戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 actor (GM-CSF) signaling based on studies in genetically modified mice.
2 erefore investigated potential mechanisms in genetically modified mice.
3 evelopmental changes in the barrel cortex of genetically modified mice.
4 ssection of placental transfer mechanisms in genetically modified mice.
5 elicited humoral responses in MV-susceptible genetically modified mice.
6 or the dominant nature of FHHt in humans and genetically modified mice.
7 uitable test for species-typical behavior in genetically modified mice.
8  article, we will highlight recent data from genetically modified mice.
9 o JNK inhibition or activation in vivo using genetically modified mice.
10 d depression-related behaviors in normal and genetically modified mice.
11 etermined by comparison of the phenotypes of genetically modified mice.
12 chanisms of graft injury and regeneration in genetically modified mice.
13 periments using LAT-deficient cell lines and genetically modified mice.
14 r assessing learning and memory abilities in genetically modified mice.
15 edative effects of ethanol in three lines of genetically modified mice.
16 emia has been greatly enhanced by the use of genetically modified mice.
17 sion in the myocardium of both wild-type and genetically modified mice.
18 mited to the use of mouse-adapted viruses or genetically modified mice.
19 n and renewal after myocardial infarction in genetically modified mice.
20 or the use of mouse-adapted virus strains or genetically modified mice.
21 es to inflammatory sites was investigated in genetically modified mice.
22 ospholamban and the ryanodine receptor using genetically modified mice.
23                                Wild type and genetically modified mice.
24 central to the cardiovascular phenotyping of genetically modified mice.
25 gic asthma model in ATX-LPA pathway-specific genetically modified mice.
26 pendent on Rab27B, as shown using acini from genetically modified mice.
27 ic smooth muscle causes thoracic aneurysm in genetically modified mice.
28 d crypt epithelia of intestinal tissues), in genetically modified mice.
29 eproducibility of some behavioral results in genetically-modified mice.
30                                   We created genetically modified mice (alpha1 H101R) with a diazepam
31 from studies utilizing rapamycin, studies in genetically modified mice also suggest that mTOR couples
32               Research has focused mostly on genetically modified mice, although other species like p
33 first targeting of the dorsal hippocampus in genetically modified mice and confirm a role for CREB in
34   Mechanistic studies were carried out using genetically modified mice and depletion of lymphocyte su
35                         Recent studies using genetically modified mice and electrophysiological recor
36                    Evidence is reviewed from genetically modified mice and human biomarker and geneti
37                      Our studies, using both genetically modified mice and human induced pluripotent
38                                 The study of genetically modified mice and improvements in the in-vit
39                              Observations in genetically modified mice and in humans with mutations i
40                                  Analyses of genetically modified mice and in vitro and in vivo precl
41                                        Using genetically modified mice and inhibitor treatments, we f
42                                           In genetically modified mice and Jurkat T cells CREMa expre
43 ent studies using the EL4 model with various genetically modified mice and macrophage-depleted mice r
44                                Here, we used genetically modified mice and molecular imaging to test
45 nation of intravital multiphoton microscopy, genetically modified mice and novel in vivo homing assay
46                                   Studies of genetically modified mice and of molecular pathways in a
47 amined the time course of colonization using genetically modified mice and pneumococci.
48 he value of screening platelet phenotypes of genetically modified mice and shed further light upon th
49 flammation and OSM-induced fibrosis, we used genetically modified mice and show that the fibrotic res
50                                        Using genetically modified mice and time-lapse video recording
51             Modeling connexin pathologies in genetically modified mice and tissue-relevant cells has
52 d Tbx1 function during OFT development using genetically modified mice and tissue-specific deletion,
53                     By analyzing cell lines, genetically modified mice, and HCC tissues, we found tha
54                       Our findings in cells, genetically modified mice, and human vascular specimens
55 ide) in genetically distinct inbred strains, genetically modified mice, and outbred mice.
56 bal and regional ischemia/reperfusion (I/R), genetically modified mice, and primary cell culture, to
57                          The two colonies of genetically modified mice-and, for purposes of controls,
58  retinol formation in the different types of genetically modified mice are in reasonable agreement wi
59 an hematopoietic stem, progenitor cells into genetically modified mice, are invaluable to study human
60 ic stem (ES) cells have been used to produce genetically modified mice as experimental models of huma
61                                Findings from genetically modified mice as well as pharmacological stu
62 n gene-related peptide were also examined in genetically modified mice (BDNF(+/-)) with reduced level
63                                  We describe genetically modified mice bearing the hypomorphic mutati
64                     In line with these data, genetically modified mice completely resistant to PAR2 c
65 , and rhodopsin kinase-deficient mice and in genetically modified mice containing unpalmitylated rhod
66 ears have seen an explosion in the number of genetically modified mice created to aid understanding o
67 rally available non-peptidic antagonists and genetically modified mice deficient in B(1) receptor exp
68          Consistent with the human data, the genetically modified mice displayed significantly more e
69                         Here we show that in genetically modified mice, disrupting the recruitment of
70 ta might be particularly useful in assessing genetically modified mice, even in laboratories not prim
71               In conclusion, Folbp1 and RFC1 genetically modified mice exhibit distinct changes in co
72                                           In genetically modified mice expressing diverse human immun
73                                We found that genetically modified mice expressing low levels of tissu
74                                              Genetically modified mice, featuring specific depletion
75 generation failure that have been made using genetically modified mice, focusing on the inhibitory in
76 annulating collecting lymphatic vessels from genetically modified mice for ex vivo study.
77   One of the major limitations in the use of genetically modified mice for studying cognitive functio
78                          Interestingly, both genetically modified mice gained significantly less body
79                                      We used genetically modified mice (germline Npy, Y1, and Y2 rece
80                                   The use of genetically modified mice has been an important model sy
81                              Sharp tuning in genetically modified mice has been attributed to decreas
82                          The availability of genetically modified mice has prompted the adaptation of
83                                              Genetically modified mice have advanced our understandin
84                                              Genetically modified mice have been extensively used for
85                                              Genetically modified mice have been used extensively to
86                        Recent experiments in genetically modified mice have demonstrated that mutatio
87                                   Studies in genetically modified mice have demonstrated that neuregu
88 t encode spatial location -in freely moving, genetically modified mice have further advanced our unde
89 rgence in mammalian reproduction, studies of genetically modified mice have identified biomarkers tha
90                        However, studies with genetically modified mice have identified the contributi
91                                     Although genetically modified mice have provided a great understa
92                         Recent studies using genetically modified mice have provided important new in
93 nd-healing assay was performed in 3 types of genetically modified mice having various Nur77 activitie
94 SG-DUC1 and mSG-PAC1 cells were derived from genetically modified mice, homozygous for floxed alleles
95 ch combined behavioral-molecular findings on genetically modified mice, human postmortem Dys expressi
96 BP in drug-induced plasticity using CBP-FLOX genetically modified mice in combination with adeno-asso
97                           We used HDAC3-FLOX genetically modified mice in combination with adeno-asso
98       To permit the use of these (and other) genetically modified mice in the analysis of venous inju
99                    Here we describe compound genetically modified mice in which expression of the end
100 onductances unique to sour cells, we created genetically modified mice in which sour cells were marke
101  ADAR2 protein expression, we have generated genetically modified mice in which the ability of ADAR2
102 is was confirmed by experiments performed in genetically modified mice in which the alpha1 subunit ha
103   We intended to address this question using genetically modified mice in which the expression of a s
104 cytes freshly isolated from adult rabbits or genetically modified mice, in combination with pharmacol
105 al, high-frequency region of the cochleae of genetically modified mice (including models of human her
106 rial myocytes and vessels from wild-type and genetically modified mice, including an inducible smooth
107  distribution of blood flow in the hearts of genetically modified mice is a phenotype of interest bec
108 that, as illustrated here, the real value of genetically modified mice is not as 'models of schizophr
109            When this technique is applied in genetically modified mice, it enables the investigator t
110 he latter fluorescence was absent in eyes of genetically modified mice lacking a functional visual cy
111                            Here we show that genetically modified mice lacking beta1 integrins in the
112               Here, we have used tissue from genetically modified mice lacking functional COX-1 (COX-
113                  In particular, screening of genetically modified mice lacking individual GRKs or bet
114                        Moreover, findings in genetically modified mice lacking macrophages have confi
115 in organotypic hippocampal slices taken from genetically modified mice lacking the GluA1 subunit.
116                                              Genetically modified mice lacking the glutamate receptor
117             Consistently, NGAL deficiency in genetically modified mice leads to an increased growth o
118                                          Two genetically modified mice models carrying Mib1 variants
119  we used both pharmacological inhibitors and genetically modified mice models to investigate the iden
120 X(2) receptors based on studies conducted on genetically modified mice needs to be viewed with cautio
121 ibroblast-cholangiocyte coculture system and genetically modified mice, Omenetti and colleagues prese
122             The study of renal miRNAs, using genetically modified mice or by perturbing endogenous mi
123        In Kras(G12D)/Pdx1-Cre/Tp53/Rosa(YFP) genetically modified mice, oral administration of SLC-01
124 te the apparent dominant role of ADAMTS-5 in genetically modified mice, our data suggest that both AD
125                            Studies employing genetically modified mice point to Hif-2alpha, one of tw
126 pectroscopy in perfused hearts isolated from genetically modified mice (PPARalpha(-/-)) that mimic th
127                          Recent studies with genetically modified mice provide important new insights
128 n proteomic screen of endothelial cells from genetically modified mice, R-Ras, known to promote vesse
129 escent imaging of kidney sections from these genetically modified mice revealed that RhoA and AQP2 ac
130                                              Genetically modified mice shed new light on how ketamine
131                                Studies using genetically modified mice suggest that Galpha q mediates
132                                    Work with genetically modified mice suggests that the viral latent
133 shown in an animal model: F-araAMP protected genetically modified mice susceptible to MV infection fr
134 vations, altogether with those obtained from genetically modified mice targeting individual CRMPs and
135 f CD103(+) DCs in cGN using several lines of genetically modified mice that allowed us to reduce the
136 ible to follow new blood vessel formation in genetically modified mice that are perinatally lethal.
137               In this study, we have created genetically modified mice that conditionally express Ht3
138                                        Using genetically modified mice that conditionally expressed g
139 egment is known as the M1' domain, and using genetically modified mice that contain the human M1' dom
140 er, irradiation, or thymectomy, we developed genetically modified mice that express diphtheria toxin
141 he same disorder, achalasia, was observed in genetically modified mice that express full-length farne
142                                     Although genetically modified mice that lack receptors for either
143 rally evoked symptoms is based on studies on genetically modified mice, the cellular localization of
144                               By using novel genetically modified mice, the current study showed that
145 tor complex has been enriched by analyses of genetically modified mice; these analyses have uncovered
146 tralizing antibodies or T cells derived from genetically modified mice, TL1A inhibition of Th17 devel
147                        In this work, we used genetically modified mice to allow conditional expressio
148                                 We also used genetically modified mice to define the roles of Chi3l1
149            For the current study we utilized genetically modified mice to delineate the relative cont
150 ntiallodynic action compared to DMI, we used genetically modified mice to gain insight into RGS prote
151                        In this study, we use genetically modified mice to investigate the role of Gal
152    To explore the nature of this deficit, we genetically modified mice to model the increase in stria
153                       In this study, we used genetically modified mice to study the light responses a
154 ide (CCl(4))-induced minipig NASH model, and genetically modified mice to unravel the landscape of th
155           Implications for studies involving genetically modified mice treated with topical agents an
156                                        Using genetically modified mice, we addressed the contribution
157              By introducing this system into genetically modified mice, we can readily manipulate the
158              Using platelets from humans and genetically modified mice, we demonstrate that binding o
159 ng expression analyses, retinal explants and genetically modified mice, we demonstrate that CXCL12 (S
160                                        Using genetically modified mice, we demonstrate that premature
161 onsistent with this information derived from genetically modified mice, we demonstrated that neutrali
162                    During our examination of genetically modified mice, we discovered a series of pro
163      Because those studies were performed on genetically modified mice, we examined whether neutraliz
164                                        Using genetically modified mice, we find that increasing the l
165                                        Using genetically modified mice, we found that imiquimod-induc
166                                        Using genetically modified mice, we found that loss of Id1 inh
167                            Using male CaMKIV genetically modified mice, we found that nicotine reward
168                                        Using genetically modified mice, we have shown that renal phos
169 nctional assays of platelets from humans and genetically modified mice, we identify layilin as the re
170                                        Using genetically modified mice, we show a dynamic role of RGS
171                       Using a combination of genetically modified mice, we show that the coordinated
172                                        Using genetically modified mice, we systematically examined th
173                                              Genetically modified mice were developed to selectively
174 L-10(-/-)) mice and several other strains of genetically modified mice were generated and used.
175                                 Studies with genetically modified mice were performed.
176                              For this study, genetically modified mice were used to determine how SMA
177 es, and electrophysiological recordings from genetically modified mice were used to show that WAVE-1
178 ugs acting on the endocannabinoid system and genetically modified mice were used.
179 ssection of the immune response to HBV using genetically modified mice whose immunoregulatory and imm
180                                           In genetically modified mice with a 50% reduction in liver
181                       In this study, we used genetically modified mice with a mutation (KR389-390AA)
182                       In this study, we used genetically modified mice with a mutation (KR389-390AA)
183                                              Genetically modified mice with approximately 10% of the
184                            We then show that genetically modified mice with CARMA3-deficient AECs hav
185 as 40% suppressed ventricular arrhythmias in genetically modified mice with catecholaminergic polymor
186                 Properties were evaluated in genetically modified mice with differing levels of TGF-b
187              Immunization of MV-susceptible, genetically modified mice with either vector induced neu
188  abnormal muscular septum, which phenocopied genetically modified mice with elevated BMP10 levels.
189 ffect of FoxO6 on hepatic gluconeogenesis in genetically modified mice with FoxO6 gain- versus loss-o
190                                           In genetically modified mice with low tissue factor activit
191                                        Using genetically modified mice with lung-specific inducible (
192                                           In genetically modified mice with reduced catechol-O-methyl
193 r phenotype of liver damage than those using genetically modified mice, with the exception of the chr
194 tes in regulating intact FGF23 production in genetically modified mice without and with adenine-induc

 
Page Top