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1 ptor tyrosine kinase expressed abundantly in glomerular mesangial cells.
2 t proinflammatory cytokine expression in rat glomerular mesangial cells.
3 up-regulate the expression of lipocalin-2 in glomerular mesangial cells.
4  the ability of collagen IV to interact with glomerular mesangial cells.
5 with proliferation and apoptosis of resident glomerular mesangial cells.
6 COL1A2 gene activation by TGF-beta1 in human glomerular mesangial cells.
7  phospholipase C-dependent, pathway in human glomerular mesangial cells.
8 al growth factor-induced Ca2+ entry in human glomerular mesangial cells.
9 ad signaling in collagen production by human glomerular mesangial cells.
10 cells have the ability to differentiate into glomerular mesangial cells.
11 cipate in the development and maintenance of glomerular mesangial cells.
12 ; also called capacitative calcium entry) in glomerular mesangial cells.
13  by platelet-derived growth factor (PDGF) in glomerular mesangial cells.
14 oncomitant release of nitric oxide (NO) from glomerular mesangial cells.
15 a concomitant release of prostaglandins from glomerular mesangial cells.
16 ition and, H3 and H4- acetylation in primary glomerular mesangial cells.
17 staglandins (PGs) and nitric oxide (NO) from glomerular mesangial cells.
18 on, bradykinin (BK) is mitogenic in cultured glomerular mesangial cells.
19 fic transactivator of MMP-2 transcription by glomerular mesangial cells.
20 duces a biphasic activation of p21ras in rat glomerular mesangial cells.
21 racellular matrix turnover, was evaluated in glomerular mesangial cells.
22 rix accumulation was explored in vitro using glomerular mesangial cells.
23            We investigated the origin of the glomerular mesangial cell, a smooth muscle-like cell tha
24          Overexpression of this component in glomerular mesangial cells, a model perivascular myofibr
25 , we investigated the effects of 9-cis-RA on glomerular mesangial cell activation induced by transfor
26 iposomes were detected in the glomerulus and glomerular mesangial cells after tail vein injection in
27 ctivation and transient proliferation of the glomerular mesangial cells and by a prominent glomerular
28 regulates microRNA-192 (miR-192) in cultured glomerular mesangial cells and in glomeruli from diabeti
29  tubular epithelial cells (HKC-8) but not in glomerular mesangial cells and interstitial fibroblasts.
30 rate that GATA3 is specifically expressed in glomerular mesangial cells and plays a critical role in
31 g pathway is present and functional in human glomerular mesangial cells and plays a role in activatin
32 creases angiotensin II (AngII) levels in rat glomerular mesangial cells and that AngII mediates the i
33 stem cell is capable of differentiating into glomerular mesangial cells and that the process does not
34 to interact physically with NF-kappaB p65 in glomerular mesangial cells and thereby to inhibit NF-kap
35            Similarly, SPARC was increased in glomerular mesangial cells and visceral epithelial cells
36    We find that kidney beta8 is localized to glomerular mesangial cells, and expression is decreased
37 lipopolysaccharide treatment in vitro induce glomerular mesangial cell apoptosis by hitherto incomple
38                                              Glomerular mesangial cells are active participants in th
39                                              Glomerular mesangial cells are exposed to pulsatile capi
40                                              Glomerular mesangial cells are key modulators of the inf
41 quires the removal of proliferating resident glomerular mesangial cells, but excessive loss of glomer
42 contractile function of both human and mouse glomerular mesangial cells by decreasing TRPC1 channel p
43 Here, we show that TGF-beta activates Akt in glomerular mesangial cells by inducing miR-200b and miR-
44  TNF-restricted M(phi)-directed apoptosis of glomerular mesangial cells can be down-regulated by M(ph
45                     In this study, a line of glomerular mesangial cells derived from normal mice was
46 oth muscle actin is known to be expressed in glomerular mesangial cells exclusively in pathologic sit
47 itioned by several cell lines and found that glomerular mesangial cells exclusively secrete a factor
48 ERK) pathway was inhibited in HMC and in rat glomerular mesangial cells expressing the dominant-negat
49                            We first isolated glomerular mesangial cells from MKK3-null (Mkk3-/-) and
50 hat TGF-beta1 induces autophagy and protects glomerular mesangial cells from undergoing apoptosis dur
51                     High glucose (HG) causes glomerular mesangial cell (GMC) growth, production of tr
52                                              Glomerular mesangial cell (GMC) proliferation and death
53                                              Glomerular mesangial cell (GMC) proliferation and matrix
54                                              Glomerular mesangial cell (GMC)-derived pleiotropic cyto
55  COX-2 expression also has been described in glomerular mesangial cells (GMC), where COX-2 is not exp
56 inhibiting the activation of p38 in cultured glomerular mesangial cells (GMC).
57                          Alteration in renal glomerular mesangial cell growth and fibronectin matrix
58                                        Human glomerular mesangial cells (HMC) are embedded in the mes
59 uced a rapid activation of p21(ras) in human glomerular mesangial cells (HMC).
60 ly decreased PEDF secretion in primary human glomerular mesangial cells (HMCs), suggesting that hyper
61 ized by in situ hybridization to vessels and glomerular mesangial cells in allogeneic and syngeneic (
62 scular smooth muscle cells and pericytes and glomerular mesangial cells in the kidney and that Tbx18-
63 tion in approximately 5000 gene promoters in glomerular mesangial cells, including those of Tgfb1, Tg
64 is study we examined the mechanisms by which glomerular mesangial cells ingest apoptotic cells and th
65 hysiologically activated human monocytes and glomerular mesangial cells (MC) by employing a cell cult
66                                Activation of glomerular mesangial cells (MCs) by angiotensin II (Ang
67                                Activation of glomerular mesangial cells (MCs) by angiotensin II (Ang
68                Here, we demonstrate that, in glomerular mesangial cells (MCs), endothelial nitric oxi
69 n rate (GFR) and the contractile function of glomerular mesangial cells (MCs).
70 s in fibronectin synthesis in cultured renal glomerular mesangial cells (MCs).
71 I) exerts contractile and trophic effects in glomerular mesangial cells (MCs).
72 high glucose (HG)- or TGF-beta-treated mouse glomerular mesangial cells (MMCs).
73  activated by 1 microm thapsigargin in human glomerular mesangial cells or A431 cells.
74 ) collagen gene (COL1A2) activation in human glomerular mesangial cells, potentially contributing to
75                                              Glomerular mesangial cells produce reactive oxygen inter
76                         These data show that glomerular mesangial cell progenitors are derived from t
77 F-B plays a pivotal role in the mediation of glomerular mesangial cell proliferation.
78           These findings demonstrate that in glomerular mesangial cells, repeated cycles of stretchin
79                                     In human glomerular mesangial cells, Smad3 protein levels were sp
80 a hyperglycemic-induced hypocontractility of glomerular mesangial cells that may be associated with d
81 -kappaB selectively sensitizes primary adult glomerular mesangial cells to TNF-induced apoptosis but
82  alpha8 integrin expressed on the surface of glomerular mesangial cells was selected as a target mole
83 tion, and collagen synthesis in isolated rat glomerular mesangial cells were evaluated in vitro.
84 might regulate HK activity and expression in glomerular mesangial cells, which constitute the princip
85 the G3YR mice showed degenerative changes in glomerular mesangial cells, which deteriorated progressi
86 f NF-kappaB on cytokine-induced apoptosis of glomerular mesangial cells, which is important in determ
87 eport we demonstrate that conditioning human glomerular mesangial cells with IL-1beta results in the
88                          Clones of recipient glomerular mesangial cells with the donor genotype were
89 scular pericytes, liver fat-storing cells or glomerular mesangial cells, with IFN-gamma dramatically