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1 ghtly regulated by 25-hydroxycholesterol and geranylgeraniol.
2 97 and augmented by the nonsterol isoprenoid geranylgeraniol.
3 s, which can be chemically complemented with geranylgeraniol.
4 ells was p53-independent and was reversed by geranylgeraniol.
5 and is augmented by the nonsterol isoprenoid geranylgeraniol.
6 d by addition of HMG-CoA, mevalonic acid, or geranylgeraniol.
7 endence of LTP on the non-sterol isoprenoid, geranylgeraniol.
8 olesterol and nonsterol isoprenoids, such as geranylgeraniol.
9 , or the prenylation precursors farnesol and geranylgeraniol.
10 ranylated by labelling J774 cells with [(3)H]geranylgeraniol.
11 whereas alendronate activation is blocked by geranylgeraniol.
12 se of the protons of esters of phytol and of geranylgeraniol.
13 ol, and alendronate effects are prevented by geranylgeraniol.
14 tially by mevalonate and more effectively by geranylgeraniol.
15 tabolically labeled with [(3)H]F-OH or [(3)H]geranylgeraniol ([(3)H]GG-OH).
16        Similar plots for 1-phosphono-(E,E,E)-geranylgeraniol, a dead-end inhibitor for GGPP, gave a c
17 tutive activity of LXRalpha was inhibited by geranylgeraniol, a metabolite of mevalonic acid.
18 vastatin, but not clodronate, are blocked by geranylgeraniol, a precursor of geranylgeranyl diphospha
19                             We conclude that geranylgeraniol acts specifically and quickly to affect
20                Time course studies show that geranylgeraniol acts within 5 min and at 2 different tim
21 nisomycin, a protein synthesis inhibitor, or geranylgeraniol, an intermediate in the cholesterol bios
22 itavastatin-induced apoptosis was blocked by geranylgeraniol and mevalonate, products of the HMGCR pa
23 tatin effects are prevented by mevalonate or geranylgeraniol, and alendronate effects are prevented b
24 nd FOLK harbor kinase activities for phytol, geranylgeraniol, and farnesol with different specificiti
25 orporation of exogenously provided farnesol, geranylgeraniol, and unnatural analogs of these isopreno
26 e certain sterols, oxysterols, farnesol, and geranylgeraniol, as well as the diphosphate derivatives
27  LTP are reversed by a 20-min treatment with geranylgeraniol but not by cholesterol.
28          This superinduction was reversed by geranylgeraniol, but not by farnesol, further confirming
29 rporation studies showed that mevalonate and geranylgeraniol, but not farnesol, released the lovastat
30      When added to lovastatin-treated cells, geranylgeraniol, but not farnesol, restored replication
31  also supplied with the 20-carbon isoprenoid geranylgeraniol, but not the 15-carbon farnesol, raising
32  adhesion was reversed by co-incubation with geranylgeraniol, but not with farnesol, with concurrent
33         This suggests that a diet containing geranylgeraniol can limit the anti-tumour activity of pi
34 that the oxysterol 25-hydroxycholesterol and geranylgeraniol combine to trigger extraction of reducta
35                                              Geranylgeraniol counteracts the antiaphrodisiac effect d
36  miriamin, characterized as a polyoxygenated geranylgeraniol derivative.
37 lesterol biosynthetic pathway, of which one, geranylgeraniol diphosphate, is required for learning in
38 s c were found to be farnesol esterified and geranylgeraniol esterified.
39  role of geranylgeranyl isoprenoids GGPP and geranylgeraniol (GGOH) in regulating Abeta production.
40 I as well as combinations of lovastatin with geranylgeraniol (GGOH) or farnesol (FOH) to investigate
41 ch utilizing a combination of lovastatin and geranylgeraniol (GGOH) to potentiate the ability of lova
42                                              Geranylgeraniol (GGOH), the alcohol derivative of GGpp,
43                                  Addition of geranylgeraniol (GGOL) in the presence of lovastatin, to
44  to sterols and to other products, including geranylgeraniol groups attached to proteins.
45    New fluorescent analogues of farnesol and geranylgeraniol have been prepared and then converted to
46 on and the counterproductive role of dietary geranylgeraniol highlight how we should assess statins i
47 te pathway and that a constant production of geranylgeraniol in a small subset of neurons is required
48 lts identify UBIAD1 as the elusive target of geranylgeraniol in reductase degradation, the inhibition
49                                              Geranylgeraniol inhibits binding of UBIAD1 to reductase,
50 as a treatment for ovarian cancer if dietary geranylgeraniol is controlled.
51                          The requirement for geranylgeraniol is independent of acute protein isopreny
52     Shorter (C10, geraniol) and longer (C20, geranylgeraniol) isoprenols were inactive.
53 ls 50- and 22-28-kDa proteins, whereas [(3)H]geranylgeraniol labels only 22-28-kDa proteins.
54 ly related compound, does not substitute for geranylgeraniol nor does another animal model of impaire
55 tent with N-BP inhibition of isoprenylation, geranylgeraniol or farnesol prevented accumulation in S
56 renoids, such as nerolidol, cis-farnesol, or geranylgeraniol, or metabolites in the cholesterol pathw
57  nude mice were maintained on a diet lacking geranylgeraniol, oral pitavastatin caused regression of
58 , when the animal diet was supplemented with geranylgeraniol, pitavastatin failed to prevent tumour g
59 sterification of bacteriochlorophyllide a by geranylgeraniol-PPi during bacteriochlorophyll a biosynt
60    This inhibition is prevented by exogenous geranylgeraniol, probably required for prenylation of GT
61 lyalkenes, including geraniol, farnesol, and geranylgeraniol, providing an efficient and stereoselect
62 led with bacteriochlorophyll esterified with geranylgeraniol, rather than with phytol, implying that
63 AS9-mediated knockout of UBIAD1 relieves the geranylgeraniol requirement for reductase degradation.
64       Exogenously supplied mevalonic acid or geranylgeraniol reversed the inhibitory effects of stati
65 eranyl acetate is progressively converted to geranylgeraniol then externalized.
66 splacement from reductase in the presence of geranylgeraniol, thereby preventing degradation of reduc
67 dation augmented by the nonsterol isoprenoid geranylgeraniol through an unknown mechanism.
68                                  Addition of geranylgeraniol to slices from 24-hydroxylase knockout m
69                        Localized delivery of geranylgeraniol to the dendritic trees of CA1 hippocampa
70  farnesyl diphosphate synthase inhibitors or geranylgeraniol transferase I inhibitors, we evaluated c
71 ated activation is blocked by mevalonate and geranylgeraniol whereas alendronate activation is blocke
72  fully by mevalonate and less effectively by geranylgeraniol whereas alendronate effects are blocked
73 f bacteriochlorophyll (BChl) a, phytol, with geranylgeraniol, which possesses three more double bonds
74                              The addition of geranylgeraniol, which restores geranylgeranylation, res