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1 holesterol, high C-reactive protein and high glycated hemoglobin).
2 e foreclosure rate per census-block group on glycated hemoglobin.
3 block group in the prior year and changes in glycated hemoglobin.
4 een the study groups were seen for change in glycated hemoglobin.
5 tion to determine levels of CRP, lipids, and glycated hemoglobin.
6 ng glucose and postload glucose but not with glycated hemoglobin.
7 in cholesterol, systolic blood pressure, and glycated hemoglobin.
8 ith adjustment for all covariates as well as glycated hemoglobin.
9 s-related traits such as fasting glucose and glycated hemoglobin.
10 f signals while varying concentration of the glycated hemoglobin.
11 o five subfractions, four of which contained glycated hemoglobins.
14 d 10.2 percentage points, respectively), and glycated hemoglobin (10.1 percentage points and 9.4 perc
15 of 1.42 (0.69-2.92) and 2.91 (1.19-7.11) for glycated hemoglobin 5.7-<6.5% and >/=6.5%, respectively,
16 of 1.12 (0.94-1.34) and 1.39 (1.04-1.85) for glycated hemoglobin 5.7-6.4% and >/=6.5%, respectively,
17 omol/L [IQR, 72-89 micromol/L], P = .61) and glycated hemoglobin (5.9% [IQR, 5.6%-6.1%] vs 5.9% [IQR,
18 97 individuals >=40 and <=80 years old with glycated hemoglobin 6.5% to 10.0%, known coronary artery
19 ss index (47.6+/-9.3 to 36.7+/-7; P<0.0001), glycated hemoglobin (6.7+/-1.5 to 5.8+/-0.6; P<0.0001),
21 y; median (IQR), 10 (5-9) y of T2D duration; glycated hemoglobin 7.0% +/- 0.8%; body mass index (in k
22 an age, 64.0 years; 2414 [39.9%] women; mean glycated hemoglobin, 7.2%; median duration of diabetes,
24 fication by sex, history of ischemic stroke, glycated hemoglobin A(1c), body mass index, blood pressu
25 llected at each visit for the assay of serum glycated hemoglobin A1c (A1c), hsCRP, d-8-iso, MMP-2, an
26 g plasma glucose >/=200 mg/dl (11.1 mmol/l), glycated hemoglobin A1c (HbA1c) >6.5%, self-reported phy
27 nfidence intervals (CIs) were calculated for glycated hemoglobin A1c (HbA1c), fasting plasma glucose
28 se [FCG] level, 2-hour CG [2-hCG] level, and glycated hemoglobin A1c [HbA1c] level) at enrollment, an
30 While adjusting for duration of diabetes, glycated hemoglobin A1c level, and other factors, we fou
33 ciation of baseline waist circumference with glycated hemoglobin A1c reduction is likely due to selec
35 are well-adhered to, whereas guidelines for glycated hemoglobin A1c testing for type 2 diabetes mell
36 s largely determined by assessment of HbA1c (glycated hemoglobin A1c) levels, which poorly reflects d
39 We collected data on histories of patients' glycated hemoglobin A1c, hypertension, hyperlipidemia, s
40 tions, pain (numeric rating scale), level of glycated hemoglobin A1c, level of C-reactive protein, bo
42 sted difference, 1.0 percentage points), and glycated hemoglobin (adjusted difference, 3.4 percentage
44 syndrome (69.2 vs. 51.9%), fast glycemia and glycated hemoglobin, albuminuria, triglycerides and uric
45 s there exists a direct relationship between glycated hemoglobin and cardiovascular disease (CVD), cl
46 y is to investigate the relationship between glycated hemoglobin and circulating levels of interleuki
49 All participants were screened for DM using glycated hemoglobin and fasting plasma glucose at TB tre
50 rvention also produced greater reductions in glycated hemoglobin and greater initial improvements in
51 erum concentrations of metabolic parameters (glycated hemoglobin and low-density lipoprotein), inflam
52 abetes Association diagnostic cut points for glycated hemoglobin and microvascular outcomes (chronic
53 ic red blood cells correlates with levels of glycated hemoglobin and that the mechanical properties o
54 inal pro-B-type natriuretic peptide), HbA1C (glycated hemoglobin), and systolic blood pressure were o
55 cholesterol, triglycerides, blood pressure, glycated hemoglobin, and fasting glucose and report the
56 ar reductions in insulin, insulin C-peptide, glycated hemoglobin, and homeostasis model assessment of
57 measured in urine, with diabetes prevalence, glycated hemoglobin, and insulin resistance in American
58 ymptoms and changes in the levels of lipids, glycated hemoglobin, and prolactin were similar in the t
59 ats maintained in poor glycemic control (PC, glycated hemoglobin approximately 11%) or in good glycem
60 mately 11%) or in good glycemic control (GC, glycated hemoglobin approximately 6%) for 6 months, or i
61 rs are modestly effective in reducing HbA1c (glycated hemoglobin) ( approximately 0.5%) and while the
62 ta add to the evidence supporting the use of glycated hemoglobin as a diagnostic test for diabetes.
63 of Germany or Austria), body mass index, and glycated hemoglobin as covariates were used to account f
64 iculated thrombocytosis that correlates with glycated hemoglobin as well as increased plasma S100A8/A
65 e variation, complicates the clinical use of glycated hemoglobin assays for the diagnosis and managem
67 ing age, absence of epiretinal membrane, and glycated hemoglobin below 9 as predictive of DR ultra-re
68 ears after baseline on the basis of either a glycated hemoglobin concentration of at least 6.5% or us
70 hod for measuring the hemoglobin A1c (HbA1c, glycated hemoglobin) concentration, hemoglobin (Hb) conc
71 moglobin glycation index (HGI), a measure of glycated hemoglobin controlled for blood glucose variati
72 ng an extended interval of separation of the glycated hemoglobin curves (hazard ratio, 0.83; 95% CI,
73 ed 1791 participants), the separation of the glycated hemoglobin curves between the intensive-therapy
75 se of new 2010 American Diabetes Association glycated hemoglobin cut points for the diagnosis of diab
76 s improved, for example, fasting glucose and glycated hemoglobin decreased from 6.1 to 5.4 mmol/mol a
78 liflozin group than in the placebo group for glycated hemoglobin (difference, -0.46 percentage points
79 e intervention group had significantly lower glycated hemoglobin, fasting plasma glucose, plaque inde
81 poor glycemic control (PC, approximately 12% glycated hemoglobin [GHb]) for 3 months followed by in g
84 oad glucose (PG), 2-h postload insulin (PI), glycated hemoglobin (Hb A1c), and homeostasis model asse
87 08) while controlling for age, sex, baseline glycated hemoglobin (HbA(1c)), baseline CMT, baseline VA
88 es diagnosis, serum fasting glucose (FG) and glycated hemoglobin (HbA(1c)), were estimated to be 51-6
90 s: colorectal screening rates; diabetes with glycated hemoglobin (HbA1c level) less than 9.0%; diabet
92 ype 2 diabetes were recruited: subjects with glycated hemoglobin (HbA1c) </=7% and subjects with HbA1
93 ; betaPFOA=1.71 pM; 95% CI: 0.72, 2.71), and glycated hemoglobin (HbA1c) (betaPFOS=0.03%; 95% CI: 0.0
94 ) concentration (-37.0 mg/dL; P < 0.001) and glycated hemoglobin (HbA1c) [-0.97% (-10.6 mmol/mol); P
95 nt increases in hippocampal FC, decreases in glycated hemoglobin (HbA1c) and body fat, and increases
96 We examined the association between baseline glycated hemoglobin (HbA1c) and high-sensitivity cardiac
98 a significant difference in postintervention glycated hemoglobin (HbA1c) between the arms (P = 0.007)
99 ulinemia, decreased C-peptide, and increased glycated hemoglobin (HbA1c) compared with sham-operated
107 lycemia from birth, resulting in an elevated glycated hemoglobin (HbA1c) level that mimics recommende
108 ex, waist circumference, fat percentage, and glycated hemoglobin (HbA1c) level were recorded chairsid
109 n leakage area at month 36 (P = 0.0137), and glycated hemoglobin (HbA1c) levels at month 36 (P = 0.00
110 ed the association between periodontitis and glycated hemoglobin (HbA1c) levels in individuals withou
111 (20-50 U) and metformin (>/=1500 mg/d) with glycated hemoglobin (HbA1c) levels of 7% to 10% and a bo
112 nfluence of periodontal status on changes of glycated hemoglobin (HbA1c) levels of patients with type
113 igated OCT-A parameters with DM duration and glycated hemoglobin (HbA1c) levels were evaluated among
116 2 spectral domain OCT (SD-OCT) tests, and 2 glycated hemoglobin (HbA1c) measures over time with a mi
117 DM prevalence was estimated using laboratory glycated hemoglobin (HbA1c) or fasting plasma glucose in
119 hievement of good glycemic control, of which glycated hemoglobin (HbA1c) remains the standard clinica
121 in or loss and glycemic control (assessed by glycated hemoglobin (HbA1c) values) in patients from the
124 rmined for fasting glucose, fasting insulin, glycated hemoglobin (HbA1c), and homeostasis model asses
125 glucose, C-reactive protein, triglycerides, glycated hemoglobin (HbA1c), and total, low-density lipo
126 tion of high- compared with low-fat dairy on glycated hemoglobin (HbA1c), body weight, and cardiovasc
127 The relationship between delayed GE and glycated hemoglobin (HbA1c), complications of DM, and ga
128 eplacement of other sugars and its effect on glycated hemoglobin (HbA1c), fasting blood glucose, insu
129 agnostic accuracies of random blood glucose, glycated hemoglobin (HBA1c), fructosamine, and Homeostas
130 glycemia (diabetes status, fasting glucose, glycated hemoglobin (HbA1c), fructosamine, glycated albu
131 triacylglycerol (TG), fasting glucose (FG), glycated hemoglobin (HbA1c), insulin resistance (HOMA-IR
132 and at follow-up, was associated with higher glycated hemoglobin (HbA1c), insulin, HOMA-IR, triglycer
133 ent of glycated blood proteins, particularly glycated hemoglobin (HbA1c), is an important diagnostic
135 ed with the diabetes GRS on fasting insulin, glycated hemoglobin (HbA1c), the homeostasis model asses
137 blood pressure (BP), fasting blood glucose, glycated hemoglobin (HbA1c), triglyceride levels, trigly
138 led trials (RCTs) that assessed the outcomes glycated hemoglobin (HbA1c), weight, body mass index (BM
142 body mass index (in kg/m(2)): 34.6 +/- 4.3; glycated hemoglobin (HbA1c): 7.3 +/- 1.1%; duration of d
143 n [n = 136; mean +/- SD age: 12.8 +/- 2.6 y; glycated hemoglobin (HbA1c): 8.1% +/- 1.0%; 69.1% using
144 .7%) was associated with a reduction in mean glycated hemoglobin (HbA1c, -1.3 +/- 1.8%, P < 0.001), f
145 d by measurement of the cutoff ratio between glycated hemoglobins (HbA1c) and total hemoglobin (Hb),
146 ol/L, 2-hr plasma glucose >/=11.1 mmol/L, or glycated hemoglobin [HbA1c] >/=6.5%) was detected in 46%
147 8 of whom had poor glycemic control (average glycated hemoglobin [HbA1c] >/=8% during the year) while
148 tensive glycemic control in type 2 diabetes (glycated hemoglobin [HbA1c] level <7%) is an established
149 cose [RBG], fasting blood glucose [FBG], and glycated hemoglobin [HbA1c]) and survival in all lung tr
150 cant between-group differences were found in glycated hemoglobin, HDL-cholesterol, or triglyceride co
152 justment for metabolic biomarkers, including glycated hemoglobin, high-density lipoprotein cholestero
154 h both incidence of T2D and increased plasma glycated hemoglobin in individuals without T2D, providin
155 and with low HDL concentrations and elevated glycated hemoglobin in obese and diabetic patients.CCK r
158 closures do not affect health in general, 2) glycated hemoglobin is insensitive to local foreclosure
159 rly high-density lipoprotein cholesterol and glycated hemoglobin, led to a greater degree of attenuat
161 dard therapy or aggressive therapy (targets: glycated hemoglobin level <6.0%, low-density lipoprotein
162 val [CI], 0.8 to 15.0) for glycemic control (glycated hemoglobin level <7.0%), 9.4 percentage points
163 ed optimal diabetes care (n = 448) (targets: glycated hemoglobin level <7.0%, low-density lipoprotein
167 wer; 95% CI, 24 to 51; P<0.001) and the mean glycated hemoglobin level (difference, -0.3%; 95% CI, -0
168 , 1.02; 95% CI, 1.01-1.04) and knowing one's glycated hemoglobin level (odds ratio, 2.00; 95% CI, 1.3
169 on of T2DM (P = 0.006), a higher presurgical glycated hemoglobin level (P = 0.019), insulin treatment
174 index greater than the 85th percentile and a glycated hemoglobin level between 7.0 and 11.0% if the p
175 eters) for 84.2% of participants and data on glycated hemoglobin level for 71.3% of participants.
176 os for death from any cause according to the glycated hemoglobin level for patients with diabetes as
178 of the primary efficacy end point, the mean glycated hemoglobin level had decreased by 0.64 percenta
181 n, the proportion of patients who achieved a glycated hemoglobin level lower than 7.0% with no severe
182 mg per deciliter [5.6 mmol per liter] and a glycated hemoglobin level of <6.5% in the absence of pha
184 was 1.0% over 5 years among patients with a glycated hemoglobin level of 6%, as compared with 4.3% o
185 point was the proportion of patients with a glycated hemoglobin level of 6.0% or less 12 months afte
188 MI of 35 or more, a BMI of 40 or more, and a glycated hemoglobin level of 6.5% or more were lower in
189 diagnosis of obstructive sleep apnea, with a glycated hemoglobin level of 6.5-8.5%, and an oxygen des
190 ients younger than 55 years of age who had a glycated hemoglobin level of 6.9% or less (</=52 mmol pe
191 ng those younger than 55 years of age with a glycated hemoglobin level of 6.9% or less, as compared w
192 trols were 2.36 (95% CI, 1.97 to 2.83) for a glycated hemoglobin level of 6.9% or lower (</=52 mmol p
193 l study, patients with type 1 diabetes and a glycated hemoglobin level of 6.9% or lower had a risk of
194 story of at least 5 years of diabetes, and a glycated hemoglobin level of 7.0% or more were randomly
195 ol improved in all three groups, with a mean glycated hemoglobin level of 7.5+/-1.8% in the medical-t
196 e was loss of glycemic control, defined as a glycated hemoglobin level of at least 8% for 6 months or
197 , but intensive glucose control, targeting a glycated hemoglobin level of less than 6.5%, did not.
198 t a dose of 1000 mg twice daily) to attain a glycated hemoglobin level of less than 8% and were rando
199 ed hemoglobin level score [calculated as the glycated hemoglobin level plus 4 times the insulin dose]
201 e patient's current state of retinopathy and glycated hemoglobin level reduced the frequency of eye e
202 esponse (defined as an insulin dose-adjusted glycated hemoglobin level score [calculated as the glyca
203 r mean percentage reduction from baseline in glycated hemoglobin level than did patients who received
204 e estimated mean change from baseline in the glycated hemoglobin level was -1.33 percentage points in
207 similar in the two groups; the mean baseline glycated hemoglobin level was 8.09% in the icodec group
208 iabetes was 16.4 years, and the mean (+/-SD) glycated hemoglobin level was 8.4+/-1.7%; 83.9% of the p
209 s 16.1 years (range, 2-36 years), and median glycated hemoglobin level was 8.8% (IQR, 7.4%-10%).
210 s was 49+/-8 years, 66% were women, the mean glycated hemoglobin level was 9.2+/-1.5%, and the mean B
212 /-8 years, 68% were women, the mean baseline glycated hemoglobin level was 9.3+/-1.5%, and the mean b
213 hich patients with type 2 diabetes mellitus (glycated hemoglobin level, >=7%), chronic kidney disease
214 ucose load, 140 to 199 mg per deciliter; and glycated hemoglobin level, 5.7 to 6.4%) and no diagnosti
215 type 2 diabetes was inadequately controlled (glycated hemoglobin level, 7.0 to 9.5%) while taking met
216 etes had poor glycemic control (mean [+/-SD] glycated hemoglobin level, 9.0+/-2.4%), and the rates of
217 s >180 mg per deciliter, mean glucose level, glycated hemoglobin level, and percentage of time that t
220 itional end points included insulin use, the glycated hemoglobin level, the number of hypoglycemic ev
221 points included the change from baseline in glycated hemoglobin level, weight, systolic blood pressu
230 greater overall 12-month improvement across glycated hemoglobin levels (difference, 0.58%), LDL chol
231 t did not continue after equalization of the glycated hemoglobin levels (hazard ratio, 1.26; 95% CI,
232 f 3.0 years, there was a small difference in glycated hemoglobin levels (least-squares mean differenc
233 The phenomenon of a discrepancy between glycated hemoglobin levels and other indicators of avera
234 loop group and 23 to the control group); the glycated hemoglobin levels at baseline ranged from 5.7 t
236 ween-group differences in blood pressure and glycated hemoglobin levels during the trial were no long
237 of glucose levels and 10,208 measurements of glycated hemoglobin levels from 2067 participants withou
239 clines in cortisol levels, blood glucose and glycated hemoglobin levels increased soon after treatmen
241 we observed no significant effect on average glycated hemoglobin levels or on the percentage of parti
242 e fully attenuated after adjustment for mean glycated hemoglobin levels over the entire follow-up.
243 sults show that interindividual variation in glycated hemoglobin levels was evident in diabetes patie
247 the greatest degree of improvement (average glycated hemoglobin levels, 7.69+/-0.57% in the medical-
248 erapy resulted in significant improvement in glycated hemoglobin levels, as compared with injection t
250 Interindividual and ethnic variation in glycated hemoglobin levels, unrelated to blood glucose v
256 ere among metabolic surgery patients (higher glycated hemoglobin levels; greater percentage of insuli
257 es included blood-pressure, cholesterol, and glycated hemoglobin levels; screening for depression; me
258 cident CKD included BP >140/90 mm Hg, higher glycated hemoglobin, lower baseline eGFR, and higher bas
259 ith type 2 diabetes in good glucose control (glycated hemoglobin < 7.5%) before and after 7 d of a VL
263 ptical coherence tomography, blood pressure, glycated hemoglobin, medications, and changes in such pa
264 /m(2)) of 39.2 (95% CI: 35.2, 43.3) and mean glycated hemoglobin of 5.3% (95% CI: 4.9%, 5.6%), were s
269 the intention-to-treat population, the mean glycated hemoglobin profile improved in the intervention
270 ceride/HDL-C ratio (r = 0.16, p = 0.03), and glycated hemoglobin (r = 0.16, p = 0.03) with pioglitazo
272 o a greater improvement in glycemic control [glycated hemoglobin reductions of -0.12% (P = 0.04), -0.
273 positive correlation of chemerin with IL-6, glycated hemoglobin, sampled-site clinical attachment le
274 adjusting for age, waist-to-hip ratio (WHR), glycated hemoglobin, smoking, education, and grip streng
275 The proportion of patients who reached the glycated hemoglobin target (<7%) was greater in the pump
276 x, diabetes, waist/hip ratios, and levels of glycated hemoglobin, the NAFLD activity score was associ
278 lar disease (CVD), clinical trials targeting glycated hemoglobin to near-normal levels using intensiv
279 corresponding reduction in the percentage of glycated-hemoglobin to levels similar to lean, healthy m
280 rotein cholesterol (HDL-C) and reductions in glycated hemoglobin, triglycerides, and C-reactive prote
281 meters controlling for sex, body mass index, glycated hemoglobin, use of oral hypoglycemic drugs, and
283 mg per deciliter (2.6 mmol per liter), and a glycated hemoglobin value of 9.0% or lower, according to
284 ological factors assessed, insulin index and glycated hemoglobin values explained 15% and 16% of the
285 periodontitis presented higher glycemia and glycated hemoglobin values in contrast to patients with
289 mination showed significant improvement when glycated hemoglobin was added to models including fastin
294 nity-based population of nondiabetic adults, glycated hemoglobin was similarly associated with a risk
295 diabetes at baseline, clinical categories of glycated hemoglobin were associated with risk of chronic
296 rular basement membrane and higher levels of glycated hemoglobin were independent predictors of progr
298 ignificant thresholds in the associations of glycated hemoglobin with kidney disease risk or retinopa
299 mm Hg; -9.0, -2.7 mm Hg) diets, and reduced glycated hemoglobin with the Mediterranean diet (-0.8 mm