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1 moting the resolution of inflammation in the gastrointestinal mucosa.
2 ecreted CA and exists in both saliva and the gastrointestinal mucosa.
3 course), which occurs across the oral and/or gastrointestinal mucosa.
4 virulence factor in EAEC colonization of the gastrointestinal mucosa.
5 mbryo, to turnover in the homeostasis of the gastrointestinal mucosa.
6 nic infection, few have explored Treg in the gastrointestinal mucosa.
7 ortion of intraepithelial lymphocytes in the gastrointestinal mucosa.
8 the interactions between EAEC and the human gastrointestinal mucosa.
9 rologists to obtain in-vivo histology of the gastrointestinal mucosa.
10 uch as the skin, respiratory tract, and oral/gastrointestinal mucosa.
11 e of inducing growth in normal and malignant gastrointestinal mucosa.
12 ities, including growth-promoting actions on gastrointestinal mucosa.
13 or, and the number of tumors per 1000 mm2 of gastrointestinal mucosa.
14 ns in terminally differentiated cells in the gastrointestinal mucosa.
15 Aminobiphosphonates can irritate the upper gastrointestinal mucosa.
16 real-time, microscopic visualization of the gastrointestinal mucosa, allowing an endoscopic approach
17 olvement in lymphocyte recruitment to normal gastrointestinal mucosa and associated lymphoid tissue.
18 A-producing plasma cell populations in human gastrointestinal mucosa and bone marrow and the specific
19 ine and etoposide in the bone marrow and the gastrointestinal mucosa and emphasize the potential for
20 ing of vaccine-elicited T lymphocytes to the gastrointestinal mucosa and for vaccine protective effic
22 so reduced proviral DNA in peripheral blood, gastrointestinal mucosa and lymph nodes without the deve
23 ressing endings were seen clearly within the gastrointestinal mucosa and myenteric plexus, respective
24 ammadelta T cells are primarily found in the gastrointestinal mucosa and play an important role in th
25 and function in blood, spleen, lymph nodes, gastrointestinal mucosa, and bronchoalveolar lavage of u
26 eptor (EGFR) triggers mitogenic signaling in gastrointestinal mucosa, and its expression is also upre
27 ed levels of proviral DNA in lymph nodes and gastrointestinal mucosa as compared with treatment at la
28 Galpha(t)-(2), also was demonstrated in the gastrointestinal mucosa as well as in STC-1 cells, as re
29 e sites of origin include the oral, lung and gastrointestinal mucosa, as data consistent with this hy
36 umor size, the proportion of the area of the gastrointestinal mucosa covered with tumor, and the numb
37 are rapidly depleted from the periphery and gastrointestinal mucosa during acute HIV-1 infection.
38 of rhIL-11 to preserve the integrity of the gastrointestinal mucosa during cancer treatment regimens
39 the poor bioavailability of 2-MeOE2, as the gastrointestinal mucosa expresses high levels of 17beta-
40 protein contributes to the protection of the gastrointestinal mucosa from injury by strengthening and
43 ary, we demonstrate that UGTs are located in gastrointestinal mucosa, have vast overlapping activitie
49 c stem cell transplantation (allo-HSCT), the gastrointestinal mucosa is damaged, and colonizing bacte
55 emokine receptor expression in the blood and gastrointestinal mucosa; mucosal cells are predominantly
56 samples of normal pancreas (n = 11), normal gastrointestinal mucosa (n = 22), resected pancreas canc
58 on was also seen on organisms colonizing the gastrointestinal mucosa of mice, indicating that the ant
59 etabolism of SCFAs affected apoptosis in the gastrointestinal mucosa of the mouse, and whether this a
60 d tissue-resident Vdelta1 T cells within the gastrointestinal mucosa of virally suppressed people wit
61 ockade involve either barrier tissues (e.g., gastrointestinal mucosa or skin) or endocrine organs, al
62 Therefore, the metabolism of SCFAs by the gastrointestinal mucosa plays a role in modulating apopt
64 conclude that, based on these findings, the gastrointestinal mucosa represents a favored target for
66 may be a common mechanism by which the upper gastrointestinal mucosa responds to noxious insults.
67 bout the effects of individual NSAIDs on the gastrointestinal mucosa, risk factors for sustaining an
69 e chemokine receptor CCR9 to localize to the gastrointestinal mucosa; their respective ligands, mucos
72 d stability of CD25(+) FOXP3(+) cells in the gastrointestinal mucosa to support immunoregulation and
73 (renal proximal tubular epithelial cells and gastrointestinal mucosa) to damage to renal glomerular t
74 press toxicity to the bone marrow and to the gastrointestinal mucosa when used at therapeutic doses,
75 owever, these neurons are not present in the gastrointestinal mucosa, where 5-HT initiates peristalti
76 the host-toxin response at the level of the gastrointestinal mucosa, where STEC infection begins.
77 understanding of prorepair mechanisms in the gastrointestinal mucosa will aid in the development of n