ライフサイエンスコーパス: glucosyl

1 tal structure of BT3312 in complex with beta-glucosyl-1,6-deoxynojirimycin revealed a TIM barrel cata

2 wly discovered roasting products 17-O-beta-d-glucosyl-11-hydroxycafestol-2-on, 11-O-beta-d-glucosyl-(

3 The new compounds 17-O-beta-d-glucosyl-11-hydroxycafestol-2-on, 11-O-beta-d-glucosyl-1

4 into several products, including 17-O-beta-D-glucosyl-11-hydroxycafestol-2-one (2), 11-O-beta-D-gluco

5 e binding modes of the compounds 11-O-beta-d-glucosyl-15,16-dehydrocafestol-2-on and 11-O-beta-d-gluc

6 )-16-desoxy-17-oxocafestol-2-on, 11-O-beta-d-glucosyl-15,16-dehydrocafestol-2-on, and 11-O-beta-d-glu

7 )-16-desoxy-17-oxocafestol-2-on, 11-O-beta-d-glucosyl-15,16-dehydrocafestol-2-on, and 11-O-beta-d-glu

8 )-16-desoxy-17-oxocafestol-2-on, 11-O-beta-d-glucosyl-15,16-dehydrocafestol-2-on, and 11-O-beta-d-glu

9 desoxy-17-oxocafestol-2-one (4), 11-O-beta-D-glucosyl-15,16-dehydrocafestol-2-one (5), 11-O-beta-D-gl

10 lucosyl-11-hydroxycafestol-2-on, 11-O-beta-d-glucosyl-16-desoxycafestol-2-on, 11-O-beta-d-glucosyl-(S

11 yl-11-hydroxycafestol-2-one (2), 11-O-beta-D-glucosyl-16-desoxycafestol-2-one (3), 11-O-beta-D-glucos

12 nosyl)hexoside and quercetin-3-O-(6''-acetyl)glucosyl-2''-sinapic acid.

13 (beta-D-glucosyl)3 and (beta-D-galactosyl)3 Yariv phenylglycosid

14 l auxiliary containing 1-(tetracetyl-alpha-D-glucosyl)-3-pivaloxymethylpyridinium perchlorate undergo

15 We have focused on the glucosyl-3-phosphoglycerate synthase (GpgS), a "retainin

16 The natural product 7-O-glucosyl-4'-hydroxyisoflavone (daidzin), isolated from t

17 deoxyglucosone-3-ene (3,4-DGE) isomers and 4-glucosyl-5,6-dihydroxy-2-oxohexanal (4-G,3-DG) were foun

18 metabolic pathway to transform the 5hmC into glucosyl-5-hydroxymethyl-2'-deoxycytidine (5-gmC) and ac

19 ATP-stimulated REase activity on T4 DNA with glucosyl-5-hydroxymethyl-cytosines (glc-5hmC) and T4gt D

20 e efficient and consistent pull-down of beta-glucosyl-5-hydroxymethylcytosine (beta-glu-5hmC), and se

21 re further modified by glycosylation to form glucosyl-5-hydroxymethylcytosine (ghmC).

22 oxybenzoic acid diglucoside, tryptophan, 6-C-glucosyl-8-C-arabinosyl-apigenin and diferulic acids.

23 igate the mechanisms of the glycosylation of glucosyl a-trichloroacetimidate with three acceptors (Et

24 y strain AB-1 is cyclodecakis-(1-->3)-beta-D-glucosyl, a cyclic beta-(1-->3)-linked decasaccharide in

25 e enzyme was specific for glucose-6-P as the glucosyl acceptor, and the Km for this substrate was app

26 effect appears to be general across various glucosyl acceptors, glucosyl donor types, and modes of a

27 Glucosyl alkyl gallates were shown previously to be bett

28 the reversible interconversion of trehalose (glucosyl-alpha,alpha-1,1-glucose) and maltose (glucosyl-

29 ucosyl-alpha,alpha-1,1-glucose) and maltose (glucosyl-alpha1-4-glucose).

30 ncluding the first reported synthesis of the glucosyl analogue of the bacterial antigen BbGL-II.

31 Coupling of 3 with the peracetylated glucosyl and galactosyl halides 12a,b and 26 afforded, a

32 little difference between the reactivity of glucosyl and galactosyl iodides.

33 evelopment of a set of compounds bearing the glucosyl and galactosyl moieties.

34 he differences in inhibitory potency between glucosyl and glucosaminyl derivatives and also for the d

35 Perbenzylated N-phenyl trifluoroacetimidate glucosyl and heptosyl derivatives served as alpha-select

36 ion between the C2-O2 and C3-O3 bonds in the glucosyl and mannosyl donors and of the influence of thi

37 he corresponding 4,6-O-benzylidene-protected glucosyl and mannosyl donors, which are alpha- and beta-

38 glycosylation systems involving benzylidene glucosyl and mannosyl donors.

39 components with a wide range of galactosyl, glucosyl and mannosyl linkages that do not directly refl

40 nclude cytochromes P450, GSH S-transferases, glucosyl and other transferases, aryl acylamidase, and o

41 se (alpha-Gal A) hydrolyze the sphingolipids glucosyl- and globotriaosylceramide, respectively, and m

42 sferases in other families with galactosyl-, glucosyl-, and xylosyltransferase activities, each with

43 ne 1-phosphate (S1P), PAF, psychosine (Psy), glucosyl-beta1'1-sphingosine (Glu-Sph), galactosyl-beta1

44 o-2-1,3-benzoxadiazol-4-yl)amino)hexanoyl)-D-glucosyl-beta1-1'-sphin gosine, a fluorescent GlcCer ana

45 quirements: we propose that spsL codes for a glucosyl-(beta1-->4)-glucuronosyl transferase in Sphingo

46 by thioglycosylation of a 6-S-acetyl-alpha-D-glucosyl bromide with the isothiouronium salt of 2,3,4,6

47 suggested that it can potentially generate a glucosyl buffer between maltose and hexose phosphate bec

48 n chains on the heteroglycan that acts as a "glucosyl buffer" to ensure a constant rate of sucrose sy

49 echanism, in which NAD(+) initially oxidizes glucosyl C4 of dTDP-glucose to NADH and dTDP-4-ketogluco

50 , a simultaneous kinetic characterization of glucosyl C5((1)H/(2)H) solvent hydrogen and C6((16)OH/(1

51 iants is due to the loss of control over the glucosyl C5-C6 bond rotation in the active site.

52 previously uncharacterized, enzyme-catalyzed glucosyl-C5 hydrogen-solvent exchange reaction of produc

53 demonstrate Asp135's role in protonating the glucosyl-C6(OH) during dehydration.

54 Our results show the glucosyl cap to have a single, well-defined conformation

55 be largely unaffected by the presence of the glucosyl cap.

56 wever, one is remarkably homologous to human glucosyl ceramidase, an enzyme involved in the ceramide

57 The mutant has an enrichment in glucosyl ceramide and cell surface glycoconjugates beari

58 se (GCase) leads to abnormal accumulation of glucosyl ceramide in lysosomes and the development of th

59 amides and a corresponding increase in their glucosyl ceramide precursors.

60 We report that inactivation of glucosyl ceramide synthase (GCS), either by RNAi or with

61 First, inhibition of glucosyl ceramide synthase by a new specific inhibitor o

62 buffers cystathionine, hexosylceramides, and glucosyl ceramides in the invasive front of both hydroge

63 ids sphingomyelin, lactosyl cerebroside, and glucosyl cerebroside tended to inhibit full pore enlarge

64 ic studies on ternary complexes in which the glucosyl component is substituted by the putative transi

65 The binding results using the O-linked glucosyl conjugate were consistent with a simple model i

66 Caffeic acid, caffeoyl-glucose, linocaffein, glucosyl-coumarate, vanillic acid, rutin and TPI values

67 nd subsequent hydrolysis of the five alpha-d-glucosyl-d-fructoses by K. pneumoniae.

68 e but also its five linkage-isomeric alpha-d-glucosyl-d-fructoses: trehalulose, turanose, maltulose,

69 nd phenyl derivatives induce weakly, but the glucosyl derivative does not.

70 nses with imino compounds (cyclo-DOPA or its glucosyl derivatives), or amines and/or their derivative

71 GBS synthesizes three major glycolipids: glucosyl-diacylglycerol (Glc-DAG), diglucosyl-DAG (Glc(2

72 nionic PG, and a novel cationic lipid, lysyl-glucosyl-diacylglycerol (Lys-Glc-DAG), from neutral glyc

73 covery of a novel cationic glycolipid, lysyl-glucosyl-diacylglycerol (Lys-Glc-DAG), which is synthesi

74 n that catalyzes the formation of galactosyl-glucosyl-diacylglycerol, a glycolipid important for cell

75 anylated polyglycerol-phosphate linked to di-glucosyl-diacylglycerol.

76 Cyclodecakis-(1-->3)-beta-D-glucosyl did not suppress the fungal beta-glucan-induced

77 Two ether glucosyl diglyceride analogs were synthesized, and their

78 ell-inhibitory profile of these ether-linked glucosyl diglycerides strengthens the hypothesis that su

79 The interaction of a C-glucosyl dihydrochalcone with a POPC membrane was modele

80 A library of C-glucosyl dihydrochalcones and their dihydrochalcone and

81 ns, while the formation of the analogous 1,3-glucosyl dioxanium ions is thwarted by a prohibitively s

82 r assay to measure the decay rate of ([(14)C]glucosyl)-diphytanylglyceroldiether (GlcDGD) as an analo

83 toluene solvent mixture, ethyl 1-thio-beta-d-glucosyl disaccharide donors having 6-O-benzyl group(s)

84 bearing the peripheral L-vancosaminyl-1,2-D-glucosyl disaccharide that contain changes to a key sing

85 hing activity when incubated with sucrose as glucosyl donor and (oligo-)dextran as acceptor, transfer

86 approximately 7 mm when UDP-glucose was the glucosyl donor and approximately 4 mm with GDP-glucose.

87 e general across various glucosyl acceptors, glucosyl donor types, and modes of activation.

88 The Km for UDP-glucose as the glucosyl donor was approximately 18 mm, and that for GDP

89 rom Thermus sp. Tapioca starch served as the glucosyl donor, and erythritol as the acceptor.

90 h cellulase and adding soluble starch as the glucosyl donor.

91 othiopyranoside were prepared and studied as glucosyl donors at -60 degrees C in dichloromethane with

92 med-disarmed coupling between two benzylated glucosyl donors by tuning their reactivity.

93 ently protected beta-linked 2-O-glycosylated glucosyl donors carrying bulky tert-butyldimethylsilyl g

94 ucleoside diphosphate glucose derivatives as glucosyl donors, i.e. ADP-glucose, CDP-glucose, GDP-gluc

95 lpha-directing effect in both galactosyl and glucosyl donors, irrespective of the configuration at C-

96 swer to why these do not form from analogous glucosyl donors.

97 banana lectin also recognizes beta1,6-linked glucosyl end groups (gentiobiosyl groups) as occur in ma

98 carbose-complexed, and trapped 5-fluoro-beta-glucosyl-enzyme intermediate forms revealed extended sub

99 y 3-fold, while accelerating turnover of the glucosyl-enzyme intermediate several hundredfold.

100 Abscisic acid glucosyl ester (ABA-GE) is a hydrolyzable ABA conjugate

101 The four glucosyl esters were synthesized and tested for the dete

102 ganelles (gut granules), as anthranilic acid glucosyl esters--not, as previously surmised, the damage

103 aring kinetic results obtained using alpha-D-glucosyl fluoride (GF) and maltooligosaccharides as subs

104 tain wild-type level activity toward alpha-D-glucosyl fluoride hydrolysis.

105 nism-based inhibitor 2-deoxy-2-fluoro-beta-D-glucosyl fluoride.

106 wild-type and Trp120-->Phe GAs using alpha-D-glucosyl fluoride.

107 -binding site in a hydrophobic cleft and the glucosyl function binding to a hydrophobic patch immedia

108 ne, or in the hydroxylysine-linked glycoside glucosyl-galactose in the diabetic kidneys.

109 +vinylcatechol, cyanidin 3-xylosyl-(feruloyl-glucosyl)-galactoside+vinylcatechol, cyanidin 3-xylosyl-

110 vinylguaiacol, cyanidin 3-xylosyl-(feruloyl-glucosyl)-galactoside+vinylguaiacol were found in the sh

111 anins including cyanidin 3-xylosyl-(caffeoyl-glucosyl)-galactoside, cyanidin 3-xylosyl-(p-hydroxybenz

112 toside, cyanidin 3-xylosyl-(p-hydroxybenzoyl-glucosyl)-galactoside, cyanidin 3-xylosyl-galactoside+vi

113 3-xylosyl-galactoside and cyanidin-3-xylosyl-glucosyl-galactoside accounted for the highest amount of

114 ecretion and a decrease in hydroxylysine and glucosyl-galactosyl hydroxylysine.

115 Several glycosphingolipids, glucosyl-, galactosyl-, lactosyl-, and galabiosylceramid

116 ding interactions between the enzyme and the glucosyl group in subsite -1, particularly with the 4'-

117 The glucosyl group of the substrate is bound to the protein

118 IroB transfers glucosyl groups from uridine-5'-diphosphoglucose to C5 o

119 that this lectin also binds to the reducing glucosyl groups of beta-1,3-linked glucosyl oligosacchar

120 de for the interconversion of galactosyl and glucosyl groups.

121 grade both alpha-glucosy-HMC T4 DNA and beta-glucosyl-HMC T4 DNA, whereas no activity was observed ag

122 fied thymidine residue termed base J (beta-d-glucosyl-HOMedU).

123 ked but not by a soluble beta-(1-->3)-linked glucosyl homopolysaccharide (pustulan and laminarin, res

124 By using various soluble glucosyl homopolysaccharides as inhibitors of SP-D carbo

125 e and proton donor, with endoglucanases from glucosyl hydrolase family 12.

126 clude infection by T4 ip1(-) phage and other glucosyl-hydroxymethylcytosine (glc-HMC) Tevens lacking

127 Brig1, a DNA glycosylase that excises alpha-glucosyl-hydroxymethylcytosine nucleobases from the bact

128 at the glucosylated thymine DNA base (beta-d-glucosyl-hydroxymethyluracil or base J) is present withi

129 Base J (beta-D-glucosyl-hydroxymethyluracil) replaces 1% of T in the Le

130 Base J (beta-D-glucosyl-hydroxymethyluracil) was discovered in the nucl

131 osynthesis and maintenance of base J (beta-d-glucosyl-hydroxymethyluracil), an epigenetic modificatio

132 Beta-D-Glucosyl-hydroxymethyluracil, also called base J, is an

133 Base J, beta-d-glucosyl-hydroxymethyluracil, is an epigenetic modificat

134 ynthesis of the modified thymine base beta-D-glucosyl-hydroxymethyluracil, or J, within telomeric DNA

135 We identified 4-O-beta-D-glucosyl-indol-3-yl formamide (4OGlcI3F) as a pathogen-i

136 lly selective Diels-Alder reaction between a glucosyl-modified alkene and an enal to set the C15-C20-

137 ipids containing alpha-linked galactosyl and glucosyl moieties have been shown to possess unique immu

138 sferase activity, DPE2, believed to transfer glucosyl moieties to a complex heteroglycan prior to the

139 site that binds cello-oligomers of up to ten glucosyl moieties.

140 ucuronate to the 2''-hydroxyl group of the 3-glucosyl moiety of cyanidin 3-O-6''-O-malonylglucoside w

141 was mainly glucosylated at the steviol C-19 glucosyl moiety.

142 W1065 provides stacking interactions to the glucosyl moiety.

143 ese pockets in stacking interaction with one glucosyl moiety.

144 s of cellobiose, CbpA releases one activated glucosyl molecule while conserving one ATP molecule per

145 at carbohydrate acceptors is achieved using glucosyl N-phenyl-trifluoroacetimidate (PTFAI) donor pro

146 of binding of the two groups (galactosyl and glucosyl) of oligosaccharides to the two respective sets

147 reducing glucosyl groups of beta-1,3-linked glucosyl oligosaccharides (e.g. laminaribiose oligomers)

148 ifies NHP by catalyzing the formation of 1-O-glucosyl-pipecolic acid in Arabidopsis thaliana.

149 preferred conformation for alpha(1-4)-linked glucosyl polymers.

150 , rhamnogalacturonans, homogalacturonans and glucosyl polysaccharides, under efficient and food-safe

151 analogues with different substituents at the glucosyl position.

152 l-15,16-dehydrocafestol-2-on and 11-O-beta-d-glucosyl-(R)-16-desoxy-17-oxocafestol-2-on and their agl

153 -15,16-dehydrocafestol-2-on, and 11-O-beta-d-glucosyl-(R)-16-desoxy-17-oxocafestol-2-on had lower bit

154 -15,16-dehydrocafestol-2-on, and 11-O-beta-d-glucosyl-(R)-16-desoxy-17-oxocafestol-2-on were detected

155 -15,16-dehydrocafestol-2-on, and 11-O-beta-d-glucosyl-(R)-16-desoxy-17-oxocafestol-2-on were isolated

156 15,16-dehydrocafestol-2-one (5), 11-O-beta-D-glucosyl-(R)-16-desoxy-17-oxocafestol-2-one (6), bengale

157 dentified as branched trisaccharides, with a glucosyl residue alpha-(1 --> 2)-linked to the acceptor'

158 ntially hydrolyzes the non-reducing terminal glucosyl residue from (1-->3)-beta-D-glucans, but also h

159 .1.25), this enzyme strictly transferred one glucosyl residue from alpha(1-->4)-glucans in disproport

160 ptor, the enzyme efficiently transferred the glucosyl residue from sucrose to linear alpha-(1-->6) de

161 dded the first xylosyl residue to the fourth glucosyl residue from the reducing end of both acceptors

162 The presence of a second alpha-linked glucosyl residue is also critical for strong inhibition

163 nteractions with the protein, whereas the +3 glucosyl residue makes relatively few contacts with the

164 forming subsite -1, involved in binding the glucosyl residue of sucrose and catalysis, are strictly

165 e I in Arabidopsis, which trims the terminal glucosyl residue of the oligosaccharide chain of nascent

166 Nss catalyzed the direct transfer of the glucosyl residue to the GlcNAc-modified Fap1 substrate i

167 rbon atom of the covalently bound subsite -1 glucosyl residue, thus explaining the unique lyase activ

168 ed affinity for the second covalently linked glucosyl residue.

169 risaccharides can be bound by their internal glucosyl residues and that binding also occurs through i

170 la showed they were composed of mannosyl and glucosyl residues and the mannosyl residues were acetyla

171 e consistent with a model in which alternate glucosyl residues are transiently or permanently twisted

172 removal may prevent the misincorporation of glucosyl residues for mannosyl residues into the glycoco

173 sidic bonds to release non-reducing terminal glucosyl residues from glycosides and oligosaccharides,

174 and a beta-(1 --> 2)-linked side chain of d-glucosyl residues in disaccharide repeating units.

175 e hydrolase family 70 were shown to transfer glucosyl residues on the non-natural acceptor.

176 Register-dependent interactions with these glucosyl residues reposition the polymer's terminal gluc

177 nt extraction, associates with it to add the glucosyl residues that complete the cellotriosyl and hig

178 d that GtfC catalyzed the direct transfer of glucosyl residues to Srr2-GlcNAc.

179 nd (oligo-)dextran as acceptor, transferring glucosyl residues to the acceptor via a ping-pong bi-bi

180 n lectins recognize internal alpha1,3-linked glucosyl residues, which occur in the linear polysacchar

181 through differential binding to nonterminal glucosyl residues.

182 the final glycosylation step by transferring glucosyl residues.

183 the final glycosylation step by transferring glucosyl residues.

184 e of unbranched (1,3)- and (1,4)-linked beta-glucosyl residues.

185 3-acetic acid and the disaccharide rutinose (glucosyl-rhamnose).

186 , myricetin 3-O-rhamnoside and myricetin 3-O-glucosyl rhamnoside.

187 MbA pathway-specific compound myricetin 3-O-glucosyl rhamnoside.

188 lactosyl-rhamnosyl-glucoside, kaempferol-3-O-glucosyl-rhamnosyl-glucoside, theaflavin, and theobromin

189 ut not in GH7 endoglucanases, at the leading glucosyl ring provide the thermodynamic driving force fo

190 ith the 2- and 3-equatorial OH groups on the glucosyl ring.

191 Roasting products 11-O-beta-d-glucosyl-(S)-16-desoxy-17-oxocafestol-2-on, 11-O-beta-d-

192 glucosyl-16-desoxycafestol-2-on, 11-O-beta-d-glucosyl-(S)-16-desoxy-17-oxocafestol-2-on, 11-O-beta-d-

193 lucosyl-11-hydroxycafestol-2-on, 11-O-beta-d-glucosyl-(S)-16-desoxy-17-oxocafestol-2-on, 11-O-beta-d-

194 syl-16-desoxycafestol-2-one (3), 11-O-beta-D-glucosyl-(S)-16-desoxy-17-oxocafestol-2-one (4), 11-O-be

195 ily as the SA 2-O-beta-D-glucoside (SAG) and glucosyl salicylate (GS).

196 ated by standard quantitative PCR (qPCR) and glucosyl-sensitive restriction enzyme digestion (gRES-qP

197 The active site accommodates a two-glucosyl side chain and provides a site for addition of

198 ear beta-(1,6)-glucan chains with beta-(1,3)-glucosyl side chain with an average of 1 branch point ev

199 hyl cellulose (CM-cellulose), with K259H (in glucosyl subsite -2) creating the highest activity (370%

200 sidic linkages of glycogen and related alpha-glucosyl substrates within lysosomes.

201 he hysteresis of freezing in the presence of glucosyl sugars, namely glucose, maltose, and trehalose.

202 Recently, the use of glucosyl sulfamate inhibitors has shown promise as selec

203 neered hCA IX-mimic in complex with selected glucosyl sulfamates and structurally rationalize mechani

204 ized metabolites (thiamine derivatives and N-glucosyl-taurine).

205 chip6 expressed in E. coli catalyses glucosyl transfer from UDP-glucose to cholesterol.

206 ich are known to serve as donors in acyl and glucosyl transfer reactions in the vacuole, where Os9BGl

207 se the plant heteroglycan as an acceptor for glucosyl transfer.

208 Glycosylation of 5-hmC residues by beta-glucosyl transferase (beta-GT) can make CCGG residues in

209 talyzed by the enzyme, UDPG-indol-3-ylacetyl glucosyl transferase (IAA-glucose-synthase).

210 yanin-related gene UDP glucose:flavonoid 3-O-glucosyl transferase (UFGT), which was dependent of the

211 Conversely, in E. amylovora, the homologous glucosyl transferase activity appears to be relatively i

212 ne, which encodes a protein with homology to glucosyl transferase enzymes, is expressed within 15 min

213 ignificantly attenuated by the inhibition of glucosyl transferase in tumor cells, suggesting that tum

214 wartii is non-functional, while the terminal glucosyl transferase is catalytically active.

215 charide glycosyl transferase G) encoding the glucosyl transferase of GC that initiates the beta chain

216 xpression of CsbB, a putative membrane-bound glucosyl transferase that is partially controlled by the

217 d 5' to 3' DNA exonuclease), alpha-gt (alpha-glucosyl transferase), gp47.1 (uncharacterized), and Nrd

218 Starch-branching enzyme (SBE), a glucosyl transferase, is required for the highly regular

219 ine at position 304 (F304S) of the alpha 1,3 glucosyl transferase.

220 id sequence are with members of family 31 of glucosyl transferase.

221 t completely lacks base J but still contains glucosyl-transferase activity.

222 An Arabidopsis mutant lacking the glucosyl-transferase, STARCH SYNTHASE 4 (SS4) is impaire

223 identified as encoding glucuronosyl-(B1-->4)-glucosyl transferases based on reciprocal genetic comple

224 hyl transfers (SAM), prenyl transfers (IPP), glucosyl transfers (UDP-glucose), and electron and ADP-r

225 >6)-linked but not alpha/beta-(1-->3)-linked glucosyl trisaccharides can be bound by their internal g

226 beta-(1-->4)-, and alpha/beta-(1-->6)-linked glucosyl trisaccharides into the SP-D carbohydrate recog

227 5-O-glucosyl-tylactone was also obtained, showing that endog

228 The extent of [4,5,6-(13)C(3)]glucosyl unit enrichment in glycogen was enhanced by ins

229 we show that DPE2 transfers the non-reducing glucosyl unit from maltose to glycogen by a ping-pong me

230 ferent glucansucrases and is close to the +1 glucosyl unit in the crystal structure of GTF180-DeltaN

231 l residues reposition the polymer's terminal glucosyl unit to form either a (1,3)- or (1,4)-beta-link

232 mylation patterns is identified: the precise glucosyl unit where hydroxycinnamic acid acylation occur

233 The glycogen (glucosyl unit) concentration was 38.1 mmol/kg wet weight

234 These calculations suggest the presence of a glucosyl unit, also inherent in the strictosidine struct

235 the nascent polysaccharide's second or third glucosyl unit.

236 red with Con (117 +/- 39 vs. 240 +/- 32 mmol glucosyl units (kg DM)(-1), respectively; P < 0.01), but

237 = 9.2 +/- 1.1 vs. 3 min = 22.3 +/- 4.0 mmol glucosyl units (kg dry muscle)(-1) min(-1), P < 0.05).

238 co-oligosaccharides in which alpha1,3-linked glucosyl units are joined sequentially to maltose.

239 eared in glycogen in carbon positions 4-6 of glucosyl units but none in positions 1-3.

240 ly 70 (GH70) that catalyzes the transfer ofd-glucosyl units from sucroseto dextrans or gluco-oligosac

241 , the transmembrane subunit MalF binds three glucosyl units from the nonreducing end of the sugar.

242 lyzes (1-->2)-, (1-->6)-, and (1-->4)-beta-D-glucosyl units in decreasing order of activity.

243 he peak intensity of the C1 resonance of the glucosyl units in muscle glycogen during a 6-h hyperglyc

244 s containing predominantly (1,4)-beta-linked glucosyl units interspersed with single (1,3)-beta-linke

245 wed to synthesize variable amounts of 1-[13C]glucosyl units of glycogen.

246 activity, capable of transferring one of the glucosyl units of maltose to glycogen or amylopectin and

247 hat consists essentially of only xylosylated glucosyl units, with no further substitutions.

248 t hydrolyzed CMC to fragments averaging 10.7 glucosyl units.

249 lZ hydrolyzed CMC to fragments averaging 3.6 glucosyl units.

250 elY hydrolyzed CMC to products averaging 2.3 glucosyl units.

251 s interspersed with single (1,3)-beta-linked glucosyl units.

252 an agent with R = isopropyl and R' = beta-D-glucosyl was prepared and shown to generate nitric oxide

253 de, delphinidin 3-O-glucoside, quercetin 3-O-glucosyl-xyloside, dihydroquercetin, and quercetin 3-O-g

254 rminated and grown in medium containing beta-glucosyl Yariv reagent (beta GlcY), a synthetic phenyl g

255 rentiating xylem of pine trees by using beta-glucosyl Yariv reagent (beta-glcY) and was recognized by

256 e arabinogalactan-protein (AGP) binding beta-glucosyl Yariv reagent (betaGlcY) that disrupts cell elo

257 l/Ara-rich motifs not recognized by the beta-glucosyl Yariv reagent but interacting with the peanut a

258 the traditional AGP-diagnostic reagent beta-glucosyl Yariv reagent, and they are also recognized by

259 -protein complex that binds poorly with beta-glucosyl Yariv reagent, and two glycoproteins.