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1 s (RMCs) constitute a population of cells in glomerular mesangium.
2 g immune complexes with proliferation of the glomerular mesangium.
3 xtraglomerular mesangium, and throughout the glomerular mesangium.
4 orter capillary route is enclosed within the glomerular mesangium.
5  galactose-deficient IgA1 [Tn(+)IgA1] in the glomerular mesangium.
6 s form in the circulation and deposit in the glomerular mesangium.
7 nown factors favoring IgA1 deposition in the glomerular mesangium.
8 s of IgA1-containing immune complexes in the glomerular mesangium.
9 glycosylation preferentially deposits in the glomerular mesangium.
10 ith abnormal O-glycosylation deposits in the glomerular mesangium.
11                 However, inflammation of the glomerular mesangium also participates in at least the e
12 e main changes in protein composition of the glomerular mesangium and basement membrane and the evide
13        These immune complexes deposit in the glomerular mesangium and induce the mesangioproliferativ
14 ultiple glomerular cell types, including the glomerular mesangium and podocytes.
15  accumulation of extracellular matrix in the glomerular mesangium and the interstitium.
16  deposition of these immune complexes in the glomerular mesangium, leading to kidney inflammation and
17  in rats on the uptake and processing by the glomerular mesangium of intravenously administered prote
18 culating immune complexes are trapped in the glomerular mesangium of the kidney where they trigger lo
19 taining immune complexes that deposit in the glomerular mesangium, producing progressive kidney injur
20                       Fibrinogen staining of glomerular mesangium was seen in kidneys with chronic re