ライフサイエンスコーパス: glomerular basement membrane

1 l expansion, and segmental thickening of the glomerular basement membrane.

2 ltration barrier on the vascular side of the glomerular basement membrane.

3 ion, mesangiosclerosis, and expansion of the glomerular basement membrane.

4 o maintain the structure and function of the glomerular basement membrane.

5 esult of defective type IV collagen in their glomerular basement membrane.

6 s present at birth, despite a grossly normal glomerular basement membrane.

7 the interaction between the podocyte and the glomerular basement membrane.

8 ocytopathy and/or segmental splitting of the glomerular basement membrane.

9 tron-dense deposits and complement C3 on the glomerular basement membrane.

10 on was induced using an antibody against the glomerular basement membrane.

11 tion of type IV collagen alpha3 chain in the glomerular basement membrane.

12 myotendinous junctions, and in kidney to the glomerular basement membrane.

13 ous NC1 domain of alpha3(IV) collagen in the glomerular basement membrane.

14 ith rabbit IgG followed by rabbit anti-mouse glomerular basement membrane.

15 racterized by ultrastructural lesions of the glomerular basement membrane.

16 angial matrix expansion or thickening of the glomerular basement membrane.

17 ter the contacts between these cells and the glomerular basement membrane.

18 at proteases are involved in damaging Alport glomerular basement membrane.

19 a exchange in disease induced by antibody to glomerular basement membrane.

20 a number of basement membranes including the glomerular basement membrane.

21 omes (group I) or DNA (group II) adherent to glomerular basement membrane.

22 nine cases showed significant IgG along the glomerular basement membrane.

23 n that forms a heterotrimer expressed in the glomerular basement membrane.

24 5 depletion nor accumulation of C3 along the glomerular basement membrane.

25 complexes along the subepithelial region of glomerular basement membrane.

26 rular epithelial cells (podocytes) along the glomerular basement membrane.

27 oding type IV collagen chains present in the glomerular basement membrane.

28 racterized by ultrastructural lesions of the glomerular basement membrane.

29 ogressive accumulation of laminin 211 in the glomerular basement membrane.

30 ded for normal formation and function of the glomerular basement membrane.

31 ubclass, along the epithelial surface of the glomerular-basement membrane.

32 the formation of podocyte foot processes and glomerular basement membranes.

33 ular matrix deposition, and thickness of the glomerular basement membranes.

34 f serum C3 levels and clearance of iC3b from glomerular basement membranes.

35 ocess effacement and irregular and thickened glomerular basement membranes.

36 ence of the limited fraction of space in the glomerular basement membrane (a concentrated gel) into w

37 Similar glomerular basement membrane abnormalities could offer a

38 lete remission, whereas endothelial cell and glomerular basement membrane abnormalities were associat

39 merulonephritis induced by heterologous anti-glomerular basement membrane Abs in wild-type (IL-4+/+)

40 ated by binding of heterologous (sheep) anti-glomerular basement membrane Abs to the glomeruli of mic

41 Trinitrophenol was planted on the glomerular basement membrane after conjugation to nephro

42 tric studies of kidneys revealed a thickened glomerular basement membrane and effaced podocytes in th

43 ratus consisting of podocyte foot processes, glomerular basement membrane and endothelial cells.

44 ease, including a thickened and disorganized glomerular basement membrane and flattened podocyte foot

45 Greater width of the glomerular basement membrane and higher levels of glycat

46 rular cells alter cell interactions with the glomerular basement membrane and lead to increased glome

47 experimental glomerulonephritis, being anti-glomerular basement membrane and lipopolysaccharide-indu

48 ith a decreased distribution of VEGFA in the glomerular basement membrane and on endothelial cells.

49 VEGF-A165 b rescues the increase in glomerular basement membrane and podocyte slit width, as

50 es them an attractive experimental model for glomerular basement membrane and possibly other extracel

51 on of extracellular matrix proteins into the glomerular basement membrane and renal mesangial cell hy

52 ention of native ferritin [corrected] in the glomerular basement membrane and systemic blood pressure

53 exogenous CFH ameliorates C3 staining of the glomerular basement membrane and triggers the appearance

54 cterized by progressive deterioration of the glomerular basement membrane and usually associated with

55 arietal epithelial cells attached to denuded glomerular basement membrane and, occasionally, disengag

56 albumin excretion, approximately 3x thicker glomerular basement membranes and severe podocyte efface

57 y was observed only outside laminin-positive glomerular basement membrane, and co-localized with neph

58 cess effacement, irregular thickening of the glomerular basement membrane, and dilated capillary loop

59 an injured endothelial morphology, thickened glomerular basement membrane, and focal foot process eff

60 e foot process effacement, thickening of the glomerular basement membrane, and FSGS-like lesions.

61 ey glomeruli was consistent with that of the glomerular basement membrane, and staining was markedly

62 is comprised of the endothelial lining, the glomerular basement membrane, and the podocyte intercell

63 ac1 also led to podocyte detachment from the glomerular basement membrane, and we detected detached p

64 ters, Ab and complement deposition along the glomerular basement membranes, and a nephrotic syndrome,

65 of HB-EGF in rat kidneys treated with anti- glomerular basement membrane (anti-GBM) antibody (Ab) to

66 Urine samples from mice with anti-glomerular basement membrane (anti-GBM) antibody-induced

67 The pathophysiology of anti-glomerular basement membrane (anti-GBM) disease before c

68 Anti-glomerular basement membrane (anti-GBM) disease is an or

69 st recruitment in renal fibrosis due to anti-glomerular basement membrane (anti-GBM) disease is unkno

70 r kidneys as a result of posttransplant anti-glomerular basement membrane (anti-GBM) disease.

71 syndrome (caused by antibodies specific for glomerular basement membrane [anti-GBM antibodies]), and

72 inct, separate disease process, such as anti-glomerular basement membrane antibodies or anti-neutroph

73 racterized by circulating and deposited anti-glomerular basement membrane antibodies, focal necrotizi

74 as been known to occur with exposure to anti-glomerular basement membrane antibody (AGBM-Ab) in the r

75 days later by injection of sheep anti-mouse glomerular basement membrane antibody and CRP or control

76 Injection of sheep anti-glomerular basement membrane antibody into preimmunized

77 In GEC(HO-1) rats with anti-glomerular basement membrane antibody mediated, compleme

78 g parameters both at baseline and after anti-glomerular basement membrane antibody treatment: blood p

79 ered GN with a subnephritogenic dose of anti-glomerular basement membrane antibody, Aire(-/-) mice ha

80 ution of NETs via DNase I did not alter anti-glomerular basement membrane antibody-induced glomerular

81 dulates iNOS expression and activity in anti-glomerular basement membrane antibody-mediated glomerulo

82 unizing mice with MPO and a low dose of anti-glomerular basement membrane antibody.

83 hritis induced by the administration of anti-glomerular basement membrane antibody.

84 re processed for matrix antigen (collagen I, glomerular basement membrane antigens, laminin, and fibr

85 Introduction of heterologous anti-glomerular basement membrane antiserum (nephrotoxic seru

86 uced with a previously described rabbit anti-glomerular basement membrane antiserum nephritis approac

87 The glomerular basement membrane appeared to be thickened an

88 sociation and distribution of SAP within the glomerular basement membrane are altered or completely d

89 The glomerular epithelial cells and the glomerular basement membrane are important constituents

90 ing complexes to dissociate and traverse the glomerular-basement membrane) are compatible with a path

91 s inhibitor prevents focal thickening of the glomerular basement membrane, but does not prevent effac

92 haracterized by accumulation of C3 along the glomerular basement membrane, but the role of properdin

93 gh glucose (HG) in cultured podocytes alters glomerular basement membrane by activating signal transd

94 alpha 3 alpha 4 alpha 5(IV) collagen in the glomerular basement membrane causes Alport syndrome, a h

95 ibodies to type IV collagen that bind to the glomerular basement membrane, causing rapidly progressin

96 inin and type IV collagen composition of the glomerular basement membrane changes during glomerular d

97 increased albuminuria and thickening of the glomerular basement membrane compared with nondiabetic F

98 of podocytes and had better integrity of the glomerular basement membrane compared with untreated Col

99 laminin alpha5 (Lama5), a major tubular and glomerular basement membrane component that is important

100 -linked hereditary nephropathy (XLHN) have a glomerular basement membrane defect that leads to progre

101 Several glomerular basement membrane defects include Alport's sy

102 crosis by light microscopy or electron-dense glomerular basement membrane deposits by electron micros

103 ted by pathogenic antibodies, including anti-glomerular basement membrane disease and lupus nephritis

104 acrophage- and T-cell-mediated model of anti-glomerular basement membrane disease in STC1 transgenic

105 phritis (APTN) is an aggressive form of anti-glomerular basement membrane disease that targets the al

106 P), 8; systemic lupus erythematosus, 3; Anti-glomerular basement membrane disease, 2; oxalosis, 2; an

107 asmic antibodies-associated vasculitis, anti-glomerular basement membrane disease, and systemic lupus

108 e glomerulonephritis after induction of anti-glomerular basement membrane disease, with more infiltra

109 roach to the treatment of patients with anti-glomerular basement membrane disease.

110 c antibodies-associated vasculitis, and anti-glomerular basement membrane disease.

111 In Goodpasture's (anti-glomerular basement membrane) disease, autoimmunity to t

112 findings included segmental splitting of the glomerular basement membrane, effacement of podocyte foo

113 owed abundant IgG deposition in the expanded glomerular basement membrane, especially in regions corr

114 er fixation of nephrotoxic antibodies to the glomerular basement membrane, even in the absence of pro

115 l role of tight adhesion of podocytes to the glomerular basement membrane for maintaining glomerular

116 ned at autopsy, glomeruli were isolated, and glomerular basement membrane fragments were prepared.

117 disease was the appearance of bare areas of glomerular basement membrane from the pulling apart of p

118 The anti-glomerular basement membrane (GBM) Ab has been regarded

119 ns that are seen at onset of albuminuria are glomerular basement membrane (GBM) alterations with a si

120 f laminin alpha5 results in breakdown of the glomerular basement membrane (GBM) and failed glomerular

121 s considerable evidence implicating both the glomerular basement membrane (GBM) and the epithelial fi

122 pecialized extracellular matrix known as the glomerular basement membrane (GBM) and the podocyte foot

123 noperoxidase assay on cell layers using anti-glomerular basement membrane (GBM) antibodies.

124 moattractant, during the progression of anti-glomerular basement membrane (GBM) antibody (Ab) GN, a m

125 Anti-glomerular basement membrane (GBM) antibody nephritis is

126 lammation and in the progression of the anti-glomerular basement membrane (GBM) antibody-induced expe

127 een immunized with a recombinant form of the glomerular basement membrane (GBM) antigen, Col4alpha3NC

128 ns of alpha3alpha4alpha5(IV) collagen in the glomerular basement membrane (GBM) are targets of Goodpa

129 Ultrastructural changes in the glomerular basement membrane (GBM) at 2 weeks of age res

130 aminin alpha5 replaces laminin alpha1 in the glomerular basement membrane (GBM) at the capillary loop

131 f the alpha3/4/5(IV) collagen network in the glomerular basement membrane (GBM) cause Alport syndrome

132 ixon, my work examined how antibodies to the glomerular basement membrane (GBM) cause disease.

133 Primary defects in either podocytes or the glomerular basement membrane (GBM) cause proteinuria, a

134 r 500-fold increase in albuminuria), loss of glomerular basement membrane (GBM) charge and foot proce

135 Here we have determined in human skin and glomerular basement membrane (GBM) collagen the levels o

136 Expression of glomerular basement membrane (GBM) collagens is reduced

137 e were less adherent than wild-type cells to glomerular basement membrane (GBM) components collagen I

138 Alport syndrome is a glomerular basement membrane (GBM) disease caused by mut

139 Anti-glomerular basement membrane (GBM) disease is a rapidly

140 Human anti-glomerular basement membrane (GBM) disease strongly asso

141 We developed a new mouse model of human anti-glomerular basement membrane (GBM) disease to better cha

142 We developed a rat model of human anti-glomerular basement membrane (GBM) disease to investigat

143 n G (IgG) and complement C3, typical of anti-glomerular basement membrane (GBM) disease.

144 Alport syndrome features a defective glomerular basement membrane (GBM) due to variants in CO

145 macrophage-mediated, cytokine-dependent anti-glomerular basement membrane (GBM) glomerulonephritis (G

146 logous phase of an accelerated model of anti-glomerular basement membrane (GBM) glomerulonephritis us

147 mmation, the development of accelerated anti-glomerular basement membrane (GBM) glomerulonephritis wa

148 star Kyoto rats by a single injection of rat glomerular basement membrane (GBM) in adjuvant.

149 gle injection of collagenase-solubilized rat glomerular basement membrane (GBM) in adjuvant.

150 yoto (WKY) rats by a single injection of rat glomerular basement membrane (GBM) in adjuvant.

151 the kidney adhere tightly to the underlying glomerular basement membrane (GBM) in order to maintain

152 The glomerular basement membrane (GBM) is a key component of

153 The glomerular basement membrane (GBM) is a specialized extr

154 of alpha3, alpha4, and alpha5 chains in the glomerular basement membrane (GBM) is speculated to invo

155 Linear binding of IgG to the glomerular basement membrane (GBM) is the hallmark of an

156 e replaced by immigration of cells along the glomerular basement membrane (GBM) is under debate.

157 ivo, we induced a model of Fc-dependent anti-glomerular basement membrane (GBM) nephritis in wild-typ

158 Alport posttransplantation anti-glomerular basement membrane (GBM) nephritis is mediated

159 monitor renal disease progression in an anti-glomerular basement membrane (GBM) nephritis mouse model

160 significant structural abnormalities in the glomerular basement membrane (GBM) of diabetic individua

161 The ultrafiltration function of the glomerular basement membrane (GBM) of the kidney is impa

162 tion with either collagenase-solubilized rat glomerular basement membrane (GBM) or the recombinant NC

163 rats immunized with collagenase-solubilized glomerular basement membrane (GBM) or the recombinant NC

164 Glomerular basement membrane (GBM) plays a crucial funct

165 Abnormal permselectivity of glomerular basement membrane (GBM) plays an important ro

166 Detachment of podocytes from the glomerular basement membrane (GBM) rather than apoptosis

167 nbred strains of mice with rabbit anti-mouse glomerular basement membrane (GBM) reactive sera.

168 psy, immunofluorescence (IF) showed granular glomerular basement membrane (GBM) staining for C4d, IgG

169 It is the kidney glomerular basement membrane (GBM) that is defective in

170 We used an ELISA employing extracts of human glomerular basement membrane (GBM) to detect, characteri

171 ul for diagnosis, quantitative evaluation of glomerular basement membrane (GBM) type IV collagen (col

172 ed in diabetic patients, but surface area of glomerular basement membrane (GBM) underlying the podocy

173 Glomerular basement membrane (GBM) width (356 +/- 52 ver

174 All three groups had increased glomerular basement membrane (GBM) width and mesangial f

175 Mesangial and matrix volume fractions and glomerular basement membrane (GBM) width were increased

176 During follow-up, glomerular basement membrane (GBM) width, mesangial frac

177 nked heparan sulfate (HS) alterations in the glomerular basement membrane (GBM) with albuminuria as a

178 r organization of proteins within the kidney glomerular basement membrane (GBM), an essential mediato

179 lial cell loss, double-contour appearance of glomerular basement membrane (GBM), and thrombus formati

180 teins and neutral dextrans permeate into the glomerular basement membrane (GBM), in general agreement

181 by deposition of immune complexes along the glomerular basement membrane (GBM), phospholipase A2 rec

182 mers of alpha3(IV) collagen that bind to the glomerular basement membrane (GBM), usually causing rapi

183 mponent of the renal filtration barrier--the glomerular basement membrane (GBM)--can disassemble cati

184 h proteinuria, decreased renal function, and glomerular basement membrane (GBM)-bound deposits in hal

185 jected to an augmented passive model of anti-glomerular basement membrane (GBM)-induced experimental

186 /HSPG2 that constitute the axial core of the glomerular basement membrane (GBM).

187 21, which is an important constituent of the glomerular basement membrane (GBM).

188 rastructural thickening and splitting of the glomerular basement membrane (GBM).

189 us-1 (NC1) domain of type IV collagen in the glomerular basement membrane (GBM).

190 nely clog with large proteins that enter the glomerular basement membrane (GBM).

191 which form the collagenous network of mature glomerular basement membrane (GBM).

192 inin beta2 (LAMB2), a major component of the glomerular basement membrane (GBM).

193 heparan sulfate proteoglycans (HSPGs) in the glomerular basement membrane (GBM).

194 rs of plasma every day through a specialized glomerular basement membrane (GBM).

195 target the alpha3(IV) collagen chain in the glomerular basement membrane (GBM).

196 glomerulogenesis associated with an abnormal glomerular basement membrane (GBM).

197 of C3 within the kidney, commonly along the glomerular basement membrane (GBM).

198 h forms the major collagen IV network of the glomerular basement membrane (GBM).

199 they remove Ig and immune complexes from the glomerular basement membrane (GBM).

200 ma1) trimer, an important constituent of the glomerular basement membrane (GBM).

201 with specific ultrastructural lesions of the glomerular basement membrane (GBM).

202 a4alpha5(IV) networks found in mature kidney glomerular basement membrane (GBM).

203 3(IV) collagen, an integral component of the glomerular basement membrane (GBM); this effect was comp

204 preparations of skin collagen (n = 110) and glomerular basement membrane (GBM, n = 28) were enzymati

205 or AGE detected in diabetic mesangium (96%), glomerular basement membranes (GBM) (42%), tubular basem

206 TEM analysis of the glomerular basement membranes (GBM) during development o

207 Laminin alpha2 chain was absent from glomerular basement membranes (GBM) in normal human, mur

208 Why glomerular basement membranes (GBM) undergo laminin tran

209 spread podocyte foot process broadening, and glomerular basement membranes (GBMs) were significantly

210 glomerulonephritis after induction with anti-glomerular basement membrane globulin, with enhanced pat

211 cal characteristics during experimental anti-glomerular basement membrane glomerulonephritis (anti-GB

212 e compared the intensity of accelerated anti-glomerular basement membrane glomerulonephritis between

213 diffuse type that is characteristic of anti-glomerular basement membrane glomerulonephritis, and a f

214 Anti-glomerular basement membrane GN involved NET formation a

215 Accelerated anti-glomerular basement membrane GN was examined in groups o

216 monomers, but not dimers, from native human glomerular basement membrane hexamers, a property that m

217 proteinuria and decreased renal function and glomerular basement membrane IgG deposits.

218 einuria and restored the architecture of the glomerular basement membrane in alpha1 integrin-null Alp

219 novel podocyte protrusions invading into the glomerular basement membrane in disease and these occurr

220 we found striking ultrastructural changes in glomerular basement membranes in FVB mice.

221 Ultrastructural abnormalities of the glomerular basement membrane, including lamellation and

222 r neutrophil accumulation and damage in anti-glomerular basement membrane-induced (anti-GBM-induced)

223 Similarly, both anti-glomerular basement membrane-induced glomerulonephritis

224 ous chronic glomerulonephritis (GN) and anti-glomerular basement membrane-induced nephritis.

225 trained to semantically segment the podocyte-glomerular basement membrane interface and filtration sl

226 ment of viable podocytes from the underlying glomerular basement membrane is an important mechanism o

227 glomerular epithelial cell attachment to the glomerular basement membrane is an important pathogeneti

228 , the improvement in the architecture of the glomerular basement membrane is associated with de novo

229 Because podocyte adhesion to the glomerular basement membrane is mediated by integrins, t

230 Podocyte adhesion to the glomerular basement membrane is required for proper func

231 Ultrastructural studies show that the glomerular basement membrane is thickened, podocyte slit

232 llagen IV networks, a major component of the glomerular basement membrane, is poorly understood.

233 onstrate that defects induced by proteins of glomerular basement membrane lead to an insidious plasma

234 ed by lipopolysaccharide and antibody to the glomerular basement membrane, led to rapid glomerular ne

235 led histopathological analysis revealed that glomerular basement membrane lesions typical of Alport s

236 te that was adsorbed to NC1 hexamer from rat glomerular basement membrane lost all reactivity to glom

237 tration slit frequency and thickening of the glomerular basement membrane, lowering computed hydrauli

238 on gel behavior show that proteins cross the glomerular basement membrane mainly by diffusion rather

239 We used lupus and anti-glomerular basement membrane models of nephritis to dete

240 nally inhibited, glomerular foot process and glomerular basement membrane morphology are primarily re

241 Impaired podocyte adhesion to the glomerular basement membrane most likely contributed to

242 lomerulonephritis, type I and II (MPG), anti-glomerular basement membrane nephritis (anti-GBM), and m

243 dedifferentiated podocytes of mice with anti-glomerular basement membrane nephritis and in human immu

244 Using anti-glomerular basement membrane nephritis in rats, we inves

245 Moreover, after induction of anti-glomerular basement membrane nephritis in young mice, iP

246 ssary for local chemokine expression in anti-glomerular basement membrane nephritis, although the dif

247 ntrol mice, were challenged with rabbit anti-glomerular basement membrane nephrotoxic sera (NTS), to

248 inin-2 and/or laminin-4) accumulating in the glomerular basement membrane of Alport mice is markedly

249 tive assembly of the alpha3(IV) chain in the glomerular basement membrane of patients with Alport syn

250 abnormal electron-dense material within the glomerular basement membrane of the kidney and often wit

251 emonstrate that severe defects in either the glomerular basement membrane or the glomerular endotheli

252 f type IV collagen, the major constituent of glomerular basement membrane, or LMX1B transcription fac

253 effacement, irregular and split areas of the glomerular basement membrane, podocyte apoptosis and dep

254 of glomerular epithelial slit diaphragm and glomerular basement membrane proteins implicated in glom

255 circulating proteins and AGE modification of glomerular basement membrane proteins may both contribut

256 y inflammation in a macrophage-mediated anti-glomerular basement membrane reactive serum-induced immu

257 Sub-threshold doses of glomerular basement membrane-reactive serum induced more

258 The interface between the podocyte and the glomerular basement membrane requires integrins, and def

259 e glomerular epithelial cell detachment from glomerular basement membrane seen in the PAN nephrosis m

260 netic model of podocyte injury and segmental glomerular basement membrane splitting due to hyposialyl

261 pathy with podocyte effacement and segmental glomerular basement membrane splitting due to hyposialyl

262 ysms, glomerular hypertrophy, podocyte loss, glomerular basement membrane splitting, and secondary fo

263 ollagen IV networks, which are essential for glomerular basement membrane stability and molecular ult

264 sting that modifier genes act by influencing glomerular basement membrane structure.

265 n the kidneys each consisting of a barrel of glomerular basement membrane surrounded by glomerular en

266 more prominent immune deposits and a thicker glomerular basement membrane than at baseline.

267 is, and changes in the ultrastructure of the glomerular basement membrane that increase in severity i

268 l development of dorsal limb structures, the glomerular basement membrane, the anterior segment of th

269 follows: 9/14 had moderate or severe diffuse glomerular basement membrane thickening and 2/14 had nod

270 A expression down-regulation and ameliorated glomerular basement membrane thickening and foot process

271 Transmission electron microscopy revealed glomerular basement membrane thickening and podocyte eff

272 gial cell expansion, glomerular hypertrophy, glomerular basement membrane thickening and podocyte foo

273 Furthermore, DAPT prevented glomerular basement membrane thickening and proteinuria

274 us with areas of foot process effacement and glomerular basement membrane thickening and wrinkling.

275 histology revealed mesangial expansion, and glomerular basement membrane thickening as determined by

276 Glomerular basement membrane thickening depended on incr

277 Diabetic glomerular basement membrane thickening was prevented in

278 hypertrophy, mesangial matrix accumulation, glomerular basement membrane thickening, albuminuria, an

279 ar thickening, tubular dilation and atrophy, glomerular basement membrane thickening, and mesangial e

280 icroalbuminuria, mesangial matrix expansion, glomerular basement membrane thickening, and podocyte lo

281 es that exhibits mesangial matrix expansion, glomerular basement membrane thickening, and renal insuf

282 d many DN features, including podocyte loss, glomerular basement membrane thickening, mesangial expan

283 inary albumin excretion, glomerulosclerosis, glomerular basement membrane thickening, mitochondrial D

284 megaly, mesangial sclerosis, cast formation, glomerular basement membrane thickening, podocyte efface

285 Diabetes-induced mesangial expansion, glomerular basement membrane thickening, podocyte foot-p

286 oteinuria, fewer IgG glomerular deposits, no glomerular basement membrane thickening, reduced levels

287 illary surface area remained stable, but the glomerular basement membrane thickness was increased and

288 lbuminuria, arteriolar hyalinosis, increased glomerular basement membrane thickness, mesangial expans

289 Compared with wild-type mice, after anti-glomerular basement membrane treatment STC1 transgenic m

290 enetic disorder characterized by a defective glomerular basement membrane, tubulointerstitial fibrosi

291 chemical and mechanical signals to/from the glomerular basement membrane upon which it elaborates, a

292 They stay attached to the glomerular basement membrane via integrin interactions t

293 The glomerular basement membrane was disorganized and glomer

294 their suitability as experimental models of glomerular basement membrane was examined by measuring t

295 Glomerular basement membrane was thickened in OVE26 mice

296 ynthetic gels were quite similar to those in glomerular basement membrane, when compared on the basis

297 tion of extracellular chromatin fragments in glomerular basement membranes where they appear in compl

298 ey diabetic subject groups, respectively, in glomerular basement membrane width (median [range] 511 n

299 uminuria, mesangial expansion, and increased glomerular basement membrane width in diabetic mice.

300 these patients with type 1 diabetes who had glomerular basement membrane widths within the normal ra