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1 by stimulating the membrane translocation of glucose transporter 4.
2 dation, and plasma membrane translocation of glucose transporter 4.
3  4 and increased cardiomyocyte expression of glucose transporter 4.
4 tty acid synthase, uncoupling protein-1, and glucose transporter 4.
5 ted in glucose transport by interaction with glucose transporter 4.
6 ycemic glucose: 1) blocked glucose uptake by glucose transporter 4; 2) inhibited cytosolic uridine di
7 ntration-dependent manner, promoted membrane glucose transporter-4 accumulation, and enhanced [(3)H]-
8 d cells results in reduction of HCMV-induced glucose transporter 4 and glucose transporter 2 expressi
9 g NFkappaB p65 attenuated CLA suppression of glucose transporter 4 and peroxisome proliferator-activa
10 ed insulin receptor substrate 1 (Ser789) and glucose transporter 4, and increased protein abundance o
11 -binding protein alpha, fatty acid synthase, glucose transporter 4, and the transcription factor sign
12 mediate the effects of GIP, we analyzed Akt, glucose transporter-4, and glucose uptake, all of which
13 in adipocytes by triggering translocation of glucose transporter 4-containg vesicles to the plasma me
14  whereas the amount of the insulin-sensitive glucose transporter 4 either remained unchanged or decre
15 g demonstrated a 60% elevation of myocardial glucose transporter 4 expression in the left ventricle a
16 of obese rats: reductions in adiponectin and glucose transporter 4 (GLUT 4) and increases in dipeptid
17 levels in the TB of insulin receptor (InsR), glucose transporter-4 (GLUT-4) and type 1 insulin-like g
18 differentiated C2C12 myotubes by stimulating glucose transporter-4 (GLUT-4) membraned translocation.
19 termined by key adipocyte markers, including glucose transporter 4 (GLUT4) and adiponectin expression
20 responsive amino peptidase (IRAP) along with glucose transporter 4 (Glut4) and sortilin, represents a
21 , Akt, and AS160, to promote the net gain of glucose transporter 4 (GLUT4) at the plasma membrane of
22 is regulated by changing the distribution of glucose transporter 4 (GLUT4) between the cell interior
23                            The mRNA level of glucose transporter 4 (GLUT4) decreased several fold in
24 ensitive to the levels of insulin-responsive glucose transporter 4 (GLUT4) expression in adipocytes.
25                                              Glucose transporter 4 (GLUT4) expression on white adipoc
26 at cells by inducing a net redistribution of glucose transporter 4 (GLUT4) from intracellular storage
27 ls are associated with reduced expression of glucose transporter 4 (GLUT4) in adipocytes, an early pa
28 ly increased plasma membrane localization of glucose transporter 4 (GLUT4) in skeletal muscle and adi
29 as a distinct role from CHC17 in trafficking glucose transporter 4 (GLUT4) in skeletal muscle and fat
30                       Mice that over-express glucose transporter 4 (Glut4) in skeletal muscle, heart,
31                                              Glucose transporter 4 (GLUT4) is sequestered inside musc
32                         In basal adipocytes, glucose transporter 4 (GLUT4) is sequestered intracellul
33                              However, higher glucose transporter 4 (GLUT4) levels were detected in mu
34 ked glucose uptake by 1 h into G(1) Of note, glucose transporter 4 (glut4) localized on the RMC surfa
35  in cardiac PPARalpha-transgenic mice, heart glucose transporter 4 (GLUT4) mRNA expression and glucos
36 e by stimulating the movement of sequestered glucose transporter 4 (GLUT4) proteins from intracellula
37 -genetics screen based on translocation of a glucose transporter 4 (Glut4) reporter expressed in muri
38       Insulin stimulates the mobilization of glucose transporter 4 (GLUT4) storage vesicles to the pl
39          Insulin-stimulated translocation of glucose transporter 4 (GLUT4) to plasma membrane of skel
40  the translocation of the insulin-responsive glucose transporter 4 (GLUT4) to the plasma membrane in
41           Insulin-dependent translocation of glucose transporter 4 (Glut4) to the plasma membrane of
42                 Recruitment of intracellular glucose transporter 4 (GLUT4) to the plasma membrane of
43           Insulin-dependent translocation of glucose transporter 4 (Glut4) to the plasma membrane pla
44       Glucose transport and translocation of glucose transporter 4 (GLUT4) to the plasma membrane wer
45 ulin receptor (IR) and the redistribution of glucose transporter 4 (GLUT4) to the plasma membrane.
46  into skeletal muscle through recruitment of glucose transporter 4 (GLUT4) to the plasma membrane.
47 uscle and adipose tissue by translocation of glucose transporter 4 (GLUT4) to the plasma membrane.
48 ing augments glucose transport by regulating glucose transporter 4 (GLUT4) trafficking from specializ
49  resulted from a Nef-dependent inhibition of glucose transporter 4 (GLUT4) trafficking, as assessed b
50 lin A disrupts all actin filaments, inhibits glucose transporter 4 (GLUT4) translocation, and causes
51 to define the mechanism of insulin-regulated glucose transporter 4 (Glut4) translocation, we have dev
52 duction of insulin-induced hexose uptake and glucose transporter 4 (GLUT4) translocation, whereas Akt
53 reased insulin-stimulated insulin-responsive glucose transporter 4 (GLUT4) translocation, while adeno
54 rms to insulin-stimulated glucose uptake and glucose transporter 4 (GLUT4) translocation.
55 lucose uptake in muscle cells by stimulating glucose transporter 4 (GLUT4) translocation.
56                                              Glucose transporter 4 (GLUT4) was lower in IUGR and IUGR
57 se transporter 1 (GLUT1) was upregulated but glucose transporter 4 (GLUT4) was unaffected, and adipos
58 ng enhanced green fluorescent protein-tagged glucose transporter 4 (GLUT4) within a zone about 100 nm
59 ng a ubiquitin regulatory X (UBX) domain for glucose transporter 4 (GLUT4)).
60 h as insulin receptor substrate 2 (IRS2) and glucose transporter 4 (GLUT4), are present in the basal
61 is factor-alpha (TNF-alpha) mRNA and induced glucose transporter 4 (GLUT4), muscle carnitine palmitoy
62 stimulated glucose uptake is mediated by the glucose transporter 4 (GLUT4), which is expressed mainly
63 mulation and subsequent glucose transport by glucose transporter 4 (GLUT4), which resides in speciali
64 activate the insulin-responsive facilitative glucose transporter 4 (GLUT4).
65 and brain natriuretic peptide (BNP) and more glucose transporter 4 (GLUT4).
66 locate in response to insulin, much like the glucose transporter 4 (GLUT4).
67 rated by genetic disruption of one allele of glucose transporter 4 (GLUT4+/-), the insulin-responsive
68 ment of the facilitative glucose transporter glucose transporter-4 (Glut4) from an intracellular comp
69 im of this study was to evaluate the role of glucose transporter-4 (GLUT4) in the anti-diabetic effec
70                The role of insulin-regulated glucose transporter-4 (GluT4) in the brain is unclear.
71 lar glucose uptake by changing the amount of glucose transporter-4 (GLUT4) in the plasma membrane thr
72                        The insulin-regulated glucose transporter-4 (GluT4) is critical for insulin- a
73 tilization at the mRNA and protein level and glucose transporter-4 (GLUT4) localization in skeletal m
74           In adipocytes, vesicles containing glucose transporter-4 (GLUT4) redistribute from intracel
75 ), activation of protein kinase B (Akt), and glucose transporter-4 (GLUT4) translocation to the plasm
76 ved in glucose and fat metabolism, including glucose transporter-4, hexokinase-2, muscle-pyruvate kin
77 ng protein that regulates the trafficking of glucose transporter 4 in response to insulin and muscle
78 sphorylated insulin receptor substrate 1 and glucose transporter 4 in the fetal heart.
79                               The content of glucose transporter 4 in the plasma membrane and basal a
80 pression of insulin receptor substrate 1 and glucose transporter 4 in the skeletal muscle, but thiazo
81 ride lipase enzymes, leptin, adiponectin and glucose transporter-4 in 3T3-L1 cells which may have con
82 ha (hypoxia-inducible factor 3a), and GLUT4 (glucose transporter 4) in male placentas but not females
83 at the effects of AMPK on gene expression of glucose transporter 4, mitochondrial genes, and PGC-1alp
84 n more markedly attenuated insulin action on glucose transporter 4 movements, hexose transport activi
85 pomyosin, intercellular adhesion molecule-4, glucose transporter-4, Na-K-ATPase, sodium/hydrogen exch
86                                       GLUT4 (glucose transporter 4) plays a pivotal role in insulin-i
87 mulated IRbeta tyrosine phosphorylation, and glucose transporter-4 protein level were each lower in b
88 rotein, IRbeta tyrosine phosphorylation, and glucose transporter-4 protein) in the epitrochlearis mus
89 ted with decreased total and plasma membrane glucose transporter 4 proteins.
90 substrate metabolism (glucose transporter 1, glucose transporter 4, pyruvate dehydrogenase kinase 4,
91            Only MEF2C regulated transcripts (glucose transporter 4, SERCA2a, and myosin heavy chain a
92 t through the translocation of intracellular glucose transporter 4 to the plasma membrane in muscle a
93 ting translocation of the insulin-responsive glucose transporter 4 to the plasma membrane.
94 gested to be required for insulin-stimulated glucose transporter 4 translocation in mouse skeletal mu
95  binds its receptor on adipocytes or muscle, glucose transporter-4 vesicles fuse with the cell membra
96  reduction of glucose transporter I, but not glucose transporter 4, was restored in HIF transgenic mo