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1 's effects on the body (eg, polycythemia and goiter).
2 ctice in nontoxic uninodular or multinodular goiter.
3 esis of Cowden Disease and sporadic nontoxic goiter.
4 drome, which is associated with deafness and goiter.
5  and with a maternal history of hepatitis or goiter.
6 ssive disorder characterized by deafness and goiter.
7 roid function) on risk of thyroid cancer and goiter.
8 ed, especially in patients with pre-existing goiter.
9 diffuse goiter and 41 patients had a nodular goiter.
10 d gland during total thyroidectomy for toxic goiter.
11 may contribute to the development of nodular goiter.
12 t hyperthyroidism and euthyroid multinodular goiter.
13 o Pendred syndrome, causing hearing loss and goiter.
14 weed was used as a source of iodine to treat goiters.
15 38%) but not in 18 follicular adenomas and 6 goiters.
16 lignant tumor and multinodular intrathoracic goiters.
17 n indications for surgery in the cohort were goiter (35.3%) and a single nodule or neoplasm (39.2%).
18  Parathyroidectomy, Hyperthyroid Conditions, Goiter, Adjuncts and Approaches Laryngology Familial Thy
19  Parathyroidectomy, Hyperthyroid Conditions, Goiter, Adjuncts and Approaches to Thyroidectomy, Laryng
20 m in newborns that, if untreated, results in goiter along with serious cognitive and growth defects.
21                                              Goiter, an enlargement of the thyroid gland, is a common
22 atients, of which 233 patients had a diffuse goiter and 41 patients had a nodular goiter.
23 mice had hypothyroidism, dwarfism, alopecia, goiter and cardiac abnormalities including hypertrophy,
24 ome is a major cause of congenital deafness, goiter and defective iodide organification.
25 hyroid abnormalities, including multinodular goiter and follicular adenomas, and are at increased ris
26  animal model of Graves disease to show that goiter and hyperthyroidism occur to a much greater exten
27 oid-specific expression of BRAFV600E induces goiter and invasive PTC, which transitions to poorly dif
28 or autonomous nodules and toxic multinodular goiter and remains a safe and effective treatment for Gr
29 se in thyroid volume, a higher prevalence of goiter, and an increased risk of hyperthyrotropinemia.
30  Graves' disease is hyperthyroidism, diffuse goiter, and exophthalmos.
31 der characterized by sensorineural deafness, goiter, and impaired iodide organification.
32 rrhythmia or ischemic heart disease, size of goiter, and severity of thyrotoxicosis.
33  at exposure and at examination, presence of goiter, and urban/rural residency.
34 d multinodular (OR = 0.69, p = 3.9 x 10(-5)) goiters, and thyrotoxicosis (OR = 0.76, p = 1.5 x 10(-3)
35          Most patients with nontoxic nodular goiter are asymptomatic or have only mild mechanical sym
36  a murine model prevented the development of goiter as well as the induction of inflammatory and fibr
37 lary thyroid carcinoma (TC) and multinodular goiter, before and after treatment with radioactive iodi
38 o were scheduled for total thyroidectomy for goiter, benign nodular disease, suspected thyroid cancer
39  found that Pten mutant mice develop diffuse goiter characterized by extremely enlarged follicles, in
40                                              Goiter development due to incomplete thyrotropin suppres
41  causes neonatal hypothyroidism and prevents goiter development.
42 tic thyrocyte cell death, preventing thyroid goiter formation in rdw/rdw rats.
43 an important cause of congenital hypothyroid goiter; further, homozygous mice expressing two cog/cog
44 High consumption of mouflon in comparison to goitered gazelle suggests that human pressure on lowland
45 on mouflon; following by ibex, cape hare and goitered gazelle.
46                                              Goitered gazelles were only detected in an area where th
47                  Patients with toxic nodular goiter had an SMR of 1.16 (95% confidence interval [CI],
48 90 papillary carcinomas, and 0 of 10 nodular goiters had 3p25 rearrangements by interphase fluorescen
49  benign thyroid disorders deals with nodular goiter, hypothyroidism, and subacute thyroiditis.
50 ich a lack of secondary thyroid enlargement (goiter) implicates death of thyrocytes as part of diseas
51 icient to eliminate or postpone the onset of goiter in individuals with DFNB4.
52 enocytes from NODCCR7(ko/ko) animals induced goiter in NOD.SCID recipients, demonstrating that autore
53 ation (MUIC) <100 ug/L, or >5% prevalence of goiter in school-age children.
54                   Treatment for multinodular goiter includes dietary iodine supplementation, surgery,
55 g transcytosis in vivo, using a rat model of goiter induced by aminotriazole, in which increased rele
56 rited tumor syndrome, featuring multinodular goiter (MNG) and rare pediatric-onset lesions.
57 al hypothyroidism, and in the cog congenital goiter mouse and rdw rat dwarf models, thyroid hormone s
58            All 16 thyroid masses were due to goiter; none of these were changed at follow-up CT 1 yea
59        Of 59 patients referred for a diffuse goiter or a multinodular gland, ultrasonography detected
60 4.3% [70 of 288], P < .001) and multinodular goiter or thyroid nodule (40.7% [50 of 123] vs 29.2% [84
61 (OR, 7.63; 95% CI, 3.49-16.69), multinodular goiter or thyroid nodule (OR, 1.82; 95% CI, 1.01-3.28),
62 oidism and hyperthyroidism in adults without goiter or thyroid nodules.
63 erparathyroidism, and in patients with large goiters or moderate to severe thyroid eye disease who ca
64 as not a significant increase in the risk of goiter (OR, 1.8; 95% CI, 0.7-4.9).
65  0.59, 0.74), a 74% reduction in the odds of goiter (OR: 0.26; 95% CI: 0.16, 0.43), and a 41% reducti
66 e to suppress TSH because of thyroid cancer, goiters, or nodules seem to have an adverse effect on bo
67 otid artery and thyroid vein of 10 euthyroid goiter patients and one patient with a toxic solitary ad
68 e TSH marker was determined against MUIC and goiter prevalence as the reference markers.
69 tion using the TSH marker and either MUIC or goiter prevalence in school-age children were included.
70   The TSH marker has a better agreement with goiter prevalence than MUIC when classifying the iodine
71 as 65% for TSH and MUIC, and 83% for TSH and goiter prevalence.
72 h MUIC, and 0.86 and 0.50 when compared with goiter prevalence.
73 to segregate with retinitis pigmentosa (RP), goiter, primary ovarian insufficiency, and mild intellec
74 lly are associated with the development of a goiter, provided that the bioactivity and action of thyr
75 n in rural and urban areas included maternal goiter (rural odds ratio (OR) = 5.14, 95% confidence int
76        She underwent total thyroidectomy for goiter several years ago, with initial iatrogenic hypoth
77 hibit polycythemia, pericardial effusion, or goiter should be evaluated for cobalt exposure.
78 , especially co-existing with an adenomatous goiter, should prompt a work-up for thyroid metastasis.
79 d a marked enlargement of the thyroid gland (goiter) that was associated with circulating autoantibod
80 h as Graves' disease and functioning nodular goiters, there are more than 20 less common causes of el
81 ism (Graves disease, GD), toxic multinodular goiter (TMNG), and toxic thyroid adenoma (TA).
82 id gland (Graves disease, toxic multinodular goiter, toxic adenoma).
83      A growth or mass (including hernias and goiters) was the most commonly reported potentially surg
84 ation > 300 ug/L on hyperthyrotropinemia And goiter were 1.77 (95% confidence interval [CI], 1.20-2.6
85 nimals suffering from congenital hypothyroid goiter with defective thyroglobulin, GRP94 and thyroglob
86 sceptibility syndrome: familial multinodular goiter with schwannomatosis.FUNDINGCanadian Institutes o