway, gastroesophageal reflux symptoms (reflux), higher body m
244 eep apnea and to determine whether nocturnal gastroesophageal reflux symptoms affect the relationship
245 estioned regarding severity of their typical gastroesophageal reflux symptoms and presence of nocturn
246 nal GERD in 1999 (>/=3 episodes of nocturnal gastroesophageal reflux symptoms per week) had an OR of
247  Barrett's esophagus patients with nocturnal gastroesophageal reflux symptoms should be further evalu
248 's esophagus was associated with more severe gastroesophageal reflux symptoms, and nocturnal reflux s
249 al reflux symptoms and presence of nocturnal gastroesophageal reflux symptoms.
250 ymptom control in up to 50% of patients with gastroesophageal reflux symptoms.
251                                  The rate of gastroesophageal reflux was comparable with prior studie
252                                              Gastroesophageal reflux was defined by abnormal pH-testi
253                                 At 6 months, gastroesophageal reflux was evaluated by 24-hour pH test
254                                              Gastroesophageal reflux was more common among patients w
255                                              Gastroesophageal reflux was reported in 50.0% (40 vs 27
256                        Objective evidence of gastroesophageal reflux was seen in 46% patients postope
257                                              Gastroesophageal reflux was the most common GI diagnosis
258                        Chronic sinusitis and gastroesophageal reflux were also associated with exacer
259         From 1995 to 2003, 461 patients with gastroesophageal reflux were enrolled in 4 randomized co
260 s have failed to demonstrate that preventing gastroesophageal reflux with antireflux surgery halts th
261 f unproven efficacy and focus on controlling gastroesophageal reflux with reflux-reducing medication
262                             Infantile colic, gastroesophageal reflux, and constipation are the most c
263 pse include neck flexion, airway secretions, gastroesophageal reflux, and sleep deprivation.
264 is used variably to prevent complications of gastroesophageal reflux, but its effectiveness is unprov
265 trophy (SMA) notes that patients suffer from gastroesophageal reflux, constipation and delayed gastri
266 discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstructive sleep apnoea, vocal
267 in the postoperative period from SSc-related gastroesophageal reflux, renal impairment, and skin fibr
268 , sex, percent predicted FEV1, self-reported gastroesophageal reflux, St. George's Respiratory Questi
269 pump inhibitors (PPIs) are popular drugs for gastroesophageal reflux, which are now available for lon
270  BACKGROUND & AIMS: Central obesity promotes gastroesophageal reflux, which may be related to increas
271  esophagus and may work synergistically with gastroesophageal reflux.
272  criteria and two with findings secondary to gastroesophageal reflux.
273  leaks and strictures, gastric dilation, and gastroesophageal reflux.
274 , abdominal pain, diarrhea, weight loss, and gastroesophageal reflux.
275  Barrett's esophagus segments and persistent gastroesophageal reflux.
276  frequently performed surgical procedure for gastroesophageal reflux.
277 fundoplication for the surgical treatment of gastroesophageal reflux.
278  choanal atresia repair, and/or treatment of gastroesophageal reflux.
279 gher scores indicating better function), and gastroesophageal reflux.
280 n, poor overall health, home oxygen use, and gastroesophageal reflux.
281                                              Gastroesophageal reflux/dysphagia and asthma/rhinitis re
282                                          For gastroesophageal resections combined, adjusted mortality
283 nclude younger age at presentation, previous gastroesophageal surgery or ulcers as an etiology, and a
284                                 Each type of gastroesophageal tumor has a unique gene expression prof
285 pes of cancer, including HER2-overexpressing gastroesophageal tumors.
286 y is common in cirrhotic patients with acute gastroesophageal variceal bleeding and is an independent
287 a lower rate of abdominal and chest varices, gastroesophageal variceal bleeding and refractory ascite
288 cotropin stimulation test in 157 episodes of gastroesophageal variceal bleeding in 143 patients with
289                                              Gastroesophageal variceal bleeding in patients with cirr
290  We present a case of a patient with massive gastroesophageal variceal bleeding refractory to numerou
291 utcomes of patients with cirrhosis and acute gastroesophageal variceal bleeding remains unknown.
292 mulation test in patients with cirrhosis and gastroesophageal variceal bleeding.
293 ich may induce lethal complications, such as gastroesophageal variceal hemorrhage and hepatic encepha
294  clinical trial to assess the development of gastroesophageal varices (GEV).
295       The accurate prediction of the risk of gastroesophageal varices could spare unnecessary endosco
296                    We assessed the course of gastroesophageal varices in a large cohort of patients w
297 eeded to eradicate varices; no bleeding from gastroesophageal varices was observed after eradication.
298 tension (PH) has been exclusively devoted to gastroesophageal varices-related events at different fra
299 ch ultimately causes portal hypertension and gastroesophageal varices.
300  children with biliary atresia and high-risk gastroesophageal varices.

 
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