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1                                              Geniculocortical afferent arbors in layer IV of area 17
2                                       First, geniculocortical afferent arbors serving either the depr
3 ation of early mGluR2/3 laminar changes with geniculocortical afferent segregation indicate that mGlu
4 consistent with a strictly Hebbian model for geniculocortical afferent segregation.
5 ative amounts of synaptic vesicle protein in geniculocortical afferents after both 2 and 7 d of MD.
6 hese results indicate that TrkB receptors on geniculocortical afferents are potential mediators of th
7 eurons express TrkB, it is not known whether geniculocortical afferents express this receptor on thei
8       This deprivation induces remodeling of geniculocortical afferents in a distribution that corres
9               Second, transneuronal label of geniculocortical afferents in flattened sections of cort
10                   The effect of shrinkage of geniculocortical afferents in layer 4C following visual
11 levels of TrkB-like immunoreactivity (IR) on geniculocortical afferents in layer IV of primary visual
12                   In primate striate cortex, geniculocortical afferents in layer IVc terminate in par
13 te afferents into eye-specific layers and of geniculocortical afferents into ocular dominance bands.
14 erely permissive, role in the segregation of geniculocortical afferents into ocular dominance columns
15                 We have anatomically labeled geniculocortical afferents of postnatal day 40 kittens w
16                    Because the patterning of geniculocortical afferents resists this dramatic change
17 lar dominance columns, initially overlapping geniculocortical afferents segregate by means of an acti
18                      In macaque monkeys, the geniculocortical afferents serving each eye segregate in
19                    In normal animals, single geniculocortical afferents serving the contralateral eye
20      However, a significant expansion of the geniculocortical afferents serving the nondeprived eye w
21 ors can also influence the morphology of the geniculocortical afferents that project from the lateral
22 on, we have studied the morphology of single geniculocortical afferents to area 17.
23 ices obtained for colocalization of TrkB and geniculocortical afferents were also compared with the e
24 pupil area eclipsed to trigger remodeling of geniculocortical afferents.
25 ge in distribution follow the development of geniculocortical afferents.
26 : a near-surround generated predominantly by geniculocortical and intra-V1 horizontal connections, an
27 nar analysis demonstrated plasticity of both geniculocortical and intracortical connections.
28 rivation (MD), the shrinkage of deprived-eye geniculocortical arbors is less than half-maximal at 4 d
29 a decrease in the total length of individual geniculocortical arbors representing the deprived eye.
30  loss of presynaptic sites from deprived-eye geniculocortical arbors.
31  candidate retrograde signaling molecule for geniculocortical axons during the formation of ocular do
32  analysis indicated that TrkB was present on geniculocortical axons for all five TrkB antibodies test
33                             More than 90% of geniculocortical axons from the dorsal lateral geniculat
34 l development did not prevent segregation of geniculocortical axons into alternating stripes with per
35 ed to involve the segregation of overlapping geniculocortical axons into eye-specific patches based o
36     We found that the density of synapses in geniculocortical axons was similar for deprived and nond
37 d showed significant colocalization with the geniculocortical axons.
38 tern to that of TrkB but is not expressed on geniculocortical axons.
39 ions, indicating active rearrangement of the geniculocortical circuit.
40 play an important role in the development of geniculocortical circuits and the emergence of cortical
41 to P40 promoted the growth of the open eye's geniculocortical connections without causing the closed
42 ise retinogeniculate connections help refine geniculocortical connections, sharpening both thalamocor
43 en found previously for retinogeniculate and geniculocortical connections.
44 two experimental conditions, we compared the geniculocortical connectivity in normal kittens with tha
45 Here, we have determined the contribution of geniculocortical feedforward and corticogeniculate feedb
46 ing, whereas beta-band interactions mediated geniculocortical feedforward processing.
47 ominent role in shaping the axonal arbors of geniculocortical fibers and the arbors of Y cells in the
48  in layers and compartments receiving direct geniculocortical innervation.
49  in the relative timing of the maturation of geniculocortical inputs and intracortical lateral connec
50       These inputs were similar in number to geniculocortical inputs and, therefore, considered robus
51 h the convergence of ipsi- and contralateral geniculocortical inputs as well as transcallosal project
52                 The mapping of eye-specific, geniculocortical inputs to primary visual cortex (V1) is
53                                  We examined geniculocortical inputs to these two sublayers labeled b
54                   The overlapping pattern of geniculocortical inputs with M2 muscarinic acetylcholine
55                                              Geniculocortical K axons were labeled via iontophoretic
56 he dLGN of ephrin-A2A5(-/-) mice, indicating geniculocortical map refinement.
57                            We show here that geniculocortical mapping is imprecise when the waves of
58                      Analysis shows that the geniculocortical miswiring is not a trivial or necessary
59 crease in cytochrome c oxidase activity, and geniculocortical neurons at the transitional, early apop
60                Transneuronal labeling of the geniculocortical pathway revealed changes in both cases,
61          We performed experiments in the cat geniculocortical pathway, in vivo, to examine how presyn
62 hypothesis that a rapid loss of deprived-eye geniculocortical presynaptic sites is responsible for th
63 rker for the koniocellular or "K" pathway of geniculocortical processing.
64 t apoptotic, oxidatively damaged neurons are geniculocortical projection neurons.
65             Previous anatomic studies of the geniculocortical projection showed that ocular dominance
66 ntially activated by one eye showed that the geniculocortical projection was already partially segreg
67                              In the afferent geniculocortical projection, LI-rTMS decreased the abnor
68                         Retinogeniculate and geniculocortical projections are privileged over modulat
69 timulation to characterize the koniocellular geniculocortical projections in nonhuman primates.
70 blood vessels leads to rewiring of the eye's geniculocortical projections, imprinting an image of the
71  dendritic spines, are largely excluded from geniculocortical recipient layers of the striate cortex.
72 omplex and subtle role in the control of the geniculocortical relay.
73 uggesting they arise from multisynaptic, non-geniculocortical sources.
74 uppression in the thalamus and depression at geniculocortical synapses.
75 lso requires a significant NMDA component in geniculocortical synapses.
76 em, retinorecipient nuclei distinct from the geniculocortical system.
77 nto V1 and V(HO); this latter phase requires geniculocortical TCA input to the nascent V1 that determ
78 cal axon (TCA) input by genetically deleting geniculocortical TCAs and showed that they drive differe
79      Moreover, we found that the majority of geniculocortical terminals in both IVa and IVb contained
80          In addition, we found that 9-10% of geniculocortical terminals in each sublamina contacted G
81 cal areas, we examined the ultrastructure of geniculocortical terminals in the tree shrew striate cor
82                       We found that layer IV geniculocortical terminals, as well as their postsynapti
83 -surround facilitatory-inhibitory effects on geniculocortical transmission via corticoreticulogenicul