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1 e brain has been attributed to the action of gonadal hormones.
2 d secretion of gonadotropins and, therefore, gonadal hormones.
3 afferents is highly responsive to changes in gonadal hormones.
4 in the arcuate nucleus, a target tissue for gonadal hormones.
5 oth odor set sizes, but was not dependent on gonadal hormones.
6 the gonads to function without mediation by gonadal hormones.
7 RFR1 is regulated by perinatal but not adult gonadal hormones.
8 nt tissues, a characteristic shared with the gonadal hormones.
9 re genes located on the sex chromosomes, and gonadal hormones.
10 al to hippocampal function and influenced by gonadal hormones.
11 pe are caused by the differential effects of gonadal hormones.
12 h organizational and activational effects of gonadal hormones.
13 and thus expression may be regulated also by gonadal hormones.
14 ex chromosome genes that are not mediated by gonadal hormones.
15 ces in risky decision making are mediated by gonadal hormones.
16 ion and behavior are regularly attributed to gonadal hormones.
17 these differences precede the influences of gonadal hormones.
18 ate regulation of the CNS by male and female gonadal hormones.
19 in the male genome, without any mediation by gonadal hormones.
20 ding attributed to the protective effects of gonadal hormones.
21 red from mice that were capable of secreting gonadal hormones.
22 dence from animal studies indicates that the gonadal hormone 17beta-estradiol (E(2)) impacts the stru
23 avioral responses to defeat, indicating that gonadal hormones act during puberty to program behaviora
26 how sex-defining factors (e.g., chromosomes, gonadal hormones) alter immune responses in health and d
27 ary outcome measure) and had blood drawn for gonadal hormone and neurosteroid levels determined on ea
29 ie a two-way interaction between circulating gonadal hormones and behavioral responses to socially sa
31 dimorphism have emphasized the importance of gonadal hormones and cell-autonomous influences in mamma
33 nadal development, separating the effects of gonadal hormones and chromosomal factors requires specif
34 e hypothalamopituitary axis and suggest that gonadal hormones and female presence independently regul
35 lished by organizational effects of neonatal gonadal hormones and highlight the role of INs as mediat
36 central nervous system (CNS) factors, genes, gonadal hormones and receptors, genitalia, and social/en
40 e organizational and activational effects of gonadal hormones and to genes on the sex chromosomes.
41 exerts its metabolic effects indirectly via gonadal hormones and/or directly via the kisspeptin rece
42 gical sex (determined by sex chromosomes and gonadal hormones) and gender (social and cultural behavi
43 mones, permanent (organizational) effects of gonadal hormones, and cell-autonomous effects of sex chr
46 alamus, brain receptors for gonadotropic and gonadal hormones are concentrated in specific subcortica
54 se dimorphisms are maintained by circulating gonadal hormones, as castration of adult male rats reduc
55 nxiety were not because of altered levels of gonadal hormones, as serum estrogen and progesterone lev
56 ese differences were not solely dependent on gonadal hormones, because they persisted in gonadectomiz
61 y and provide a potential mechanism by which gonadal hormones could regulate the maturation of the as
64 ned for a female phenotype unless exposed to gonadal hormones during a perinatal sensitive period.
65 brain can be attributed to known effects of gonadal hormones during development or adulthood, and fa
66 l nervous system to low levels of endogenous gonadal hormones during development, the central nervous
70 halves of the brain were exposed to a common gonadal hormone environment, the lateral differences ind
71 An emerging hypothesis is that fluctuating gonadal hormones, especially estrogen, in the menstrual
72 inatal sensitive period, when organizational gonadal hormones establish the sexually dimorphic brain,
73 llowing experiments examined the role of the gonadal hormone estrogen in comparison to the neurotroph
78 (HUVECs) were propagated in phenol red-free gonadal hormone-free medium and pretreated with 17 beta-
79 trogen receptor-positive) were propagated in gonadal hormone-free medium and were E2-pretreated for 4
81 ted status, suggesting that dysregulation of gonadal hormone function may be a characteristic trait o
82 pothalamus become sexually differentiated by gonadal hormones giving rise to sexually differentiated
83 is more common in males than females; thus, gonadal hormones have been a focal point for research in
85 specting the breadth and depth of the impact gonadal hormones have on brain functioning and its rich
86 involving neonatal organizational effects of gonadal hormones have previously been shown to profoundl
89 astrocytes persisted even in the absence of gonadal hormones in adulthood, suggesting that androgens
90 in these sites is influenced by circulating gonadal hormones in adults, which may be related to chan
92 sidering sex chromosomes and sex-independent gonadal hormones in evaluating potential sex-specific di
93 discriminable, suggesting opposite roles for gonadal hormones in influencing male and female olfactor
94 ssion is sexually dimorphic and modulated by gonadal hormones in lateral regions of the rat dorsal st
95 J and SJL/J mice to assess the role of adult gonadal hormones in regulating clinical and histopatholo
96 nessed increased confusion as to the role of gonadal hormones in the development of neuroeffectors fo
98 ffects of stress, where the rapid decline of gonadal hormones in women combined with cellular aging p
100 PR expression, suggesting the presence of a gonadal hormone-independent PR regulatory mechanism.
103 tradiction between the dependence on ERK for gonadal hormone-induced neuroprotection and the lack of
106 ontext of genetic heterogeneity, circulating gonadal hormones influence both clinical and histopathol
109 Rats were used to determine how sex and gonadal hormones influence choices between small, immedi
110 onitase activity, suggesting that endogenous gonadal hormones influence mitochondrial ROS production
112 nt work illustrating how sex differences and gonadal hormones influence sleep and circadian rhythms t
113 uggest a limited organizational-activational gonadal hormone interaction in the mediation of systemic
114 e (ERbeta effect), while having no effect on gonadal hormone levels (ERalpha effect) at 10x the effic
118 of these sex differences, but whether adult gonadal hormones maintain the increased number and compl
121 ded that prenatal, organizational effects of gonadal hormones may play a role in the development of A
122 ng vehicle treatment, the presence of female gonadal hormones may underlie the sex differences in mor
123 as genetic sex and developmental exposure to gonadal hormones, may also contribute to altered vulnera
124 Although studies indicate that circulating gonadal hormones might underlie the observed sex differe
126 involving the adult activational effects of gonadal hormones minimally alter these analgesic sex dif
129 ural mechanisms underlying the influences of gonadal hormones on human behavior are beginning to be i
132 here is also a sex difference independent of gonadal hormones: OVX females exhibit a greater magnitud
133 emales: reversible (activational) effects of gonadal hormones, permanent (organizational) effects of
134 t experimental data in rodents, suggest that gonadal hormones play a role in modulating this system.
135 nd gonadectomized male rats, suggesting that gonadal hormones play an important role in modulating th
138 printed genes is most likely attributable by gonadal hormones rather than by sex chromosome complemen
141 neurotrophic and angiogenic growth factors, gonadal hormone receptors, and neurotransmitters such as
143 al steroid hormones and is a likely site for gonadal hormone regulation of sexually dimorphic social
144 to characterize the chemical composition and gonadal hormone regulation of these sexually dimorphic C
145 Together, these data indicate a stress and gonadal hormone responsive nucleus that is unique to fem
146 Are sex differences in levels of circulating gonadal hormones robust enough to account for the full s
147 changes during puberty that may be driven by gonadal hormone secretion during this developmental peri
148 x chromosomes and the consequent sex-typical gonadal hormone secretions may play important roles in t
150 ed that in addition to well-known effects of gonadal hormones, sex chromosome complement influences c
151 t of the interaction of three major factors: gonadal hormones, sex chromosomes, and the environment.
157 ence indicates that prenatal exposure to the gonadal hormone, testosterone, influences the developmen
158 rentiation of the gonads, which then secrete gonadal hormones that act directly on tissues to induce
159 data provide a substantive mechanism linking gonadal hormones to cellular excitability and anhedonia-
160 ver the contributions of sex chromosomes and gonadal hormones to sex differences in demyelination and
161 y circuits was used to test contributions of gonadal hormones to sex differences in vHPC afferents.
163 onding and in other hypothalamic sites after gonadal hormone treatments sufficient to activate lordos
164 s in model systems have revealed that, while gonadal hormones undoubtedly play an important role in s
165 adotropins without the concomitant loss of a gonadal hormone, we crossed INH(-/-) mice with a transge
166 s were not the consequence of the actions of gonadal hormones, we induced gonadal sex reversal to alt
167 , cortisol, adrenocorticotropic hormone, and gonadal hormones were analyzed at baseline and week 52.
168 ral to neurodevelopment and are sensitive to gonadal hormones, which increase during adolescence.