ライフサイエンスコーパス: gingivitis

1 etween health and periodontal disease (i.e., gingivitis).

2 perceived oral health and less self-reported gingivitis.

3 ease chances of having poor oral hygiene and gingivitis.

4 ong sex, digit sucking, OHS, and presence of gingivitis.

5 a (TRP) mouthwash in reduction of plaque and gingivitis.

6 with thalassemia major (TM) with or without gingivitis.

7 rs in a possible association between GDM and gingivitis.

8 evidence of generalized, moderate-to-severe gingivitis.

9 to clarify the relationship between PCOS and gingivitis.

10 isite for the development of both caries and gingivitis.

11 ed with healthy individuals or patients with gingivitis.

12 d dentifrice for the treatment of plaque and gingivitis.

13 either a clinically healthy periodontium or gingivitis.

14 evels had the highest frequency of pregnancy gingivitis.

15 variation was found in cases with health and gingivitis.

16 ation required for microbial development and gingivitis.

17 e and gingival inflammation in patients with gingivitis.

18 enting severe periodontitis and longstanding gingivitis.

19 ta of WSL, accounting for confounding due to gingivitis.

20 moglobin values in contrast to patients with gingivitis.

21 nd either clinically healthy periodontium or gingivitis.

22 rmation for the therapeutic effects of CT on gingivitis.

23 microbial interactions and clinical signs of gingivitis.

24 associated with increased odds of having BGI gingivitis.

25 d as having biofilm-gingival interface (BGI) gingivitis.

26 may play a significant role in patients with gingivitis.

27 givalis increased under conditions emulating gingivitis.

28 severe diarrhoea, and necrotising ulcerative gingivitis.

29 ssified as controls and 164 as children with gingivitis.

30 ults with naturally occurring plaque-induced gingivitis.

31 stically significant for clinically measured gingivitis.

32 ave a higher prevalence of dental caries and gingivitis.

33 althy controls in the presence or absence of gingivitis.

34 els of MMP-8 especially when associated with gingivitis.

35 indicated a significant impact of obesity on gingivitis.

36 served clinical improvement in patients with gingivitis.

37 ential therapeutic agent in the treatment of gingivitis.

38 ased chances of having poor oral hygiene and gingivitis.

39 of 351 sites, including periodontitis (109), gingivitis (115), and healthy (127) sites.

40 significantly elevated in subjects with BGI gingivitis (136.2 +/- 112.9 ng/ml and 277.2 +/- 187.2 ng

41 plotypes were more frequent in children with gingivitis (27% and 23% versus 19% and 21% in controls).

42 e frequent in controls than in children with gingivitis (36% versus 23%) (P=0.036).

43 Eighty participants (40 with gingivitis, 40 healthy) provided saliva at baseline and

44 evere periodontitis (88%) and the lowest for gingivitis (55%).

45 e frequent in controls than in children with gingivitis (60% versus 50%).

46 riodontal disease (periodontitis = 85.2% and gingivitis = 60.2%).

47 luded: chronic periodontitis, plaque-induced gingivitis, aggressive periodontitis, acute gingival and

48 <3 mm and bleeding on probing (BOP) <10%; 2) gingivitis, all PD <3 mm and BOP >/=10%; 3) periodontiti

49 these data from children with obesity alone, gingivitis alone, both, and neither.

50 aggressive periodontitis than in those with gingivitis, although not significantly higher than in he

51 cking habits, oral hygiene status (OHS), and gingivitis among a group of Nigerian children.

52 level of serum IL-6, is associated with BGI gingivitis among non-smoking patients with diabetes.

53 ration of healthy patients and patients with gingivitis and chronic periodontitis (CP) and correlates

54 However, the full relationship between gingivitis and COHRQoL has been assessed by only a small

55 Differences between subjects with gingivitis and controls in the frequency of haplotypes w

56 s were significantly higher in patients with gingivitis and CP compared with healthy controls (P <0.0

57 cytokines may suggest an interaction between gingivitis and GDM.

58 lpha, VEGF, and TNF-alpha total amounts than gingivitis and healthy groups (P < 0.05).

59 he GO+ and GO- groups compared with both the gingivitis and healthy groups (P <0.008).

60 gP, than those in the group of patients with gingivitis and in the group that was healthy (P <0.001).

61 at are significantly associated with health, gingivitis and mild periodontitis (<25% attachment loss)

62 ollected from 223 dogs with healthy gingiva, gingivitis and mild periodontitis with 72 to 77 samples

63 scenarios ranging from periodontal health to gingivitis and mild, moderate, and severe periodontitis.

64 features, however, were also identified for gingivitis and mucositis.

65 tions was positively associated with age and gingivitis and negatively associated with lifetime numbe

66 Data suggest that interaction between gingivitis and obesity may exhibit disease reciprocity i

67 ldren afflicted by different degrees of both gingivitis and obesity.

68 No significant association was found between gingivitis and obesity.

69 insult and inflammatory responses leading to gingivitis and peri-implant mucositis.

70 nas gingivalis results in the development of gingivitis and periapical bone loss, which apparently ar

71 ur discovery of the presence of ILCs both in gingivitis and periodontitis and concomitant expression

72 diabetic cohorts having periodontal health, gingivitis and periodontitis could reveal microbial sign

73 ean amounts of GCF TGF-beta1 were greater in gingivitis and periodontitis sites than in healthy sites

74 We collected gingivitis and periodontitis soft tissue and characteriz

75 tissues, comparing samples from experimental gingivitis and periodontitis subjects to those from heal

76 Mouse models of acute gingivitis and periodontitis were used to assess the ear

77 Periodontal diseases, such as gingivitis and periodontitis, are characterized by bacte

78 readouts which distinguished between heath, gingivitis and periodontitis, correlated identically wit

79 plaque, a highly complex biofilm that causes gingivitis and periodontitis, requires specific adherenc

80 depth were used as measures of the extent of gingivitis and periodontitis, respectively.

81 studies, the two major periodontal diseases, gingivitis and periodontitis, were combined and consider

82 maintaining chronic inflammation, including gingivitis and periodontitis.

83 limit caries, as well as halt progression to gingivitis and periodontitis.

84 h and without HIV who presented with chronic gingivitis and periodontitis.

85 fferent ILC subsets (ILC1, ILC2 and ILC3) in gingivitis and periodontitis.

86 tudies reporting measures of heritability of gingivitis and periodontitis.

87 ILCs were detected both in gingivitis and periodontitis.

88 iterature on the yet-unclear heritability of gingivitis and periodontitis.

89 elated negatively to IL-34, in patients with gingivitis and periodontitis.

90 Predictors of presence of gingivitis and poor oral hygiene were determined using m

91 n the same direction after onset of clinical gingivitis and returned to baseline levels after resolut

92 paste is effective in controlling plaque and gingivitis and slowing progression of periodontitis; how

93 ydroxyapatite) of oral pathogens involved in gingivitis and tooth decay.

94 385) and 11% (n = 80) of the sample had BGI gingivitis and type 2 diabetes, respectively.

95 microbial biofilm, and the host response in gingivitis and upon resolution following clinical interv

96 the anteromandibular sextant with or without gingivitis and with or without periodontitis at the lowe

97 to oral biofilms associated with "health", "gingivitis" and "periodontitis".

98 .31 years), 15 with generalized AgP, 15 with gingivitis, and 10 who were periodontally healthy.

99 females with PCOS, 30 females with PCOS and gingivitis, and 12 systemically and periodontally health

100 althy periodontium, 30 females with PCOS and gingivitis, and 12 systemically and periodontally health

101 zed aggressive periodontitis (GAgP), 20 with gingivitis, and 20 classified as healthy (H).

102 ithout GO [GO-; n = 20], 20 individuals with gingivitis, and 20 healthy participants).

103 his study: 31 periodontally healthy, 27 with gingivitis, and 31 with CP.

104 nts: 8 periodontally healthy, 9 experimental gingivitis, and 9 periodontitis subjects.

105 y determines prevalence of digit sucking and gingivitis, and association among age, sex, socioeconomi

106 us (attachment level, probing depth, plaque, gingivitis, and bleeding on probing scores) and signific

107 ations in periodontal assessment for health, gingivitis, and mild, moderate, and severe periodontitis

108 evicular fluid (GCF) collected from healthy, gingivitis, and periodontitis sites in humans, by liquid

109 were performed to compare data from healthy, gingivitis, and periodontitis sites.

110 val crevicular fluid collected from healthy, gingivitis, and periodontitis sites.

111 groups were established: periodontal health, gingivitis, and periodontitis.

112 nts (29 patients with periodontitis, 12 with gingivitis, and seven healthy patients) and related to t

113 evicular fluid samples from six healthy, six gingivitis, and three periodontitis sites were collected

114 e interval [CI]: 0.20 to 0.35; P <0.001) and gingivitis (AOR: 0.21; 95% CI: 0.14 to 0.31; P <0.001) w

115 (OR = 0.87, 95% CI = 0.10 to 7.84), nor was gingivitis associated with the ESS (OR = 1.25, 95% CI =

116 ng incubation with epithelium stimulated by "gingivitis-associated" biofilm.

117 ficant reduction of plaque and resolution of gingivitis at all post baseline time points (P < 0.0001)

118 n age, 23.3 +/- 5.0 [SD] years) with minimal gingivitis at baseline.

119 periodontitis (BGI-P1), health (BGI-H), and gingivitis (BGI-G) (P = 0.005).

120 uped as BGI-healthy (14.3% of sample) or BGI-gingivitis (BGI-G, 15.1%).

121 Plaque, gingivitis, bleeding on probing, suppuration, probing de

122 bohydrate metabolism network in experimental gingivitis but not in periodontitis.

123 h certain microbial taxa not associated with gingivitis by a previous study or the current one.

124 via wiggsiae (p = 0.04) Taxa associated with gingivitis by microarray included: Gemella sanguinis (p

125 ed with both healthy patients and those with gingivitis, by 2.60-fold and 2.77-fold, respectively (P

126 Plaque-induced gingivitis can be alleviated by various treatment regime

127 GI), and plaque index (PI) were monitored in gingivitis cases among systemically healthy patients (n

128 ted the ED with a primary diagnosis of acute gingivitis, chronic gingivitis, gingival recession, aggr

129 ons following SIV infection in macaques with gingivitis compared to controls.

130 serum IL-6 was elevated in subjects with BGI gingivitis compared to subjects with gingival health onl

131 res brought an additional risk of developing gingivitis compared with a high VPI score alone.

132 PO levels were higher in women with PCOS and gingivitis compared with periodontally healthy women wit

133 igher in the systemically healthy women with gingivitis compared with periodontally healthy women wit

134 ger than those in the group of patients with gingivitis, consistent with significantly greater ADAM8

135 unities during the resolution of established gingivitis continues to evolve as additional knowledge i

136 the increased volume of GCF associated with gingivitis could potentially dilute macrolides.

137 atients with periodontal diseases, including gingivitis, CP, and AgP, in comparison to control partic

138 subject at baseline (day 0), at the peak of gingivitis (day 28), and at resolution (day 35) and proc

139 Participants with gingivitis demonstrated significantly higher bleeding on

140 Bacterial levels, gingivitis, dental plaque, and caries experience were as

141 Desquamative gingivitis (DG) is a common clinical manifestation of or

142 described in the literature as desquamative gingivitis (DG).

143 ntitis] and 39 natural teeth [19 healthy, 12 gingivitis, eight periodontitis] in 15 systemically heal

144 Salivary PGE2 and MIP-1alpha discriminate gingivitis from health, and patients with gingivitis who

145 ation with MIP-1alpha, readily discriminated gingivitis from health.

146 hy controls (C group, n = 20), patients with gingivitis (G group, n = 20), and patients with chronic

147 ve periodontitis (GAgP), 18 individuals with gingivitis (G), and 19 periodontally healthy (H) partici

148 ur groups, including periodontal health (H), gingivitis (G), chronic periodontitis (CP), and aggressi

149 20 healthy (H) individuals; 20 patients with gingivitis (G); 20 CsA-medicated patients with GO (CsA G

150 25), chronic periodontitis (CP, n = 14), and gingivitis (G, n = 18) were tested for the presence of a

151 Seventy-one women with GDM and gingivitis (Gg), 30 women with GDM and healthy periodont

152 imary diagnosis of acute gingivitis, chronic gingivitis, gingival recession, aggressive or acute peri

153 s were lower in the mucositis group than the gingivitis group (P <0.05).

154 GDM with gingivitis group than non-GDM with gingivitis group (P = 0.044).

155 Healthy group (H, n = 20) and periodontitis/gingivitis group (PG, n = 70) according to their periodo

156 The gingivitis group consisted of 28 patients, 41-70 years o

157 bjects were equally recruited into a healthy/gingivitis group or a periodontitis population.

158 The PCOS + gingivitis group revealed significantly higher GCF, sali

159 The PCOS + gingivitis group revealed significantly higher insulin c

160 ANKL (P <0.0001) were higher in the GDM with gingivitis group than GDM without gingivitis group.

161 Saliva IL-6 level was higher in the GDM with gingivitis group than non-GDM with gingivitis group (P =

162 were significantly higher (2.8 times) in the gingivitis group than the healthy group (P </=0.02).

163 index was significantly higher in the PCOS + gingivitis group than the PCOS + healthy group (P = 0.03

164 Salivary HIF-1alpha concentrations in gingivitis group were significantly higher than G-AgP an

165 CF HIF-1alpha and TNF-alpha total amounts in gingivitis group were significantly higher than the heal

166 decrease significantly from baseline in the gingivitis group, although concentrations of IL-1beta, I

167 e GDM with gingivitis group than GDM without gingivitis group.

168 infected with multiple viral variants in the gingivitis group.

169 higher in the CP group than the healthy and gingivitis groups (P <0.001).

170 th chronic periodontitis (CP), patients with gingivitis (GV), and individuals with no periodontal dis

171 only obesity had 5.2% and children with only gingivitis had 19.16%.

172 the CPQ11-14; those with extended levels of gingivitis had a 1.20 times higher mean score than those

173 (Hh), and 37 systemically healthy women with gingivitis (Hg) were evaluated.

174 Gingivitis, however, showed a stronger positive correlat

175 alivary glucose levels on the development of gingivitis in a prospective longitudinal study of Kuwait

176 ompounds, on the development of experimental gingivitis in beagle dogs.

177 d have an active role in the pathogenesis of gingivitis in Caucasian children.

178 tigated the relationship between obesity and gingivitis in children by focusing on plasma and salivar

179 es the association of parenting practices on gingivitis in children using path analysis to evaluate i

180 y demographic factors that could account for gingivitis in children, with a focus on the mediational

181 the induction and resolution of experimental gingivitis in humans were not previously explored using

182 g frequency, oral hygiene effectiveness, and gingivitis in low social status adolescents.

183 rate periodontitis as compared to those with gingivitis in mid and late stages of pregnancy.

184 during onset and resolution of experimental gingivitis in smokers.

185 may exacerbate preconditions associated with gingivitis in susceptible individuals.

186 tered, may significantly reduce experimental gingivitis in the beagle dog.

187 ould exist between children with and without gingivitis in the distribution of IL-10 alleles and hapl

188 s, only a few investigations have focused on gingivitis in this at-risk population.

189 e association between the -1082*A allele and gingivitis in white Caucasian children.

190 being selectively modulated in experimental gingivitis, including neural processes, epithelial defen

191 Gingivitis increased over time in children who had short

192 oral SIV infection rates were similar in the gingivitis-induced and control groups (5 infections foll

193 giene practices ceased for 21 days to induce gingivitis (induction), after which home care was reinst

194 pants were enrolled in a 21-day experimental gingivitis investigation and grouped according to clinic

195 Ligneous gingivitis is a rare periodontal disorder closely associ

196 membranous periodontal disease, or ligneous gingivitis, is a rare condition involving nodular gingiv

197 arger in periodontitis than in long-standing gingivitis lesions.

198 L were minimally changed with adjustment for gingivitis level.

199 ically significant differences in plaque and gingivitis levels would help to determine whether oral h

200 matory immune modulators in the SIV-infected gingivitis macaques could also be observed in blood plas

201 ate that the presence of extensive levels of gingivitis might be negatively associated with how child

202 m accumulation, and GCF exudation in a human gingivitis model were examined.

203 ccumulation over 21 days in the experimental gingivitis model would elicit systemic inflammatory resp

204 s during a 28-day stent-induced experimental gingivitis model, followed by treatment, and resolution

205 The aim of this study, using an experimental gingivitis model, was to examine the development and res

206 healthy individuals (n = 15), patients with gingivitis (n = 15), and patients with severe chronic pe

207 ically healthy, non-smoking individuals with gingivitis (n = 20) or chronic periodontitis (CP) (n = 2

208 25); and 4) systemically healthy women with gingivitis (n = 20).

209 n additional separate group of patients with gingivitis (n = 21) and some of the patients with period

210 n with PCOS (n = 45); 2) women with PCOS and gingivitis (n = 35); 3) systemically and periodontally h

211 n-surgical treatment (N = 58), patients with gingivitis (N = 54) and periodontally healthy volunteers

212 use was associated with an increased risk of gingivitis (odds ratio [OR] =1.7; 95% confidence interva

213 Concerning the chronic gingivitis of the individuals with HIV, both strong and

214 the relationship between sleep duration and gingivitis on 3 levels: within schools, among children,

215 The effects of gingivitis on obesity were in the same direction but gen

216 ge: 46.0 +/- 8.8 years) and 16 patients with gingivitis only (mean age: 31.5 +/- 7.5 years) were inve

217 e results with serum levels of patients with gingivitis only.

218 patients with generalized AgP and those with gingivitis or a healthy periodontium.

219 aggressive periodontitis than in those with gingivitis or in healthy persons.

220 t were significantly associated with health, gingivitis or mild periodontitis.

221 Individuals with gingivitis or mild-moderate periodontitis (n=36) were in

222 patients having the basic clinical signs of gingivitis or periodontal disease).

223 e possible association between the extent of gingivitis or periodontitis and an index of gingival mic

224 , respectively, the clinical predominance of gingivitis or periodontitis in such a site.

225 hout HIV, both groups presented with chronic gingivitis or periodontitis, and no statistically signif

226 All individuals presented gingivitis or periodontitis.

227 ho had more decayed or filled teeth had more gingivitis ( P < 0.05).

228 alivary glucose levels >1.13 mg/dL predicted gingivitis ( P < 0.05).

229 n TM gingivitis than in systemically healthy gingivitis (P <0.001).

230 patients with CP compared with patients with gingivitis (P <0.01).

231 ontally healthy women or women with PCOS and gingivitis (P <0.05).

232 ts with GCP when compared with patients with gingivitis (P = 0.007, P = 0.004, P = 0.033, and P = 0.0

233 An additional sample was collected from gingivitis participants 10 to 30 days after dental proph

234 s (G-AgP), 20 chronic periodontitis (CP), 26 gingivitis patients, and 21 periodontally healthy indivi

235 nd -1beta in a sample of naturally occurring gingivitis patients.

236 have an affect on BOP in naturally occurring gingivitis patients.

237 ants presenting with either clinical health, gingivitis/peri-implant mucositis, or chronic periodonti

238 significant, but smaller than the effect of gingivitis, periodontal disease, smoking, caries, and ot

239 ifferentiate retinopathy; age, T1D duration, gingivitis, periodontitis at P < 0.001, sex, and serum G

240 us of individuals affected by plaque-induced gingivitis (pGI).

241 During the experimental gingivitis phase (days 0 to 21), the rate of change in g

242 ntal problems, such as gingival enlargement, gingivitis, poor oral hygiene, dental hypoplasia, and ca

243 wing gums or mouthwashes for both caries and gingivitis prevention.

244 derate gingivitis was designed with two anti-gingivitis regimens: the brush-alone treatment and the b

245 al signatures of the three treatments during gingivitis relieve indicate distinct mechanisms of actio

246 1 to 5 years had poor oral hygiene and mild gingivitis, respectively.

247 6 to 12 years had poor oral hygiene and mild gingivitis, respectively.

248 d that differences between periodontitis and gingivitis samples remained after adjusting for smoking,

249 The corresponding figure for gingivitis samples was 15%.

250 pression was higher in periodontitis than in gingivitis samples.

251 regnancy histories, risk factors, plaque and gingivitis scores, and current pregnancy outcomes.

252 ntally healthy individuals and patients with gingivitis showed similar HBD-2 levels, the CP group dis

253 (GI >0; PD > or =5 mm; AL > or =3 mm), three gingivitis sites (GI >0; PD < or =3 mm; AL = 0), and two

254 mycin concentrations are similar in GCF from gingivitis sites and healthy sites, suggesting that the

255 ine, lysine, putrescine, and xanthine at the gingivitis sites as early as week 1.

256 Clarithromycin levels at control and gingivitis sites were higher than serum by 5.7- and 7.0-

257 romycin was compared in GCF from healthy and gingivitis sites.

258 crevicular fluid (GCF) could be increased at gingivitis sites.

259 ls participated in an experimentally induced gingivitis study, and gingival biopsies were collected a

260 olism are markedly different in experimental gingivitis subjects compared with healthy controls.

261 suggest that among never and former smokers, gingivitis susceptibility is higher among men with perio

262 d IL-6 and IL-8 (P <0.005) were higher in TM gingivitis than in systemically healthy gingivitis (P <0

263 MP-9 levels were lower in healthy women with gingivitis than systemically and periodontally healthy w

264 centrations were significantly higher in the gingivitis than the mucositis group (P = 0.004).

265 Many taxa showed a stronger association with gingivitis than with WSL.

266 5 for each group: the group of patients with gingivitis, the group with aggressive periodontitis [AgP

267 serum YKL-40 and IL-6 levels increased from gingivitis to periodontitis.

268 Heritability for the self-reported gingivitis trait was estimated at 0.29 (95% CI, 0.22 to

269 Response to gingivitis treatment in patients with diabetes can sligh

270 ed clinical trial compared the response to a gingivitis treatment protocol that combined mechanical p

271 To probe the impacts of various anti-gingivitis treatments on plaque microflora, here a doubl

272 the induction and resolution of experimental gingivitis using bioinformatic tools.

273 ildren were assessed for OHS and severity of gingivitis using the simplified oral hygiene index and t

274 The prevalence of gingivitis was 53.9% for females who reported having use

275 odel of supragingival plaque associated with gingivitis was characterized by traditional culture tech

276 Because improvement in gingivitis was comparable with that of CHX mouthwash, TR

277 nical examination of oral hygiene status and gingivitis was conducted using simplified oral hygiene a

278 controlled trial of 91 adults with moderate gingivitis was designed with two anti-gingivitis regimen

279 The level of gingivitis was different among the 6 governorates of Kuw

280 Gingivitis was evaluated as the percent of sites conside

281 Gingivitis was found in 68.2%, periodontitis in 21.2%, a

282 ically applied to the gingiva after moderate gingivitis was identified through clinical and immunolog

283 The odds of having gingivitis was increased in children with low socioecono

284 Experimental gingivitis was induced in 15 smokers for 21 days, follow

285 A localized experimental gingivitis was induced in 9 patients with APE (test grou

286 Experimental gingivitis was induced in eight healthy subjects at one

287 Experimental gingivitis was induced in one maxillary posterior sextan

288 In conclusion, while moderate gingivitis was not associated with increased susceptibil

289 Onset of clinical gingivitis was preceded by significant changes in the ma

290 Experimental gingivitis was then induced, with cessation of plaque co

291 those with healthy periodontal tissues/mild gingivitis were included.

292 Ninety individuals with chronic generalized gingivitis were randomly assigned to three groups: 1) gr

293 patients diagnosed with chronic generalized gingivitis were selected and randomly divided into three

294 Clinical features of health and gingivitis were stable at both baseline visits.

295 r resolution phase of experimentally induced gingivitis, which represented a reversible periodontal l

296 IL-17E were lowest in females with PCOS and gingivitis who also had the highest FGS.

297 te gingivitis from health, and patients with gingivitis who return to clinical health continue to pro

298 were reported using 10 different indices and gingivitis with nine indices.

299 regression models fitted the association of gingivitis with overall and domain-specific CPQ11-14 sco

300 estigation is to explore the relationship of gingivitis with salivary biomarkers, periodontal pathoge