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1 had mucosal lesions (with 54 having a single genital lesion).
2 viral shedding that develops into recurrent genital lesions.
3 ductions in the frequency of HSV shedding or genital lesions.
4 . haematobium and a dynamic evolution of the genital lesions.
5 tients received the same local treatment for genital lesions.
6 s greatly reduced the frequency of recurrent genital lesions.
7 sing clinically indistinguishable facial and genital lesions.
8 x acts occurring when the source partner had genital lesions.
9 tudinally track these clonotypes in PBMC and genital lesions.
10 ermine the specific HPV types present in the genital lesions.
11 nts, both HSV-1 and HSV-2 was recovered from genital lesions.
12 marily from sacral ganglia causing recurrent genital lesions.
13 es, typically causing characteristic oral or genital lesions.
14 1), which typically causes recurrent oral or genital lesions.
17 nced rash, with 6 (46.2%) having significant genital lesions and 1 (7.7%) having a severe perianal le
19 nfection presented as a prolonged episode of genital lesions and a marked increase in the frequency o
20 cy in preventing the development of external genital lesions and anogenital HPV infection in boys and
22 ein C (gC2) and glycoprotein D (gD2) reduced genital lesions and asymptomatic HSV-2 genital shedding
24 on was associated with a reduced severity of genital lesions and lower levels of viral shedding in th
26 a sexually transmitted pathogen that causes genital lesions and spreads to the nervous system to est
28 protection against infection, as measured by genital lesions and vaginal virus titers 2 days after in
32 se, vaginal titers, DRG infection, recurrent genital lesions, and recurrent vaginal shedding of HSV-2
33 areas for HSV culture, maintained a diary of genital lesions, and were examined at the time of recurr
36 were detected in all biopsies, together with genital lesion-associated HPV types 6, 16, and 52, (as w
39 difference is attributable to less frequent genital lesions because lesions are accompanied by frequ
40 rated HIV-1 DNA in cells derived from healed genital lesion biopsies than in cells from control skin
42 ivity was assessed by testing 112 ulcerative genital lesions by the HC II assay and comparing the res
45 Human papillomavirus (HPV) causes external genital lesions (EGLs) in men, including condyloma and p
46 wice daily for HSV-2 detection and monitored genital lesions for 28-day periods at baseline and at in
49 ex viruses (HSV) cause painful cold sores or genital lesions in many people; less often, they affect
50 f people worldwide, causing painful oral and genital lesions, in addition to a multitude of more seve
52 n occur, most commonly during delivery; when genital lesions or prodromal symptoms are present, cesar
55 ng (RR, 1.7 [95% CI, 1.1-2.7]; P = .01), and genital lesions (RR, 2.1 [95% CI, 1.2-3.4]; P = .005), c
57 we examined the women with a colposcope for genital lesions, tested endocervical specimens for gonor
59 f HSV-specific CD4(+) cells that localize to genital lesions, we developed a sensitive and highly spe
66 e intention-to-treat population, 36 external genital lesions were seen in the vaccine group as compar
67 Both vaccines protected against death and genital lesions when infected 1 month after immunization
70 ogens significantly reduced the frequency of genital lesions, with the bivalent vaccine showing the g