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1 nia and/or systemic hypoglycemia (2.8 mmol/l glucose clamp).
2 els were similar among all groups during the glucose clamp.
3 cemic (5.0 mmol/L)-hypoglycemic (2.8 mmol/L) glucose clamp.
4 th a stepped hyperinsulinaemic hypoglycaemic glucose clamp.
5 liver and the peripheral tissues during the glucose clamp.
6 ng recovery (5-6 mmol/L) by hyperinsulinemic glucose clamp.
7 mmol/L) hypoglycemic (2.80 +/- 0.12 mmol/L) glucose clamp.
8 three periods of hypoglycemia by an insulin-glucose clamp.
9 Insulin sensitivity was assessed by glucose clamps.
10 ovement in glucose disposal rates during the glucose clamps.
11 ormed on the day before the second and third glucose clamps.
12 ximately 3.6 mmol/l, approximately 65 mg/dl) glucose clamps.
14 during a 100-min hyperinsulinemic-euglycemic glucose clamp (40 mU x m(-2) x min(-1)) before and after
19 in sensitivity (hyperinsulinemic-isoglycemic glucose clamp) and insulin-stimulated changes in brachia
20 intravenous (ivGTT) glucose tolerance tests, glucose clamps, and body composition assessed between 15
21 er changes in HK-II expression seen during a glucose clamp are likely to be physiologically relevant,
22 tudies were conducted with maternal arterial glucose clamped at 5 micromol ml(-1) and fetal glucose a
23 emission tomography during hyperinsulinemic glucose clamps at nominal plasma glucose concentrations
24 . m(-2). min(-1) hyperinsulinemic-euglycemic glucose clamp before and after 3 months' treatment with
25 was assessed by hyperinsulinemic-euglycemic glucose clamp before and after intranasal application of
29 n contrast, insulin action (hyperinsulinemic glucose clamp) did not correlate with the age at onset o
30 l range (2.0-2.5-microm wavelength) during a glucose clamp experiment in order to identify the presen
32 ype 1 diabetes underwent 4 separate stepwise glucose clamps (five 30-min steps from fasting level to
36 lucose uptake by euglycemic-hyperinsulinemic glucose clamp in 15 normal-weight and 15 obese participa
37 mpared with the tolbutamide protocol and the glucose clamp in 35 nondiabetic subjects (age 38 +/- 2 y
39 is, we performed euglycemic-hyperinsulinemic glucose clamps in conscious dogs (n = 8) in which FFA we
40 and plasma FFA concentrations throughout the glucose clamps in control (r = 0.996) and GDM (r = 0.995
41 lin therapy displayed a significantly higher glucose clamp infusion rate posttreatment (9.1 +/- 1.3 i
42 onostatic potency of GLP-1 during a stepwise glucose clamp is preserved in patients with type 2 diabe
43 e test (OGTT) and an isoglycemic intravenous glucose clamp (iso-IVGC) in: 1) 16 severely obese patien
44 blood glucose levels were manipulated using glucose clamp methodologies with continuous basal insuli
45 nsitivity during late pregnancy, but neither glucose clamp nor minimal model measures of insulin sens
47 those based on an intravenous test (e.g., a glucose clamp or an intravenous glucose tolerance test).
49 few data using direct measures of IR such as glucose clamps or frequently sampled intravenous glucose
50 t and exercise using a pancreatic clamp with glucose clamped (PC/GC; n = 5), a pancreatic clamp with
52 mmol/l) or hypoglycemic (2.9 +/- 0.1 mmol/l) glucose clamps (prolonged hypoglycemia) were carried out
53 old (by intralipid infusion during 11 mmol/l glucose clamp) resulted in a robust, approximate twofold
54 was assessed by hyperinsulinemic-isoglycemic glucose clamp (SI(Clamp)) and endothelial function evalu
55 rnoon 2-h hyperinsulinemic (528+/-30 pmol/l) glucose clamp studies of 5.3+/-0.1 mmol/l (euglycemic co
57 ests (ITTs), and hyperinsulinemic euglycemic glucose clamp studies, we demonstrated that mice lacking
58 essed during a hyperinsulinemic-hypoglycemic glucose clamp study in chronically catheterized awake ma
59 g field data (Raman spectra) acquired from a glucose clamping study on an animal model subject, we pe
60 in intracerebral glucose levels during a 2-h glucose clamp (target glucose concentration 220 mg/dL).
61 Insulin sensitivity was measured using the glucose clamp technique (40 mU.m-2.min-1), in conjunctio
63 dy and individual skeletal muscles using the glucose clamp technique combined with D-[3-3H]glucose in
72 mmol/L) and hypoglycemic (3 mmol/L) [1-(13)C]glucose clamps were performed in eight healthy subjects
73 hyperinsulinemic euglycemic and hypoglycemic glucose clamps were performed on separate days, using [1
74 and type 2 diabetic volunteers (n = 8 each); glucose clamps were used to assess insulin sensitivity.