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1 colabeled with antibodies against VGluT2, a glutamate transporter.
2 otential rather than acting as a presynaptic glutamate transporter.
3 by rodents but was shown to be a functional glutamate transporter.
4 ty that the parasite activates the host cell glutamate transporter.
5 amate and are not known to have a functional glutamate transporter.
6 majority of GLT-1, the brain's most abundant glutamate transporter.
7 In addition, GltS was identified as a glutamate transporter.
8 l binding of two Na(+) ions in Na(+)-coupled glutamate transporters.
9 ls near astrocytes and tightly gated by Glt1 glutamate transporters.
10 1), voltage-gated sodium channels (Nav ) and glutamate transporters.
11 ly via expression and function of astroglial glutamate transporters.
12 proteins, synapsin 1, unique microRNAs, and glutamate transporters.
13 eir highly branched morphologies and express glutamate transporters.
14 h necessitates the proper function of active glutamate transporters.
15 domain movements in a bacterial homologue of glutamate transporters.
16 r glutamate, which is regulated primarily by glutamate transporters.
17 into mechanisms of allosteric modulation for glutamate transporters.
18 smission due to their abundant expression of glutamate transporters.
19 substance, cytochrome oxidase, and vesicular glutamate transporters.
20 ine-induced suppression of the high-affinity glutamate transporter 1 (EAAT2/GLT-1) in the nucleus acc
21 e via specialized transporters such as glial glutamate transporter 1 (excitatory amino-acid transport
22 seeking while increasing the function of the glutamate transporter 1 (GLT-1) and system xC- (Sxc) in
23 his behavioral effect has been attributed to glutamate transporter 1 (GLT-1) and xCT (a catalytic sub
24 t TX enhanced the expression and function of glutamate transporter 1 (GLT-1) in rat astrocytes, an ef
25 ea, to promote the upregulation of the glial glutamate transporter 1 (GLT-1) on astrocytes and to red
26 hat astroglial glutamate transporter subtype glutamate transporter 1 (GLT1) and glutamate uptake is s
28 illary acidic protein, glutamine synthetase, glutamate transporter 1 (GLT1), aquaporin-4, aldehyde de
29 g the tibial nerve (TN), and using Vesicular GLUtamate Transporter 1 (VGLUT1) and the 65 kDa isoform
30 ectors to target expression of the vesicular glutamate transporter 1 (VGLUT1) following injection int
31 or NL2 after IUEP does not affect vesicular glutamate transporter 1 (vGlut1) in the glutamatergic co
32 hR2) is conditionally expressed in vesicular glutamate transporter 1 (Vglut1) sensory neurons (Vglut1
34 ere identified in both nuclei: (a) vesicular glutamate transporter 1 (vGluT1)-positive terminals form
35 shown by using antibodies against vesicular glutamate transporter 1 [labeling all ON and OFF bipolar
37 porter Sv2 (now known as SV2A) and Vesicular glutamate transporter 1 in the outer molecular layer of
38 ate transporter levels were higher and glial glutamate transporter 1 levels were lower in the DH of f
39 droxylase in periglomerular cells, vesicular glutamate transporter 1, a presynaptic protein, in mitra
40 microscopic level, the density of vesicular glutamate transporter 1-positive (i.e. cortico-subthalam
41 y synapses, defined by overlapping vesicular glutamate transporter 1-positive (VGlut1+) and postsynap
42 ory synapses (i.e., the overlap of vesicular glutamate transporter 1-positive [VGlut1+] puncta and po
43 studies showing that the number of vesicular glutamate transporter 1-positive terminals and of axon t
45 oxide synthase, somatostatin, and vesicular glutamate transporters 1 and 2 accounted for a combined
46 imary afferents immunoreactive for vesicular glutamate transporters 1 and 2 and by intraspinal neuron
47 utamatergic terminals express both vesicular glutamate transporters 1 and 2 denoting a specific sourc
48 glutamate-aspartate transporter (GLAST) and glutamate transporter-1 (GLT-1), which are essential for
51 and (1)H-MRS methods, lower levels of glial glutamate transporter-1 and ATP-alpha, but increased lev
53 glutamatergic neurons that express vesicular glutamate transporter 2 (VGlut2) and are located in subc
54 by their immunoreactivity for the vesicular glutamate transporter 2 (VGluT2) and performed unbiased
55 ea dopamine (DA) neurons expresses vesicular glutamate transporter 2 (VGluT2) and releases glutamate
56 the adult, healthy brain expresses vesicular glutamate transporter 2 (VGluT2) and thus releases gluta
57 Cre-expressing neurons or inducing vesicular glutamate transporter 2 (VGLUT2) deficiency in Trpv1-Cre
58 -Cre expressing neurons or induced vesicular glutamate transporter 2 (Vglut2) deficiency in Trpv1-Cre
59 rminals, which are associated with vesicular glutamate transporter 2 (Vglut2) expression, in the dors
60 ffect of deleting the gene for the vesicular glutamate transporter 2 (Vglut2) from neurons in the PB.
61 tive to previous expression of the vesicular glutamate transporter 2 (Vglut2) gene, coupled with immu
62 s, including the expression of the vesicular glutamate transporter 2 (VGluT2) mRNA in Ipc neurons, ha
63 r bouton size, and the presence of vesicular glutamate transporter 2 (Vglut2) or parvalbumin (PV).
65 he VTA has many neurons expressing vesicular glutamate transporter 2 (VGluT2) that also project to LH
66 onprimary boutons that express the vesicular glutamate transporter 2 (VGLUT2), and form asymmetrical
67 N-->CeA CGRP projections coexpress vesicular glutamate transporter 2 (VGLUT2), providing evidence tha
68 rojected to MDmc and expressed the vesicular glutamate transporter 2 (VGLUT2), which is found in high
69 atic rings of terminals expressing vesicular glutamate transporter 2 (VGLUT2)--to subdivide IC GABAer
71 a, with or without the presence of vesicular glutamate transporter 2 (VGLUT2)-mediated glutamatergic
74 markers [neurofilament, NeuN, and vesicular glutamate transporter 2 (VGlut2)], and cultures exhibite
75 the VP [VP neurons expressing the vesicular glutamate transporter 2 (VP(VGluT2))], whose activation
76 lation of dopamine neurons express vesicular glutamate transporter 2 and make glutamatergic connectio
77 ar layer, while immunostaining for vesicular glutamate transporter 2 and neurofilament H indicate tha
78 re GABAergic and were contacted by vesicular glutamate transporter 2-containing somatic terminals, as
80 kers for both glutamate signaling (vesicular glutamate transporter 2; VGluT2) and GABA signaling (glu
81 with glutamate neurons (expressing vesicular glutamate transporter 2; VGluT2), which play roles in re
83 glutamatergic neurons--expressing vesicular glutamate transporter-2 (VGluT2)--project to limbic and
84 2 signals alongside an increase in vesicular glutamate transporter-2 signals on PV+ cells in MeCP2-de
85 rformed antibody stainings against vesicular glutamate transporter-2, which suggested that cytochrome
86 K neurons is composed of transient vesicular glutamate transporter 3 (tVGLUT3) neurons, which convey
88 -originating pathway consisting of vesicular glutamate transporter 3 (VGluT3) containing neurons that
91 dings to characterize responses of vesicular glutamate transporter 3 (VGluT3)-expressing amacrine cel
94 transporter 1(+), somatostatin(+), vesicular glutamate transporter 3 (VGLUT3)/cholecystokinin/CB(1) c
98 ner hair cells in mice lacking the vesicular glutamate transporter-3 (Vglut3(KO) ), at 9-11 weeks, ap
102 ociated with reduced placental glutamine and glutamate transporter activity and expression, and propo
103 suggest that the Kir4.1 conductance affects glutamate transporter activity in a dual manner: (1) by
104 esis and suggest that abnormal glutamine and glutamate transporter activity is part of the spectrum o
109 symporter, Glt(Ph) is an archaeal homolog of glutamate transporters and has been extensively used to
112 vesicle transport at neuronal synapses), the glutamate transporter, and a voltage-gated calcium chann
113 none oxidoreductase 1 (NQO1), Bach1, cystine/glutamate transporter, and glutamate cysteine ligase.
115 pic and metabotropic glutamate receptors and glutamate transporters, and altered neuronal excitabilit
116 /H(+) exchanger, ClC-7 H(+)/Cl(-) exchanger, glutamate transporters, and neutral amino acid transport
117 ound in episodic ataxia of the dual-function glutamate transporter/anion channel EAAT1, and discovere
125 tamate binding can be isolated in the mutant glutamate transporter because reactions, such as glutama
126 d EPSCs in afferent fibers are unaffected by glutamate transporter blockade, suggesting that transmit
128 ncreased (or decreased) in the presence of a glutamate transporter blocker (or a competitive glutamat
129 n thus prompts spatial retreat of astroglial glutamate transporters, boosting glutamate spillover and
130 DVGLUT functions not only as a vesicular glutamate transporter but also serves as an acid-extrudi
137 or voltage-clamped astrocytes and respective glutamate transporter currents (GTCs) were induced by ph
138 examined intracellular Na(+) transients and glutamate transporter currents as the most telling indic
142 oduced two cysteine residues in the neuronal glutamate transporter EAAC1 at positions predicted to be
143 g debate on the contribution of the neuronal glutamate transporter EAAC1 to the onset of compulsive b
144 plicate Slc1a1, a gene encoding the neuronal glutamate transporter EAAC1, with obsessive-compulsive d
145 increase the expression and activity of the glutamate transporter (EAAT(2)) on glial cells, blocks m
146 e up-regulated proteins (GFAP, high affinity glutamate transporter (EAAT-2), apo-J (Clusterin), and p
148 ncluding AMPA and kainate receptor subunits, glutamate transporters EAAT1 and EAAT2, and the GABA(A)
151 Previous literature has indicated that glial glutamate transporter EAAT2 plays an essential role in c
153 els are tightly controlled by the astrocytic glutamate transporter EAAT2, influencing synaptic functi
156 -181A > C) SNP in the promoter of astroglial glutamate transporter (EAAT2) and the same approach was
159 c vesicle machinery, including the vesicular glutamate transporter eat-4/VGLUT, induction of neuropep
160 ture of glutamatergic neurons, the vesicular glutamate transporter EAT-4/VGLUT, is expressed in 38 of
161 Perisynaptic astrocytes express important glutamate transporters, especially excitatory amino acid
162 ous glutamate levels controlled by astrocyte glutamate transporters, evokes a transient and reversibl
163 st the effects of K(+) and Na(+) on neuronal glutamate transporter excitatory amino acid carrier 1 (E
164 uated glutamate uptake and expression of the glutamate transporter excitatory amino acid transporter
166 mily 1 (SLC1), which also includes the human glutamate transporters (excitatory amino acid transporte
167 an excitatory neuronal marker, the vesicular glutamate transporter, expanding the possible roles of S
169 ssociated membrane protein 1), and astrocyte glutamate transporter expression (glutamate/aspartate tr
170 gnaling is a positive regulator of astrocyte glutamate transporter expression and function, an essent
171 a brain tumor stem cells with low astrocytic glutamate transporter expression are dependent on GLS to
172 dition, ENT1 inhibition or knockdown reduces glutamate transporter expression in cultured astrocytes.
174 vation and lysosomal pathology, and restored glutamate transporter expression to levels observed in W
178 te earlier developmental roles in regulating glutamate transporter function.SIGNIFICANCE STATEMENT In
179 s study enhances our understanding as to how glutamate transporters function as both amino-acid trans
180 Additionally, the expression of two known glutamate transporters, genderblind and excitatory amino
181 dates include non-recurrent deletions at the glutamate transporter gene SLC1A1, a CNV locus recently
182 The gene for EAAT2, the major astrocytic glutamate transporter, generates two carrier isoforms (E
183 ontrast levels of the predominant cerebellar glutamate transporter GLAST, expressed in Bergmann glia,
184 The compound did not affect the astrocytic glutamate transporter GLAST, nor did it block glutamate
186 s that increase expression of the astroglial glutamate transporter GLT-1 (N-acetylcysteine and ceftri
187 te carrier family 1 member 2), also known as glutamate transporter GLT-1 and excitatory amino acid tr
188 ining, we assessed surface expression of the glutamate transporter GLT-1 and glutamate efflux in the
189 ed to increased expression of the astrocytic glutamate transporter GLT-1 and to attenuated changes in
192 e; then, using an antisense strategy against glutamate transporter GLT-1, we found that restored tran
196 efrontal cortical blockade of the astrocytic glutamate transporter (GLT-1) induces anhedonia and c-Fo
198 g glutamate spillover by blocking astroglial glutamate transporters (GLT-1) had no effect on reinstat
199 eurotrophic factors, BDNF and IGF-1, and the glutamate transporter, GLT-1 after ischemic brain damage
202 , presumably through dysregulated astroglial glutamate transporter GLT1 and impaired glutamate uptake
203 -124-3p further up-regulates the predominant glutamate transporter GLT1 by suppressing GLT1-inhibitin
204 , likely through the dysregulated astroglial glutamate transporter GLT1 expression and impaired gluta
207 ficantly reduced expression of the major CNS glutamate transporter, GLT1, in superficial dorsal horn
210 utward- and inward-facing conformations of a glutamate transporter homolog from archaebacterium Pyroc
212 e fluorescence imaging of the archaeal model glutamate transporter homologue Glt(Ph) from Pyrococcus
213 y proteins defining glutamatergic signaling (glutamate transporter-I [GLT-I], N-methyl-D-aspartate re
214 Ceftriaxone modulated the expression of the glutamate transporter in a critical region of the cortic
217 mino acid transporter 2 (EAAT2) is the major glutamate transporter in the brain expressed predominant
219 results indicate a role for neuron-specific glutamate transporters in AMPAR synaptic localization an
220 Here we elucidated the roles of these two glutamate transporters in cerebellar pathogenesis mediat
221 gulates the trafficking of both dopamine and glutamate transporters in dopamine neurons by increasing
223 ssion for the typically astroglial-localized glutamate transporters in the mediodorsal and ventral ti
224 se was tightly controlled by plasma membrane glutamate transporters, indicating that clearance of syn
225 ed with LTP magnitude following nonselective glutamate transporter inhibition but not following selec
227 orters, the reverse dialysis of ammonia, the glutamate transporter inhibitor, DL-threo-beta-benzyloxy
228 nion conducting conformation of the neuronal glutamate transporter is associated with an early step o
231 opose that the switch to the dynamic mode in glutamate transporters is due to separation of the trans
233 uptake is mediated by members of a family of glutamate transporters known as "excitatory amino acid t
234 ate signaling dynamics, increased astrocytic glutamate transporter levels and alleviated multiple sig
235 uggests that abnormalities in the astroglial glutamate transporter localization and function may unde
236 the peptide transporter CstA, PEB1 aspartate/glutamate transporter, LutABC lactate dehydrogenase and
239 itochondria and suggest a mechanism by which glutamate transporters might retain mitochondria at site
240 erlie their capacity to upregulate the glial glutamate transporter on astrocytes through the vascular
241 relapse to opioid use, and downregulation of glutamate transporters on astroglial processes adjacent
243 lar glutamate levels influenced by astrocyte glutamate transporters resulted in a significant inhibit
244 calcium pump SERCA, leucine transporter and glutamate transporter shows that ANMPathway yields resul
245 responding serine residue, Ser-364, of human glutamate transporter SLC1A2 (solute carrier family 1 me
246 id application of glutamate to the wild-type glutamate transporter subtype EAAC1 (excitatory amino ac
248 ate in glutathione biosynthesis, the cystine/glutamate transporter (system xc(-)) represents a potent
250 rter (GLAST) in rodents, is one of two glial glutamate transporters that are responsible for removing
251 mate receptors, GABA receptors (GABARs), and glutamate transporters that have been implicated in pain
252 ino acid transporters (EAATs) are a class of glutamate transporters that terminate glutamatergic syna
254 sociated protein 25, syntaxin, and vesicular glutamate transporter type 1), together with an increase
259 ate of glutamate clearance through astrocyte glutamate transporters under high-frequency stimulation
260 ed the accumulation of presynaptic vesicular glutamate transporter (VGlut) and increased spontaneous
263 study with probes for the lamprey vesicular glutamate transporter (VGLUT) provides an anatomical bas
264 ded into synaptic vesicles via the vesicular glutamate transporter (VGLUT), a mechanism conserved acr
268 xide synthase as well as the three vesicular glutamate transporters (VGLUT 1-3) in the locus coeruleu
270 transporter [vGAT]) or glutamate (vesicular glutamate transporter [vGLUT]) into vesicles, as well as
271 fore mapped boutons expressing the vesicular glutamate transporters VGluT1 and VGluT2, together with
273 rents and of central neurons (with vesicular glutamate transporters VGLUT1 or VGLUT2, respectively),
274 utamate decarboxylase (GAD67), and vesicular glutamate transporters (vGLUT1 and vGLUT2) in postmortem
275 ing in situ hybridization to label vesicular glutamate transporters (vglut1, vglut2.1, vglut3), gluta
276 e reported expression of all three vesicular glutamate transporters (VGLUT1-3) by astrocytes suggests
279 unopositive for met-enkephalin and vesicular glutamate transporter VGLUT2, but not for GABAergic mark
280 e terminals were demonstrated to express the glutamate transporter VGlut2, the projections are presum
282 ed on coexpression of mRNA for the vesicular glutamate transporter (vGlut2) and the GABA synthetic en
283 most neurons in the GT express the vesicular glutamate transporter (VGluT2) mRNA, indicating a glutam
284 ecarboxylase 65 (GAD65) and type 2 vesicular glutamate transporter (VGLUT2) mRNAs, respectively.
285 r the glutamatergic marker, type 2 vesicular glutamate transporter (VGLUT2), and the GABA synthetic e
291 release machinery mediated through vesicular glutamate transporters (VGLUTs) that ultimately dictate
292 apse in NTS, we determined whether vesicular glutamate transporters (VGLUTs) were differentially dist
293 ecord currents associated with the vesicular glutamate transporters (VGLUTs), we characterize a chlor
294 T), which is a major component of astrocytic glutamate transporters, was reduced by TNR knockdown.
295 for Nissl, cytochrome oxidase, and vesicular glutamate transporters, we investigated the primary soma
298 hrin-A2 in the cortex colocalized with glial glutamate transporters, which were significantly downreg