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1 h-sensitivity cardiac troponin T (cTnT), and genetic analysis.
2 atory DNA maps and integrated epigenomic and genetic analysis.
3 ws for continuous innovation in multivariate genetic analysis.
4 ; 78%), including patients with inconclusive genetic analysis.
5 significant new opportunities for functional genetic analysis.
6 ir subcellular localization and, critically, genetic analysis.
7 had potential to benefit from more extensive genetic analysis.
8 Fragman is a valuable new tool for genetic analysis.
9 rdalis Linnaeus 1758 [5], were included in a genetic analysis.
10 to deduce the minimum cellularity needed for genetic analysis.
11 nknown genes to identify targets for reverse genetic analysis.
12 tion, and their correlations using bivariate genetic analysis.
13 rom those reported for patients referred for genetic analysis.
14 recovery and near 100% purity), followed by genetic analysis.
15 y relationships from quantitative functional genetic analysis.
16 rting the dissection of SLE heterogeneity by genetic analysis.
17 e cases of cTTP were diagnosed, confirmed by genetic analysis.
18 rom genotype data plays an important role in genetic analysis.
19 are standard genetic markers for population genetic analysis.
20 accuracy of the predictions through reverse genetics analysis.
22 s proven to be a highly tractable system for genetic analysis and for studying adaptive evolution.
23 cal interpretation of the genome, population genetic analysis and functional genomic analysis of alle
26 ction of blood samples, which will allow for genetic analysis and interrogation of gene-environment i
28 uld overcome many issues related to accurate genetic analysis and mitigate issues with processing sma
32 treous hemorrhage, biopsy yield was 100% for genetic analysis, and no patients showed recurrence or i
33 ilage include those previously identified by genetic analysis, and several mucilage proteins are redu
35 est level of genome coverage for genomic and genetic analysis because of their abundance and relative
37 nd also review the evidence that genome wide genetic analysis can substantially help in the predictio
38 howed evidence of intracellular storage, and genetic analysis confirmed a GLA A gene mutation (p.Asn2
42 es on the single-cell scale lag behind their genetic analysis counterparts and typically report on ex
43 Curiously, this is consistent with previous genetic analysis defining MYB33 and MYB65 as the major f
56 fecal microbial composition was analyzed by Genetic Analysis GA-map Dysbiosis test, targeting >=300
58 Furthermore, fitting an LMM that utilized a genetic-analysis group rather than a self-identified bac
59 i-dimensional clustering method to define a "genetic-analysis group" variable that retains many prope
60 familial relatedness, PCs for ancestry, and genetic-analysis groups for additional group-associated
69 first discovered, technological advances in genetic analysis have made finding genomic variation a m
71 identified through sequence alignment and/or genetic analysis; however, few have been studied in vitr
82 use results of forward genetic screening and genetic analysis in our new model to demonstrate that al
86 and advances researchers' ability to perform genetic analysis in this pathogen; the protocol also has
89 nalysis of the PCSK6 locus combining further genetic analysis, in silico predictions and molecular as
90 r and bioinformatic techniques that underpin genetic analysis, including PCR amplification, hybridiza
91 ples were obtained from all participants for genetic analysis, including whole-exome sequencing and m
94 time maltose levels are reduced in lsf1, and genetic analysis indicated that the starch-excess phenot
99 plant development we performed an extensive genetic analysis involving the generation and characteri
103 tive complex (CSC) of the fungal retromer by genetic analysis, live cell imaging and immunological as
104 has been used as a quantitative phenotype in genetic analysis looking for Huntington's disease modifi
107 an integrated approach featuring conditional genetic analysis, massively parallel reporter assays (MP
108 s appears to be highly heritable and further genetic analysis may help identify new biological pathwa
109 tudied using both quantitative and molecular genetic analysis methods, both approaches lack studies s
110 LMNA-related cardiomyopathy, suggesting that genetic analysis might be useful for diagnosis and risk
112 ulf of California fin whales in a population genetic analysis of 18 nuclear microsatellite genotypes
115 emerging technique which enables systematic genetic analysis of a cellular or molecular phenotype in
124 thermore, by combining the genomic data with genetic analysis of an additional 800 isolates and envir
133 s of nuclear envelope and ER fission and, by genetic analysis of cdc42 mutant yeast, uncover a unique
134 e use single-molecule optical microscopy and genetic analysis of chimeric protein constructs to evalu
139 method represents a blueprint for systematic genetic analysis of complex tissue hierarchies at single
142 ions should greatly accelerate the molecular genetic analysis of development in V. carteri, and of de
144 the degree of EGFR-signaling inhibition nor genetic analysis of EGFR was sufficient to predict sensi
147 ve developed a method for spatially resolved genetic analysis of formalin-fixed paraffin-embedded (FF
148 report on HistoMosaic, a novel technique for genetic analysis of formalin-fixed, paraffin-embedded ti
149 rphic mutations are a valuable tool for both genetic analysis of gene function and for synthetic biol
152 evidence of an exoticN. bombiorigin based on genetic analysis of globalNosemapopulations; the widespr
154 make use of genealogic information can favor genetic analysis of highly polygenic traits, but not gen
156 ore platform to be used in the diagnosis and genetic analysis of influenza virus and other respirator
164 as important methodological implications for genetic analysis of omics traits across a range of genom
165 e now describe the first detailed population genetic analysis of P. acnes isolates recovered from pai
166 this is the largest report of a clinical and genetic analysis of patients affected with CSNB due to T
168 IGNIFICANCE STATEMENT This report provides a genetic analysis of primary nociceptive neuron mechanism
169 Combining proteomics, in vivo imaging and genetic analysis of proteins linked to planar cell polar
170 ause AGD is related to weight, we included a genetic analysis of pup weight at birth and weaning.
176 w comparative genome analysis and a thorough genetic analysis of SSV1 using both specific and random
180 For in vivo functional verification and genetic analysis of TBX6 compound inheritance, we genera
183 mulation in plants and describe a population-genetic analysis of the Alyssum serpyllifolium (Brassica
184 validated by several methods that include a genetic analysis of the conserved PACT domain that recru
185 By comprehensive molecular, cellular, and genetic analysis of the EPSIN gene family, we identify E
188 Here we first performed a comprehensive genetic analysis of the mouse auditory epithelium to sho
192 has sufficient tumor-derived DNA to perform genetic analysis of the tumor, including DNA copy number
193 ignin regulators began with the quantitative genetic analysis of the xylem transcriptome and resulted
204 e diagnosis should be confirmed by molecular genetic analysis or measurement of levels of fibroblast
207 lets based on cellular phenotype upstream of genetic analysis, particularly for large and complex cel
209 ould be determined by the planned downstream genetic analysis, PicoPLEX and RepliG performing better
217 ent was specific for escaped germ cells, and genetic analysis revealed it did not depend on pathways
234 ncurrent copy-number data, our comprehensive genetic analysis reveals the primacy of NF1 loss as the
241 ll divisions, combined with marker lines and genetic analysis, showed that the spch-5 leaf phenotype
245 e emerging of high-dimensional genomic data, genetic analysis such as genome-wide association studies
251 erapy was successful, and histopathology and genetic analysis supported a diagnosis of extraocular uv
252 data mining, data reanalysis, and networked genetic analysis that lead to the generation of new know
254 signaling, we conducted a systematic forward genetic analysis through reporter-based screens in haplo
255 use of structural variants for quantitative genetic analysis through whole-genome shotgun sequencing
256 will allow the extensive infrastructure for genetic analysis to be applied to proteomic assays, whic
257 se coimmunoprecipitation studies paired with genetic analysis to define the cullin-RING ligase compon
258 show that Cryo-OrbiSIMS can be combined with genetic analysis to identify enzymes regulating SVOC met
260 genetic HoFH, highlighting the importance of genetic analysis to improve familial hypercholesterolemi
261 formed laser microdissection and single cell genetic analysis to investigate the relationship between
263 The iBAC library represents an important new genetic analysis tool openly available to the research c
267 We demonstrate our method on an imaging genetic analysis using data from the Alzheimer's Disease
268 quaternary structures made it amenable to a genetic analysis using in-frame insertions and deletions
269 informatic techniques have been proposed for genetic analysis using networks, based on random walks,
276 illustrate the utility of this approach for genetic analysis, we demonstrate spatially resolved geno
279 n hemiganglia of the same individual and, by genetic analysis, we identify the bithorax-complex genes
283 The GWAS data were used to guide reverse genetic analysis, which found effectors of ABA accumulat
284 ations include the small sample size for the genetic analysis, which was underpowered to detect genom
285 veral processes for functional phenomics and genetic analysis, which will lead to a greater understan
286 ic susceptibilities in individual disorders, genetic analysis with increased numbers of variants and
289 n data from 20 pedigrees, the members of our Genetic Analysis Workshop (GAW) 19 gene expression group
290 sults and application to gene DNAH9 from the Genetic Analysis Workshop 16 for associated with Anti-cy
291 sed method is demonstrated by applying it to Genetic Analysis Workshop 16 rheumatoid arthritis GWAS d
293 on studies and applications to data from the Genetic Analysis Workshop 17 and the Ocular Hypertension
296 The Pathway-based Analyses Group of the Genetic Analysis Workshop 19 (GAW19) sought reduction of
299 current literature and developments from the Genetics Analysis Workshop 19 (GAW19) Collapsing Rare Va