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1                             Importantly, the genioglossus activating effects of these interventions w
2 ry units [mean +/- SEM], p < 0.01) and tonic genioglossus activation (36.3 +/- 5.3 to 20.7 +/- 3.9 ar
3 a number of respiratory variables, including genioglossus activation under both nasal and tracheal st
4  the frequency and amplitude of the sporadic genioglossus activations occurring during REM sleep.
5 haryngeal negative pressure itself modulates genioglossus activity both within breaths and between br
6                                              Genioglossus activity during wakefulness and sleep, geni
7 e that intrapharyngeal pressure may modulate genioglossus activity during wakefulness, with a fall in
8 esipramine abolished the normal reduction of genioglossus activity from wakefulness to non-REM sleep
9 normal individuals during stable NREM sleep, genioglossus activity rises above baseline as PCO2 rises
10 0 mg prevents the state-related reduction in genioglossus activity that occurs during sleep and there
11 lossal motor pool prevents the inhibition of genioglossus activity throughout REM sleep; likewise, wi
12 50-92] wakefulness; P = 0.01) but not phasic genioglossus activity was higher with desipramine compar
13 sed respiratory rate and respiratory-related genioglossus activity, and increased the frequency and a
14 e sought to determine the stimuli modulating genioglossus activity, dissociating the influences of ph
15 sal motor nucleus (MoXII) restores REM sleep genioglossus activity, highlighting the importance of ch
16 obese mice and examined the effect of J60 on genioglossus activity, pharyngeal patency, and breathing
17 gh flow also showed strong correlations with genioglossus activity, there was a significant change in
18 tructive apnea in NREM sleep augments phasic genioglossus activity.
19                                              Genioglossus and diaphragm activities were recorded in 3
20 at 3T the fanlike configuration of the human genioglossus and the laterally positioned merging fibers
21 ruitment of four major upper airway muscles (genioglossus, digastric, sternohyoid, and omohyoid) and
22 vity of the moving-time average (MTA) of the genioglossus electromyogram (EMG-GG) and the esophageal
23 females) during stable NREM sleep, measuring genioglossus electromyogram, epiglottic/choanal pressure
24                     The correlations between genioglossus electromyography (GGEMG) and epiglottic pre
25 , with an epiglottic catheter, intramuscular genioglossus electromyography, nasal mask and pneumotach
26 ity (ventilation at eupneic drive), baseline genioglossus EMG activity, or responsiveness.
27 ), ventilation (oronasal "ventilation"), and genioglossus EMG activity.
28                                 In 6-8 weeks genioglossus EMG and dynamic MRI of the upper airway wer
29                                              Genioglossus EMG signals were analyzed offline by automa
30 cord pharyngeal dilator muscle activity (the genioglossus [EMGgg] normalized to the wakeful baseline)
31 cord pharyngeal dilator muscle activity (the genioglossus [EMGgg]), we evaluated the muscle, ventilat
32                                              Genioglossus (GG) activation in response to upper airway
33     We therefore determined waking levels of genioglossus (GG) and tensor palatini (TP) muscle activi
34                          Reflex increases in genioglossus (GG) muscle activity in response to negativ
35                                          The genioglossus (GG) muscle is considered the principal pro
36 rvating the superior longitudinalis (SL) and genioglossus (GG) muscles exhibit distinct biophysical p
37 astric (LAD, RAD), masseter, buccinator, and genioglossus (GG) muscles within the rat's face primary
38 control the superior longitudinalis (SL) and genioglossus (GG) muscles, which retract and protrude th
39 bialis anterior (TA)) and a deep muscle (the genioglossus (GG)) during contractions at various forces
40 ance and electromyographic (EMG) activity of genioglossus (GG), hyoglossus (HG) and inspiratory inter
41  dilator muscle electromyograms (EMGs, i.e., genioglossus [GG-an inspiratory phasic muscle], tensor p
42 ion, plus activation of two dilator muscles (genioglossus [GG] and tensor palatini [TP]) were monitor
43 st the hypothesis that the tongue protrudor (genioglossus, GG) and retractor (styloglossus, SG and hy
44 eviously shown that the activity of both the genioglossus (GGEMG) and tensor palatini (TPEMG) are dec
45 during wakefulness, the activity of both the genioglossus (GGEMG) and tensor palatini (TPEMG) is grea
46                                          The genioglossus is an upper airway dilator muscle, the leng
47  we showed that muscarinic inhibition of the genioglossus is functionally linked to GIRK channel acti
48 noid, and posterior cricoarytenoid), tongue (genioglossus), jaw (digastric), and respiration (diaphra
49 nerve branches innervating tongue protrudor (genioglossus; medial XIIth nerve branch) and retractor (
50 Immunocytochemical results revealed that the genioglossus motoneuron pool, comprising the ventrolater
51 BP), heart rate (HR), diaphragm (D(EMG)) and genioglossus muscle (GG(EMG)) activity were recorded in
52 sodic hypoxia evokes persistent increases of genioglossus muscle (GG) activity, termed long-term faci
53              It is well established that the genioglossus muscle (tongue protrudor) has a role in pro
54 ormoxic hypercapnia alone leads to increased genioglossus muscle activation.
55 drenergic and antimuscarinic effects improve genioglossus muscle activity and upper airway patency du
56 mine reduces the state-related drop in tonic genioglossus muscle activity that occurs from wakefulnes
57 s that, during both REM and REM-like states, genioglossus muscle activity was strongly depressed and
58 ximum inspiratory flow, oronasal resistance, genioglossus muscle activity, and arterial blood pressur
59 tration of AAV9-DREADD, J60 can activate the genioglossus muscle and improve pharyngeal patency and b
60    In DREADD-treated mice, CNO activated the genioglossus muscle and markedly dilated the pharynx, wh
61 drugs (DREADD) could be used to activate the genioglossus muscle as a potential novel treatment strat
62 ration, HFPOs caused tonic activation of the genioglossus muscle EMG and inhibition of inspiratory mo
63  14; AHI, 4 +/- 1/h) were extracted from the genioglossus muscle EMG signals.
64 ratory time (TE) and tonically activated the genioglossus muscle EMG.
65                   A robust activation of the genioglossus muscle in all lean and obese rats was assoc
66                  These data suggest that the genioglossus muscle is less responsive to either chemica
67  synaptic contacts with retrogradely labeled genioglossus muscle motoneuronal dendrites and perikarya
68 DOR terminals contacted retrogradely labeled genioglossus muscle motoneurons.
69 of these processes with retrogradely labeled genioglossus muscle motoneurons.
70 Syn-hM3(Gq)-mCherry or control AAV9 into the genioglossus muscle of diet-induced obese mice and exami
71 of pharyngeal dilator muscles, including the genioglossus muscle of the tongue, is required to mainta
72 lossal (XII) motoneurons (MNs) innervate the genioglossus muscle of the tongue, which plays an import
73 h peroxidase (CTB-HRP) was injected into the genioglossus muscle on the right side of four isoflurane
74 ughout all sleep stages (p = 0.010), whereas genioglossus muscle responsiveness did not change.
75 otal of 36% of patients with OSA had minimal genioglossus muscle responsiveness during sleep, 37% had
76 ossus activity during wakefulness and sleep, genioglossus muscle responsiveness to negative epiglotti
77                                  The AHI and genioglossus muscle responsiveness to negative esophagea
78 f the upper airway [Pcrit]) and nonanatomic (genioglossus muscle responsiveness, arousal threshold, a
79 he major protruder muscle of the tongue, the genioglossus muscle, are modulated by terminals containi
80 ng masseter muscle and the tongue-protruding genioglossus muscle.
81 tioxidant enzyme activity in the omohyoid or genioglossus muscle.
82 ss of sleep stage and despite an increase in genioglossus-muscle activity.
83                             In six patients, genioglossus-muscle electromyograms (EMGs) were recorded
84     While these results demonstrate that the genioglossus musculature is targeted by ENK inputs, they
85 anized differentially for the control of the genioglossus musculature whose activity is essential in
86  significantly reduced glucose uptake in the genioglossus of patients with sleep apnea in comparison
87  significantly reduced glucose uptake in the genioglossus (P = 0.03) in comparison with obese normal
88 e nerves - which innervate the diaphragm and genioglossus respectively - that we propose contributes
89                     Hypnotics did not affect genioglossus responsiveness (high QoE).
90 a-hypopnea index [AHI]; primary outcome) and genioglossus responsiveness (secondary outcome) in peopl
91 zolpidem increases the arousal threshold and genioglossus responsiveness in people with and without o
92                                              Genioglossus responsiveness increased approximately thre
93  25 for AHI, 11 for arousal threshold, 4 for genioglossus responsiveness), hypnotics minimally raised
94 a raising the arousal threshold and possibly genioglossus responsiveness.
95 tics on arousal threshold, OSA severity, and genioglossus responsiveness.
96 action, whereas independently activating the genioglossus resulted in tongue protrusion.
97 esults: Compared with control, J60 increased genioglossus tonic activity by greater than sixfold and