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1 pact of CovS on CovR phosphorylation and GAS global gene expression.
2 in genes, with minimal off-target changes in global gene expression.
3 rnative to microarray-based methods to study global gene expression.
4  of human genomes and their association with global gene expression.
5 d the effects of single copy mutant K-Ras on global gene expression.
6 imilar effects to overexpressing HP1alpha in global gene expression.
7 romatin and effects of Gbeta(2) depletion on global gene expression.
8 oral program integrating DNA replication and global gene expression.
9 its expression in NIH 3T3 cells and analyzed global gene expression.
10  of diverse transcription factors that drive global gene expression.
11 es, thereby acting as a positive modifier of global gene expression.
12 SC lots from each center were compared using global gene expression.
13 logy to characterize the temporal changes in global gene expression after contusive SCI in mice.
14 neural crest-derived cells (CNCCs) to define global gene expression along the dorsoventral axis of th
15 called RNA-seq has been widely used to study global gene expression, alternative exon usage, new exon
16                             Quantitative and global gene expression analyses (microarray and RNAseq)
17                                     Unbiased global gene expression analyses demonstrate that MECP2 f
18                                              Global gene expression analyses following Setd8 knockdow
19                                              Global gene expression analyses reveal that trisomy 12 p
20                                 Furthermore, global gene expression analyses show that GW9662 treatme
21                                              Global gene expression analyses suggested that Rb mainta
22                                              Global gene expression analyses were conducted using spl
23 s that integrates comparative genomics data, global gene expression analyses, and intrinsic propertie
24 terol metabolism were assessed together with global gene-expression analyses in peripheral blood mono
25              RNA-sequencing (RNA-seq) allows global gene expression analysis at the individual transc
26                                              Global gene expression analysis identified a molecular s
27                                              Global gene expression analysis identified ablated Tiam2
28                                              Global gene expression analysis identified that PELP1-cy
29                                              Global gene expression analysis in AD-HIES patient skin
30                                              Global gene expression analysis indicated that 47% of an
31                                            A global gene expression analysis of AS versus empty vecto
32                           Here, we performed global gene expression analysis of human dermal papilla
33                                              Global gene expression analysis of infected hNPCs reveal
34                                              Global gene expression analysis of KDM3A-depleted cells
35                                              Global gene expression analysis of knockout hearts befor
36                                              Global gene expression analysis of mesenchymal stem cell
37                                            A global gene expression analysis of S100A12-activated mon
38 cale experimental approach that involved the global gene expression analysis of strains D23580 and 4/
39 sequencing, X-chromosome inactivation study, global gene expression analysis on Epstein-Barr virus (E
40                                              Global gene expression analysis on liver identified Tpcn
41                                        While global gene expression analysis profiles the average exp
42                                         This global gene expression analysis provides new insights in
43                                              Global gene expression analysis revealed changes in gene
44                                              Global gene expression analysis revealed marked differen
45                                              Global gene expression analysis revealed that genes upre
46                                              Global gene expression analysis revealed that infection-
47       Consistent with methylome alterations, global gene expression analysis reveals that the vast ma
48 x5 and other B-lineage-associated genes, and global gene expression analysis suggested that the reduc
49 ation assays, surface antigen profiling, and global gene expression analysis to identify the lines ex
50 h infection assays, genome resequencing, and global gene expression analysis to study the effect of t
51  To test for MR function in skeletal muscle, global gene expression analysis was conducted on human m
52                                              Global gene expression analysis was performed on HMEC li
53                                              Global gene expression analysis, data intersection, path
54                                              Global gene-expression analysis has shown remarkable dif
55                                              Global gene-expression analysis of the transgenic plants
56                                              Global gene-expression analysis showed that the steroid
57 xpression was defined by Hic-5 depletion and global gene-expression analysis.
58  early muscle marker genes are reprogrammed, global gene expression and accessibility changes are sti
59                               By integrating global gene expression and computational analyses, we di
60                      Based on comparisons of global gene expression and DNA methylation between the w
61                                        Using global gene expression and DNA methylation profiling, we
62 nduced DNA methylation, we analyzed parallel global gene expression and DNA methylation using normal
63 igate this effect, we have characterized the global gene expression and epigenetic profiles of multip
64 ts that overexpressing these proteins had on global gene expression and freezing tolerance.
65         MicroRNAs (miRNAs) are regulators of global gene expression and function in a broad range of
66                                      We used global gene expression and genome-wide binding analyses
67 nd loss-of-function approaches combined with global gene expression and genome-wide transcription fac
68 e findings indicate that lincRNA-p21 affects global gene expression and influences the p53 tumor supp
69  in longevity across mammals at the level of global gene expression and metabolite levels and reveals
70 te the validity of this concern, we compared global gene expression and methylation profiles between
71                          Here, we integrated global gene expression and proteomic analyses and identi
72                                              Global gene expression and secretome analysis reveals th
73 ounteract the effects of ATF3 or CSL loss on global gene expression and suppress CAF tumor-promoting
74 hereas filaggrin haploinsufficiency affected global gene expression and was characterized by a type 1
75                                              Global gene-expression and methylome analyses of TKO EBs
76  (PEV), and the link among rDNA copy number, global gene expression, and chromatin regulation.
77 ing system to control SpeB production, alter global gene expression, and enhance virulence.
78            Although average telomere length, global gene expression, and microbiome changes returned
79 f the role of the Y chromosome in modulating global gene expression, and suggest a link with modifica
80                            Using an unbiased global gene expression approach in conditional p120-cate
81 s, there are also fundamental differences in global gene expression as pathogenic filamentous fungi u
82 sis was employed to determine alterations in global gene expression associated with miR-10a in embryo
83    In the present study, we investigated the global gene expression associated with stomach carcinoge
84                                              Global gene expression at 8 and 24 h was analyzed by dee
85                          Our previous map of global gene expression, based on ~400K single nucleotide
86  vaccine and performed a timed assessment of global gene expression before and after vaccination.
87                              A comparison of global gene expression between severe and mild disease c
88 red the relationship between sexual mode and global gene expression by comparing two selfing species,
89 te a statistical model for the regulation of global gene expression by multiple regulatory programs a
90 RNAi and further determination of changes in global gene expression by RNAseq, and composition of pri
91 can alleviate shRNA-mediated perturbation of global gene expression by specifically de-repressing off
92                                              Global gene expression can be quantified from the number
93 ssess a highly homogeneous morphology, size, global gene expression, cellular composition, and struct
94 all relationship between cell commitment and global gene expression changes are still unclear.
95                                              Global gene expression changes associated with upregulat
96                          Further analysis of global gene expression changes during reprogramming reve
97 about the effects of caffeine stimulation on global gene expression changes in neurons.
98                                  We analyzed global gene expression changes in seedlings grown in med
99 is study provides clinical trial evidence of global gene expression changes occurring in the human co
100 n this work, we describe the biochemical and global gene expression changes of epimastigotes under hy
101 sion of endogenous Oct4, Nanog and Klf4, and global gene expression changes that enriched for transcr
102                                We determined global gene expression changes using Illumina HiSeq-base
103 process and identification of the associated global gene expression changes.
104 itro differentiation conditions and measured global gene-expression changes using gene expression mic
105 n entire developmental pathway, we generated global gene expression, chromatin accessibility, histone
106                                              Global gene expression comparisons between Bintje and up
107 ecent hypothesis-driven studies that utilize global gene expression data for elucidating the molecula
108                                  Analyses of global gene expression data from adipose tissue, skeleta
109                                  Analysis of global gene expression data from HOXA5-depleted MCF10A b
110 ociated with the hippocampus, I analyzed the global gene expression data from post-mortem hippocampal
111  parameters in cervical cancer combined with global gene expression data to reveal their underlying m
112 ys and Affymetrix HTA 2.0 arrays to generate global gene expression data with doxycycline induction.
113 l practices can lead to misinterpretation of global gene expression data.
114                            Both targeted and global gene expression differences in the transcriptome
115      Biological pathways were compared using global gene expression differences.
116                                              Global gene-expression differences in ectopic T1 vs. T2/
117                    Depletion of NusA altered global gene expression directly and indirectly via readt
118 al. (2014) uncover extensive oscillations in global gene expression during C. elegans development, in
119 ription factor participates in reprogramming global gene expression during Cu insufficiency in order
120                             We have profiled global gene expression during optic fissure morphogenesi
121 s to monitor their environment and reprogram global gene expression during specific stages of infecti
122 er significantly from primary mDA neurons in global gene expression, especially in genes related to n
123 omparable to adult articular chondrocytes in global gene expression, extracellular matrix production,
124 rray analysis of a SS cell line, we surveyed global gene expression following combined PKCbeta-GSK3 t
125        We assayed peripheral blood leucocyte global gene expression for a prospective discovery cohor
126 d metal ions and induce distinct patterns of global gene expression from established LRAs.
127               We investigated alterations to global gene expression (GE) in nontransformed human smal
128  This analysis reveals that the magnitude of global gene expression heterogeneity is regulated in res
129 ovo steroidogenic immune cells, defining the global gene expression identity of these steroid-produci
130 ole of poly(ADP-ribose) polymerase (PARP) on global gene expression in a lymphoblastoid B cell line.
131  RNA-Seq has provided valuable insights into global gene expression in a wide variety of organisms.
132 nal regulators with the ability to influence global gene expression in addition to modulating gene ex
133                             We then profiled global gene expression in blood from individual mice usi
134 engers, carboxy-PTIO and DMSO, on changes in global gene expression in cultured normal human fibrobla
135                              We analyzed the global gene expression in epithelium collected intraoper
136 te-limiting steps have been shown to control global gene expression in eukaryotes: preinitiation comp
137 rk highlights the hierarchical patterning of global gene expression in floral development, and suppor
138  of VP35 and VP24 with the IRADs disabled on global gene expression in human DC.
139 n in CF following TLR signaling, we profiled global gene expression in immortalized human CF and non-
140                                              Global gene expression in mouse HBCs reveals broad upreg
141 ecific neuronal genes, while dispensable for global gene expression in murine ES cells.
142 ct of genome-wide cardiac DNA methylation on global gene expression in myocardial samples from end-st
143 s complex are critical for the regulation of global gene expression in ocular cells, making LOXL1-AS1
144 lie skin specific manifestation, we analyzed global gene expression in peripheral blood of a small co
145 d sequencing for the time-course analysis of global gene expression in practically any organism.
146            In the present study, we compared global gene expression in primitive, fetal definitive, a
147 his study provides a detailed description of global gene expression in seven successive developmental
148 n, ISGF3-independent signaling in regulating global gene expression in STAT1-, STAT2-, or IRF9-defici
149 echanism by which satellite repeats regulate global gene expression in trans via piRNA-mediated gene
150 y defects in ribosome synthesis by analyzing global gene expression in various cellular models of DBA
151 dy, we utilized RNA-Seq technology to assess global gene expression in white lupin cluster roots, nor
152                                 In comparing global gene expression in wild-type, TLR9-, or STAT3-def
153 and in vivo and caused widespread changes in global gene expression, including up-regulation of myoge
154 bisphenol A (BPA) has been reported to alter global gene expression, induce epigenetic modifications,
155 e to the difficulty in embryo isolation, the global gene expression involved in plant embryogenesis,
156                   We presently describe that global gene expression is similar in cytokine-treated an
157 these resources for exploring the balance of global gene expression levels between excitatory AMPA re
158 ch is accompanied by a substantial change in global gene expression levels.
159                   We aimed to assess whether global gene expression measured in whole blood of health
160 a major contributing factor to the change of global gene expression, metabolic pathways and activatio
161 ma by an unbiased, detailed interrogation of global gene expression.Methods: BAL cell expression was
162   To investigate the causes, we compared the global gene expression of aged and young bone marrow-der
163 the histopathology, genome architecture, and global gene expression of donor tumors.
164                          To directly compare global gene expression of hPSC-PCs with developing mouse
165 hroughput-sequencing approach to compare the global gene expression of Rice black-streaked dwarf viru
166 bacterial factors in H. pylori pathogenesis, global gene expression of six H. pylori isolates was ana
167 can obviously have a strong influence on the global gene expression of the bacterium that produces it
168 llect incipient PCT cells and analyzed their global gene expression on Affymetrix Mouse Genome 430A m
169 o bind to individual genes can customize the global gene expression output of cells.
170 rient limitation, we examined transitions in global gene expression over short time scales, induced b
171  introgression lines, as well as a divergent global gene expression pattern between the low-PEV and h
172         To address this gap, we compared the global gene expression pattern of primary human hepatocy
173 HC), to determine whether they show the same global gene-expression pattern and high ESR1 mRNA expres
174 e workflow identifies four subtypes based on global gene expression patterns and ontologies.
175  g of the Earth, has been shown to influence global gene expression patterns and protein levels in cu
176  1 x g, microgravity has been shown to alter global gene expression patterns and protein levels in cu
177  end, we examined differentiation potential, global gene expression patterns and Sp1 target regions i
178                 In this study we showed that global gene expression patterns are very similar between
179       For many years, the relationship among global gene expression patterns associated with determin
180                             Here, we examine global gene expression patterns in corals and their intr
181 studies on injured epidermis, we showed that global gene expression patterns in highly occluded versu
182                                  We compared global gene expression patterns in livers from wildtype,
183                   In this study, we compared global gene expression patterns in progeny of damaged an
184 merged as a powerful methodology to quantify global gene expression patterns in various contexts from
185                         Here we compared the global gene expression patterns of CTBs from PAS cases t
186                                  Analysis of global gene expression patterns of soybean CIPK family r
187 signaling induces large scale changes in the global gene expression patterns of vascular tumors, incl
188 at H2B monoubiquitination does not influence global gene expression patterns, but instead ensures sel
189 d phytohormones, specialized metabolites and global gene expression patterns, in combination with het
190 vides a general method for understanding how global gene-expression patterns are choreographed.
191 sess shifts in cyanobacterial abundances and global gene-expression patterns in response to natural a
192                        In the present study, global gene expression prior to and after the onset of l
193       iOPCs exhibit a bipolar morphology and global gene expression profile consistent with bona fide
194                      In both cases, a common global gene expression profile is observed, but downstre
195                           For evaluating the global gene expression profile of cells carrying p.T120A
196                                    Herein, a global gene expression profile of Mycobacterium leprae-i
197                                              Global gene expression profile of stem cells reveals sig
198 propensity, cellular proliferative rate, and global gene expression profile when compared with unalte
199 ith an efficiency of up to 70% in 28 d and a global gene-expression profile comparable to primary hum
200     We also derived a fusion-type-associated global gene-expression profile.
201  the transcription factors which control the global gene expression profiles and consequently the cel
202 ld not be used to predict outcome but rather global gene expression profiles and epigenetic status of
203 activated preferentially by NGF, we compared global gene expression profiles between cells treated wi
204 systems biology network analysis approach to global gene expression profiles derived from common in v
205                              We investigated global gene expression profiles from HCC arising in diff
206              Here, we compared pathology and global gene expression profiles in lung tissue from BALB
207 ontribute to its pathogenesis, we determined global gene expression profiles in muscles of rats aged
208                                  Analysis of global gene expression profiles in nondiseased primary p
209  expressed in female germ cells, we analyzed global gene expression profiles in perinatal ovaries fro
210                We employ network analysis of global gene expression profiles in tumors derived from a
211 is study, we analyzed the effects of IL-4 on global gene expression profiles of Ag-induced memory CD8
212              This study aimed to investigate global gene expression profiles of BK viremia and nephro
213 the first in-depth comparison of DOX-induced global gene expression profiles of hearts and PBMCs.
214                              We examined the global gene expression profiles of human CD8(+) TN and T
215                In light of this, we compared global gene expression profiles of impaired epitope-spec
216 ow that there are significant differences in global gene expression profiles of isolated osteosarcoma
217       The differences between the respective global gene expression profiles of macrophages, derived
218    The objective of this study was to define global gene expression profiles of NDCs at key stages of
219                    Therefore, in this study, global gene expression profiles of ovary and brain of fe
220                                              Global gene expression profiles of PBMCs from 43 drug-na
221                                  Remarkably, global gene expression profiles of PS34CD45(-) cells cor
222 RNA sequencing to generate comprehensive and global gene expression profiles of this gland at differe
223 archical clustering of participants based on global gene expression profiles revealed that participan
224                                              Global gene expression profiles showed similarities betw
225                                              Global gene expression profiles were determined and comp
226 n dsk2 mutants accumulate BES1, have altered global gene expression profiles, and have compromised st
227  stem cells, including cell surface markers, global gene expression profiles, and in vivo pluripotenc
228                 We assessed iHep maturity by global gene expression profiles, hepatic polarity, secre
229  similar cytotoxic function, cell death, and global gene expression profiles, these cells had greater
230                              On the basis of global gene expression profiling and chromatin immunopre
231                                         Both global gene expression profiling and quantitative revers
232                                    Moreover, global gene expression profiling did not distinguish lar
233       This study represents the first use of global gene expression profiling from healthy human brai
234                                              Global gene expression profiling identified the transcri
235                                              Global gene expression profiling identifies clear featur
236 d each of 100 TFs with shRNA and carried out global gene expression profiling in mouse embryonic stem
237                              A major goal of global gene expression profiling in plant seeds has been
238                                              Global gene expression profiling in skin and peripheral
239                                          Our global gene expression profiling indicates that the non-
240                                              Global gene expression profiling of Ccn6(fl/fl) mammary
241                                              Global gene expression profiling of EML1 cells at differ
242                                              Global gene expression profiling of HPAIV-infected endot
243                                              Global gene expression profiling of human and mouse isle
244                                              Global gene expression profiling of infected mouse lungs
245                                              Global gene expression profiling of MCF7 cells revealed
246                  In this study, we performed global gene expression profiling of mouse tumor-infiltra
247                                    Recently, global gene expression profiling of patient samples led
248                                              Global gene expression profiling of post-ischemic kidney
249 ection against type 1 diabetes, we performed global gene expression profiling of splenocytes using hi
250                                              Global gene expression profiling of the lungs of infecte
251 n in breast cancer cells in combination with global gene expression profiling revealed p53 (TP53) sig
252                                              Global gene expression profiling revealed that more than
253                                              Global gene expression profiling revealed that the expre
254                                  Single-cell global gene expression profiling showed that undifferent
255                                              Global gene expression profiling suggested that Smchd1 n
256                                      We used global gene expression profiling to evaluate changes in
257                                              Global gene expression profiling was performed for 4 nor
258 cal melanoma samples, normal melanocytes and global gene expression profiling we have investigated th
259    Furthermore, molecular studies, including global gene expression profiling, have provided evidence
260                                        Using global gene expression profiling, we show that KDM3A pos
261           In the present study, we performed global gene-expression profiling and ChIP coupled with d
262 etween transcription factor (TF) binding and global gene expression programs as multipotent cells dif
263 w that as cells enter G0, their survival and global gene expression programs become increasingly depe
264 ta sets to elucidate the regulatory logic of global gene expression programs in mouse embryonic stem
265 ns of computational normalization to compare global gene expression programs in steady-state and dyna
266 cle provide the structural framework for the global gene expression programs of the individual chromo
267 ng sites; all of which play immense roles in global gene expression regulation.
268                       wt EBOV induced a weak global gene expression response, including markers of DC
269 sing a time-course transcriptomics approach, global gene expression responses of each cultivar were a
270                           Here, we report on global gene expression responses to VOR and examine the
271  whereas single-cell RNA-seq, which measures global gene expression, separates cells from their nativ
272 nhibition of known MSI-targets, and a shared global gene expression signature similar to shRNA deplet
273 l- and vitamin D(3)-treated PrP/SCs revealed global gene expression signatures consistent with induct
274                                Comparison of global gene expression signatures showed that transcript
275 ediated synthesis of eIF4G imposes increased global gene expression stochasticity and reduced viabili
276                                              Global gene expression studies demonstrated that, while
277 sent a novel approach to this issue by using global gene expression studies in conjunction with a sys
278                                              Global gene expression studies suggest that RD26 can act
279                Using ontological analysis of global gene expression studies, we demonstrate that many
280 ll-type proportion or pathway activity, from global gene expression studies.
281                   We performed a comparative global gene expression study using data from morphologic
282                                         In a global gene expression study, we found higher expression
283  and hypermutator phenotypes, in addition to global gene expression subtypes.
284 ide arrays are now routinely used to profile global gene expression, there is still a lack of tools f
285  a host, Pseudomonas aeruginosa orchestrates global gene expression to adapt to the host environment
286                                              Global gene expression (transcriptome) profiling reveale
287 estigate chromatin accessibility changes and global gene expression under extended darkness and contr
288     First, we look at how fungi change their global gene expression upon recognition of the host envi
289      Here we study post-SCI mRNA editing and global gene expression using massively parallel sequenci
290                                              Global gene expression was assayed using human microarra
291                                              Global gene expression was compared between PDGF-stimula
292 med cell lines on cellular functionality and global gene expression, we report that TFIIB is dispensa
293 in global histone acetylation and changes in global gene expression were observed in microarray analy
294                             Here, changes in global gene expression were studied in root, male and fe
295 ase in cell viability and alterations in the global gene expression with a broad range of biochemical
296                            GapR also affects global gene expression with a chromosomal bias from orig
297  We studied non-heart-beating donor rats for global gene expression with Affymetrix microarrays, hepa
298 roglioma display a high degree of overlap in global gene expression with no clear distinctions betwee
299 on or improvement has significantly affected global gene expression, with many genes targeted by sele
300 e sequences calls for quantitative models of global gene expression, yet predicting gene expression p

 
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