ライフサイエンスコーパス: glycine receptor

1 logous pair of cysteines on the human alpha1 glycine receptor.

2 ysis of a novel activation mechanism for the glycine receptor.

3 glutamate-gated Cl(-) channel and the alpha1 glycine receptor.

4 aracterizing high-affinity modulators of the glycine receptor.

5 280M, induced spontaneous activity in alpha1 glycine receptors.

6 positive allosteric modulators of inhibitory glycine receptors.

7 eby enabling complex formation of functional glycine receptors.

8 entify the stoichiometry of GluK2 and alpha1 glycine receptors.

9 e phyla but are closely related to mammalian glycine receptors.

10 ed that TXA and EACA inhibit the activity of glycine receptors.

11 metabotropic GABA(B) and ionotropic GABA(A)/glycine receptors.

12 se measured in the CSF of patients inhibited glycine receptors.

13 ors and decreases this probability acting at glycine receptors.

14 addition of GABA, a weak partial agonist of glycine receptors.

15 motif prevented PKC modulation of wild-type glycine receptors.

16 yric acid (GABA(A)), serotonin (5-HT(3)) and glycine receptors.

17 suggesting they were mediated by GABAa/c and glycine receptors.

18 ined phorbol ester modulation of GABA(A) and glycine receptors.

19 y influence from CVLM to RVLM is mediated by glycine receptors.

20 e A (GABA(A)), serotonin type 3 (5-HT3), and glycine receptors.

21 d significantly more charge than GABA(A) and glycine receptors.

22 spartate modulation of native or recombinant glycine receptors.

23 nisms include direct antagonistic actions on glycine receptors.

24 on, such as ring-like structures composed of glycine receptors.

25 ition are mediated by heteromeric alpha/beta glycine receptors.

26 laevis oocytes expressing recombinant human glycine receptors.

27 receptor activation in homo- and heteromeric glycine receptors.

28 ed by the activation of GABA, glutamate, and glycine receptors.

29 teins, and gamma-aminobutyric acid (GABA) or glycine receptors.

30 ge in biophysical properties of postsynaptic glycine receptors.

31 of a low-affinity competitive antagonist at glycine receptors [2-(3-carboxypropyl)-3-amino-6-(4-meth

32 ypoxia alters the expression of the GABA and glycine receptors; 2) inhibitory amino acids change the

33 bitory contributions of GABA(A), GABA(C) and glycine receptors across the BC pathways.

34 e cerebral cortex does not appear to require glycine receptor activity for proper development, as Glr

35 This glycine receptor activity inhibits an activity-dependent

36 In the developing retina, inhibition of glycine receptor activity prevents proper rod photorecep

37 uroactive steroid 5alpha-THDOC and classical glycine receptor agonists beta-alanine and taurine direc

38 using a mixture comprising GABAA, GABAB, and glycine receptor agonists caused an immediate and tempor

39 gonist SR-95531 (gabazine) is known to block glycine receptors, albeit with low affinity.

40 The favourable properties of the glycine receptor allowed the first detection of a confor

41 Recently, glycine receptor alpha 1 antibodies have been described

42 Glycine receptor alpha 1 antibodies were found in serum

43 with rigidity and myoclonus associated with glycine receptor alpha 1 antibodies, a potentially sever

44 e generated mice with a targeted deletion of glycine receptor alpha 2 (Glra2).

45 During development, glycine receptor alpha 2 (GlyRalpha2) is expressed in th

46 put analysis showed glycineric synapses with glycine receptor alpha1 expression between AII cells and

47 in-oligodendrocyte glycoprotein (MOG) or the glycine receptor alpha1 subunit (GlyR) is unclear.

48 an disease mutation in the M2-M3 loop of the glycine receptor alpha1 subunit (K276E) using direct fit

49 t the intracellular domain of the inhibitory glycine receptor alpha1 subunit contributes to trafficki

50 mutations, and large deletions in the human glycine receptor alpha1 subunit gene (GLRA1) are the maj

51 Thus, the proline-rich stretch from the glycine receptor alpha1 subunit represents a multifuncti

52 henotype, has a point mutation (A52S) in the glycine receptor alpha1 subunit.

53 Glycine receptor alpha1-IgG aids identification of autoi

54 ved from the transmembrane M2 segment of the glycine receptor alpha1-subunit (M2GlyR) have been desig

55 Glycine receptor alpha1-transfected cells to test serum

56 co-localizing with monoclonal antibodies to glycine receptor-alpha1.

57 ; 3alphaCOOH5betaP) on receptors composed of glycine receptor alpha3 subunits, expressed in Xenopus o

58 present a 3.0 A X-ray structure of the human glycine receptor-alpha3 homopentamer in complex with a h

59 omain and the transmembrane domain of alpha1 glycine receptor (alpha7-GlyR).

60 d bipolar cell axon terminals where GABA and glycine receptors also mediate light-evoked inhibition.

61 es are directed at inhibition of GABA(A) and glycine receptors, although GABA-independent effects hav

62 mains (TM23) of the alpha-1 subunit of human glycine receptor and its truncated form, both with the w

63 s mentioned above, and other targets such as glycine receptors and mediators of neurotransmitter rele

64 prises a key energetic pathway in activating glycine receptors and other pLGICs.

65 ing the established activation mechanism for glycine receptors and our measurements of SR-95531 bindi

66 lly exclude calcium-gated chloride channels, glycine receptors and the chloride current associated wi

67 pressing GAD65, GAD67, alpha1-subunit of the glycine receptor, and a repertoire of known cell surface

68 1 and contactin-associated protein-like 2), glycine receptor, and gamma-aminobutyric acid-A receptor

69 ) between this pair of residues misfolds the glycine receptor, and in consequence, the protein is ret

70 es, alpha1, which forms most of the synaptic glycine receptors, and alpha3.

71 e actions of glycine at strychnine-sensitive glycine receptors, and therefore GlyT1 antagonists also

72 cle nicotinic acetylcholine receptor and the glycine receptor-and find that the open-shut reaction is

73 The glycine receptor antagonist strychnine (4 microM) and th

74 Microinjection of the glycine receptor antagonist strychnine (4 mm, 100 nL) in

75 The glycine receptor antagonist strychnine also prevented Et

76 BAa/c receptor antagonist picrotoxin and the glycine receptor antagonist strychnine did not affect ch

77 tor antagonists AP5 and CGP78608 but not the glycine receptor antagonist strychnine inhibited the CBF

78 eviously been shown to be a weak GABA(A) and glycine receptor antagonist.

79 receptors are inhibited, and is blocked by a glycine receptor antagonist.

80 were identified in 12 patients (11%), mainly glycine receptor antibodies (9 cases), which predominate

81 The glycine receptor antibodies activated complement on glyc

82 Glycine receptor antibodies are strongly associated with

83 Glycine receptor antibodies but not other cell surface a

84 The presence of glycine receptor antibodies did not correlate with any s

85 The clinical associations of glycine receptor antibodies have not yet been described

86 d levels; there was intrathecal synthesis of glycine receptor antibodies in each of the six pairs ava

87 Glycine receptor antibodies occur frequently in P-OMS wi

88 The presence of glycine receptor antibodies should help to identify a di

89 r; P = .02); however, a similar frequency of glycine receptor antibodies was found in patients with l

90 sin, GABA(A)-receptor-associated protein, or glycine receptor antibodies), neuromyotonia (Caspr2 anti

91 The 3 patients with glycine receptor antibody all improved with immunotherap

92 Serum glycine receptor antibody endpoint titres ranged from 1:

93 7 nmol/L; normal value, </= 0.02 nmol/L) and glycine receptor antibody in 3 cases.

94 Ten glycine receptor antibody positive samples were also ide

95 Glycine receptors are chloride-permeable, ligand-gated i

96 is known regarding the mechanism(s) by which glycine receptors are endocytosed.

97 man immunoglobulin G (IgG) autoantibodies to glycine receptors are found in a substantial proportion

98 Glycine receptors are ligand-gated chloride channels tha

99 type 3, gamma-amminobutyric acid type A, and glycine receptors are major players of human neuronal co

100 Inhibitory glycine receptors are pentameric ligand-gated ion channe

101 both the channel activity of NMDARs and the glycine receptors are pre-inhibited.

102 ration when their GABA(A), glutamate, and/or glycine receptors are stimulated, use glutamate and GABA

103 Glycine receptors are, in several ways, the member of th

104 rites that express the alpha3 subunit of the glycine receptor, as well as a subclass of unidentified

105 so identified glycine, acting via alpha-cell glycine receptors, as the predominant amino acid stimula

106 rnalization and lysosomal degradation of the glycine receptors at 37 degrees C.

107 ine and produces near-complete inhibition of glycine receptors at concentrations commonly employed to

108 We conclude that human glycine receptor autoantibodies disrupt glycinergic neur

109 Demonstrating a pathogenic role of glycine receptor autoantibodies would help justify the u

110 and myoclonus or stiff person syndrome, and glycine receptor autoantibodies, were observed to disrup

111 ons are also found in the genes encoding the glycine receptor beta subunit (GLRB) and the presynaptic

112 Thus, the spastic allele of the murine glycine receptor beta subunit gene is a two-hit mutation

113 t (LINE1) retrotransposon in intron 6 of the glycine receptor beta subunit gene, Glrb(spa).

114 circuit activity was induced using GABA and glycine receptor blockers, which produced three rates of

115 he ON pathway and required gap junctions and glycine receptors but not ionotropic glutamate or GABA(A

116 Loss-of-function mutations in human glycine receptors cause hyperekplexia, a rare inherited

117 Since reduced function of glycine receptors causes seizures, we hypothesized that

118 binantly expressed homomeric and heteromeric glycine receptor channels, including their splice varian

119 ls a physio-molecular signature of homomeric glycine receptor channels, which provides unprecedented

120 Taurine, an agonist of glycine receptor chloride (GlyR Cl(-)) channels, was fou

121 it prevents surface expression of the alpha1 glycine receptor chloride channel.

122 glutamate, AP5, and NMDA positively modulate glycine receptor currents.

123 cytosis and also prevented PMA inhibition of glycine receptor currents.

124 The glycine receptor-deficient mutant mouse spastic carries

125 ade of GABA(A) receptors (GABA(A) Rs) and/or glycine receptors demonstrated no GABAergic synaptic com

126 acutely, EtOH may reduce lOFC function via a glycine receptor dependent process and this may trigger

127 h GABAA (gamma aminobutyric acid type A) and glycine receptors depends on the presence of the neurona

128 calyx of Held synapse, in which presynaptic glycine receptors depolarize presynaptic terminals, elev

129 nates zinc potentiation of alpha1-containing glycine receptors develop severe sensorimotor deficits c

130 To investigate bilobalide's interaction with glycine receptors directly, we determined 36chloride flu

131 with the truncated N-terminal and C-terminal glycine receptor domains, functionality of the glycine r

132 sed protein-protein interaction between both glycine receptor domains.

133 s demonstrate that PKC activation stimulates glycine receptor endocytosis, that both constitutive end

134 examined the endocytosis of homomeric alpha1 glycine receptors expressed in HEK 293 cells using immun

135 ct of SR-95531 on rat recombinant alpha1beta glycine receptors expressed in human embryonic kidney (H

136 byproduct inhibits or activates human alpha1 glycine receptors expressed on the surface of HEK 293 ce

137 We found that axonal GABA but not glycine receptor expression depends on neurotransmission

138 ry amino acids change the course of GABA and glycine receptor expression; and 3) there are any differ

139 lycine KI mouse lines with markedly impaired glycine receptor function.

140 dependent domain demonstrated restoration of glycine receptor functionality in vitro.

141 members, nicotinic acetylcholine receptors, glycine receptors, GABA(A) receptors, serotonin-3 (5-HT(

142 rose by genomic duplication of exon 5 in the glycine receptor gene Glra1.

143 porter 1; the anchoring protein for GABA and glycine receptors, gephyrin; the calcium binding protein

144 Ivermectin (Ivm), a glycine receptor (GLR) agonist, was used to modulate cel

145 ons in the alpha-1 subunit of the inhibitory glycine receptor (GLRA1).

146 for subunits of the postsynaptic inhibitory glycine receptor, GLRA1 encoding the alpha1 subunit and

147 el (VGKC)-complex antigens in four patients, glycine receptor (GLY-R) in 5 patients, N-methyl-d-aspar

148 regulation and release (GAD65, amphiphysin); glycine receptors (GLY-R); water channels (AQP4).

149 udies, taurine has been reported to activate glycine receptors (Gly-Rs) at moderate concentrations (>

150 glycinergic neurotransmission, including the glycine receptor (GlyR) alpha1 and beta subunits, gephyr

151 ssive hyperekplexia indicate disturbances in glycine receptor (GlyR) alpha1 biogenesis.

152 ominant and recessive mutations in the human glycine receptor (GlyR) alpha1 gene (GLRA1) are the majo

153 utations in the gene encoding the inhibitory glycine receptor (GlyR) alpha1 subunit (GLRA1).

154 ed inhibition was apparent for the homomeric glycine receptor (GlyR) alpha1 subunit compared to eithe

155 c alpha(1)-subunit (GLRA1) of the inhibitory glycine receptor (GlyR) and the cognate presynaptic glyc

156 The effect was blocked by the glycine receptor (GLyR) antagonist strychnine and mimick

157 systems, the GABA(A) receptor (GABA(A)R) and glycine receptor (GlyR) are described.

158 The glycine receptor (GlyR) exists either in homomeric alpha

159 way is largely blocked due to a reduction of glycine receptor (GlyR) expression, were quantitatively

160 Glycine receptor (GlyR) gating is initiated by agonist b

161 esence of several subunits of the inhibitory glycine receptor (GlyR) in the reward system, specifical

162 The glycine receptor (GlyR) is a member of the Cys-loop supe

163 The glycine receptor (GlyR) is a pentameric ligand-gated ion

164 The strychnine-sensitive glycine receptor (GlyR) mediates inhibitory synaptic tra

165 junctions, and provide inhibitory input via glycine receptor (GlyR) subunit alpha1 to OFF cone bipol

166 In contrast, the role of glycine receptor (GlyR) subunit-specific inhibition is l

167 an startle disease is caused by mutations in glycine receptor (GlyR) subunits or in other proteins as

168 GLRB genes, which encode the alpha1 and beta glycine receptor (GlyR) subunits, are the major cause.

169 rd transmembrane domains (TM23) of the human glycine receptor (GlyR) was achieved in low-q bicelles (

170 ically modified knock-in (KI) mouse having a glycine receptor (GlyR) with phenotypical silent mutatio

171 line receptor (nAChR) and the anionic alpha1 glycine receptor (GlyR), based on the structure of Torpe

172 e anion channels, such as CFTR, ANO1 and the glycine receptor (GlyR), by changing pore size.

173 , most commonly in the alpha1 subunit of the glycine receptor (GlyR), cause the startle disease/hyper

174 or (CFTR), anoctamin-1(ANO1/TMEM16A) and the glycine receptor (GlyR), revealed that the ion selectivi

175 ed that ethanol enhances the activity of the glycine receptor (GlyR), thus enhancing inhibitory neuro

176 o N-methyl-d-aspartate receptor (NMDAR), the glycine receptor (GlyR), voltage-gated potassium channel

177 tracellular domain (ECD) of the human alpha1 glycine receptor (GlyR), with amino acids from the corre

178 und to contact principal output neurons, via glycine receptor (GlyR)-enriched synapses, virtually dev

179 ed, using whole-cell patch-clamp recordings, glycine receptor (GlyR)-mediated currents in spinal moto

180 environment of Cu(2)(+) in the human alpha1-glycine receptor (GlyR).

181 receptor (5-HT3AR) that is not found in the glycine receptor (GlyR).

182 he alpha3beta4 AChR and the homomeric alpha1 glycine receptor (GlyR).

183 Glycine receptors (GlyR) are inhibitory Cys-loop ion cha

184 Strychnine-sensitive glycine receptors (GlyR) play a major role in the excita

185 s studies indicate that ethanol can modulate glycine receptors (GlyR), in part, through Gbetagamma in

186 We find that human islet beta-cells express glycine receptors (GlyR), notably the GlyRalpha1 subunit

187 GABAAR) and, to a lesser extent, by synaptic glycine receptors (GlyR).

188 tionally enhance the function of recombinant glycine receptors (GlyR).

189 n this case, the rat vanilloid (Trpv1 or the glycine receptor (GlyRalpha1), can allow selection of es

190 B1 bipolar cell axon terminals expressed the glycine receptor, GlyRalpha1, at sites of contact with A

191 eas a low-affinity competitive antagonist of glycine receptors (GlyRs) accelerated IPSC decay.

192 2 can markedly affect ethanol sensitivity in glycine receptors (GlyRs) and gamma-aminobutyric acid ty

193 sting propofol modulation of homomeric human glycine receptors (GlyRs) and nematode glutamate-gated c

194 Glycine receptors (GlyRs) are anion-permeable pentameric

195 Inhibitory glycine receptors (GlyRs) are composed of homologous alp

196 Glycine receptors (GlyRs) are found in most areas of the

197 Glycine receptors (GlyRs) are inhibitory ligand-gated io

198 Previous studies suggested that glycine receptors (GlyRs) are involved in the regulation

199 Glycine receptors (GlyRs) are key players in mediating f

200 Glycine receptors (GlyRs) are ligand-gated chloride chan

201 Glycine receptors (GlyRs) are major mediators of inhibit

202 Glycine receptors (GlyRs) are potentiated by ethanol and

203 Glycine receptors (GlyRs) are structurally related to GA

204 Glycine receptors (GlyRs) are the major mediators of fas

205 Although postsynaptic glycine receptors (GlyRs) as alphabeta heteromers attrac

206 ARs), GABA type B receptors (GABA(B)Rs), and glycine receptors (GlyRs) can be identified in patients

207 bition through the activation of heteromeric glycine receptors (GlyRs) composed primarily of alpha1 a

208 The properties of glycine receptors (GlyRs) depend upon their subunit comp

209 receptors (GABAARs) and strychnine-sensitive glycine receptors (GlyRs) expressed by excitatory cortic

210 icrotoxin antagonism of the alpha1 homomeric glycine receptors (GlyRs) has been shown to be non-use-d

211 irments or trafficking defects of inhibitory glycine receptors (GlyRs) have been linked to human hype

212 Functional glycine receptors (GlyRs) have been repeatedly detected

213 have demonstrated the presence of functional glycine receptors (GlyRs) in hippocampus.

214 s (AMPARs) and increasing that of inhibitory glycine receptors (GlyRs) in synapses.

215 required to achieve a high concentration of glycine receptors (GlyRs) in the postsynaptic membrane,

216 Glycine receptors (GlyRs) mediate fast inhibitory neurot

217 Glycine receptors (GlyRs) mediate inhibitory neurotransm

218 Strychnine-sensitive glycine receptors (GlyRs) mediate synaptic inhibition in

219 sidues in the extracellular loop 2 region of glycine receptors (GlyRs) or gamma-aminobutyric acid typ

220 face expression and regulated endocytosis of glycine receptors (GlyRs) play a critical function in ba

221 ereas both GABA(A) receptors (GABA(A)Rs) and glycine receptors (GlyRs) play a role in control of dors

222 is that several residues in Loop 2 of alpha1 glycine receptors (GlyRs) play important roles in mediat

223 ent phosphorylation of a specific subtype of glycine receptors (GlyRs) that contain alpha3 subunits.

224 nonpsychoactive cannabinoids can potentiate glycine receptors (GlyRs), an important target for nocic

225 ensitization of Cys-loop GABAA (GABAARs) and glycine receptors (GlyRs), which both mediate fast inhib

226 hronic pain and startle disease by targeting glycine receptors (GlyRs).

227 s area express Cl(-) permeable extrasynaptic glycine receptors (GlyRs).

228 ma-aminobutyric acid receptors (GABAARs) and glycine receptors (GlyRs).

229 potential (AP) firing by activating neuronal glycine receptors (GlyRs).

230 omeric to predominantly mature alpha(1)/beta glycine receptors (GlyRs).

231 ous cannabinoids can allosterically modulate glycine receptors (GlyRs).

232 e and a competitive antagonist at ionotropic glycine receptors (GlyRs).

233 of molecular mechanisms and pharmacology of glycine receptors has been hindered by a lack of high-re

234 Chloride-permeable glycine receptors have an important role in fast inhibit

235 nsmitter receptors (GABA, nACh, 5-HT(3), and glycine receptors) have resulted in a six-loop (A-F) mod

236 e kinetic properties of rat homomeric alpha3 glycine receptors heterologously expressed in HEK293 cel

237 mutations in the alpha1 subunit of the human glycine receptor (hGlyR) gene (GLRA1).

238 The human glycine receptors (hGlyRs) are chloride-selective ion ch

239 ole of the gain-of-function mutations of the glycine receptor in development of neural pathologies.

240 order of magnitude lower than the ionotropic glycine receptor in the same retina.

241 nd western blotting revealed the presence of glycine receptors in lOFC.

242 no butyric acid (GABA) receptor subtypes and glycine receptors in mediating CVLM sympathoinhibition.

243 TXA and EACA are competitive antagonists of glycine receptors in mice.

244 clude that bilobalide does not interact with glycine receptors in neurochemical assays but it signifi

245 ially regulate the expression of GABA(A) and glycine receptors in rat cortical neurons under normoxic

246 of breathing pattern by blocking GABA(A) and glycine receptors in the preBotzinger complex (preBotC),

247 role in forming the mature heteropentameric glycine receptors in these brain stem nuclear groups.

248 A T258F mutation of the glycine receptor increases the receptor affinity to endo

249 ion of gamma-aminobutyric acid (GABA)(A) and glycine receptors inhibits neurons as a result of low in

250 demonstrate that constitutive endocytosis of glycine receptors is blocked by the dominant negative dy

251 inhibitory central nervous system GABAA and glycine receptors, is believed to interact with residues

252 Because ws-LYNX1 was inactive against glycine receptor, its "non-classical" binding sites on a

253 ce, and it persisted while blocking GABA and glycine receptors, K((Ca)) channels, or mGluRs.

254 Inherited defects in glycine receptors lead to hyperekplexia, or startle dise

255 s necessary for the depolarizing response to glycine receptor ligands.

256 is the first time that two TM domains of the glycine receptor linked by the important 23 loop have ev

257 To determine whether GABA(A), GABA(C) and glycine receptors made different contributions to light-

258 and suggest that TXA-mediated inhibition of glycine receptors may underlie the effect.

259 at GlyRalpha2, the developmentally expressed glycine receptor, may play an important role in neuronal

260 Glycine receptors mediate fast synaptic inhibition in sp

261 We show that GABA(A), GABA(C) and glycine receptors mediate functionally distinct inhibiti

262 extent propofol, reverses TXA inhibition of glycine receptor-mediated current, suggesting that these

263 ors and involved an increase in postsynaptic glycine receptor-mediated currents.

264 eurons are cut off from GABA(B), GABA(A) and glycine receptor-mediated inhibition.

265 ntrast, the frequency of spontaneous GABA(A)/glycine receptor-mediated inhibitory postsynaptic synapt

266 gamma-aminobutyric acid type A receptor- and glycine receptor-mediated signaling that characterizes n

267 G93A) mice to investigate the development of glycine-receptor-mediated synaptic currents recorded fro

268 e examine the properties of a range of novel glycine receptor mutants identified in human hyperekplex

269 ycinergic transmission by acting directly on glycine receptors, narrowing the time window for effecti

270 Tonic conductance through glycine receptors of cerebellar granule cells is a yet u

271 te their relative scarcity, tonically active glycine receptors of cerebellar granule cells make a sig

272 Glycine receptors of cerebellar granule cells play their

273 Our results suggest that glycine receptors on ventral horn neurones in the juveni

274 (two nicotinic acetylcholine receptors, two glycine receptors, one GABA receptor, two AMPA-type glut

275 t position -1' of the anion-selective alpha1 glycine receptor open-state structure-instead of the fiv

276 r GluN2B subunits AMPA, kainate, and GABA or glycine receptors or a variety of other potential target

277 In both muscle nicotinic and glycine receptors, partial agonists are as good as full

278 Analysis of the glycine receptor properties mediating inhibition of parv

279 napses on RCs correlates well with increased glycine receptor recruitment to large gephyrin patches,

280 including the ginkgolides-some of which are glycine-receptor-selective antagonists(8).

281 ease and postsynaptic expression of GABA and glycine receptor subtypes.

282 is correlates with a change in glutamate and glycine receptor subunit composition quantified via mRNA

283 that the inhibition is marked by a switch in glycine receptor subunits from neonatal to adult form ar

284 gamma-aminobutyric acid type A (GABA(A)) and glycine receptor subunits.

285 he shared stoichiometry for phasic and tonic glycine receptors suggests pharmacology is unlikely to b

286 uggest that a UCA-induced down-regulation of glycine receptor synthesis may have occurred via reduced

287 Truncated glycine receptors that have been found in human patients

288 Ames medium and after blocking GABA(A/C) and glycine receptors, that PCP2-null rod bipolar cells were

289 hibitory amino acids is mediated by GABA and glycine receptors, the expression of these receptors is

290 synaptic currents can be mediated by alpha3 glycine receptors, they are likely to be very close in t

291 tly determine the subunit stoichiometry of a glycine receptor to be 3alpha1:2beta.

292 hibitory synapses can directly interact with glycine receptors to enhance inhibitory signalling.

293 ression and correct orientation according to glycine receptor topology.

294 receptor antibodies activated complement on glycine receptor-transfected human embryonic kidney cell

295 Gephyrin-mediated clustering of GABA(A) and glycine receptors underlies fast inhibitory signaling at

296 ycine receptor domains, functionality of the glycine receptor was again restored.

297 By applying glycine to native or recombinant glycine receptors, we show that response decay times are

298 c response was not found although functional glycine receptors were demonstrated at E16.

299 Calyces express presynaptic glycine receptors whose activation depolarizes the synap

300 The fast unbinding rate of SR-95531 from the glycine receptor will make it useful for establishing th