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1 nous PGCs migrate through circulation to the genital ridge.
2 lacking Nanog fail to mature on reaching the genital ridge.
3 y due to the action of WNT4 within the early genital ridge.
4 erm cells begin to migrate to the developing genital ridge.
5 erm cells shortly after their arrival in the genital ridge.
6 initiates just before translocation into the genital ridge.
7  in the allantois to the time they enter the genital ridges.
8 ation, followed by directed migration to the genital ridges.
9 GCs), resulting in fewer PGCs colonizing the genital ridges.
10 approximately 10.5 dpc as the PGCs enter the genital ridges.
11  in which they migrate out of the gut to the genital ridges.
12 ctionally from the dorsal body wall into the genital ridges.
13 exit the gut, but do not migrate towards the genital ridges.
14  the gut mesentery at E10.5 migrate into the genital ridges.
15 al and migration as these cells colonize the genital ridges.
16  both around the dorsal aorta and in the uro-genital ridges.
17 e identify a possible PGC niche in the mouse genital ridges.
18  expressed in cells located centrally in the genital ridge and then later in cells located at the cra
19  find that chicken Dmrt1 is expressed in the genital ridge and Wolffian duct prior to sexual differen
20 romotes mesonephric cell survival within the genital ridges and that these cells support correct deve
21 nitial specification until they colonize the genital ridges, and suggest the existence of a ;spatio-t
22 reduced survival of PGCs that migrate to the genital ridge around embryonic day 11.5 (E11.5).
23 entifies the most striking difference in the genital ridge at early stages of its development between
24     SLC progenitors are localized within the genital ridge at the interface with the mesonephros and
25 s within the coelomic epithelium (CE) of the genital ridge, but from cells also able to give rise to
26  reactivation is thought to emanate from the genital ridge, but it is unclear whether it is specific
27  no longer migrate directionally towards the genital ridges, but instead rapidly fragment and disappe
28 to the masculinising environment of the male genital ridge, defining a temporal window during which P
29                            The complexity of genital ridge development obscures potential connections
30 bility that is restricted to PGCs within the genital ridge during a narrow temporal window.
31 ells (PGCs) fail to migrate correctly to the genital ridges early in organogenesis.
32 s CXCR4, whilst the body wall mesenchyme and genital ridges express the ligand SDF1.
33 s migrate normally, but somatic cells of the genital ridge fail to proliferate and a discrete gonad f
34 n, irrespective of the phenotypic sex of the genital ridge from which the EGCs had been derived.
35           The numbers of PGCs populating the genital ridge in TIAR-deficient embryos are severely red
36 ather than ovary from the indifferent gonad (genital ridge) in mammalian embryos.
37 uced to minimal levels in the Lhx9-deficient genital ridge, indicating that Lhx9 may lie upstream of
38   C-met expression is not detectable in male genital ridges isolated from embryos at 11.5 days postco
39                                         Male genital ridges isolated from embryos at 11.5 dpc are mor
40 t of Wt1 in mice results in apoptosis of the genital ridge, it is unknown whether WT1 is required for
41  cells derived after PGC colonisation of the genital ridge, "late" and embryonic stem (ES) cell lines
42             Reduced BMP signaling within the genital ridges led to increased somatic cell death withi
43 ac and the para-aortic splanchnopleura/aorta-genital ridges-mesonephros (P-Sp/AGM) region are the mai
44 tral aspect of the dorsal aorta in the aorta/genital ridge/mesonephros (AGM) region.
45 e Dmrt1 mRNA is expressed exclusively in the genital ridge of early XX and XY embryos.
46  of Sry in the undifferentiated, bipotential genital ridges of mammalian XY fetuses initiates testis
47                                  In a female genital ridge, or in a non-gonadal environment, PGCs dev
48                              Using rat organ genital ridge organ cultures, we found that inhibition o
49 o track through endoderm on their way to the genital ridge, our work raises the possibility that cons
50                                  Once in the genital ridge, PGCs cease dividing and differentiate acc
51 to be expressed exclusively in the mammalian genital ridge prior to sexual differentiation.
52 as deleted before sex determination and most genital ridge somatic cells differentiated into steroido
53 mous, and is not due to any influence of the genital ridge somatic cells upon the PGCs.
54 haracteristics in both sexes at indifferent (genital ridge) stages and that these persist, becoming m
55 stitution of the EGCs or with the sex of the genital ridge (testis versus ovary) from which the PGCs
56       In the mouse Dmrt1 is expressed in the genital ridge (the gonadal primordium) in both sexes and
57 cation in the extraembryonic mesoderm to the genital ridge, the gonadal anlage.
58 e number of PGCs reaching and populating the genital ridges, the molecular identity of only two of th
59 mitive vasculature is identical in XX and XY genital ridges until 11.5 days postcoitum (dpc), by 12.5
60  the developing hindgut, and traverse to the genital ridge where they cluster and ultimately inhabit
61 e through somatic tissues to the presumptive genital ridges, where they proliferate and differentiate