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1 sting or reducing anginal episodes or use of glyceryl trinitrate.
2 ere was no significant change in response to glyceryl trinitrate.
3 onduit artery selectivity similar to that of glyceryl trinitrate (0.013-4.4 nmol/min) over resistance
4 achidonic acid: 0.2, 2, and 20 nmol/min; and glyceryl trinitrate 1, 2, and 4 pmol/min) were construct
5 f albuterol (5 microg/min, DeltaRI(ALB)) and glyceryl trinitrate (5 microg/min, DeltaRI(GTN)).
6  The metabolism of nitrovasodilators such as glyceryl trinitrate and nitroprusside provides the activ
7 P=0.045) after adjusting for the response to glyceryl trinitrate and was independent of conventional
8 , D-S-nitrosocysteine, S-nitrosoglutathione, glyceryl trinitrate, and sodium nitroprusside produced m
9     In addition, exogenous acetylcholine and glyceryl trinitrate both increase arterial distensibilit
10 ependent dilation, EDD) and after sublingual glyceryl trinitrate (causing endothelium-independent dil
11    There was no significant change in FMD or glyceryl trinitrate during the placebo phase.
12 .01% (P=0.036) with no change in response to glyceryl trinitrate (endothelial-independent dilatation)
13 ers of each sex, we assessed FMD response to glyceryl trinitrate (GTN) at baseline and at 8 hours and
14                                 The NO donor glyceryl trinitrate (GTN) caused an increase in cGMP in
15 donor drugs isosorbide mononitrate (ISMN) or glyceryl trinitrate (GTN) for stable angina.
16                  Nitric oxide donors such as glyceryl trinitrate (GTN) have been shown to cause a rev
17 n each group, a migraine was induced using a glyceryl trinitrate (GTN) infusion.
18                                  Infusion of glyceryl trinitrate (GTN) into patients with migraine pr
19     Therefore, we investigated the effect of glyceryl trinitrate (GTN) on the efficacy of cell engraf
20                                 The NO donor glyceryl trinitrate (GTN) provokes delayed migraine atta
21                   Albuterol (salbutamol) and glyceryl trinitrate (GTN) were administered locally by b
22                               The effects of glyceryl trinitrate (GTN), amlodipine, and atenolol were
23                                        Using glyceryl trinitrate (GTN), an agonist of nitric oxide (N
24 ing pacing and after intragraft injection of glyceryl trinitrate (GTN).
25 h after infusing the NO donor nitroglycerin [glyceryl trinitrate (GTN)] to migraineurs.
26 ent) and response to 50 microg of sublingual glyceryl trinitrate (GTN, endothelium independent) were
27 abolism of the cornerstone nitrovasodilator, glyceryl trinitrate (GTN; 0.1-100 mg/kg), in the rat in
28 (BK) and the endothelium-independent dilator glyceryl-trinitrate (GTN).
29 mediated dilatation [FMD]) and -independent (glyceryl trinitrate [GTN]) changes in brachial artery ar
30 yl dinitrate and nitrite from nitroglycerin (glyceryl trinitrate [GTN]) within mitochondria, leading
31 ular mechanisms through which nitroglycerin (glyceryl trinitrate, GTN) elicits nitric oxide (NO)-base
32 atalyzes the bioactivation of nitroglycerin (glyceryl trinitrate, GTN) in blood vessels, resulting in
33                               Nitroglycerin (glyceryl trinitrate, GTN), originally manufactured by Al
34 ry flow-mediated (endothelium-dependent) and glyceryl trinitrate-induced (endothelium-independent) di
35                                 In contrast, glyceryl trinitrate-induced dilatation was similar in th
36 15) and lower FMD percentage (P = 0.026) and glyceryl trinitrate-induced dilation (P = 0.012) than he
37  pattern was identical in migraineurs during glyceryl trinitrate-induced migraine attacks as well as
38 ix control subjects at rest and during acute glyceryl trinitrate-induced migraine attacks using posit
39 ins tight for dihydroergotamine during acute glyceryl trinitrate-induced migraine attacks.
40                                      We used glyceryl trinitrate (nitroglycerin) to trigger premonito
41 thysmography with acetylcholine, bradykinin, glyceryl trinitrate, norepinephrine, and l-NG-monomethyl
42    Sublingual administration of the NO donor glyceryl trinitrate normalized platelet VASP phosphoryla
43 harged on the same day with diclofenac, 0.2% glyceryl-trinitrate ointment, lactulose, a telephone num
44 rently available nitric oxide donors such as glyceryl trinitrate or isosorbide mononitrate could be u
45 educed responses to bradykinin (P=0.005) and glyceryl trinitrate (P=0.006).
46  been short-term; a new topical, easy-to-use glyceryl trinitrate preparation has been shown to improv
47 inistration of the nitric oxide (NO) mimetic glyceryl trinitrate prevented altered uteroplacental per
48                Infusion of acetylcholine and glyceryl trinitrate reduced PWV significantly, by 6+/-4%
49                         In in vitro studies, glyceryl trinitrate, sodium nitroprusside, and 3'-morpho
50 ts, intravenous infusion of three NO donors, glyceryl trinitrate, sodium nitroprusside, or 3'-morphol
51 pothalamus, seen in the premonitory phase of glyceryl trinitrate-triggered migraine attacks can expla
52                      Responses to sublingual glyceryl trinitrate were unchanged.