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1 and ordering contigs into pseudomolecules by genetic linkage analysis.
2 and 3 unaffected family members, followed by genetic linkage analysis.
3 g locus in the M. smegmatis chromosome using genetic linkage analysis.
4 ed LINKAGE package, to perform more powerful genetic linkage analysis.
5 order had been mapped to chromosome 11q22 by genetic linkage analysis.
6 ilies of line crosses for quantitative trait genetic linkage analysis.
7 rectly linked polymorphism (MLP1) useful for genetic linkage analysis.
8 se on chromosome 6p21.1-p12 were excluded by genetic linkage analysis.
9 nd six candidate genes have been excluded by genetic linkage analysis.
10 ly been localised to chromosome 17q12-q21 by genetic linkage analysis.
11 n D11S1765 and UGB (uteroglobin) in 11q13 by genetic linkage analysis.
12 ons is less than the 45 percent predicted by genetic-linkage analysis.
13 Two partly parallel approaches were used: genetic linkage analysis (19 large families) and the pro
14 y using an interspecific crossing design and genetic linkage analysis, a major quantitative trait loc
18 festing with calf weakness, we identified by genetic linkage analysis and exome sequencing a heterozy
22 paleoarcheology, genetic disease diagnosis, genetic linkage analysis, and genetic diversity studies.
24 Three genetic locl have been identified by genetic linkage analysis at chromosomes 8q24.1, 11p11-13
25 vary among individuals of a species and that genetic linkage analysis can be used to identify quantit
27 ission in the year following infection using genetic linkage analysis despite apparently increased ri
41 ed in controlling hair texture, we performed genetic linkage analysis in six families of Pakistani or
45 rted to combine gene expression studies with genetic linkage analysis, leading to a new synergy betwe
48 17, 19, and X were constructed by multipoint genetic linkage analysis of 22 polymorphic markers in 40
51 variation in gene expression, we carried out genetic linkage analysis of genomewide expression patter
52 mating in Leishmania, opening the way toward genetic linkage analysis of important traits and providi
55 ve gene, NKX2-5, has been identified through genetic linkage analysis of pedigrees with non-syndromic
59 ine skin model of chemical carcinogenesis in genetic linkage analysis of three independent Mus muscul
62 ed with those of the new kindreds (HP2), and genetic linkage analysis, screening for mutations throug
64 r describes MLIP, a multiprocessor two-point genetic linkage analysis system that supports statistica
65 , a HSL susceptibility gene), we conducted a genetic linkage analysis that included serotyping and ph
66 These results provide the groundwork for a genetic linkage analysis to identify the genes that infl
67 ial breast cancer has recently been shown by genetic linkage analysis to map to chromosome l3q12.
71 To map the CPP locus we performed molecular genetic linkage analysis using microsatellite markers in
72 locate the disease-causing gene we completed genetic linkage analysis using short tandem repeat polym
77 arch and for understanding genome evolution, genetic linkage analysis was used to identify chromosome
80 described by Virgil Haws in 1963, and using genetic linkage analysis, we localized the susceptibilit
82 sults from the somatic deletion analysis and genetic linkage analysis, we were able to further narrow
85 e analyzed all 49 heritable phenotypes using genetic linkage analysis, with special emphasis on pheno