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1 6J mice to flurothyl until they experience a generalized seizure.
2 enia, diarrhea, vomiting, and syncope with a generalized seizure.
3 f two hospitals in Boston after a witnessed, generalized seizure.
4 ffects on spontaneous subclinical and evoked generalized seizures.
5 ell excitability, and is sufficient to cause generalized seizures.
6  of the time with ictal SPECT in secondarily generalized seizures.
7 th fast polyspike activity during convulsive generalized seizures.
8 splayed sustained protection from developing generalized seizures.
9 energy depletion in the brain resulting from generalized seizures.
10 - mice appear ataxic and display spontaneous generalized seizures.
11 ey role of the thalamus in the generation of generalized seizures.
12 epsy, a history of febrile convulsions or of generalized seizures.
13 linergic neurons influence the expression of generalized seizures.
14 neurons may participate in the modulation of generalized seizures.
15 odialysis before and during the induction of generalized seizures.
16 pilepsy of unknown cause, and 66 percent had generalized seizures.
17 associated with waking, slow-wave sleep, and generalized seizures.
18 ctal discharges or behaviour in mice without generalized seizures.
19 entate gyrus caused by PTEN loss, as well as generalized seizures.
20 reated with deep brain stimulation (DBS) for generalized seizures.
21 nical trials of brain stimulation to control generalized seizures.
22 phy, lower seizure frequency and secondarily generalized seizures.
23 mal dominant complex partial and secondarily generalized seizures, a greatly reduced threshold for hi
24 cur in response to the repeated induction of generalized seizure activity.
25                  By measuring the latency to generalized seizures after beta-CCM administration to A/
26  new disease entity hallmarked by idiopathic generalized seizure and atypical RLS.
27                                  Secondarily generalized seizures and complex partial seizures exhibi
28 alities, both in mice developing spontaneous generalized seizures and in mice only exhibiting interic
29  for the R1648H mutation exhibit spontaneous generalized seizures and premature death between P16 and
30 e Sandhoff disease that was hospitalized for generalized seizures and psychomotor retardation.
31 onsequence of bilateral insults (potentially generalized seizures and/or medications).
32 nuous partial seizures and even new bouts of generalized seizures, and (3) development of mild sponta
33  mutation in the L-ferritin gene, idiopathic generalized seizures, and atypical restless leg syndrome
34  immunodeficiency, neurodevelopmental delay, generalized seizures, and impaired hearing but with no m
35 of humans and macaques across normal waking, generalized seizure, anesthesia, and psychedelic states.
36 n-1B(Open) mice were viable but succumbed to generalized seizures at 2 to 3 months of age.
37         The progression of flurothyl-induced generalized seizure behaviors was significantly altered
38 s interpretation, ictal SPECT in secondarily generalized seizures can help localize the region of sei
39 -46 mSMEI had myoclonic seizures followed by generalized seizures caused by elevated core body temper
40 e of a more diffuse disorder with additional generalized seizures, cognitive and behavioural disturba
41 Inhibition of cell bodies in SNpr suppressed generalized seizures evoked by pentylenetetrazole (PTZ),
42 the stimulation-free interval on the type of generalized seizure expressed.
43  frequency bands was highest for secondarily generalized seizures, followed by complex partial seizur
44 yr-old woman experienced several episodes of generalized seizures following 2 wk of immunosuppressive
45 l blood flow (CBF) changes in 59 secondarily generalized seizures from 53 patients.
46 ance of these findings for the triggering of generalized seizures from the deep piriform region are d
47 taA mRNA induction, after the first stage 5 (generalized) seizure, had spread to the contralateral am
48                 Ictal SPECT from secondarily generalized seizures has not been thoroughly investigate
49 ker CD68 and focal to bilateral tonic-clonic generalized seizure history.
50 , duration of epilepsy, history of secondary generalized seizures, history of head injury, handedness
51 s adults displayed susceptibility to induced generalized seizures, hyperactivity, repetitive and redu
52 ple partial, complex partial and secondarily generalized seizures in 28 patients with temporal lobe s
53 niques in the bicuculline model of sustained generalized seizures in newborn pigs.
54 correlated with the frequency of secondarily generalized seizures in the 3-5 days preceding the recor
55 such as glia in the genesis and spreading of generalized seizures in the brain.
56 hippocampal neuronal loss despite decades of generalized seizures, including status epilepticus.
57 the hippocampus are critical to the onset of generalized seizures, indicating that the hippocampus pl
58       Here, we examined effects of secondary generalized seizures induced by kainate, on dendritic st
59 ker of acid-sensing ion channels, suppresses generalized seizures induced by maximal electroshock and
60 izures, but whether this occurs in prolonged generalized seizures is not known.
61 2 Aldh5a1-/- mice display ataxia and develop generalized seizures leading to rapid death.
62  modified tau score and history of secondary generalized seizures (likelihood-ratio chi(2), P < 0.05)
63   New insights into these different types of generalized seizures may lead eventually to new therapie
64 e entire brain, indicating that the onset of generalized seizures may originate locally rather than d
65              Here, we characterize a primary generalized seizure model of epilepsy following repeated
66 elop additional extreme hyper-reactivity and generalized seizures not seen in any other genotypes.
67                                          The generalized seizures of status epilepticus (SE) trigger
68 ork site for mediating analgesia and also in generalized seizures, particularly in genetically epilep
69 rozygous mice exhibit infrequent spontaneous generalized seizures, reduced threshold and accelerated
70  several brain sites, including the PAG, and generalized seizures result in endocannabinoid release.
71 ol and loss of consciousness consistent with generalized seizure semiology.
72 e, with severe nocturnal focal and secondary generalized seizures starting in early childhood with de
73 an seizure onset age of 24 months, focal and generalized seizures, status epilepticus (n = 5), severe
74 ple partial, complex partial and secondarily generalized seizures suggest a possible connection betwe
75 nist caused a dose-dependent increase in the generalized seizure threshold (GST) of these seizure sus
76 sulted in a progressive decrease in both the generalized seizure threshold and the latency to the fir
77 and to proceed independent of changes in the generalized seizure threshold.
78                                              Generalized seizure thresholds (GSTs) were subsequently
79        CLON significantly elevated focal and generalized seizure thresholds, whereas IDA significantl
80 hibition and thus increase susceptibility to generalized seizures through a reduction of alphabetagam
81                                Each of these generalized seizures typically lasts <30 s.
82  electrical current is used to elicit brief, generalized seizures under general anesthesia.
83 e-3-carboxylate (beta-CCM)]-induced model of generalized seizures was genetically dissected in mice.
84 leads to their hyperactivity at the onset of generalized seizures, which have low-voltage-fast featur
85  extrinsic and intrinsic caspase pathways in generalized seizures, which produce morphologically necr
86 ng intractable complex partial and secondary generalized seizures, whose magnetic resonance imaging s
87 hat are associated with febrile seizures and generalized seizures with febrile seizures plus pedigree
88  with AAV Nrf2 displayed significantly fewer generalized seizures, with profound reduction in microgl