ライフサイエンスコーパス: go on to

1 not infrequently seen in some patients that went on to achieve CR.

2 D4(+) T cells that formerly expressed IL-17A go on to acquire an anti-inflammatory phenotype.

3 nguished European ancestry case subjects who went on to acquire a schizophrenia diagnosis from those

4 e salt signal to detach from the biofilm and go on to activate virulence.

5 ht that is not absorbed in the film does not go on to add a deleterious energy loading to the substra

6 We go on to address the relative importance of monomer geom

7 a mother cell generates daughter cells that go on to adopt different fates because of differential s

8 ion as a mode to prevent intubation and then go on to airway pressure release ventilation, high-frequ

9 ized this system with firefly luciferase and went on to apply it to members of three important classe

10 We go on to argue that the superior colliculus is a candida

11 We went on to ask whether AC are able to modulate the cours

12 ot always differentiate between subjects who go on to attempt suicide, suicidal subjects who never at

13 r, Seymour Benzer, and Francois Jacob, would go on to be among the founders of modern cell biology, d

14 Results suggest that infants who go on to be diagnosed with autism exhibit early differen

15 Since that time, the ASC has gone on to be one of the most popular adult stem cell po

16 owing and still they come." Eliot, who would go on to become an influential American pediatrician and

17 Unsurprisingly, all have gone on to become leaders in the fields of molecular cel

18 illard Hotel in Washington, DC, in 1909, and went on to become some of the leaders of American medici

19 d biopsy-proven HER2-positive metastases and went on to benefit from HER2-targeted therapy.

20 lopment were assessed in 45 adolescents, who went on to binge-drink (but were alcohol-naive at baseli

21 A is processed within these complexes, which go on to capture the target DNA and integrate the viral

22 ted DCDD offers were divided into donors who went on to cardiac arrest and those who did not.

23 e individual coronary lesions do not in fact go on to cause clinical events.

24 ansmitted to humans via the food supply, and go on to cause potentially life-threatening infections.

25 ells that survive chemotherapy treatment and go on to cause tumor regrowth, which we refer to as tumo

26 or in the fabrication of its main mirror, it went on to change forever many fields of astronomy, and

27 We go on to characterize the mitosis-specific interaction o

28 We go on to classify an array of equilibrium and nonequilib

29 enzymes can dissociate upon DNA cleavage and go on to cleave further DNA molecules (they can 'turnove

30 e tumors fully infiltrated by CD8(+) T cells went on to complete resolution.

31 east conservation, whereas 4 patients (5.1%) went on to completion mastectomy.

32 The second paper in the series then goes on to construct reversible models of energy and inf

33 The authors went on to correlate CEACAM1 levels with postreperfusion

34 oughout pregnancy, could predict which women go on to deliver preterm.

35 We go on to demonstrate for the first time a submicron patt

36 ncy analysis and dynamic causal modeling, we go on to demonstrate functional integration between thes

37 ling tumour cells with CpG adjuvant, we then go on to demonstrate in vitro that labelling is preserve

38 ly restricted variants of these proteins, we go on to demonstrate that all known host shutoff-related

39 Using both in vitro and in vivo models, we go on to demonstrate that hydralazine has neuroprotectiv

40 stem of LPS-treated RAW264.7 macrophages, we go on to demonstrate that OPN increases STAT1 ubiquitina

41 We go on to demonstrate that TGF-beta induces the formation

42 We then go on to demonstrate that the antagonistic functions of

43 is indeed the receptor interaction site and go on to demonstrate that the conformational changes tha

44 We go on to demonstrate that the failure of ICP22 protein t

45 eproductive success as a fitness measure, we go on to demonstrate that these status changes can have

46 We go on to demonstrate that this nuclear activity reflects

47 We go on to demonstrate the utility of this small molecule

48 We have gone on to demonstrate that an intact microtubule networ

49 One patient went on to demonstrate progressive ocular disease that w

50 We went on to demonstrate the utility of this approach by c

51 We go on to describe methods for the detection of individua

52 We go on to describe the algorithm RNAborMEA, which for an

53 icle reviews the theoretical basis of WE and goes on to describe successful applications to a number

54 nditure ushered in a decade of research that went on to describe not only the specific nuclei and cel

55 We go on to determine that this channel inhibition by Ucn1

56 We go on to determine the biochemical and structural basis

57 th these improved models, it was possible to go on to determine the location of the B atoms in the fr

58 Based on this study, we go on to develop a mathematical explanation of how ULFO

59 identifying individuals who will eventually go on to develop a particular disease and by the inacces

60 We go on to develop an automated approach involving an in s

61 ctories in infants at high familial risk who go on to develop ASD.

62 om infants who were not colonized and do not go on to develop asthma symptoms in childhood.

63 ssed before developing AUD predict those who go on to develop AUD.

64 corpus callosum may be larger in infants who go on to develop autism spectrum disorder.

65 whether brain volume differs in infants who go on to develop autism.

66 that are elevated preclinically in women who go on to develop breast cancer can be discovered and val

67 ability to reliably predict who exactly will go on to develop cardiomyopathy and heart failure in the

68 n with these extreme measures, many patients go on to develop chronic infection or sustain disease co

69 patterns, many children with high BI do not go on to develop clinical, or even subclinical, anxiety

70 ls are protected from infection while others go on to develop disease are unclear.

71 Only about 2% to 3% of patients go on to develop epilepsy.

72 eferrals, and earlier referrals in those who go on to develop ESRD.

73 D psychosis risk factor data in patients who go on to develop illusions or hallucinations within 3-4

74 curately determined which PAD patients would go on to develop major adverse cardiac and cerebrovascul

75 dict whether an individual at high risk will go on to develop psychosis on the basis of their present

76 CHRs who would go on to develop psychosis were found to show abnormal m

77 an one third of patients with incident bleed go on to develop recurrent GI bleeding.

78 of AA patients and 2% to 6% of PNH patients go on to develop secondary MDS/AML by 10 years of follow

79 eded to determine how many of these patients go on to develop systemic manifestations of SS.

80 , many individuals with these factors do not go on to develop the disorder.

81 These PS1 subjects will go on to develop the first symptoms of the disease aroun

82 fferentially expressed genes in patients who go on to develop VAP compared to similar patients who do

83 implications for patients with, or who will go on to develop, dementia-related visual impairment, in

84 elp for cognitive symptoms neither have, nor go on to develop, dementia.

85 nt had flares while on originator infliximab went on to develop a single flare on infliximab-abda.

86 sted case-control study of HIV+ subjects who went on to develop AIDS-NHL, as well as HIV+ subjects wh

87 We found that the microbiome of infants who went on to develop allergic sensitization later in child

88 healthy individuals (n = 374), of which half went on to develop breast or colon cancer.

89 tland Primary Prevention Study (WOSCOPS) who went on to develop coronary heart disease events with th

90 gnificantly higher (p < 0.0001) in those who went on to develop DHF compared to DF.

91 % of patients with an MA of 65 mm or greater went on to develop E-HAT (hazard ratio, 5.28; 95% confid

92 All 5 of these nonexudative CNV went on to develop exudation in subsequent visits.

93 Mice that went on to develop GVHD showed signs of rapid proliferat

94 People who went on to develop ICB had more severe anxiety, worse au

95 itis and in preterm infants who subsequently went on to develop lung disease.

96 oviral therapy and at VF in participants who went on to develop M184V/I.

97 tiation of ART and at VF in participants who went on to develop M184V/I.

98 ases of MDD who were well at recruitment but went on to develop MDD at a later assessment (beta = 0.1

99 In addition, patients that went on to develop metastasis showed increased expressio

100 Patients with baseline sensory deficits went on to develop more symptom complaints during chemot

101 egativicutes class (p=0.0051) in infants who went on to develop necrotising enterocolitis compared wi

102 disease (N) matched against 10 patients who went on to develop overt nephropathy (DN), 10 patients w

103 on (2.01, 1.62-2.49) was higher in those who went on to develop Parkinson's disease (n=1680) than in

104 pared with controls (n=25,544), patients who went on to develop Parkinson's disease (n=4769) had a hi

105 pain or stiffness was higher in patients who went on to develop Parkinson's disease (n=7232) than in

106 cy, distinctive immune response in women who went on to develop preeclampsia.

107 antigen were significantly higher in men who went on to develop prostate cancer, up to 6 years prior

108 Before treatment, patients who went on to develop secondary autoimmunity had more than

109 Analyses of biomarkers in patients who later went on to develop secondary infections revealed a more

110 ial function, was lowest in the patients who went on to develop severe dengue (median 1.54, IQR 1.36-

111 ts who were screened for active tuberculosis went on to develop tuberculosis compared with 1099 (1%)

112 the greatest number of macrophages at day 5 went on to develop tumors.

113 e onset of the disease in those patients who went on to develop type 1 diabetes.

114 cted critically-injured trauma patients that went on to develop VAP (VAP+) and those that did not (VA

115 organs or tissues had occurred in people who went on to develop vCJD.

116 s in critically injured trauma patients that went on to develop ventilator-associated pneumonia (n=10

117 sies who have impaired development and often go on to die of their disease respond poorly to anticonv

118 an acute myocardial infarction diagnosis but went on to die of acute myocardial infarction as the und

119 ise to multiple progenitor cell domains that go on to differentiate unique classes of neurons and gli

120 We went on to discover that Aurora B directly phosphorylate

121 We go on to discuss a number of correction procedures that

122 We then go on to discuss how the critical periods for these gene

123 We go on to discuss how these inactive and active phases mi

124 We go on to discuss the theoretical and methodological limi

125 We go on to discuss their growing potential for advancing b

126 We go on to discuss when in the epidemic such a high ratio

127 These cells then go on to divide at least once to become OSNs, demonstrat

128 Cells with amplified centrioles can go on to divide, with centrioles clustered at each pole.

129 ecological release of tyrannosauroids, which went on to dominate end-Cretaceous food webs.

130 the cellularly diverse airway epithelium and goes on to drive development of most chronic lung diseas

131 I went on to earn a Master's degree from the University of

132 These tissues go on to elongate, fold, segment, and form appendages.

133 els determine which insular cortical neurons go on to encode a given conditioned taste memory.

134 We went on to engineer point mutations that stimulated IgG

135 enriched in samples taken from patients who went on to enter complete remission (CR), whereas MDR(+)

136 ight later determine why EBV infected blasts go on to establish transformed cell lines, we performed

137 dents and postdoctoral fellows, many of whom went on to establish successful careers of their own.

138 They went on to evaluate the tryptase inhibitor nafamostat me

139 We go on to examine our predictions of molecular interactio

140 We go on to examine the available evidence for the Mpemba e

141 spects of neural processing in this task, we go on to examine the BOLD correlates with the within tri

142 The review goes on to examine how well exogenously supplied vitamin

143 The authors went on to examine samples derived from patient lymphoid

144 ogenesis in a developing zebrafish heart and went on to examine the functions of cardiac troponin T (

145 We went on to examine the genetic architecture of resistanc

146 tant glands overcame these early delays, but went on to exhibit enlarged and chaotic ductal morpholog

147 sleep spindles during a quiet night of sleep went on to exhibit higher tolerance for noise during a s

148 that survive the initial ischemic insult but go on to experience a delayed axonal degeneration driven

149 number of people with negative test results go on to experience adverse cardiac events.

150 lly had completely normal fundi in both eyes went on to experience moderately decreased vision, altho

151 seizure developed in 86 patients, 42 of whom went on to experience recurrent seizures.

152 scientific and political endeavors, Franklin went on to experiment with oil, spreading monomolecular

153 al role in whole body energy homeostasis, we went on to explore the function of SIRT2 in adipose tiss

154 These ectopic domains go on to express other parathyroid markers but do not mi

155 duce monomethyl arginine (MMA), type 1 PRMTs go on to form asymmetrically dimethylated arginine (ADMA

156 The initial Cu(II)-Abeta complexes go on to form Cu(II)-containing Abeta fibrils.

157 on, the UPS is more impaired in neurons that go on to form IBs than in those that do not.

158 Furthermore, these T cells go on to form memory cells that are phenotypically and f

159 l minority of cells that express mutant Kras go on to form PanINs.

160 a1, or Kin4 can return to mitotic growth and go on to form polyploid cells.

161 e collaborations with the public sector that go on to form the basis of national vector control progr

162 yrosine phosphatase 1 recruitment and do not go on to form the KIR microclusters induced by high-affi

163 er (tau(ex) ~ 30 mus, K (diss) ~ 0.1 M) that goes on to form a tetramer ([Formula: see text] mus; K (

164 nriched all Mn(IV) form of the cofactor that goes on to form the O-O bond, after the next light-induc

165 express typical tip markers, with these tips going on to form epithelial trees, at high frequency.

166 The authors then went on to formulate guidelines, both short-term and lon

167 ontinue to exhibit subthreshold symptoms, or go on to fully remit.

168 arbour relatively few species, some of which go on to generate endemic radiations.

169 al standards useful in MALDI imaging studies goes on to get not only lateral distribution but also re

170 C, especially in patients whose tumors would go on to have a pathological complete response (pCR), bu

171 rs with high fecundities train proteges that go on to have fecundities 29% higher than expected.

172 rs with high fecundities train proteges that go on to have fecundities 31% lower than expected.

173 birth outcomes, the majority of adolescents go on to have full-term, healthy babies.

174 w mentorship fecundities train proteges that go on to have mentorship fecundities 37% higher than exp

175 it is the offspring with family history who go on to have recurrences and a poor outcome as they mat

176 ents, with approximately a third of patients going on to have deferred treatment.

177 and 28,464 had a planned VBAC, 28.4% of whom went on to have an in-labor nonelective repeat cesarean

178 in the 3-month stool samples of children who went on to have atopic wheeze.

179 anisms involved and how they collaborate and go on to highlight contexts of curvature that are exciti

180 We go on to highlight the types of ecological and evolution

181 the onset of seismicity and in regions that went on to host moderate magnitude earthquakes within 6

182 We then go on to identify a novel motif in Rsc2 BAH that is crit

183 We go on to identify an interaction between decreasing wate

184 We go on to identify cell envelope proteins that are necess

185 We go on to identify Irgm1 as an IFNgamma-regulated gene in

186 ice use of the 'systematic review' label, we go on to identify methodological solutions to mitigate t

187 Using molecular dynamics simulations, we go on to identify unique paralogs in M. stadtmanae and M

188 We went on to identify a common TBX6 haplotype as the secon

189 We went on to identify a critical Wnt3a signaling threshold

190 We went on to identify a more potent PAM for use in native

191 We went on to identify a novel subpopulation of Ag-binding

192 We went on to identify that the cells expressing IL-17 were

193 We went on to identify the AMPK-nuclear factor-erythroid 2

194 etition between demixing and crystallization goes on to impact the growth process, as shown by the la

195 did not meet response criteria after step 2 went on to in-person physical therapy visits (step 3).

196 cultural beliefs of a given individual, and go on to include a mechanism of cultural inheritance fro

197 To explain resurgences of the epidemic, we go on to include waning immunity and a mechanism explici

198 VMPFC during exposure to health messages and went on to increase their objectively measured activity

199 ition of zinc(II), and the ejected copper(I) went on to induce the self-assembly of a Cu(I)(2) helica

200 a major outbreak (i.e. an epidemic that will go on to infect a large number of individuals) can be pr

201 the number of progeny per infected cell that go on to infect new cells are needed.

202 on average, less than one progeny virus that goes on to infect a new cell.

203 odels within the biomedical domain, and then go on to introduce a number of prototypical data integra

204 The study went on to investigate the effect of conditioning dentin

205 ct prey and regurgitates Photorhabdus, which goes on to kill the insect.

206 te a device to obtain food in the laboratory go on to lay more eggs in the wild.

207 We go on to localize the site of block as a defect in lysos

208 d more than 2 prior regimens; and 6 patients went on to maintenance.

209 f oral health researchers, many of whom have gone on to make major contributions to dental research a

210 wever, nearly 60% of children with BI do not go on to meet criteria for social anxiety disorder.

211 Of 77 patients with CLE, 13 (17%) went on to meet criteria for SLE, with a mean (SD) time

212 metabolite measurements; from this, we then go on to obtain a dynamical view of the hierarchical reg

213 We go on to perform the first global DNA promoter methylati

214 demethylase of N6-methyladenosine (m6A), we went on to perform genome-wide m6A profiling and observe

215 The authors went on to perform invasive procedures for the treatment

216 ere symptoms and high transmission rate, has gone on to pose a control challenge for healthcare syste

217 The model goes on to predict that the ratio of steady-state [H2O2]

218 We go on to propose a two-stage analysis of RNA-Seq data in

219 Karin et al. went on to provide a sufficient condition to test whethe

220 We go on to quantify the vessel distribution heterogeneity

221 alities are not present in infants who later go on to receive a diagnosis of autism; rather, abnormal

222 Fifty-two patients who did not go on to receive transplants received continued therapy

223 rt of cardiothoracic transplant patients who went on to receive a sequential kidney transplant, a rep

224 durations were shortest in those infants who went on to receive an ASD diagnosis at 36 months.

225 All patients went on to receive platinum-based chemotherapy; platinum

226 Based on the results obtained, the authors go on to recommend the deployment of batteries with high

227 s into Galpha and Gbetagamma subunits, which go on to regulate various effectors involved in a panopl

228 r target variant frequencies in patients who go on to relapse, demonstrating the potential for MASQ t

229 29 patients went on to relapse and 38 remained well.

230 We go on to replace the missing LDLR in familial hyperchole

231 Almost 60% (434/734) went on to report a further episode as a young adult.

232 with none of these factors at baseline, <5% went on to report persistent LBP.

233 More than 20% of cardiac transplant patients go on to require permanent pacing.

234 icosteroids for many months, yet all of them went on to require methotrexate.

235 patients who were PRP naive at baseline who went on to require PRP, experienced more clinical events

236 He subsequently went on to residency and fellowship at Children's Hospit

237 icipal waste stocks (8.5 billion tonnes) and go on to resolve these stocks at the county level, refle

238 Substantial research is going on to reveal the functions of piRNAs in the epigen

239 We go on to show a very similar pattern of chronic allocent

240 We go on to show how DNA packaging can induce spontaneous r

241 We go on to show in cultured Perforin-2(-/-)cells that the

242 The authors go on to show intriguing parallels between compensation

243 We go on to show that a large percentage of primary NSCLC s

244 We go on to show that activation of Src42A is necessary to

245 We go on to show that apart from being LTM defective, creb1

246 We go on to show that biofilm induction appears to be a gen

247 We go on to show that blue light evokes higher Fos inductio

248 roscopy and ratiometric light microscopy, we go on to show that bulk membrane proteins are depleted w

249 We go on to show that by silencing these neurons, we can ch

250 We go on to show that complete degradation of Rpb1 in alpha

251 We go on to show that direct electron transfer from KOtBu c

252 ith ferroquadrupole order of the Yb ions and go on to show that elastoresistivity measurements can in

253 We go on to show that Eomes(hi) NK cells can be recruited f

254 We go on to show that H3K79me2/3 is essential for maintaini

255 We go on to show that insertion of an ultrathin CTE buffer

256 We go on to show that it is the overlap of AID hotspots at

257 We go on to show that recombinant TWEAK activates DRs and t

258 olution, signifying a selective benefit, but go on to show that similar SNPs are rare in the genome d

259 We go on to show that Spen, recruited through the Xist A-re

260 We go on to show that the alpha.beta dimers differ in key p

261 We go on to show that the metafluid obeys the Einstein rela

262 We go on to show that the timing of the onset of tail-spike

263 We go on to show that the Y32A mutant stimulates the actin

264 We go on to show that these connections impact olfactory pr

265 After discussing examples, we go on to show that this effect is generally to be expect

266 to 3'-nuclease activity of Dna2; however, we go on to show that this same protein inhibits the 3'- to

267 We go on to show that this signaling defect translates to t

268 We go on to show that ubiquitin is attached to hD4R through

269 We go on to show that universal TERMs provide an effective

270 We go on to show that, even with these methods, standard te

271 ced adaptation to facial identity in ASD and goes on to show that reduced adaptation is not unique to

272 We went on to show that a great deal of electrical signalin

273 We then went on to show that anodal transcranial direct current

274 The authors went on to show that dual inhibition of JAK and the MAP

275 (ALKs) are necessary for Smad activation, we went on to show that knockdown of ALK2, but not TGFbetaR

276 in folding and molecular chaperones, and she went on to show that protein folding can have profound a

277 tor of IFN-induced STAT1 phosphorylation, we went on to show that RING finger protein 4 depletion sta

278 We went on to show that this common pathway involves Trio,

279 Having elucidated the charging mechanism, we go on to study the factors that affect the rate of ionic

280 We go on to study the molecular mechanisms leading to TrkA

281 We went on to study two of them in detail, the very closely

282 -22BP in both rat and mouse models of MS and go on to suggest a pathogenic mechanism involving lack o

283 We have also gone on to support the hypothesis that CRBP may also bin

284 ctus abdominis insertion were most likely to go on to surgical pelvic floor repair.

285 ntial chemoselective ligation strategies, we go on to tag these analytes with a series of labels, all

286 further explore this inconsistency, we have gone on to test whether DmHMT-1, an HMT-1 from a new sou

287 We went on to test if 14 out of the 18 genes, those which w

288 We therefore went on to test if therapeutically relevant changes in t

289 We then went on to test whether or not this phenotype occurred i

290 the reactive Pd(pi-allyl) intermediate that goes on to the desired coupling product after attack by

291 laxes back to P(r) in 3 ps, whereas the rest goes on to the Lumi-R photoproduct consistent with the 1

292 Following this analysis, we go on to trace how these differences in natural images t

293 hronic obstructive hydrocephalus, 12 of whom went on to undergo ETV, were imaged with phase-contrast

294 esponse rate was 90%; of these patients, 45% went on to undergo stem-cell transplantation.

295 We go on to use a recently developed modeling approach, MUS

296 With this knowledge, we go on to use therapies targeted at T cells to investigat

297 We go on to use these antibodies to assess PDH phosphorylat

298 a limited number of diffraction images, and went on to use these techniques to obtain a complete dat

299 Yoon and colleagues go on to validate their observations by extensively docu

300 We went on to validate the findings of WES using 151 lepros