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1 s derive from a common precursor pool during gonadogenesis.
2 ng that lines represses hub cell fate during gonadogenesis.
3 nd identified twenty-four genes required for gonadogenesis.
4 t for testis differentiation at the onset of gonadogenesis.
5  that is required for Sox9 expression during gonadogenesis.
6  lineages and have distinct roles in somatic gonadogenesis.
7 rphic and initiates sex-specific programs of gonadogenesis.
8 howed that C. elegans gon-14 is required for gonadogenesis.
9  plays several distinct roles during somatic gonadogenesis.
10  primary focus has been the role of hnd-1 in gonadogenesis.
11 ate morphogenic processes are at work during gonadogenesis.
12  well as their migration and guidance during gonadogenesis.
13 ut does not appear to have any role in later gonadogenesis.
14 ng functions with hnd-1 in embryogenesis and gonadogenesis.
15 partially penetrant defects in viability and gonadogenesis.
16 b on a Numbl mutant background just prior to gonadogenesis.
17 with FOG co-factors exhibit abnormalities in gonadogenesis.
18 on-4 in an attempt to learn how it regulates gonadogenesis.
19 he regulation of both hermaphrodite and male gonadogenesis.
20 ee posterior parasegments at early stages in gonadogenesis.
21 1 to early L2 stage hermaphrodites to permit gonadogenesis.
22 ns hermaphrodite distal tip cell (DTC) leads gonadogenesis.
23 al differentiation, which can guide in vitro gonadogenesis.
24 tic vpr-1 expression is sufficient to induce gonadogenesis and fertility.
25 rk demonstrates a role for DLC3/Cv-c in male gonadogenesis and highlights a novel StART domain mediat
26 e ubiquitously expressed HDA-1 in C. elegans gonadogenesis and place hda-1 in the Notch signaling pat
27 r findings shed light on the complexities of gonadogenesis and the genetic regulation required for pr
28 e that the lag-2 gene, which plays a role in gonadogenesis and vulval development and encodes a Notch
29 results in specific postembryonic defects in gonadogenesis and vulval development.
30 ay in which germ cell pressure may influence gonadogenesis, and also predicts that adult germ cells m
31                           Although embryonic gonadogenesis appeared normal, primordial follicles were
32 the C. elegans gonad is defined during early gonadogenesis by two somatic gonadal precursor cells, Z1
33  we describe a novel regulator of Drosophila gonadogenesis, clift, mutations in which abolish gonad f
34 (q645) allele is fully penetrant for the Sys gonadogenesis defect in hermaphrodites, but affects male
35 ch has also been called a gon-10 mutant (for gonadogenesis defective 10) and show that loss of HDA-1
36 We find that sys-1 is haplo-insufficient for gonadogenesis defects and that sys-1 and pop-1 mutants d
37           Inactivation of gem-1 enhances the gonadogenesis defects of gon-2 hypomorphic mutations, su
38     In this article, we demonstrate that the gonadogenesis defects of gon-2 loss-of-function mutants
39 pression is sufficient to prevent the severe gonadogenesis defects.
40 nd mice have hampered our knowledge of human gonadogenesis, despite its importance in gonadal conditi
41  are maternal effect sterile due to arrested gonadogenesis following embryo hatching.
42 nimals, GSCs are established during preadult gonadogenesis following the proliferation and migration
43             We conclude that fkh-6 regulates gonadogenesis in both sexes, but is male specific in est
44 is of distal tip cell (DTC) migration during gonadogenesis in Caenorhabditis elegans has revealed the
45 blish a link between ribosome biogenesis and gonadogenesis in Caenorhabditis elegans that affects ger
46 racellular matrix component fibulin-1 rescue gonadogenesis in the absence of these proteases.
47                                              Gonadogenesis in the Drosophila embryo is a complex proc
48               The gon-4 gene is required for gonadogenesis in the nematode Caenorhabditis elegans.
49  Neuronal vpr-1 expression is sufficient for gonadogenesis induction during a limited time period sho
50                              Instead, global gonadogenesis is mildly delayed relative to development
51                            In gon-2 mutants, gonadogenesis is severely impaired; in some animals, non
52 of Pitx2 on the right side before and during gonadogenesis is sufficient to transform the right gonad
53 or 1 (Sf1), a nuclear receptor essential for gonadogenesis, is reduced to minimal levels in the Lhx9-
54 ing other genes that mediate early stages of gonadogenesis, Lhx9 mutants do not exhibit additional ma
55 pecified at disparate positions during early gonadogenesis, must assemble into one collective at the
56 cell (LC), a migratory cell crucial for male gonadogenesis of C. elegans.
57 tissues, involvement in sex determination or gonadogenesis pathways, or conserved sex-biased expressi
58 or 1 (WT1) genes are essential for mammalian gonadogenesis prior to sexual differentiation.
59 to the seminiferous tubules of the testis as gonadogenesis proceeds, and both Sertoli cells and germ
60                                 During early gonadogenesis, proliferating cells in the coelomic epith
61  of these male-biased genes were involved in gonadogenesis, spermatogenesis, testicular determination
62  determination system, but genes involved in gonadogenesis, spermatogenesis, testicular determination
63 cell (AC/VU) fate decision during C. elegans gonadogenesis, two "alpha cells," each with equal potent
64  antagonistic effect on fkh-6 expression and gonadogenesis, which is relieved by cyclin D activity.