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1 , abdominal pain, diarrhea, weight loss, and gastroesophageal reflux.
2 esophagus and may work synergistically with gastroesophageal reflux.
3 criteria and two with findings secondary to gastroesophageal reflux.
4 leaks and strictures, gastric dilation, and gastroesophageal reflux.
5 Barrett's esophagus segments and persistent gastroesophageal reflux.
6 frequently performed surgical procedure for gastroesophageal reflux.
7 fundoplication for the surgical treatment of gastroesophageal reflux.
8 choanal atresia repair, and/or treatment of gastroesophageal reflux.
9 gher scores indicating better function), and gastroesophageal reflux.
10 ergone a fundoplication for the treatment of gastroesophageal reflux.
11 The symptoms are often confused for those of gastroesophageal reflux.
12 esophageal diseases result from uncontrolled gastroesophageal reflux.
13 hincter relaxation is the main mechanism for gastroesophageal reflux.
14 sociated with a two-to threefold increase in gastroesophageal reflux.
15 n, poor overall health, home oxygen use, and gastroesophageal reflux.
16 point), obstructive sleep apnea (+1 point), gastroesophageal reflux (+1 point), and depression (+1 p
17 omorbidities were allergic rhinitis (62.4%), gastroesophageal reflux (42.1%), sinusitis (37.9%), nasa
20 tissue remodeling is associated with chronic gastroesophageal reflux and constitutes a premalignant l
23 also significant improvements in symptoms of gastroesophageal reflux and rhinitis, bronchial reversib
24 wn a positive correlation between pathologic gastroesophageal reflux and those with hiatal hernia or
25 index greater than 4 on pH monitoring and/or gastroesophageal reflux and/or herniated wrap on upper g
27 y causes symptoms similar to those seen with gastroesophageal reflux, and is characterized by increas
29 ralized anxiety disorder, diabetes mellitus, gastroesophageal reflux, bacterial infection, and bone m
30 is used variably to prevent complications of gastroesophageal reflux, but its effectiveness is unprov
31 ment of patients who fail medical therapy of gastroesophageal reflux, but recurrent gastroesophageal
34 trophy (SMA) notes that patients suffer from gastroesophageal reflux, constipation and delayed gastri
39 gus (n = 1059) with those from subjects with gastroesophageal reflux disease (gastroesophageal reflux
40 .1; 95% confidence interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI,
41 lin, leptin, and ghrelin are associated with gastroesophageal reflux disease (GERD) and Barrett's eso
42 We examined the incidence and predictors of gastroesophageal reflux disease (GERD) and dyspepsia and
43 and evaluate esophageal disorders, including gastroesophageal reflux disease (GERD) and eosinophilic
45 might represent an alternative treatment of gastroesophageal reflux disease (GERD) and may provide d
46 has enormous potential for the treatment of gastroesophageal reflux disease (GERD) and other esophag
48 agement of extraesophageal manifestations of gastroesophageal reflux disease (GERD) and to compare th
49 Lung transplant recipients with documented gastroesophageal reflux disease (GERD) are at increased
51 hageal eosinophilia who were thought to have gastroesophageal reflux disease (GERD) but who did not r
53 t clear why only a minority of patients with gastroesophageal reflux disease (GERD) develop Barrett's
54 .4)] were included; 70% had been treated for gastroesophageal reflux disease (GERD) during infancy.
56 surgery, highly variable rates of recurrent gastroesophageal reflux disease (GERD) have been reporte
57 The histologic changes associated with acute gastroesophageal reflux disease (GERD) have not been stu
60 RAs) are frequently used in the treatment of gastroesophageal reflux disease (GERD) in children; howe
61 ull-thickness plication for the treatment of gastroesophageal reflux disease (GERD) in comparison wit
62 n (TF) can decrease or eliminate features of gastroesophageal reflux disease (GERD) in some patients
76 illustrated by the Montreal classification, gastroesophageal reflux disease (GERD) is much more than
78 ump inhibitor (PPI) therapy in patients with gastroesophageal reflux disease (GERD) is reported in up
83 f an underlying acid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it pre
87 has been developed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite
90 phagus (cases) were matched to subjects with gastroesophageal reflux disease (GERD) without Barrett's
91 tailed critique of objective measurements of gastroesophageal reflux disease (GERD) would improve man
93 cluding Crohn disease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic
94 frequency of irritable bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive b
95 e upper airways dysfunction syndrome (RUDS), gastroesophageal reflux disease (GERD), and rare cases o
96 rm use of PPIs for three common indications: gastroesophageal reflux disease (GERD), Barrett's esopha
97 this study was to appraise the prevalence of gastroesophageal reflux disease (GERD), esophagitis, and
98 n several pathophysiological states, such as gastroesophageal reflux disease (GERD), functional dyspe
101 ients, who had a previous fundoplication for gastroesophageal reflux disease (GERD), underwent reoper
102 ed the accuracy of frequency and duration of gastroesophageal reflux disease (GERD), using data from
105 can individuals have a similar prevalence of gastroesophageal reflux disease (GERD), yet esophageal a
106 defined as a decrease of 50% or more in the Gastroesophageal Reflux Disease (GERD)-Health Related Qu
124 cations in adults were dysphagia (70.1%) and gastroesophageal reflux disease (GERD)/heartburn (27.1%)
126 n 2002 and 2005 were matched to persons with gastroesophageal reflux disease (GERD; n = 316) and to p
128 nths after fundoplication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF
129 (n = 296 cases) were matched to persons with gastroesophageal reflux disease (n = 308) without Barret
130 ad EoE (n = 17), indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esoph
132 onitoring on therapy in patients with proven gastroesophageal reflux disease [GERD]), to document phy
133 ether patient-identified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting,
134 uency, severity, and duration of symptoms of gastroesophageal reflux disease among randomly selected
135 er improvement in quality of life related to gastroesophageal reflux disease and a 50% or greater red
136 rs to result in decreased prevalence of both gastroesophageal reflux disease and adenocarcinoma of th
138 ulosis is associated with conditions such as gastroesophageal reflux disease and diabetes mellitus, a
139 allenges to diagnosis, including the role of gastroesophageal reflux disease and proton pump inhibito
140 75-year-old woman with a medical history of gastroesophageal reflux disease and type II diabetes pre
141 e mechanisms involved in the pathogenesis of gastroesophageal reflux disease are complex and multifac
143 pump inhibitors and has focused attention on gastroesophageal reflux disease as a causative factor in
144 We prospectively assessed 100 patients with gastroesophageal reflux disease before and after sphinct
145 iteria consisted of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophagea
146 , screening 50-year-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost ef
147 emerging disease that is distinguished from gastroesophageal reflux disease by the expression of a u
151 search into the use of these technologies in gastroesophageal reflux disease continues to accelerate,
152 95% confidence interval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95%
153 bjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and
155 es for detailed drugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive
156 ugs in 8 drug classes (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, a
157 ecome available in the last 2 years for many gastroesophageal reflux disease endotherapies, providing
159 rm outcome of ARS in pediatric patients with gastroesophageal reflux disease have shown good to excel
160 months improved in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality o
161 imary laparoscopic antireflux surgery due to gastroesophageal reflux disease in adults (>18 years).
163 ed strong genetic correlations of BE/EA with gastroesophageal reflux disease in male individuals and
164 re of the height in meters - and symptoms of gastroesophageal reflux disease in persons of normal wei
165 related to the acidic environment caused by gastroesophageal reflux disease in the gastroesophageal
170 esophageal sphincter (LES) in patients with gastroesophageal reflux disease often has a low resting
171 is (EoE) was historically distinguished from gastroesophageal reflux disease on the basis of histolog
172 ated with EoE (or indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and
175 ted with a relatively high rate of recurrent gastroesophageal reflux disease requiring treatment, dim
177 racticed, its value in patients with chronic gastroesophageal reflux disease symptoms is of unproven
178 ng upper endoscopy for patients with chronic gastroesophageal reflux disease symptoms to assess for B
179 men in the United Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the p
183 screening ages for patients with symptomatic gastroesophageal reflux disease were older (58 for men a
184 as chronic sinusitis, allergic rhinitis, and gastroesophageal reflux disease were only associated wit
186 structive sleep apnea may be associated with gastroesophageal reflux disease, a strong risk factor fo
187 allergic rhinitis, chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep
189 ibly due to increased abdominal pressure and gastroesophageal reflux disease, although this pathogeni
190 lization for asthma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other
191 lation), and prior diagnoses (eg, pneumonia, gastroesophageal reflux disease, and other comorbidities
192 the individual to irritable bowel syndrome, gastroesophageal reflux disease, and peptic ulcer diseas
193 treat a variety of gastric disorders such as gastroesophageal reflux disease, autoimmune gastritis, g
194 factors for EAC have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, ob
195 shown to be a safe and effective therapy for gastroesophageal reflux disease, but its effect on the L
198 sing and treating chest pain associated with gastroesophageal reflux disease, correlates abnormal amb
199 terms: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
200 lowing: heartburn, regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, lary
201 l gastrointestinal complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Ba
202 This issue provides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis,
203 of the lower esophagus epithelium related to gastroesophageal reflux disease, is the strongest known
204 a diligent search for an esophageal etiology-gastroesophageal reflux disease, motility abnormalities,
205 itant chemical carcinogen treatment leads to gastroesophageal reflux disease, multilayered epithelium
207 on airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunction
208 , including history of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal ble
211 premorbid prevalence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infect
212 44 patients (52%) had objective findings of gastroesophageal reflux disease, such as esophagitis.
214 ic conditions (hypertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabet
216 ministered a modified version of a validated gastroesophageal reflux disease-specific QOL tool to pat
242 the AGA Institute on "Endoscopic Therapy for Gastroesophageal Reflux Disease." It was approved by the
243 is a laryngeal symptom that can be caused by gastroesophageal reflux disease; however, treatment outc
244 cough may have abnormal proximal exposure to gastroesophageal reflux documented by HMII that would ha
246 ers, including nonalcoholic steatohepatitis, gastroesophageal reflux, gallstones, and increased risk
252 f a disease engendered more controversy than gastroesophageal reflux (GER), a highly prevalent condit
254 sts with erosive tooth wear have significant gastroesophageal reflux (GERD), despite minor reflux sym
255 n the temporal association between cough and gastroesophageal reflux (GOR) in patients in whom non-GO
256 laparoscopically but failure with recurrent gastroesophageal reflux has resulted in the development
257 tocompatibility locus antigen antibodies and gastroesophageal reflux have been associated with poor o
258 sphincter (UES) to simulated or spontaneous gastroesophageal reflux have shown conflicting results.
261 rtburn with and without regurgitation due to gastroesophageal reflux in the absence of esophageal muc
262 py of gastroesophageal reflux, but recurrent gastroesophageal reflux is a persistent problem that req
264 he diagnosis of cough, LPR, or asthma due to gastroesophageal reflux is difficult, as no criterion st
266 allograft injury from donor-directed Abs or gastroesophageal reflux led to new ColV and KAT Abs post
267 discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstructive sleep apnoea, vocal
270 rative complications, sleep difficulties and gastroesophageal reflux progressively worsened during fo
271 in the postoperative period from SSc-related gastroesophageal reflux, renal impairment, and skin fibr
272 ireflux surgery, 17.7% experienced recurrent gastroesophageal reflux requiring long-term medication u
273 poorly described, and the risk of worsening gastroesophageal reflux requiring revision may be higher
275 , sex, percent predicted FEV1, self-reported gastroesophageal reflux, St. George's Respiratory Questi
276 presenting with extra-esophageal symptoms of gastroesophageal reflux such as chronic cough and asthma
278 based on the modified Rome III criteria) and gastroesophageal reflux symptoms (GERS) in a population-
280 eep apnea and to determine whether nocturnal gastroesophageal reflux symptoms affect the relationship
281 estioned regarding severity of their typical gastroesophageal reflux symptoms and presence of nocturn
282 nal GERD in 1999 (>/=3 episodes of nocturnal gastroesophageal reflux symptoms per week) had an OR of
283 Barrett's esophagus patients with nocturnal gastroesophageal reflux symptoms should be further evalu
284 's esophagus was associated with more severe gastroesophageal reflux symptoms, and nocturnal reflux s
296 pump inhibitors (PPIs) are popular drugs for gastroesophageal reflux, which are now available for lon
297 BACKGROUND & AIMS: Central obesity promotes gastroesophageal reflux, which may be related to increas
298 s have failed to demonstrate that preventing gastroesophageal reflux with antireflux surgery halts th
299 f unproven efficacy and focus on controlling gastroesophageal reflux with reflux-reducing medication