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1 moting the resolution of inflammation in the gastrointestinal mucosa.
2 ecreted CA and exists in both saliva and the gastrointestinal mucosa.
3 course), which occurs across the oral and/or gastrointestinal mucosa.
4 virulence factor in EAEC colonization of the gastrointestinal mucosa.
5 mbryo, to turnover in the homeostasis of the gastrointestinal mucosa.
6 nic infection, few have explored Treg in the gastrointestinal mucosa.
7 ortion of intraepithelial lymphocytes in the gastrointestinal mucosa.
8  the interactions between EAEC and the human gastrointestinal mucosa.
9 rologists to obtain in-vivo histology of the gastrointestinal mucosa.
10 uch as the skin, respiratory tract, and oral/gastrointestinal mucosa.
11 e of inducing growth in normal and malignant gastrointestinal mucosa.
12 ities, including growth-promoting actions on gastrointestinal mucosa.
13 or, and the number of tumors per 1000 mm2 of gastrointestinal mucosa.
14 ns in terminally differentiated cells in the gastrointestinal mucosa.
15   Aminobiphosphonates can irritate the upper gastrointestinal mucosa.
16  real-time, microscopic visualization of the gastrointestinal mucosa, allowing an endoscopic approach
17 olvement in lymphocyte recruitment to normal gastrointestinal mucosa and associated lymphoid tissue.
18 A-producing plasma cell populations in human gastrointestinal mucosa and bone marrow and the specific
19 ine and etoposide in the bone marrow and the gastrointestinal mucosa and emphasize the potential for
20 ing of vaccine-elicited T lymphocytes to the gastrointestinal mucosa and for vaccine protective effic
21 6 appears to be most highly expressed in the gastrointestinal mucosa and in kidney and lung.
22 so reduced proviral DNA in peripheral blood, gastrointestinal mucosa and lymph nodes without the deve
23 ressing endings were seen clearly within the gastrointestinal mucosa and myenteric plexus, respective
24 ammadelta T cells are primarily found in the gastrointestinal mucosa and play an important role in th
25  and function in blood, spleen, lymph nodes, gastrointestinal mucosa, and bronchoalveolar lavage of u
26 eptor (EGFR) triggers mitogenic signaling in gastrointestinal mucosa, and its expression is also upre
27 ed levels of proviral DNA in lymph nodes and gastrointestinal mucosa as compared with treatment at la
28  Galpha(t)-(2), also was demonstrated in the gastrointestinal mucosa as well as in STC-1 cells, as re
29 e sites of origin include the oral, lung and gastrointestinal mucosa, as data consistent with this hy
30 viding a point-of-care diagnostic method for gastrointestinal mucosa associated illnesses.
31                   Functional analysis of the gastrointestinal mucosa-associated lymphoid tissue revea
32                  The organisms penetrate the gastrointestinal mucosa by unknown mechanisms and are ph
33 gnificant accumulation of eosinophils in the gastrointestinal mucosa compared with control mice.
34                                          The gastrointestinal mucosa contains a complex network of ly
35                                          The gastrointestinal mucosa contains an intact immune system
36 umor size, the proportion of the area of the gastrointestinal mucosa covered with tumor, and the numb
37  are rapidly depleted from the periphery and gastrointestinal mucosa during acute HIV-1 infection.
38  of rhIL-11 to preserve the integrity of the gastrointestinal mucosa during cancer treatment regimens
39  the poor bioavailability of 2-MeOE2, as the gastrointestinal mucosa expresses high levels of 17beta-
40 protein contributes to the protection of the gastrointestinal mucosa from injury by strengthening and
41                                          The gastrointestinal mucosa harbors the majority of the body
42                                          The gastrointestinal mucosa has proven to be an interesting
43 ary, we demonstrate that UGTs are located in gastrointestinal mucosa, have vast overlapping activitie
44  glands, lymphoid organs, bone, bone marrow, gastrointestinal mucosa, heart, and kidneys.
45 IV-specific cellular immune responses to the gastrointestinal mucosa in a primate model.
46                                          The gastrointestinal mucosa is a major lymphoid tissue reser
47                                              Gastrointestinal mucosa is an early target of HIV and a
48                                       As the gastrointestinal mucosa is an important site of HIV tran
49 c stem cell transplantation (allo-HSCT), the gastrointestinal mucosa is damaged, and colonizing bacte
50                                    The human gastrointestinal mucosa is exposed to a diverse normal m
51                                    The upper gastrointestinal mucosa is exposed to endogenous and exo
52                                          The gastrointestinal mucosa is exposed to numerous chemical
53                                          The gastrointestinal mucosa is the primary site where human
54 (HIV) in peripheral blood, but the effect in gastrointestinal mucosa is uncertain.
55 emokine receptor expression in the blood and gastrointestinal mucosa; mucosal cells are predominantly
56  samples of normal pancreas (n = 11), normal gastrointestinal mucosa (n = 22), resected pancreas canc
57                               Imaging of the gastrointestinal mucosa of mice infected with differenti
58 on was also seen on organisms colonizing the gastrointestinal mucosa of mice, indicating that the ant
59 etabolism of SCFAs affected apoptosis in the gastrointestinal mucosa of the mouse, and whether this a
60 d tissue-resident Vdelta1 T cells within the gastrointestinal mucosa of virally suppressed people wit
61 ockade involve either barrier tissues (e.g., gastrointestinal mucosa or skin) or endocrine organs, al
62    Therefore, the metabolism of SCFAs by the gastrointestinal mucosa plays a role in modulating apopt
63            Epithelial and other cells of the gastrointestinal mucosa rely on both luminal and bloodst
64  conclude that, based on these findings, the gastrointestinal mucosa represents a favored target for
65 (BaP) and the MCP in the bone marrow and the gastrointestinal mucosa, respectively.
66 may be a common mechanism by which the upper gastrointestinal mucosa responds to noxious insults.
67 bout the effects of individual NSAIDs on the gastrointestinal mucosa, risk factors for sustaining an
68                                          The gastrointestinal mucosa, structurally formed by the epit
69 e chemokine receptor CCR9 to localize to the gastrointestinal mucosa; their respective ligands, mucos
70                                       In the gastrointestinal mucosa they provide the priming cytokin
71  responsible for the unique tolerance of the gastrointestinal mucosa to proinflammatory stimuli.
72 d stability of CD25(+) FOXP3(+) cells in the gastrointestinal mucosa to support immunoregulation and
73 (renal proximal tubular epithelial cells and gastrointestinal mucosa) to damage to renal glomerular t
74 press toxicity to the bone marrow and to the gastrointestinal mucosa when used at therapeutic doses,
75 owever, these neurons are not present in the gastrointestinal mucosa, where 5-HT initiates peristalti
76  the host-toxin response at the level of the gastrointestinal mucosa, where STEC infection begins.
77 understanding of prorepair mechanisms in the gastrointestinal mucosa will aid in the development of n
78  that induces potent immune responses in the gastrointestinal mucosa would be highly desirable.