ライフサイエンスコーパス: glutamic

1 nd kinetics of (S)-4-(3-[18F]fluoropropyl)-l-glutamic acid ((18)F FSPG) in healthy volunteers and to

2 radiotracer, (S)-4-(3-[(18)F]fluoropropyl)-L-glutamic acid ([(18)F]FSPG), decreases in proportion to

3 , aspartic acid (coded by GAY [Y = U or C]), glutamic acid (coded by GAZ [Z = A or G]), glycine (code

4 K66 in the SMG7 14-3-3-like domain with the glutamic acid (E) abolishes interactions with its client

5 y via a Ca(2+) binding pocket with conserved glutamic acid (E) and aspartic acid (D) residues.

6 We found that another rare substitution, glutamic acid (E) at position 206, significantly reduced

7 acid residues, glycine (G), serine (S), and glutamic acid (E) at positions 172, 207, and 212, respec

8 However, mutation that substituted glutamic acid (E) for glutamine (Q) at amino acid positi

9 The tyrosine-to-glutamic acid (E) mutant Y131E, which may mimic phosphot

10 The tyrosine to glutamic acid (E) mutant Y138E, which can mimic phosphot

11 C-terminal domain of connexin43 (Cx43) into glutamic acid (E) or alanine (A) residues.

12 o additional amino acids (tryptophan (W) and glutamic acid (E)) at the C-terminus of the mature ligan

13 A glutamic acid (E)-to-glycine (G) difference at position

14 e show that a single hydrogen bond between a glutamic acid (E90) and an asparagine (N258) residue suf

15 Poly-gamma-glutamic acid (gamma-PGA) is an important biochemical pr

16 e pgsBCA cluster (responsible for poly-gamma-glutamic acid (gamma-PGA) synthesis), were intentionally

17 Four of them contained a gamma-carboxyl glutamic acid (Gla) domain, a calcium-binding module, an

18 Acidic amino acids, aspartic acid (Asp) and glutamic acid (Glu) can enhance the solubility of many p

19 ve methods for measuring glutamine (Gln) and glutamic acid (Glu) in cell cultures and other biologica

20 Plasma levels of glutamic acid (GLU), glutamine, glycine, proline (PRO),

21 of an unusually located and highly conserved glutamic acid (Glu-176) within the beta3 transmembrane r

22 xidase (GmOx) microelectrode for measuring l-glutamic acid (GluA) in oxygen-depleted conditions, whic

23 rtic acid (IAA-Asp) and indole-3-acetic acid glutamic acid (IAA-Glu) of 438- and 240-fold, respective

24 Natural mutations such as lysine 255 to glutamic acid (K to E), threonine 259 to isoleucine (T t

25 replacing this lysine with alanine (K265A), glutamic acid (K265E) or glutamine (K265Q), and the func

26 transmitters glutamate and N-acetyl-aspartyl-glutamic acid (NAAG) and their precursor glutamine.

27 mug/mL histidine (p < 0.001), 100 mug/mL of glutamic acid (p < 0.05) and 200 mug/mL of glutamic acid

28 The poly-gamma-glutamic acid (PGA) capsule produced by Bacillus anthrac

29 A skin prick test for poly-gamma-glutamic acid (PGA) which is a component of jellyfish st

30 terized a pH-responsive biodegradable poly-L-glutamic acid (PGA)-fluocinolone acetonide (FLUO) conjug

31 eimer's disease, covalently linked to poly-l-glutamic acid (PGA).

32 is work, we demonstrate that proline-proline-glutamic acid (PPE)17 protein of Mycobacterium tuberculo

33 In animal experiments knocking in a glutamic acid (S1530E) in DEPDC5, a phospho mimic, in tu

34 ated either to the potential phospho-mimetic glutamic acid (Y102E) or to the nonphosphorylated homolo

35 ive mutagenesis analysis, we identified that glutamic acid 14 (E14) of vBcl-2 is critical for KSHV ly

36 to a chemical reaction, the deprotonation of glutamic acid 148 (E148).

37 he lumen-exposed residues, threonine 162 and glutamic acid 173, form stabilizing hydrogen bonds betwe

38 acids, L-aspartic acid 4-methyl ester and L-glutamic acid 5-methyl ester, is a convenient and sensit

39 External glutamic acid 623 (E623) is key for TMEM16A's ability to

40 ngs we propose that external protons titrate glutamic acid 623, which enables voltage activation of T

41 erred a small-plaque avirulent phenotype and glutamic acid a large-plaque virulent phenotype.

42 We show that protonation of the conserved glutamic acid alters the peptide insertion depth in the

43 Glutamic acid and alanine make up more than 60 per cent

44 f glutamic acid (p < 0.05) and 200 mug/mL of glutamic acid and aspartic acid (p < 0.001) without affe

45 d lower plasma concentrations of citrulline, glutamic acid and carnitine at 24 hrs after enrolment an

46 e biosynthetic pathway to kainic acid from l-glutamic acid and dimethylallyl pyrophosphate in red mac

47 Free glutamic acid and free aspartic acid found in the PPI hy

48 m, were conjugated quickly and directly with glutamic acid and glutamine, and further with peptides,

49 PELP1 (proline, glutamic acid and leucine rich protein 1) is a nuclear r

50 containing multi-layers of short alternating glutamic acid and lysine (EK) peptides as a facile, high

51 TPs calculated from delta (15)N values of glutamic acid and phenylalanine, which range from 8.3-33

52 s from wet solids were significantly rich in glutamic acid and proline.

53 ng 98% of ascorbic acid and 100% of glycine, glutamic acid and uric acid.

54 ghest regression coefficients were found for glutamic acid and valine, with regards to blood orange j

55 as compared to adsorption of soy protein and glutamic acid as common ingredients.

56 e of F-III was due to the presence of free l-glutamic acid at 6 times, while FII and FIV were due to

57 For rat Glut5, a change of glutamine to glutamic acid at codon 166 (p.Q166E) has been reported t

58 n of the AIF-MIF interaction, or mutation of glutamic acid at position 22 in the catalytic nuclease d

59 with previous studies, we demonstrate that a glutamic acid at position 296 results in attenuation.

60 thin the C terminus of LukA, we identified a glutamic acid at position 323 that is critical for LukAB

61 In this study we found that KIR2DL2/L3 with glutamic acid at position 35 (E(35)) are functionally st

62 rily linked to HLA-DPB1 alleles possessing a glutamic acid at position 69 of the beta-chain.

63 ions not previously identified, specifically glutamic acid at positions 10 or 11 or lysine at positio

64 71 mutant, containing lysine, glutamine, and glutamic acid at the respective residues 98, 145, and 16

65 tonia is caused by an in-frame deletion of a glutamic acid codon in the gene encoding the AAA+ ATPase

66 ter enrolment and significantly lower plasma glutamic acid concentrations (74.4 versus 98.2 mumol/L)

67 nd non-coding regions explained aspartic and glutamic acid consumption differences, likely due to a p

68 tic amino acid aspartic acid could fully and glutamic acid could partially reconstitute the level of

69 predicted absolute growth rates of the alpha-glutamic acid crystal at lower supersaturations are in r

70 ved DC were pulsed with preproinsulin (PPI), glutamic acid decarboxylase (65-kDa isoform; GAD65), and

71 gamma aminobutyric acid synthesizing enzyme glutamic acid decarboxylase (GAD) and choline acetyltran

72 High levels of antibodies against glutamic acid decarboxylase (GAD) are observed in patien

73 is is controlled by enzymes derived from two glutamic acid decarboxylase (GAD) genes, GAD1 and GAD2,

74 Antibodies to glutamic acid decarboxylase (GAD) have been associated w

75 of activation (Fos) of GABAergic neurons and glutamic acid decarboxylase (GAD) mRNA expression in the

76 pocampal slices and activity measurements of glutamic acid decarboxylase (GAD), a PLP-dependent enzym

77 dinucleotide phosphate-diaphorase (NADPH-d), glutamic acid decarboxylase (GAD), cytochrome oxidase (C

78 e and some were parvalbumin-, calbindin-, or glutamic acid decarboxylase (GAD)-67-positive.

79 hibitory neurotransmitter, is synthesized by glutamic acid decarboxylase (GAD).

80 nobutyric acid (GABA) transporter (vGAT) and glutamic acid decarboxylase (GAD)65 in the GABAergic con

81 Additionally, glutamic acid decarboxylase (GAD)65-loaded tolDCs from w

82 Here, we study the distribution of glutamic acid decarboxylase (GAD)67 and GLY transporter

83 tibodies against insulin, the 65-kDa form of glutamic acid decarboxylase (GAD65), insulinoma-associat

84 Gad1 gene-encoded 67-kDa protein isoform of glutamic acid decarboxylase (GAD67) is a hallmark of sch

85 ntain normal levels of the 67 kDa isoform of glutamic acid decarboxylase (GAD67) protein, the enzyme

86 to lower expression of the 67-kDa isoform of glutamic acid decarboxylase (GAD67), a key enzyme for GA

87 in cells that contain the 67 kDa isoform of glutamic acid decarboxylase (GAD67-GFP), or Cre-recombin

88 P), or Cre-recombinase in cells that contain glutamic acid decarboxylase (GAD; GAD2-cre).

89 ext, we produced conditional null alleles of Glutamic acid decarboxylase 1 (Gad1) and Resistant to di

90 nerated transgenic mouse lines that suppress glutamic acid decarboxylase 1 (GAD1) in either cholecyst

91 Levels of gamma-aminobutyric acid (GABA) and glutamic acid decarboxylase 1 (GAD1), the enzyme that sy

92 hypothalamus activated a small population of glutamic acid decarboxylase 2 (GAD2)-expressing neurons

93 The glutamic acid decarboxylase 2 but not the parvalbumin su

94 n of T1D-related autoantigens [proinsulin or glutamic acid decarboxylase 65 (GAD)] delayed T1D onset,

95 tis Abs as well as thyroperoxidase (TPO) and glutamic acid decarboxylase 65 (GAD65) Abs.

96 Glutamic acid decarboxylase 65 (GAD65) and autoantibodie

97 been identified, including orexin cells and glutamic acid decarboxylase 65 (GAD65) cells, but their

98 brane protein (PMP) antibody positivity; and glutamic acid decarboxylase 65-kDa isoform (GAD65) antib

99 atus of the second GABA-synthesizing enzyme, glutamic acid decarboxylase 65-kDa isoform (GAD65), rema

100 pendent expression levels of parvalbumin and glutamic acid decarboxylase 67 (GAD67) in schizophrenia

101 dies have consistently found lower levels of glutamic acid decarboxylase 67 (GAD67) messenger RNA (mR

102 of DNMT1 to psychiatric candidate promoters (glutamic acid decarboxylase 67, Reelin, and brain-derive

103 Levels of the GABA-synthesizing enzyme glutamic acid decarboxylase 67-kDa isoform (GAD67) in th

104 Four patients also had high glutamic acid decarboxylase antibodies (>1000 U/ml), and

105 ms of pathology, many patients with SPS have glutamic acid decarboxylase antibodies (GAD-ab), but the

106 Little is known of glutamic acid decarboxylase antibodies (GAD-abs) in the

107 riodontal conditions, retinopathy, and serum glutamic acid decarboxylase antibody (GADA) titers in re

108 balanced for age, sex, disease duration, and glutamic acid decarboxylase autoantibody titers.

109 ain development through direct activation of glutamic acid decarboxylase enzyme isoforms that convert

110 he presence of the GABA-synthesizing enzyme, glutamic acid decarboxylase in EC were confirmed by immu

111 brillary acidic protein-immunocytochemistry, glutamic acid decarboxylase in situ hybridization, and p

112 hat specific promoter regulatory elements of glutamic acid decarboxylase isoforms (Gad1 and Gad2), wh

113 IgE responses to insulin, autoantibodies to glutamic acid decarboxylase or insulinoma-associated ant

114 We also used immunohistochemistry for glutamic acid decarboxylase to distinguish GABAergic fro

115 gic neurons expressing different isoforms of glutamic acid decarboxylase were found to have different

116 ith SPS have antibodies directed against the glutamic acid decarboxylase, the rate-limiting enzyme fo

117 lear layer and in the ganglion cell layer is glutamic acid decarboxylase-positive and shows the morph

118 ve for GABA and the GABA-synthesizing enzyme glutamic acid decarboxylase.

119 o patients with novel de novo Tpm3.12 single glutamic acid deletions at positions DeltaE218 and Delta

120 A beta-glutamic acid dendron anchor was used to attach a PEG ch

121 With a protected glutamic acid derivate as the starting material, the pro

122 toes resulted in robust GABA production from glutamic acid derived from blood protein digestion.

123 d-Alanine and d-glutamic acid derived from peptidoglycan decomposition e

124 10(4) M(-1)), whereas sucrose, aspartame and glutamic acid did not bind at all.

125 reported that the HPV-31 E2 Y138 mutation to glutamic acid did not bind to the Brd4 C-terminal motif

126 Arginine-glutamic acid dipeptide repeats (RERE) is located in the

127 at binding did not require the gamma-carboxy glutamic acid domain.

128 w potatoes and also thermally generated from glutamic acid during frying.

129 ase structural fold for the N-prenylation of glutamic acid during the biosynthesis of the potent neur

130 protein depends on the protonation state of glutamic acid E163 (Ci1), one of the counterions of the

131 e engaged, despite the lack of aspartic acid/glutamic acid encoded in the mouse repertoire.

132 uvignon, the light body of Xinomavro and the glutamic acid for Malvasia.

133 line for enamine formation on one side and a glutamic acid for nitronate protonation on the other sid

134 for Ser2 and/or Ser7 and the phosphomimetic glutamic acid for Ser7.

135 Substitution of glutamic acid for tyrosine between the Syk SH2 domains (

136 /pvdN double mutant produced exclusively the glutamic acid form of pyoverdine.

137 the capabilities is demonstrated by mapping glutamic acid from a cryosectioned chicken breast with a

138 This mutation ('DeltaE') removes a single glutamic acid from the encoded protein, torsinA.

139 Several studies have advocated the role of glutamic acid in cancer therapy.

140 transamination of the three acids to produce glutamic acid in cancerous cells.

141 rearrangements that expressed aspartic acid/glutamic acid in CDR L2.

142 he E2 envelope glycoprotein (lysine in SFV4, glutamic acid in SFV6).

143 Replacement of a glutamic acid in the central gate with a positively char

144 Importantly, determination of free glutamic acid in the daily diet could also prevent vario

145 Point mutation of either glutamic acid in the Galpha13-binding (767)EKE motif in

146 The oral introduction of glutamic acid increased virus acquisition by mosquitoes

147 2, one cancer-derived ECRG2 mutant harboring glutamic acid instead of valine at position 30 (V30E) fa

148 The data suggest that glutamic acid is a nitrogen acceptor while alanine, aspa

149 red 35 intracellular metabolites involved in glutamic acid metabolism and the gamma-glutamyl cycle in

150 eight pathways, including D-glutamine and D-glutamic acid metabolism; linoleic acid metabolism; alph

151 The hydration number of the glutamic acid molecule also fluctuates due to the rapid

152 uration, the average hydration number of the glutamic acid molecule decreases and can reach an asympt

153 ylalanine mutant E2 Y138F and phosphomimetic glutamic acid mutant Y138E.

154 ations involved replacements by glutamine or glutamic acid of E2 glycoprotein amino acids in the acid

155 sp70EEVD was restored upon substitution of a glutamic acid of the J-domain.

156 er for A(3)R binders, when it was mutated to glutamic acid or alanine, the activity of IB-MECA increa

157 activation is reversed by the removal of the glutamic acid penultimate to the tyrosine.

158 nts in complex with L-aspartic acid versus L-glutamic acid provide insights into their differential s

159 acid substitutions at this highly conserved glutamic acid residue and illustrates the value of syste

160 found that transient protonation changes of glutamic acid residue E141 and, most notably, arginine R

161 ous missense substitutions in the paralogous glutamic acid residue in TWIST2 (p.Glu75Ala, p.Glu75Gln

162 , and a strictly conserved fluorophore-bound glutamic acid residue is converted to a range of variant

163 17Val and p.Glu117Gly) at a highly conserved glutamic acid residue located in the basic DNA binding d

164 ing a calcium ion-binding site and chelating glutamic acid residue that mediate the formation of HC.T

165 304 was replaced with either an alanine or a glutamic acid residue was also affected.

166 enesis of any of the two conserved catalytic glutamic acid residues (Glu(200) and Glu(414)) of the ac

167 by a central tryptophan flanked by aspartic/glutamic acid residues (W-acidic).

168 Interestingly, introduction of three glutamic acid residues alone was not sufficient to estab

169 ned oligoarginine peptides equipped with six glutamic acid residues and an anionic pyranine at the N-

170 arboxylic groups of various key aspartic and glutamic acid residues by monitoring their C=O stretchin

171 Three glutamic acid residues in epsin UIM were found to intera

172 ouble dehydration and decarboxylation of two glutamic acid residues in the 30-residue precursor PaaP.

173 etween histidine 64 in CAII and a cluster of glutamic acid residues in the C terminus of the transpor

174 of different species with varying numbers of glutamic acid residues in the side chain ranging from 12

175 lanine, arginine, glycine, aspartic acid and glutamic acid residues represented the major amino acids

176 Notably, the glutamic acid residues were not solely gamma-linked, as

177 Replacing a cluster of glutamic acid residues with a glutamine-rich motif on th

178 ted by the gamma-carboxylase (GGCX) on three glutamic acid residues, a cellular process requiring red

179 Adding NLS, replacing aspartic acid by glutamic acid residues, or changing the l- to d-aspartic

180 rod cGMP-gated cation channel and associated glutamic acid rich proteins (GARPs) are required for pho

181 y, p66 with Thr(206) and Ser(213) mutated to glutamic acid showed a gain-of-function phenotype with s

182 ysines in UCP1 to acyl-mimetic glutamine and glutamic acid significantly decreases its stability and

183 Charge reversal by glutamic acid substitution at Arg-I or Arg-II has opposi

184 The simulations also show why glutamic acid substitution at either serine does not con

185 However, lysine to glutamic acid substitutions at the KTKEGV repeat domain

186 The alanine and glutamic acid substitutions reduced actin-activated ATPa

187 ociation interfaces and a putative catalytic glutamic acid that is conserved in both bacterial TIR NA

188 cation as a transformation of the N-terminal glutamic acid to a succinamide.

189 to be responsible for the conversion of the glutamic acid to alpha-ketoglutaric acid.

190 olism as it catalyses the decarboxylation of glutamic acid to form GABA.

191 GLUL encoding glutamine synthase, converting glutamic acid to glutamine.

192 A glutamic acid to lysine (E40K) residue substitution in s

193 two amino acid mutations in the PB2 protein (glutamic acid to lysine at position 627 and aspartic aci

194 n of histone 3 lysine 36 (H3K36), exhibits a glutamic acid to lysine mutation at residue 1099 (E1099K

195 an internal proton transfer from a conserved glutamic acid to the proton-loading site of the pump.

196 a revealed three novel mutations including a glutamic acid to valine substitution (E1338D), a glutami

197 ulting variant, which has cysteine-histidine-glutamic acid triads on each helix, hydrolyses p-nitroph

198 atural language (NL) largely untapped (e.g. 'glutamic acid was substituted by valine at residue 6').

199 the S4-S5 helix of the chicken receptor to a glutamic acid was sufficient to endow it with capsaicin

200 ythro-beta-d-methylaspartic acid and gamma-d-glutamic acid were key for an isomerization-free synthes

201 ne could effectively extract about 3.5 mg of glutamic acid with 95% enantiomeric excess in 24 h.

202 ydroxyethyl methacrylate modified poly(gamma-glutamic acid) (gamma-PGA-HEMA), generating hybrid HRP@g

203 We conjugate doxorubicin to poly(L-glutamic acid) by means of a pH-sensitive cleavable link

204 el B(12)-targeted poly(ethylene glycol)-poly(glutamic acid) copolymers as excipients suitable to be f

205 2)-targeted poly(ethylene glycol)-block-poly(glutamic acid) copolymers.

206 d quantification of the adsorbed amino acid (glutamic acid) demonstrated that 1 mg of homochiral poly

207 His6-OPH) and poly(ethylene glycol)-b-poly(l-glutamic acid) diblock copolymer.

208 hybrid hydrogels consisting of a poly(gamma-glutamic acid) polymer network physically cross-linked v

209 D-aas such as D-Asp (aspartic acid), D-Glu (glutamic acid), combined D-[Asp/Glu] and others were eac

210 he DOTAGA (1,4,7,10-tetraazacyclododecane-1-(glutamic acid)-4,7,10-triacetic acid) conjugate PSMA I&T

211 k copolymers, poly(ethylene glycol)-b-poly(L-glutamic acid)-b-poly(L-phenylalanine), which effectivel

212 eptidase in which the third zinc ligand is a glutamic acid).

213 me-modified lysine, C' = citrulline, and E = glutamic acid).

214 ne, aspartic acid, asparagine, tyrosine, and glutamic acid).

215 were prepared using amphiphilic PEG-b-poly(L-glutamic acid)/SN38 conjugates and subsequently loaded w

216 lyceraldehyde 3-phosphate dehydrogenase with glutamic acid, a malonyllysine mimic, suppressed its enz

217 feri and Burkholderia thailandensis bound to glutamic acid, a TrpRS from the eukaryotic pathogen Ence

218 nificant effects were noticed in the case of glutamic acid, alanine, aspartic acid and proline betwee

219 es involved in glutamine catabolism, such as glutamic acid, alanine, glycine, pyrimidine, and creatin

220 l interference from ascorbic acid, cysteine, glutamic acid, and glucose was also studied, and the obt

221 Here, we identify proline, glutamic acid, and leucine-rich protein 1 (PELP1), a chr

222 d with nontumor tissues (proline, threonine, glutamic acid, arginine, N1-acetylspermidine, xanthine,

223 here is no interference from d-glucose and l-glutamic acid, ascorbic acid and o-nitrophenol.

224 ant amino acids that contributed to flavour (glutamic acid, aspartic acid and alanine) were present a

225 chemically well-defined amphoteric carriers, glutamic acid, aspartyl-histidine (Asp-His), cycloserine

226 The peptide, which contains a photocaged glutamic acid, forms a solid-like gel in a syringe and c

227 the source (phenylalanine, Phe) and trophic (glutamic acid, Glu) AAs were 4.1 (muscle) and 5.4 (red b

228 spartic acid, cystathionine, total cysteine, glutamic acid, glutamine, glycine, histidine, total homo

229 rus, cancer, human papillomavirus, dopamine, glutamic acid, IgG, IgE, uric acid, ascorbic acid, acetl

230 Cytoplasmic localization of proline, glutamic acid, leucine-rich protein 1 (PELP1) is observe

231 side chains amino acids that were aspartate, glutamic acid, lysine and tyrosine on the negative side

232 n sources (L-Asparagine, L-Aspartic Acid, L- Glutamic Acid, m- Erythritol, D-Melezitose, D-Sorbitol)

233 acids and thiol precursors (especially with glutamic acid, r <= -0.73) rather than free thiols.

234 Glutamic acid, serine, and glycine concentrations are kn

235 Mechanically, a typical proline, glutamic acid, serine, and threonine motif specifically

236 appaBalpha C-terminal PEST (rich in proline, glutamic acid, serine, and threonine residues) sequence

237 ontains PEST sequences (enriched in proline, glutamic acid, serine, and threonine) and is normally su

238 amily contains an unnatural 4,4-disubstitued glutamic acid, the synthesis of which provides a key cha

239 Rather, amino acids alanine, phenylalanine, glutamic acid, valine, and leucine increased in samples

240 s anthracis is composed entirely of d-isomer glutamic acid, whereas nonpathogenic Bacillus species pr

241 n metabolisms, glutathione, guanosine, and L-glutamic acid, which are implicated in protection agains

242 Molecular modeling analysis reveals that the glutamic acid, which is negatively charged, interacts wi

243 Structural analysis revealed that this key glutamic acid, which is not present in Ydj1, forms a sal

244 with 3-aminophenyl boronic acid (APB) and L-glutamic acid-(2,2,2)-trichloroethyl ester (GTE).

245 ein tyrosine phosphatases from the proline-, glutamic acid-, serine- and threonine-rich (PEST) family

246 mutations causing deletions of the proline-, glutamic acid-, serine-, and threonine-rich (PEST) domai

247 nT, tyrosine-43, lysine-69, arginine-254 and glutamic acid-493, were required for activity.

248 -DOTA-E[c(RGDfK)](2), where E[c(RGDfk)](2) = glutamic acid-[cyclo(arginyl-glycyl-aspartic acid-D-phen

249 tin-1, QHREDGS (glutamine-histidine-arginine-glutamic acid-aspartic acid-glycine-serine), as a therap

250 sporter 1 (VGLUT1) and the 65 kDa isoform of glutamic acid-decarboxylase (GAD65) as markers of, respe

251 d allylation of serine-, aspartic acid-, and glutamic acid-derived organozinc reagents, followed by c

252 les were prepared by self-assembly of poly(L-glutamic acid-L-tyrosine) co-polymer with hematoporphyri

253 Here, we examined the role of non-glutamic acid-leucine-arginine CXC chemokine CXCL9 for T

254 dominant negative termed A-ZIP53 that has a glutamic acid-rich amphipathic peptide sequence attached

255 ghly-diverged structure consisting of a long glutamic acid-rich C-terminal extension of ~70 residues

256 screening strategy to identify P falciparum glutamic acid-rich protein (PfGARP) as a secreted ligand

257 overy of Src homology 3 (SH3) domain-binding glutamic acid-rich-like protein (SH3BGRL), a novel c-Src

258 MA-targeted hybrid tracers were synthesized: glutamic acid-urea-lysine (EuK)-Cy5-mas(3), EuK-(SO(3))C

259 from glucose, guanine, and p-aminobenzoyl-l-glutamic acid.

260 s associated with consumption of excess free glutamic acid.

261 caspases can also hydrolyze substrates after glutamic acid.

262 ycine, l-proline, l-serine, l-alanine, and l-glutamic acid.

263 r in the literature for a racemic mixture of glutamic acid.

264 of tRNAs specific for lysine, glutamine and glutamic acid.

265 tion of HMF from fructose in the presence of glutamic acid.

266 e digestion motifs flanked with aspartic and glutamic acid.

267 or charged residues tyrosine, histidine, and glutamic acid.

268 ially available methyl (R)-(+)-lactate and l-glutamic acid.

269 either the wild type (WT) or with alanine or glutamic acid/aspartic acid substitutions at the phospho

270 the CBP/p300-interacting transactivator with glutamic acid/aspartic acid-rich carboxyl-terminal domai

271 tudy, we examined the efficacy of poly-gamma-glutamic acid/chitosan (PC) nanogel as an adjuvant for t

272 amino acids, aspartic acid/asparagine (Asx), glutamic acid/glutamine and alanine are positively corre

273 CBP/p300-interacting transactivators with E [glutamic acid]/D [aspartic acid]-rich-carboxylterminal d

274 In 09HA_mut, a lysine-to-glutamic-acid mutation leads to the loss of both salt br

275 arum parasites(2), we identify P. falciparum glutamic-acid-rich protein (PfGARP) as a parasite antige

276 Host-adaptive mutations, particularly a glutamic-acid-to-lysine mutation at amino acid residue 6

277 used significantly higher levels (p<0.05) of glutamic acids (343.0+/-22.09mg/100g), total FAAs (1720.

278 Regarding total amino acids, aspartic and glutamic acids (9-10 mg/g) were the major compounds.

279 n the soluble structure that comprised three glutamic acids (Glu92, Glu94, and Glu97) that we hypothe

280 t was replaced with either phospho-mimicking glutamic acids (mdxS3E) or nonphosphorylatable alanines

281 f DMD cardiomyopathy in which phosphomimetic glutamic acids are substituted for serines at these resi

282 complex with the Nix LIR peptide containing glutamic acids as phosphomimetic residues and NMR experi

283 molecular baskets 1(6-), each with three (S)-glutamic acids at its rim, were found (NMR, ITC) to comp

284 essive tumors, drives the protonation of the glutamic acids in ATRAM, leading to the membrane translo

285 Interaction between three glutamic acids in the N-terminal domain of RsbR and the

286 s, and evolutionarily conserved, whereas the glutamic acids surrounding phosphosites significantly de

287 nin still can form pores, but mutating these glutamic acids to glutamines rendered the toxin pH-insen

288 served when acidic amino acids, aspartic and glutamic acids, are present near the cleavage site.

289 arity to predict the effect of mutating each glutamic and aspartic acid located in MTBD domain to ala

290 Glycine, glutamic and aspartic acids accounted for 40% of total a

291 in the presence of acidic amino acids (i.e. glutamic and aspartic acids).

292 cubation with another NAD-dependant enzyme L-glutamic dehydrogenase.

293 sequestering nitrogen from aspartate through glutamic-oxaloacetic transaminase 1 (GOT1).

294 hypertension and liver expression levels of glutamic-oxaloacetic transaminase 2 (GOT2) messenger RNA

295 t the time of admission and revealed a serum glutamic-oxaloacetic transaminase level of 9 U/L [0.15 m

296 uding mitochondrial malate dehydrogenase and glutamic-oxaloacetic transaminase, predominantly in DCD

297 ange, 5-40 U/L [0.08-0.67 mukat/L]), a serum glutamic-pyruvic transaminase level of 34 U/L [0.57 muka

298 carbonylation (alpha-amino adipic and gamma-glutamic semialdehyde).

299 red, while alpha-aminoadipic (AAS) and gamma-glutamic semialdehydes (GGS) increased when cooking at 6

300 carbonylation (alpha-amino adipic and gamma-glutamic semialdehydes) and Schiff base cross-links.