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1 induction of autoantibodies directed against glucose-6-phosphate isomerase.
2 nt mice had elevated serum concentrations of glucose-6-phosphate isomerase.
3 ine RNase 42-56, on I-Ak and an autoantigen, glucose-6-phosphate isomerase 282-294, on I-Ag7.
4 se and reduced deposition of pathogenic anti-glucose-6-phosphate isomerase Abs in the joint (with a r
5 r domain serves as a scaffold for recruiting glucose-6-phosphate isomerase and dehydrogenase.
6    These genes encode enzymes in glycolysis (glucose-6-phosphate isomerase and phosphoglycerate mutas
7 inefficient at taking up the key autoantigen glucose-6-phosphate isomerase and that Msr1-deficient mi
8 to [6,6'-(3)H]Fru-2,6-P(2) using hexokinase, glucose-6-phosphate isomerase, and 6-phosphofructo-2-kin
9 elping B cells to produce arthritogenic anti-glucose-6-phosphate isomerase (anti-GPI) autoantibodies.
10 6.TCR.Calpha(-/-)H-2(b/g7) mice induced anti-glucose 6-phosphate isomerase antibody-dependent chronic
11 uld not be reproduced by increasing the anti-glucose-6-phosphate isomerase antibody load, which demon
12 nase, glucose 6-phosphate dehydrogenase, and glucose 6-phosphate isomerase are reduced by 11-13 kcal/
13 mber of the IL-1 family, can exacerbate anti-glucose-6-phosphate isomerase autoantibody-induced arthr
14 e model of arthritis, autoantibodies against glucose-6-phosphate isomerase cause joint-specific infla
15 peroxiredoxin-5, secretoglobin family 1D and glucose-6-phosphate isomerase characterized the LF pheno
16 osure, twice-a-day treatment with 17l in the glucose-6-phosphate isomerase chronic in vivo mouse mode
17                       Finally, two proteins (glucose-6-phosphate isomerase [EC 5.3.1.9] and isoflavon
18 induction of autoantibodies directed against glucose-6-phosphate isomerase (G6PI).
19                 Analysis of the chimerism by glucose- 6-phosphate isomerase (GPI) assay revealed that
20 atoid arthritis, autoantibodies specific for glucose-6-phosphate isomerase (GPI) can transfer joint-s
21  the transfer of autoantibodies specific for glucose-6-phosphate isomerase (GPI) into naive mice rapi
22                                              Glucose-6-phosphate isomerase (GPI) is the target autoan
23 ceptors (BCRs) with different affinities for glucose-6-phosphate isomerase (GPI) were examined in the
24 vealed a point mutation, Gly-189 --> Glu, in glucose-6-phosphate isomerase (GPI), a glycolytic enzyme
25 om pathogenic immunoglobulins that recognize glucose-6-phosphate isomerase (GPI), a glycolytic enzyme
26 high titers of antibodies against the enzyme glucose-6-phosphate isomerase (GPI), promoted by CD4(+)
27 e displays oral, in vivo efficacy in a mouse glucose-6-phosphate isomerase (GPI)-induced paw swelling
28 e displays oral, in vivo efficacy in a mouse glucose-6-phosphate isomerase (GPI)-induced paw swelling
29 autoimmune arthritis by tracking the fate of glucose-6-phosphate isomerase (GPI)-reactive CD4(+) T ce
30             Here we identify this protein as glucose-6-phosphate isomerase (GPI).
31 ctivity to a ubiquitously expressed antigen, glucose-6-phosphate isomerase (GPI).
32  recognize the ubiquitous cytoplasmic enzyme glucose-6-phosphate isomerase (GPI).
33  sustained by antibodies to the self-antigen glucose-6-phosphate isomerase (GPI).
34 B cells both recognize the glycolytic enzyme glucose-6-phosphate-isomerase (GPI) as an autoantigen.
35 nize the ubiquitously expressed self-antigen glucose-6-phosphate-isomerase (GPI) initiates an anti-GP
36 thritis is induced by autoantibodies against glucose-6-phosphate-isomerase (GPI).
37                      Immunization with human glucose-6-phosphate isomerase (hG6PI) protein or with se
38 s lead demonstrated oral efficacy in a mouse glucose-6-phosphate isomerase-induced paw swelling model
39 y passive transfer of autoantibodies against glucose 6-phosphate isomerase is transient and therefore
40 ormation and autoantibody production against glucose-6-phosphate isomerase, leading to joint inflamma
41 thal phenotype of RNAi-mediated depletion of glucose-6-phosphate isomerase (PGI) in the glucose-deple
42 tive pentose phosphate (OPP) pathway and the glucose-6-phosphate isomerase (PGI) reaction.
43 .5 +/- 0.5 %ID/g), as early as 1 d after the glucose-6-phosphate-isomerase serum injection, a time po
44 hosphate dehydrogenase, pyruvate kinase, and glucose-6-phosphate isomerase showed IgE-binding for 6%-
45 icro-positron emission tomography that these glucose-6-phosphate isomerase-specific autoantibodies ra
46                                              Glucose-6-phosphate isomerase-specific plasma cells did
47       We injected mice with serum containing glucose-6-phosphate-isomerase-specific antibodies to ind
48 e in the titer of serum antibodies targeting glucose-6-phosphate isomerase, the relevant autoantigen,
49 associated with increased arthritis and anti-glucose-6-phosphate isomerase titers.
50 (2) died in galactose medium as well as when glucose-6-phosphate isomerase was knocked down, suggesti
51 ransgenic mice specific for the self-antigen glucose-6-phosphate isomerase, we show that autoreactive