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1                                   Forty-five glycated, 48 advanced glycation endproduct (AGE-) modifi
2 herapeutic approaches against neurotoxic CEL-glycated Abeta1-42.
3 eins, such as glycated hemoglobin (HbA1c) or glycated albumin (GA) in the blood, are essential indica
4 an HbA1c in meta-analysis (P = 0.0225), mean glycated albumin (P = 0.0029), and glyoxal hydroimidazol
5                                  We measured glycated albumin and fructosamine in 11 104 participants
6 ifferences were found in the relationship of glycated albumin and fructosamine levels with the mean g
7                             Fructosamine and glycated albumin are markers of short-term glycemic cont
8               We found that fructosamine and glycated albumin were associated with vascular outcomes
9  baseline concentrations of fructosamine and glycated albumin were significantly associated with each
10 e evaluated associations of fructosamine and glycated albumin with risk of coronary heart disease, is
11 , glycated hemoglobin (HbA1c), fructosamine, glycated albumin), and a latent variable for kidney func
12  monitoring and compared by race with HbA1c, glycated albumin, and fructosamine values.
13  FK is the epsilon-fructosyl amino acid from glycated albumin, another glycated protein, whereas gluc
14 d fasting glucose, insulin, adiponectin, and glycated albumin, as well as body mass index (BMI), use
15 enabling separation of positional isomers of glycated alpha- and beta-chains on the intact level.
16                            The percentage of glycated alpha-Hb and beta-Hb was calculated from the mi
17 case that repairs methylglyoxal- and glyoxal-glycated amino acids and proteins by acting on early gly
18 nes, arginines, and lysines (the three major glycated amino acids) of serum albumin, glyceraldehyde-3
19 nchmark dataset was extracted containing 235 glycated and 303 non-glycated lysine residues.
20  based on support vector machine, to predict glycated and non-glycated lysine residues using structur
21   The proposed predictor manages to classify glycated and non-glycated lysine residues with promising
22 nine sites, as well as by converting already-glycated arginine residues into citrulline.
23                                     A second glycated beta-Hb isomer that is partially resolved from
24                               The values for glycated beta-Hb were found to correlate well with the H
25 n is achieved between alpha-Hb, beta-Hb, and glycated beta-Hb.
26 ected in extracted ion electropherograms for glycated beta-Hb.
27                               Measurement of glycated blood proteins, particularly glycated hemoglobi
28  glycated chaperone-client complexes than in glycated but uncomplexed protein mixtures.
29  backbone conformation from A to B form when glycated, but does not induce any final transition in DN
30                                Proteins were glycated by incubation with sugars (glucose, methylglyox
31 protein isolates that had been denatured and glycated by thermal treatment.
32 inking AGEs were significantly higher in the glycated chaperone-client complexes than in glycated but
33                                              Glycated collagen 1 and AGE-modified ECM regulate adipoc
34 en fibrils and, using KFM and FLiM, that R5P-glycated collagen fibrils have a more negative surface c
35 e extracellular matrix structure surrounding glycated collagen fibrils.
36  indicated the formation of hydrolysates and glycated compounds with different molecular weight distr
37 on products (MRPs), protein hydrolysates and glycated compounds.
38                                   To prepare glycated cowpea protein isolate (GCPI) the cowpea flour
39 By using ESI-QTOF-MS technique, formation of glycated cytochrome C containing up to 12 glucose moieti
40 terial mutants displayed increased levels of glycated DNA and RNA and exhibited strong mutator phenot
41 e for a rapid and reliable identification of glycated DNA in a reagent-free manner.
42 in contrast to guanine oxidation repair, how glycated DNA is repaired remains undetermined.
43 ws a clear discrimination between native and glycated DNA samples.
44 depleted cells displayed increased levels of glycated DNA, DNA strand breaks, and phosphorylated p53.
45 e process where proteins accumulate advanced glycated end products (AGEs).
46  indicated a significant decrease in advance glycated end products and protection against glycoxidati
47       Hyperglycemia and exposure to advanced glycated end products inactivated MFG-E8, recognizing a
48 lysates showed reduced fluorescence advanced glycated end-products (AGE) and a reduced amount of alph
49  insulin signaling, accumulation of advanced glycated end-products, altered autophagy, changes in myo
50 e-associated molecular patterns and advanced glycated endproducts and can trigger cell activation.
51 Amadori rearrangements and the free and mono-glycated guanidine also formed imidazolinone derivatives
52  diabetes remission at 2 years, defined as a glycated haemaglobin A1c (HbA1c) concentration of 6.5% o
53 ive treatments for type 2 diabetes, lowering glycated haemoglobin (HbA(1c)) and weight, but are curre
54 years, diagnosed with type 2 diabetes with a glycated haemoglobin (HbA(1c)) concentration of 7.5% or
55                                              Glycated haemoglobin (HbA(1c)) is considered the gold st
56 erol <=4 mmol/L, triglycerides <=1.7 mmol/L, glycated haemoglobin (HbA1c) <=53 mmol/mol (<=7.0%), sys
57  more, and remission of diabetes, defined as glycated haemoglobin (HbA1c) of less than 6.5% (<48 mmol
58 lucose (FBS), random blood glucose (RBS), or glycated haemoglobin (HbA1c) were reported.
59                              Mean changes in glycated haemoglobin (HbA1c), and mean changes in fastin
60 lthough associations between multimorbidity, glycated haemoglobin (HbA1c), and mortality in people wi
61 nge glucose and insulin measures, as well as glycated haemoglobin (HbA1c), are used to diagnose and m
62  and intensification: age, sex, deprivation, glycated haemoglobin (HbA1c), body mass index (BMI), smo
63 lso associated with improvements in glucose, glycated haemoglobin (HbA1c), weight, waist circumferenc
64 n delivery (pump or injections) and baseline glycated haemoglobin (HbA1c).
65 density-lipoprotein cholesterol (HDL-C), and glycated haemoglobin (HbA1c).
66 eins [LDL and HDL], triglycerides [TGs], and glycated haemoglobin [HbA1c]).
67 abetes at high cardiovascular risk with high glycated haemoglobin A(1c) (HbA(1c)) concentrations.
68  prior to enrolment, and centrally confirmed glycated haemoglobin A1c (HbA(1c)) of 48-58 mmol/mol (6.
69          The primary outcome was a change in glycated haemoglobin A1c (HbA1c) from baseline to week 2
70  function, lipid profile, glucose tolerance, glycated haemoglobin A1c, salivary cortisol, sitting hei
71 , systolic and diastolic blood pressures and glycated haemoglobin in the 1958 British Birth Cohort (1
72  and additional cardiovascular risk factors, glycated haemoglobin of up to 9.5% (80 mmol/mol) on a ma
73               Changes in fasting glucose and glycated haemoglobin were not different between groups a
74                               The changes in glycated haemoglobin, as the primary outcome, in the pro
75 = 32), and were correlated with the level of glycated haemoglobin, glycaemic level, and time of disea
76 iles (longer duration of diabetes and higher glycated haemoglobin, non-HDL cholesterol, and waist-to-
77  least two creatinine, thyrotropin, calcium, glycated haemoglobin, or lithium measurements between Oc
78 sure, body-mass index, total cholesterol and glycated-haemoglobin levels.
79  It was observed that patients with elevated glycated Hb levels also had higher levels of HSA glycati
80                            Quantification of glycated Hb was in good correlation with the results obt
81              In clinical routine analysis of glycated Hb, sequence variants or other Hb proteoforms c
82 nctive test lines when challenged with HbA0, glycated HbA0 and HbA2.
83  points, P<0.001) and had similar control of glycated hemoglobin (0.3 percentage points, P=0.63).
84 ss index (47.6+/-9.3 to 36.7+/-7; P<0.0001), glycated hemoglobin (6.7+/-1.5 to 5.8+/-0.6; P<0.0001),
85 liflozin group than in the placebo group for glycated hemoglobin (difference, -0.46 percentage points
86                                     Improved glycated hemoglobin (Hb A1c) delays the progression of m
87 oad glucose (PG), 2-h postload insulin (PI), glycated hemoglobin (Hb A1c), and homeostasis model asse
88                                              Glycated hemoglobin (HbA(1c)) is an important measure of
89 08) while controlling for age, sex, baseline glycated hemoglobin (HbA(1c)), baseline CMT, baseline VA
90 es diagnosis, serum fasting glucose (FG) and glycated hemoglobin (HbA(1c)), were estimated to be 51-6
91 om among 321 people with type 2 diabetes and glycated hemoglobin (HbA1c) >58 mmol/mol.
92 ype 2 diabetes were recruited: subjects with glycated hemoglobin (HbA1c) </=7% and subjects with HbA1
93 ; betaPFOA=1.71 pM; 95% CI: 0.72, 2.71), and glycated hemoglobin (HbA1c) (betaPFOS=0.03%; 95% CI: 0.0
94 ) concentration (-37.0 mg/dL; P < 0.001) and glycated hemoglobin (HbA1c) [-0.97% (-10.6 mmol/mol); P
95 a significant difference in postintervention glycated hemoglobin (HbA1c) between the arms (P = 0.007)
96 ulinemia, decreased C-peptide, and increased glycated hemoglobin (HbA1c) compared with sham-operated
97 d the association between food label use and glycated hemoglobin (HbA1c) concentrations.
98                             After 12 months, glycated hemoglobin (HbA1c) decreased from 10.3+/-2.4% t
99  has been developed for the determination of glycated hemoglobin (HbA1c) in human blood samples.
100                                 The level of Glycated hemoglobin (HbA1c) is accordingly examined for
101                                              Glycated hemoglobin (HbA1c) is used to diagnose type 2 d
102 ex, waist circumference, fat percentage, and glycated hemoglobin (HbA1c) level were recorded chairsid
103 n leakage area at month 36 (P = 0.0137), and glycated hemoglobin (HbA1c) levels at month 36 (P = 0.00
104  (20-50 U) and metformin (>/=1500 mg/d) with glycated hemoglobin (HbA1c) levels of 7% to 10% and a bo
105 nfluence of periodontal status on changes of glycated hemoglobin (HbA1c) levels of patients with type
106 igated OCT-A parameters with DM duration and glycated hemoglobin (HbA1c) levels were evaluated among
107                                              Glycated hemoglobin (HbA1c) levels were measured, and pr
108  2 spectral domain OCT (SD-OCT) tests, and 2 glycated hemoglobin (HbA1c) measures over time with a mi
109 DM prevalence was estimated using laboratory glycated hemoglobin (HbA1c) or fasting plasma glucose in
110                   Glycated proteins, such as glycated hemoglobin (HbA1c) or glycated albumin (GA) in
111 hievement of good glycemic control, of which glycated hemoglobin (HbA1c) remains the standard clinica
112              An immunoassay for detection of glycated hemoglobin (HbA1c) that can reveal a patient's
113 in or loss and glycemic control (assessed by glycated hemoglobin (HbA1c) values) in patients from the
114                   In multivariable analyses, glycated hemoglobin (HbA1c) was inversely associated wit
115                             Higher levels of glycated hemoglobin (HbA1c) were associated with all-cau
116 rmined for fasting glucose, fasting insulin, glycated hemoglobin (HbA1c), and homeostasis model asses
117 tion of high- compared with low-fat dairy on glycated hemoglobin (HbA1c), body weight, and cardiovasc
118      The relationship between delayed GE and glycated hemoglobin (HbA1c), complications of DM, and ga
119 eplacement of other sugars and its effect on glycated hemoglobin (HbA1c), fasting blood glucose, insu
120 agnostic accuracies of random blood glucose, glycated hemoglobin (HBA1c), fructosamine, and Homeostas
121  glycemia (diabetes status, fasting glucose, glycated hemoglobin (HbA1c), fructosamine, glycated albu
122  triacylglycerol (TG), fasting glucose (FG), glycated hemoglobin (HbA1c), insulin resistance (HOMA-IR
123 and at follow-up, was associated with higher glycated hemoglobin (HbA1c), insulin, HOMA-IR, triglycer
124 ent of glycated blood proteins, particularly glycated hemoglobin (HbA1c), is an important diagnostic
125                                        Blood glycated hemoglobin (HbA1c), reflecting the average bloo
126 ed with the diabetes GRS on fasting insulin, glycated hemoglobin (HbA1c), the homeostasis model asses
127                               Measurement of glycated hemoglobin (HbA1c), the most widely accepted in
128  blood pressure (BP), fasting blood glucose, glycated hemoglobin (HbA1c), triglyceride levels, trigly
129 led trials (RCTs) that assessed the outcomes glycated hemoglobin (HbA1c), weight, body mass index (BM
130  sensor was developed for the recognition of glycated hemoglobin (HbA1c).
131  body mass index (in kg/m(2)): 34.6 +/- 4.3; glycated hemoglobin (HbA1c): 7.3 +/- 1.1%; duration of d
132 n [n = 136; mean +/- SD age: 12.8 +/- 2.6 y; glycated hemoglobin (HbA1c): 8.1% +/- 1.0%; 69.1% using
133 .7%) was associated with a reduction in mean glycated hemoglobin (HbA1c, -1.3 +/- 1.8%, P < 0.001), f
134         In 2006, 56% of incident cases had a glycated hemoglobin (hemoglobin A1c) test as one of the
135                      Levels of blood glucose/glycated hemoglobin (International Federation of Clinica
136                    How these risks vary with glycated hemoglobin (or hemoglobin A1c [HbA1c]) levels i
137 ion was associated with a higher increase in glycated hemoglobin (P = 0.027).
138 luble leptin receptor (reduction of 43%) and glycated hemoglobin (reduction of 28%).
139  97 individuals >=40 and <=80 years old with glycated hemoglobin 6.5% to 10.0%, known coronary artery
140 y; median (IQR), 10 (5-9) y of T2D duration; glycated hemoglobin 7.0% +/- 0.8%; body mass index (in k
141 8 of whom had poor glycemic control (average glycated hemoglobin [HbA1c] >/=8% during the year) while
142 tensive glycemic control in type 2 diabetes (glycated hemoglobin [HbA1c] level <7%) is an established
143 cose [RBG], fasting blood glucose [FBG], and glycated hemoglobin [HbA1c]) and survival in all lung tr
144 fication by sex, history of ischemic stroke, glycated hemoglobin A(1c), body mass index, blood pressu
145 g plasma glucose >/=200 mg/dl (11.1 mmol/l), glycated hemoglobin A1c (HbA1c) >6.5%, self-reported phy
146 se [FCG] level, 2-hour CG [2-hCG] level, and glycated hemoglobin A1c [HbA1c] level) at enrollment, an
147 agnosis, sex, race/ethnicity, net worth, and glycated hemoglobin A1c fraction (HbA1c).
148    While adjusting for duration of diabetes, glycated hemoglobin A1c level, and other factors, we fou
149                                The mean (SD) glycated hemoglobin A1c of the 50 patients (26 men and 2
150 ciation of baseline waist circumference with glycated hemoglobin A1c reduction is likely due to selec
151            The only significant predictor of glycated hemoglobin A1c reduction was waist circumferenc
152  are well-adhered to, whereas guidelines for glycated hemoglobin A1c testing for type 2 diabetes mell
153 s largely determined by assessment of HbA1c (glycated hemoglobin A1c) levels, which poorly reflects d
154                                 Weight, BMI, glycated hemoglobin A1c, fasting glucose, and insulin we
155  We collected data on histories of patients' glycated hemoglobin A1c, hypertension, hyperlipidemia, s
156 tions, pain (numeric rating scale), level of glycated hemoglobin A1c, level of C-reactive protein, bo
157 s there exists a direct relationship between glycated hemoglobin and cardiovascular disease (CVD), cl
158 y is to investigate the relationship between glycated hemoglobin and circulating levels of interleuki
159  All participants were screened for DM using glycated hemoglobin and fasting plasma glucose at TB tre
160 ic red blood cells correlates with levels of glycated hemoglobin and that the mechanical properties o
161 of Germany or Austria), body mass index, and glycated hemoglobin as covariates were used to account f
162 iculated thrombocytosis that correlates with glycated hemoglobin as well as increased plasma S100A8/A
163 ing age, absence of epiretinal membrane, and glycated hemoglobin below 9 as predictive of DR ultra-re
164 ears after baseline on the basis of either a glycated hemoglobin concentration of at least 6.5% or us
165 studies that have demonstrated reductions in glycated hemoglobin concentration.
166 ng an extended interval of separation of the glycated hemoglobin curves (hazard ratio, 0.83; 95% CI,
167 ed 1791 participants), the separation of the glycated hemoglobin curves between the intensive-therapy
168 nly during the prolonged period in which the glycated hemoglobin curves were separated.
169 s improved, for example, fasting glucose and glycated hemoglobin decreased from 6.1 to 5.4 mmol/mol a
170                                              Glycated hemoglobin decreased significantly from 8.2 +/-
171 h both incidence of T2D and increased plasma glycated hemoglobin in individuals without T2D, providin
172 and with low HDL concentrations and elevated glycated hemoglobin in obese and diabetic patients.CCK r
173 closures do not affect health in general, 2) glycated hemoglobin is insensitive to local foreclosure
174 52.5 vs 54.8 years), BMI (45.1 vs 42.6), and glycated hemoglobin level (7.1% vs 7.1%).
175 wer; 95% CI, 24 to 51; P<0.001) and the mean glycated hemoglobin level (difference, -0.3%; 95% CI, -0
176 , 1.02; 95% CI, 1.01-1.04) and knowing one's glycated hemoglobin level (odds ratio, 2.00; 95% CI, 1.3
177 , sulfonylurea) with stable body weight, and glycated hemoglobin level 7.0% to 10.0%.
178 uration of diabetes 34.6 years, and the mean glycated hemoglobin level 8.2%.
179 nd point was the change from baseline in the glycated hemoglobin level after 26 weeks.
180              The mean adjusted difference in glycated hemoglobin level after 6 months was -0.33 perce
181 index greater than the 85th percentile and a glycated hemoglobin level between 7.0 and 11.0% if the p
182      The primary end point was the change in glycated hemoglobin level from baseline to week 26.
183  of the primary efficacy end point, the mean glycated hemoglobin level had decreased by 0.64 percenta
184                  The primary end point was a glycated hemoglobin level lower than 7.0% at week 24, wi
185 n, the proportion of patients who achieved a glycated hemoglobin level lower than 7.0% with no severe
186 with 4.3% over 3 years among patients with a glycated hemoglobin level of 10%.
187  was 1.0% over 5 years among patients with a glycated hemoglobin level of 6%, as compared with 4.3% o
188                    The primary outcome was a glycated hemoglobin level of 6.0% or less with or withou
189 diagnosis of obstructive sleep apnea, with a glycated hemoglobin level of 6.5-8.5%, and an oxygen des
190 ients younger than 55 years of age who had a glycated hemoglobin level of 6.9% or less (</=52 mmol pe
191 ng those younger than 55 years of age with a glycated hemoglobin level of 6.9% or less, as compared w
192 l study, patients with type 1 diabetes and a glycated hemoglobin level of 6.9% or lower had a risk of
193 ed hemoglobin level score [calculated as the glycated hemoglobin level plus 4 times the insulin dose]
194  of the patients was 14 to 71 years, and the glycated hemoglobin level ranged from 5.4 to 10.6%.
195 e patient's current state of retinopathy and glycated hemoglobin level reduced the frequency of eye e
196 esponse (defined as an insulin dose-adjusted glycated hemoglobin level score [calculated as the glyca
197 r mean percentage reduction from baseline in glycated hemoglobin level than did patients who received
198 e estimated mean change from baseline in the glycated hemoglobin level was -1.33 percentage points in
199                                The mean (SD) glycated hemoglobin level was 7.4% (0.5%).
200                       At 24 months, the mean glycated hemoglobin level was 7.5+/-1.2% in each group,
201 similar in the two groups; the mean baseline glycated hemoglobin level was 8.09% in the icodec group
202 iabetes was 16.4 years, and the mean (+/-SD) glycated hemoglobin level was 8.4+/-1.7%; 83.9% of the p
203 s 16.1 years (range, 2-36 years), and median glycated hemoglobin level was 8.8% (IQR, 7.4%-10%).
204 s was 49+/-8 years, 66% were women, the mean glycated hemoglobin level was 9.2+/-1.5%, and the mean B
205 hich patients with type 2 diabetes mellitus (glycated hemoglobin level, >=7%), chronic kidney disease
206 ucose load, 140 to 199 mg per deciliter; and glycated hemoglobin level, 5.7 to 6.4%) and no diagnosti
207 type 2 diabetes was inadequately controlled (glycated hemoglobin level, 7.0 to 9.5%) while taking met
208 etes had poor glycemic control (mean [+/-SD] glycated hemoglobin level, 9.0+/-2.4%), and the rates of
209 s >180 mg per deciliter, mean glucose level, glycated hemoglobin level, and percentage of time that t
210 itional end points included insulin use, the glycated hemoglobin level, the number of hypoglycemic ev
211  points included the change from baseline in glycated hemoglobin level, weight, systolic blood pressu
212  C-peptide levels, insulin requirements, and glycated hemoglobin level.
213 lycemia, and, in adults, resulted in a lower glycated hemoglobin level.
214 1.13; P = .003) for each unit of increase in glycated hemoglobin level.
215 counting for glycemic control in the form of glycated hemoglobin level.
216 significant difference between the groups in glycated hemoglobin level.
217 t did not continue after equalization of the glycated hemoglobin levels (hazard ratio, 1.26; 95% CI,
218 f 3.0 years, there was a small difference in glycated hemoglobin levels (least-squares mean differenc
219      The phenomenon of a discrepancy between glycated hemoglobin levels and other indicators of avera
220 loop group and 23 to the control group); the glycated hemoglobin levels at baseline ranged from 5.7 t
221                            The difference in glycated hemoglobin levels between the intensive-therapy
222  previously been found to reduce glucose and glycated hemoglobin levels in humans.
223 e fully attenuated after adjustment for mean glycated hemoglobin levels over the entire follow-up.
224                                              Glycated hemoglobin levels were lower with pump therapy
225                  Secondary outcomes included glycated hemoglobin levels, insulin dose, and body mass
226 men, C-reactive protein, lipoprotein(a), and glycated hemoglobin levels.
227 progression was also closely related to mean glycated hemoglobin levels.
228 al history, antidiabetic medication use, and glycated hemoglobin levels.
229 c blood pressures, and high triglyceride and glycated hemoglobin levels.
230 ere among metabolic surgery patients (higher glycated hemoglobin levels; greater percentage of insuli
231 amer-based microfluidic system for automatic glycated hemoglobin measurements.
232 /m(2)) of 39.2 (95% CI: 35.2, 43.3) and mean glycated hemoglobin of 5.3% (95% CI: 4.9%, 5.6%), were s
233                 Adults with type 2 diabetes, glycated hemoglobin of 6.5% to 8.5%, and elevated cardio
234                            We used admission glycated hemoglobin to estimate premorbid baseline blood
235 lar disease (CVD), clinical trials targeting glycated hemoglobin to near-normal levels using intensiv
236 ological factors assessed, insulin index and glycated hemoglobin values explained 15% and 16% of the
237  mm Hg; -9.0, -2.7 mm Hg) diets, and reduced glycated hemoglobin with the Mediterranean diet (-0.8 mm
238 rs are modestly effective in reducing HbA1c (glycated hemoglobin) ( approximately 0.5%) and while the
239 inal pro-B-type natriuretic peptide), HbA1C (glycated hemoglobin), and systolic blood pressure were o
240                   Thirteen people with T2DM (glycated hemoglobin, 6.9+/-1.0%) and 12 age-matched heal
241 an age, 64.0 years; 2414 [39.9%] women; mean glycated hemoglobin, 7.2%; median duration of diabetes,
242 ysis (OCT) over a 4-y period, independent of glycated hemoglobin, age, and sex.
243 syndrome (69.2 vs. 51.9%), fast glycemia and glycated hemoglobin, albuminuria, triglycerides and uric
244  cholesterol, triglycerides, blood pressure, glycated hemoglobin, and fasting glucose and report the
245 ar reductions in insulin, insulin C-peptide, glycated hemoglobin, and homeostasis model assessment of
246 ymptoms and changes in the levels of lipids, glycated hemoglobin, and prolactin were similar in the t
247 e intervention group had significantly lower glycated hemoglobin, fasting plasma glucose, plaque inde
248 cant between-group differences were found in glycated hemoglobin, HDL-cholesterol, or triglyceride co
249 justment for metabolic biomarkers, including glycated hemoglobin, high-density lipoprotein cholestero
250 rly high-density lipoprotein cholesterol and glycated hemoglobin, led to a greater degree of attenuat
251                          For every 1% higher glycated hemoglobin, left ventricular mass was higher by
252 cident CKD included BP >140/90 mm Hg, higher glycated hemoglobin, lower baseline eGFR, and higher bas
253 ptical coherence tomography, blood pressure, glycated hemoglobin, medications, and changes in such pa
254  positive correlation of chemerin with IL-6, glycated hemoglobin, sampled-site clinical attachment le
255 adjusting for age, waist-to-hip ratio (WHR), glycated hemoglobin, smoking, education, and grip streng
256 e foreclosure rate per census-block group on glycated hemoglobin.
257 block group in the prior year and changes in glycated hemoglobin.
258 een the study groups were seen for change in glycated hemoglobin.
259 s-related traits such as fasting glucose and glycated hemoglobin.
260 f signals while varying concentration of the glycated hemoglobin.
261 corresponding reduction in the percentage of glycated-hemoglobin to levels similar to lean, healthy m
262 d by measurement of the cutoff ratio between glycated hemoglobins (HbA1c) and total hemoglobin (Hb),
263 ntified peptides in the enriched fraction as glycated, high sensitivity for detection of glycated pep
264 1c levels did not have the highest levels of glycated HSA.
265  of glycated peptides quantified in in vitro glycated human plasma increased more than 3-fold using t
266 and modified DNA aptamers specifically bound glycated human serum albumin (GHSA), which is an interme
267                                          The glycated hydrolysates showed reduced fluorescence advanc
268                    We chemically synthesized glycated IAPP (AGE-IAPP) to mimic the consequence of thi
269                  We also noted a decrease in glycated lysine residues in collagen, indicating that th
270  vector machine, to predict glycated and non-glycated lysine residues using structural properties of
271 edictor manages to classify glycated and non-glycated lysine residues with promising results consiste
272 xtracted containing 235 glycated and 303 non-glycated lysine residues.
273 trast, only 168 polypeptides contained early glycated lysines, which did not resemble the sites of ad
274 YajL could repair methylglyoxal- and glyoxal-glycated nucleotides and nucleic acids.
275 e 2D-LC-HCD-MS/MS platform for comprehensive glycated peptide quantification.
276 line platform to human plasma identified 376 glycated peptides from 10 mug of protein digests.
277                                The number of glycated peptides quantified in in vitro glycated human
278  glycated, high sensitivity for detection of glycated peptides with LOD and LOQ at 1.2 and 2.4 pg, re
279 ility with interday CVs < 20% for 80% of the glycated peptides.
280 ool for identification and quantification of glycated peptides.
281 rabidopsis thaliana The absence of the early glycated precursors of the AGE-modified protein residues
282 oducible online 2D platform is promising for glycated protein analysis of complex clinical samples.
283 n in exposed splenocyte, were evident in the glycated protein treated mice as compared to its native
284       Thereafter, allergic behaviour of this glycated protein was compared with its native form, usin
285                 The reduced allergenicity of glycated protein was observed as lesser allergic phenoty
286 yl amino acid from glycated albumin, another glycated protein, whereas glucose is naturally present a
287                                              Glycated proteins are emerging as good indicators for di
288                     Although the patterns of glycated proteins were only minimally influenced by plan
289 hree aging-specific and eight differentially glycated proteins, four of which were modified in cataly
290                                              Glycated proteins, such as glycated hemoglobin (HbA1c) o
291        However, the platform for analysis of glycated proteome has been relatively less well establis
292 -related changes in the Arabidopsis thaliana glycated proteome, including the proteins affected and s
293  to 12 glucose moieties were observed, while glycated species containing 6 and 8 glucose moieties wer
294 rameters with the levels of glycosylated and glycated species in a series of small scale experiments,
295 proteoforms, including positional isomers of glycated species in a single run.
296                                          The glycated species of cytochrome C, lysozyme, and beta-cas
297     Cytochrome C had multiple charges in non-glycated state, primarily changing from +13 to +17 posit
298          In its absence, NRF2 is extensively glycated, unstable, and defective at binding to small MA
299 ocess (ISP) was hydrolysed with Alcalase and glycated with glucosamine (GlcN) at moderate temperature
300 rmal stability and antioxidative capacity of glycated WP were increased, especially in the presence o

 
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